38 results on '"Kuranaga, Yuki"'
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2. Targeting Integrin α3 Blocks β1 Maturation, Triggers Endoplasmic Reticulum Stress, and Sensitizes Glioblastoma Cells to TRAIL-Mediated Apoptosis
3. Targeting HIF-activated collagen prolyl 4-hydroxylase expression disrupts collagen deposition and blocks primary and metastatic uveal melanoma growth
4. CSIG-26. TARGETING HYPOXIC CANCER CELLS IN MEDULLOBLASTOMA
5. Generation and initial characterization of mice lacking full‐length BAI3 (ADGRB3) expression
6. Anti-Oncogenic gem-Dihydroperoxides Induce Apoptosis in Cancer Cells by Trapping Reactive Oxygen Species
7. Perturbation of energy metabolism by fatty-acid derivative AIC-47 and imatinib in BCR-ABL-harboring leukemic cells
8. CSIG-35. TARGETING HYPOXIC CANCER CELLS IN MEDULLOBLASTOMA
9. Petasin potently inhibits mitochondrial complex I–based metabolism that supports tumor growth and metastasis
10. Uptake of MicroRNAs from Exosome-Like Nanovesicles of Edible Plant Juice by Rat Enterocytes
11. The therapeutic potential of exosomal miR-22 for cervical cancer radiotherapy
12. Induced miR‐31 by 5‐fluorouracil exposure contributes to the resistance in colorectal tumors
13. MicroRNA‐143/Musashi‐2/ KRAS cascade contributes positively to carcinogenesis in human bladder cancer
14. Synthetic miR-143 Exhibited an Anti-Cancer Effect via the Downregulation of K-RAS Networks of Renal Cell Cancer Cells In Vitro and In Vivo
15. Synthetic miR-143 Inhibits Growth of HER2-Positive Gastric Cancer Cells by Suppressing KRAS Networks Including DDX6 RNA Helicase
16. Analysis of Extracellular Vesicles in Gastric Juice from Gastric Cancer Patients
17. Potent antiproliferative effect of fatty‐acid derivative AIC ‐47 on leukemic mice harboring BCR ‐ ABL mutation
18. SRSF3, a Splicer of the PKM Gene, Regulates Cell Growth and Maintenance of Cancer-Specific Energy Metabolism in Colon Cancer Cells
19. Organ-Specific MicroRNAs (MIR122, 137, and 206) Contribute to Tissue Characteristics and Carcinogenesis by Regulating Pyruvate Kinase M1/2 (PKM) Expression
20. Impairment of K‐Ras signaling networks and increased efficacy of epidermal growth factor receptor inhibitors by a novel synthetic miR‐143
21. Oncogene RNA helicase DDX6 promotes the process of c-Myc expression in gastric cancer cells
22. Cancer-Specific Energy Metabolism in Rhabdomyosarcoma Cells Is Regulated by MicroRNA
23. Regulated Polarization of Tumor-Associated Macrophages by miR-145 via Colorectal Cancer–Derived Extracellular Vesicles
24. MiR-145 negatively regulates Warburg effect by silencing KLF4 and PTBP1 in bladder cancer cells
25. A Novel Combination RNAi toward Warburg Effect by Replacement with miR-145 and Silencing of PTBP1 Induces Apoptotic Cell Death in Bladder Cancer Cells
26. MicroRNA‐143/Musashi‐2/KRAS cascade contributes positively to carcinogenesis in human bladder cancer.
27. Anti-Oncogenic gem-Dihydroperoxides Induce Apoptosis in Cancer Cells by Trapping Reactive Oxygen Species
28. 表面処理を施した繊維強化熱可塑性樹脂の接着接合部における限界エネルギー解放率
29. A Novel Role of Dickkopf-Related Protein 3 in Macropinocytosis in Human Bladder Cancer T24 Cells
30. Perturbation of the Warburg effect increases the sensitivity of cancer cells to TRAIL-induced cell death
31. PKM1 is involved in resistance to anti-cancer drugs
32. Colorectal cancer cell-derived extracellular vesicles induce phenotypic alteration of T cells into tumor-growth supporting cells with transforming growth factor-β1-mediated suppression
33. Anti-Oncogenic gem-Dihydroperoxides Induce Apoptosis in Cancer Cells by Trapping Reactive Oxygen Species
34. Determination of Critical Energy Release Rates of Adhesively Bonded Joints for Surface Treated Fiber Reinforced Thermoplastics
35. Synthetic miR-143 Exhibited an Anti-Cancer Effect via the Downregulation of K-RAS Networks of Renal Cell Cancer Cells In Vitroand In Vivo
36. MicroRNA-214 and MicroRNA-126 Are Potential Biomarkers for Malignant Endothelial Proliferative Diseases
37. Synthetic miR-143 Inhibits Growth of HER2-Positive Gastric Cancer Cells by Suppressing KRAS Networks Including DDX6 RNA Helicase.
38. Petasin potently inhibits mitochondrial complex I-based metabolism that supports tumor growth and metastasis.
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