532 results on '"Koyanagi, Satoru"'
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2. Oncogenic accumulation of cysteine promotes cancer cell proliferation by regulating the translation of D-type cyclins
3. Inhibition of G protein-coupled receptor 68 using homoharringtonine attenuates chronic kidney disease-associated cardiac impairment
4. Implications of biological clocks in pharmacology and pharmacokinetics of antitumor drugs
5. Modulation of cell physiology by bispecific nanobodies enabling changes in the intracellular localization of organelle proteins
6. Chronopharmacology of immune-related diseases
7. RNA editing enzyme ADAR1 controls miR-381-3p-mediated expression of multidrug resistance protein MRP4 via regulation of circRNA in human renal cells
8. Circadian rhythms in CYP2A5 expression underlie the time-dependent effect of tegafur on breast cancer
9. Prostaglandin F2α Affects the Cycle of Clock Gene Expression and Mouse Behavior
10. Suppression of neuropathic pain in the circadian clock–deficient Per2m/m mice involves up-regulation of endocannabinoid system
11. Alteration of circadian machinery in monocytes underlies chronic kidney disease-associated cardiac inflammation and fibrosis
12. RNA editing enzyme ADAR2 regulates P-glycoprotein expression in murine breast cancer cells through the circRNA-miRNA pathway
13. Time-dependent differences in vancomycin sensitivity of macrophages underlie vancomycin-induced acute kidney injury
14. Optimizing the dosing schedule of l-asparaginase improves its anti-tumor activity in breast tumor-bearing mice
15. Inhibition of Tumor-Derived C-C Motif Chemokine Ligand 2 Expression Attenuates Tactile Allodynia in NCTC 2472 Fibrosarcoma-Inoculated Mice
16. Dietary supplementation with essence of chicken enhances daily oscillations in plasma glucocorticoid levels and behavioral adaptation to the phase-shifted environmental light–dark cycle in mice
17. Diurnal expression of MRP4 in bone marrow cells underlies the dosing-time dependent changes in the oxaliplatin-induced myelotoxicity
18. Contribution of the clock gene DEC2 to VEGF mRNA upregulation by modulation of HIF1α protein levels in hypoxic MIO-M1 cells, a human cell line of retinal glial (Müller) cells
19. The scaffold protein PDZK1 governs diurnal localization of CNT2 on the plasma membrane in mouse intestinal epithelial cells
20. Senescence-induced alteration of circadian phagocytic activity of retinal pigment epithelium cell line ARPE-19
21. Supplementary Figure from Diurnal Expression of PD-1 on Tumor-Associated Macrophages Underlies the Dosing Time-Dependent Antitumor Effects of the PD-1/PD-L1 Inhibitor BMS-1 in B16/BL6 Melanoma-Bearing Mice
22. Supplementary Data from Diurnal Expression of PD-1 on Tumor-Associated Macrophages Underlies the Dosing Time-Dependent Antitumor Effects of the PD-1/PD-L1 Inhibitor BMS-1 in B16/BL6 Melanoma-Bearing Mice
23. Data from Diurnal Expression of PD-1 on Tumor-Associated Macrophages Underlies the Dosing Time-Dependent Antitumor Effects of the PD-1/PD-L1 Inhibitor BMS-1 in B16/BL6 Melanoma-Bearing Mice
24. Fig. S11-S13 from Optimized Dosing Schedule Based on Circadian Dynamics of Mouse Breast Cancer Stem Cells Improves the Antitumor Effects of Aldehyde Dehydrogenase Inhibitor
25. Table S3 from Optimized Dosing Schedule Based on Circadian Dynamics of Mouse Breast Cancer Stem Cells Improves the Antitumor Effects of Aldehyde Dehydrogenase Inhibitor
26. Data from Optimized Dosing Schedule Based on Circadian Dynamics of Mouse Breast Cancer Stem Cells Improves the Antitumor Effects of Aldehyde Dehydrogenase Inhibitor
27. Supplementary methods from Optimized Dosing Schedule Based on Circadian Dynamics of Mouse Breast Cancer Stem Cells Improves the Antitumor Effects of Aldehyde Dehydrogenase Inhibitor
28. Supplementary Data 6 from Circadian Regulation of mTOR by the Ubiquitin Pathway in Renal Cell Carcinoma
29. Supplementary Table 3 from Stress-Regulated Transcription Factor ATF4 Promotes Neoplastic Transformation by Suppressing Expression of the INK4a/ARF Cell Senescence Factors
30. Supplementary Data 1 from Circadian Regulation of mTOR by the Ubiquitin Pathway in Renal Cell Carcinoma
31. Data from Circadian Rhythm of Transferrin Receptor 1 Gene Expression Controlled by c-Myc in Colon Cancer–Bearing Mice
32. Supplementary Data 4 from Circadian Regulation of mTOR by the Ubiquitin Pathway in Renal Cell Carcinoma
33. Supplementary Data 3 from Circadian Regulation of mTOR by the Ubiquitin Pathway in Renal Cell Carcinoma
34. Supplementary Table 1 from Stress-Regulated Transcription Factor ATF4 Promotes Neoplastic Transformation by Suppressing Expression of the INK4a/ARF Cell Senescence Factors
35. Supplementary Figure 4 from Circadian Rhythm of Transferrin Receptor 1 Gene Expression Controlled by c-Myc in Colon Cancer–Bearing Mice
36. Supplementary Data 5 from Circadian Regulation of mTOR by the Ubiquitin Pathway in Renal Cell Carcinoma
37. Supplementary Figure 1 from Circadian Rhythm of Transferrin Receptor 1 Gene Expression Controlled by c-Myc in Colon Cancer–Bearing Mice
38. Supplementary Figure 1 from Rhythmic Control of the ARF-MDM2 Pathway by ATF4 Underlies Circadian Accumulation of p53 in Malignant Cells
39. Supplementary Figures 1-5 from Stress-Regulated Transcription Factor ATF4 Promotes Neoplastic Transformation by Suppressing Expression of the INK4a/ARF Cell Senescence Factors
40. Supplementary Figure 3 from Circadian Rhythm of Transferrin Receptor 1 Gene Expression Controlled by c-Myc in Colon Cancer–Bearing Mice
41. Supplementary Figure 2 from Rhythmic Control of the ARF-MDM2 Pathway by ATF4 Underlies Circadian Accumulation of p53 in Malignant Cells
42. Data from Circadian Regulation of mTOR by the Ubiquitin Pathway in Renal Cell Carcinoma
43. Supplementary Figure 5 from Circadian Rhythm of Transferrin Receptor 1 Gene Expression Controlled by c-Myc in Colon Cancer–Bearing Mice
44. Supplementary Table 2 from Stress-Regulated Transcription Factor ATF4 Promotes Neoplastic Transformation by Suppressing Expression of the INK4a/ARF Cell Senescence Factors
45. Data from Stress-Regulated Transcription Factor ATF4 Promotes Neoplastic Transformation by Suppressing Expression of the INK4a/ARF Cell Senescence Factors
46. Supplementary Methods, Figure Legends from Rhythmic Control of the ARF-MDM2 Pathway by ATF4 Underlies Circadian Accumulation of p53 in Malignant Cells
47. Data from Rhythmic Control of the ARF-MDM2 Pathway by ATF4 Underlies Circadian Accumulation of p53 in Malignant Cells
48. Supplementary Figure 3 from Rhythmic Control of the ARF-MDM2 Pathway by ATF4 Underlies Circadian Accumulation of p53 in Malignant Cells
49. Supplementary Figure 4 from Rhythmic Control of the ARF-MDM2 Pathway by ATF4 Underlies Circadian Accumulation of p53 in Malignant Cells
50. Supplementary Figure 2 from Circadian Rhythm of Transferrin Receptor 1 Gene Expression Controlled by c-Myc in Colon Cancer–Bearing Mice
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