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1. Inhibition of mTORC2 promotes natriuresis in Dahl salt-sensitive rats via the decrease of NCC and ENaC activity.

11. Metabolic Responses of Normal Rat Kidneys to a High Salt Intake.

18. Divergent roles of angiotensin II upon the immediate and sustained increases of renal blood flow following unilateral nephrectomy.

23. Effects of long-term vasopressin receptor stimulation on medullary blood flow and arterial pressure

24. Effects of daily sodium intake and ANG II on cortical and medullary renal blood flow in conscious rats

26. Inhibition of mTORC1 attenuates salt-induced hypertension and kidney injury in Dahl salt-sensitive rats

27. NOX4/H2O2/mTORC1 Pathway in Salt-Induced Hypertension and Kidney Injury.

30. Transcriptomic analysis reveals inflammatory and metabolic pathways that are regulated by renal perfusion pressure in the outer medulla of Dahl-S rats

34. NULL MUTATION OF THE NADPH OXIDASE SUBUNIT p67PHOXPROTECTS THE DAHL-S RAT FROMSALT-INDUCED REDUCTIONS IN MEDULLARY BLOOD FLOW AND GFR

35. Breaking the Cycle

37. Abstract P136: Dietary Salt and Non-Salt Components Have Substantial Effects on Genome-Wide DNA Methylation Patterns in Dahl SS Rats

42. Maternal Diet During Gestation and Lactation Modifies the Severity of Salt-Induced Hypertension and Renal Injury in Dahl Salt-Sensitive Rats

44. Characterization of biological pathways associated with a 1.37 Mbp genomic region protective of hypertension in Dahl S rats

47. Null mutation of the nicotinamide adenine dinucleotide phosphate-oxidase subunit p67phox protects the Dahl-S rat from salt-induced reductions in medullary blood flow and glomerular filtration rate.

48. Null Mutation of the Nicotinamide Adenine Dinucleotide Phosphate-Oxidase Subunit p67phox Protects the Dahl-S Rat From Salt-Induced Reductions in Medullary Blood Flow and Glomerular Filtration Rate.

49. Abstract 665.

50. Abstract 78.

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