166 results on '"Kriegs M"'
Search Results
2. P08 Radiosensitivity and impaired DNA double-strand break repair in HPV-positive OPSCC: The role of smoking and the ATM kinase as observed in ex vivo and in vitro studies
3. Radiation DNA damage and use in cancer/therapeutics-translation of radiation modifiers
4. p53 modulates homologous recombination at I-SceI-induced double-strand breaks through cell-cycle regulation
5. Radiosensitization of HPV Positive Head and Neck Squamous Cell Carcinomas (HNSCC) by Chk1 Inhibition: ID 301
6. Kinase activity profiles as prognostic marker for immunotherapy in head and neck squamous cell carcinoma (HNSCC)
7. Kinaseaktivitätsprofile als prognostischer Marker für die Immuntherapie bei Kopf-Hals-Tumoren (HNSCC)
8. Nachweis eines DNA-Reparaturdefekts HPV-positiver OPSCC mittels ex vivo kultivierter, humaner Tumorschnitte
9. Patient derived e x vivo slice cultures demonstrate a profound DNA double-strand break repair defect in HPV-positive OPSCC
10. A lack of effectiveness in the ATM-orchestrated DNA damage response contributes to the enhanced radiation sensitivity of HPV-positive HNSCC cell lines
11. Eingeschränkte Effektivität der ATM-gesteuerten DNA Schadensantwort als Ursache der erhöhten zellulären Strahlenempfindlichkeit HPV-positiver HNSCC Zellen
12. Molekulare Virologie am Beispiel des Hepatitis-B-Virus: Arbeiten der Nachwuchsgruppe “Hepatitis B und C: virale Genfunktionen” des Robert Koch-Institutes
13. Kinase activity profiles as prognostic marker for immunotherapy in head and neck squamous cell carcinoma (HNSCC)
14. Kinaseaktivitätsprofile als prognostischer Marker für die Immuntherapie bei Kopf-Hals-Tumoren (HNSCC)
15. Ex-vivo-Kulturen humaner HNSCC Tumoren bestätigen eine eingeschränkte DNA-Doppelstrangbruch-Reparatur HPV-positiver OPSCC und weisen auf einen Defekt der ATM-abhängigen Schadensantwort hin
16. Ex-vivo slice cultures of tumor tissues confirm the DNA double-strand break repair defect of HPV-positive OPSCC and suggest a defect in the ATM-mediated DNA damage response
17. Variation in EGFR protein expression is correlated with cellular and tumour radiosensitivity, except for tumours with extreme EGFR gene amplification: FV413
18. Characterization of human head and neck squamous cell carcinoma (HNSCC) on a kinomic level
19. A tissue micro array analysis of Src kinase expression and activity in HNSCC
20. Tissue Micro-Array basierte Analyse der Expression und Aktivität von Src-Kinasen in HNSCC
21. Charakterisierung von Kopf-Hals-Tumoren (HNSCC) auf Kinomebene
22. Chapter 11 - Radiation DNA damage and use in cancer/therapeutics-translation of radiation modifiers
23. Duale Inhibition von PARP1 und dem intra-S/G2-Zellzykluskontrollpunkt als neue Strategie zur hoch wirksamen Strahlensensitivierung HPV+ HNSCC
24. OC-0490: Targeting PARP1 and the intra-S/G2 checkpoints for highly effective radiosensitization of HPV+ HNSCC
25. Dual inhibition of PARP1 and the intra-S/G2 cell cycle checkpoints as a novel strategy for highly effective radiosensitization of HPV+ HNSCC
26. Kinomprofiling bei Kopf-Hals-Tumoren: Identifizierung neuer Ziele für die molekulare Therapie
27. Identifizierung funktioneller Proteomsignaturen in HPV positiven Kopf-Hals Karzinomen
28. Radiosensitivierung von Kopf-Halstumorzellen durch Sorafenib in Kombination mit einer cisplatinbasierten Chemotherapie
29. G2-checkpoint targeting and radiosensitization of HPV/p16-positive HNSCC cells through the inhibition of Chk1 and Wee1
30. P06.20 EGFRvIII: a predictive marker for Temozolomide response in O6-methylguanine-DNA methyltransferase negative glioblastoma cells and tumor xenografts
31. Der Multikinase Inhibitor Sorafenib sensitiviert Kopf-Hals-Tumorzellen gegenüber einer cisplatinbasierten Radiochemotherapie
32. Radiosensitization of NSCLC cells by EGFR inhibition is the result of an enhanced p53-dependent G1 arrest.
33. Der Einfluss EGFR-abhängiger Signalwege auf die Radiosensitivität von HNSCC-Zellen
34. 2 Effects of sorafenib treatment on HNSCC cells in combination with cisplatin based-chemoradiation
35. Radiosensitivierung von HPV-positiven Kopf-Hals-Karzinomen durch PARP- und EGFR-Inhibition
36. SP-0557: Radiobiology of HPV-positive HNSCC cells: Mechanisms of radiosensitivity and implications for therapy
37. Sorafenib Behandlung von HNSCC führt zu einer Radiosensitivierung
38. Strahlensensibilisierung durch Chk1 Inhibitor bei HPV positiven Kopf-Hals-Karzinomen
39. Inactivation of HNSCC Cells by 90Y-Labeled Cetuximab Strictly Depends on the Number of Induced DNA Double-Strand Breaks
40. 173 - G2-checkpoint targeting and radiosensitization of HPV/p16-positive HNSCC cells through the inhibition of Chk1 and Wee1
41. OC-0499: HNSCC cell lines positive for HPV and p16 possess high cellular radiosensitivity due to impaired DSB repair capacity
42. PD-0094: EGFR inhibition radiosensitizes NSCLC cells via permanent G1 arrest but only when p53/p21 signaling is intact
43. OC-0071 INACTIVATION OF HNSCC CELLS BY 90Y-LABELED CETUXIMAB STRICTLY DEPENDS ON NUMBER OF INDUCED DNA DOUBLE-STRAND BREAKS
44. p53 modulates homologous recombination at I-SceI-induced double-strand breaks through cell-cycle regulation
45. 843 A SYNTHETIC CODON-OPTIMIZED HCV NON-STRUCTURAL 5A DNA VACCINE PRIMES POLYFUNCTIONAL CD8+ T CELL RESPONSES IN WILDTYPE AND NS5A-TRANSGENIC MICE
46. 378 HNSCC CELL LINES POSITIVE FOR HPV AND P16 POSSESS AN EXCEPTIONALLY HIGH RADIO-SENSITIVITY
47. 74 INHIBITION OF EPIDERMAL GROWTH FACTOR RECEPTOR ENHANCES RADIATION-INDUCED PERMANENT G1 ARREST SOLELY IN TUMOR CELLS WITH INTACT P53/P21 CELL CYCLE REGULATION
48. 252 EFFECT OF EGFR ON DOUBLE-STRAND BREAK REPAIR
49. EGFR Inhibition Radiosensitizes NSCLC Cells via DNA Double-strand Break Inducible Senescence
50. Raf‐1 kinase associates with Hepatitis C virus NS5A and regulates viral replication
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