34 results on '"Kroll, Todd G."'
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2. PAX8-PPARγ1 Fusion in Oncogene Human Thyroid Carcinoma
3. Supplementary Figure 2 from Peroxisome Proliferator-Activated Receptor-δ Induces Cell Proliferation by a Cyclin E1–Dependent Mechanism and Is Up-regulated in Thyroid Tumors
4. Data from CREB3L2-PPARγ Fusion Mutation Identifies a Thyroid Signaling Pathway Regulated by Intramembrane Proteolysis
5. Supplementary Figure 1 from CREB3L2-PPARγ Fusion Mutation Identifies a Thyroid Signaling Pathway Regulated by Intramembrane Proteolysis
6. Supplementary Figure 1 from Peroxisome Proliferator-Activated Receptor-δ Induces Cell Proliferation by a Cyclin E1–Dependent Mechanism and Is Up-regulated in Thyroid Tumors
7. Data from Peroxisome Proliferator-Activated Receptor-δ Induces Cell Proliferation by a Cyclin E1–Dependent Mechanism and Is Up-regulated in Thyroid Tumors
8. Well-Differentiated Thyroid Follicular Carcinoma
9. Heterochromatin protein 1 expression is reduced in human thyroid malignancy
10. Molecular Events in Follicular Thyroid Tumors
11. Molecular Detection of PPARγ Rearrangements and Thyroid Carcinoma in Preoperative Fine-Needle Aspiration Biopsies
12. The PAX8/PPARγ fusion oncoprotein transforms immortalized human thyrocytes through a mechanism probably involving wild-type PPARγ inhibition
13. The PAX8/PPARγ fusion oncoprotein transforms immortalized human thyrocytes through a mechanism probably involving wild-type PPARγ inhibition
14. RAS Point Mutations and PAX8-PPARγ Rearrangement in Thyroid Tumors: Evidence for Distinct Molecular Pathways in Thyroid Follicular Carcinoma
15. Molecular Events in Follicular Thyroid Tumors
16. CREB3L2-PPARγ Fusion Mutation Identifies a Thyroid Signaling Pathway Regulated by Intramembrane Proteolysis
17. Peroxisome Proliferator-Activated Receptor-δ Induces Cell Proliferation by a Cyclin E1–Dependent Mechanism and Is Up-regulated in Thyroid Tumors
18. PIKE (Phosphatidylinositol 3-Kinase Enhancer)-A GTPase Stimulates Akt Activity and Mediates Cellular Invasion
19. RASPoint Mutations and PAX8-PPARγ Rearrangement in Thyroid Tumors: Evidence for Distinct Molecular Pathways in Thyroid Follicular Carcinoma
20. Genetic and Biological Subgroups of Low-Stage Follicular Thyroid Cancer
21. PAX8-PPARγ Rearrangement in Thyroid Tumors
22. Molecular Rearrangements and Morphology in Thyroid Cancer
23. BRD4 Bromodomain Gene Rearrangement in Aggressive Carcinoma with Translocation t(15;19)
24. PAX8-PPAR γ 1 Fusion in Oncogene Human Thyroid Carcinoma
25. PIKE-A is amplified in human cancers and prevents apoptosis by up-regulating Akt.
26. RAS Point Mutations and PAX8-PPARγ Rearrangement in Thyroid Tumors: Evidence for Distinct Molecular Pathways in Thyroid Follicular Carcinoma.
27. Transcriptional repression and developmental functions of the atypical vertebrate GATA protein TRPS1.
28. PAX8-PPAR Gamma 1 Fusion in Oncogene Human Thyroid Carcinoma.
29. BRD4Bromodomain Gene Rearrangement in Aggressive Carcinoma with Translocation t(15;19)
30. DIFFERENTIAL OCCURRENCE OF FREE BETA AND FREE ALPHA SUBUNITS OF HUMAN CHORIONIC GONADOTROPIN (hCG) IN PREGNANCY SERA*
31. PIKE (Phosphatidylinositol 3-Kinase Enhancer)-A GTPase Stimulates Akt Activity and Mediates Cellular Invasion.
32. Peroxisome proliferator-activated receptor-delta induces cell proliferation by a cyclin E1-dependent mechanism and is up-regulated in thyroid tumors.
33. Molecular events in follicular thyroid tumors.
34. RAS point mutations and PAX8-PPAR gamma rearrangement in thyroid tumors: evidence for distinct molecular pathways in thyroid follicular carcinoma.
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