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1. Limited oxygen in standard cell culture alters metabolism and function of differentiated cells

2. Author Correction: Limited oxygen in standard cell culture alters metabolism and function of differentiated cells

4. Dissecting the biology of mTORC1 beyond rapamycin

11. Defining the Nutritional and Metabolic Context of FGF21 Using the Geometric Framework

13. Phosphoproteomics reveals rewiring of the insulin signaling network and multi-nodal defects in insulin resistance

14. Limited oxygen availability in standard cell culture alters metabolism and function in terminally-differentiated cells

17. Correction: Corrigendum: 14-3-3ζ regulates the mitochondrial respiratory reserve linked to platelet phosphatidylserine exposure and procoagulant function

18. Trafficking regulator of GLUT4-1 (TRARG1) is a GSK3 substrate

20. Phosphoproteomics reveals rewiring of the insulin signaling network and multi-nodal defects in insulin resistance

23. Genome-wide analysis in Drosophila reveals diet-by-gene interactions and uncovers diet-responsive genes

24. Trafficking regulator of GLUT4-1 (TRARG1) is a GSK3 substrate.

27. Acute mTOR inhibition induces insulin resistance and alters substrate utilization in vivo

28. Mitochondrial oxidants, but not respiration, are sensitive to glucose in adipocytes

29. Trafficking regulator of GLUT4-1 (TRARG1) is a GSK3 substrate

30. Insulin signaling requires glucose to promote lipid anabolism in adipocytes

34. Lactate production is a prioritized feature of adipocyte metabolism

35. Mitochondrial oxidative stress causes insulin resistance without disrupting oxidative phosphorylation

41. Symposium12-4

42. High throughput protein-protein interaction data: clues for the architecture of protein complexes

45. High dietary fat and sucrose result in an extensive and time-dependent deterioration in health of multiple physiological systems in mice

46. Mitochondrial CoQ deficiency is a common driver of mitochondrial oxidants and insulin resistance

49. Author response: Mitochondrial CoQ deficiency is a common driver of mitochondrial oxidants and insulin resistance

50. Dynamic Metabolomics Reveals that Insulin Primes the Adipocyte for Glucose Metabolism

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