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1. Stearoyl-CoA desaturase inhibition is toxic to acute myeloid leukemia displaying high levels of the de novo fatty acid biosynthesis and desaturation

2. The selective prolyl hydroxylase inhibitor IOX5 stabilizes HIF-1α and compromises development and progression of acute myeloid leukemia

3. Mannose metabolism inhibition sensitizes acute myeloid leukaemia cells to therapy by driving ferroptotic cell death

5. Hypoxia shapes the immune landscape in lung injury and promotes the persistence of inflammation

7. MYC sensitises cells to apoptosis by driving energetic demand

9. JMJD6 promotes self-renewal and regenerative capacity of hematopoietic stem cells

10. A time- and single-cell-resolved model of murine bone marrow hematopoiesis

11. Author Correction: Hypoxia shapes the immune landscape in lung injury and promotes the persistence of inflammation

16. Systemic silencing of Phd2 causes reversible immune regulatory dysfunction

17. SCD inhibition eradicates AML displaying high de novo fatty acid desaturation and synergizes with chemotherapy

24. Inhibition of Stearoyl-CoA Desaturase Has Anti-Leukemic Properties in Acute Myeloid Leukemia

26. Targeting HIF-Hydroxylases Compromises Disease Initiation and Propagation, and Synergises with Current Therapies to Eliminate Cancer Stem Cells in AML

27. Haemogenic Gastruloids Recapitulate Developmental Haematopoiesis and Provide an Ontogeny-Relevant Context to Dissect the Origins of Infant Leukemia

28. Dissecting infant leukemia developmental origins with a hemogenic gastruloid model

30. MYC sensitises cells to apoptosis by driving energetic demand

32. Mannose metabolism inhibition sensitizes acute myeloid leukemia cells to cytarabine and FLT3 inhibitor therapy by modulating fatty acid metabolism to drive ferroptotic cell death.

35. Hypoxia shapes the immune landscape in lung injury promoting inflammation persistence

37. CITED2 coordinates key hematopoietic regulatory pathways to maintain the HSC pool in both steady-state hematopoiesis and transplantation

38. Elevated 2HG does not cause features of tumorigenesis

42. The mRNA m6A reader YTHDF2 suppresses proinflammatory pathways and sustains hematopoietic stem cell function

44. T Cell-Mediated Autoimmune Disease Due to Low-Affinity Crossreactivity to Common Microbial Peptides

46. Metabolic and innate immune cues merge into a specific inflammatory response via unfolded proteinresponse (UPR)

48. Metabolic and Innate Immune Cues Merge into a Specific Inflammatory Response via the UPR

49. CpG binding protein (CFP1) occupies open chromatin regions of active genes, including enhancers and non-CpG islands

50. Catapulting HSCs into action

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