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1. Ca2+/calmodulin-dependent protein kinase II-based regulation of voltage-gated Na+ channel in cardiac disease.

2. Catecholamine-independent heart rate increases require Ca2+/calmodulin-dependent protein kinase II.

4. MIRO1 controls energy production and proliferation of smooth muscle cells.

5. Short-Term Statin Treatment Reduces, and Long-Term Statin Treatment Abolishes, Chronic Vascular Injury by Radiation Therapy.

6. Short-term statin treatment reduces, and long-term statin treatment abolishes chronic vascular injury by radiation therapy.

7. Regulation of Smooth Muscle Cell Proliferation by Mitochondrial Ca2+ in Type 2 Diabetes.

8. Mechanisms by which statins protect endothelial cells from radiation-induced injury in the carotid artery.

9. The mitochondrial regulation of smooth muscle cell proliferation in type 2 diabetes.

10. Mitochondrial Ca 2+ Uptake Drives Endothelial Injury By Radiation Therapy.

11. Retraction notice to Corrigendum to "Inhibition of CaMKII in mitochondria preserves endothelial barrier function after irradiation" [Free Radical Biol. Med. 146, January (2020) 287-298].

12. Sex-Specific Differences in Endothelial Function Are Driven by Divergent Mitochondrial Ca 2+ Handling.

13. Knockout of Sorbin And SH3 Domain Containing 2 (Sorbs2) in Cardiomyocytes Leads to Dilated Cardiomyopathy in Mice.

14. Retraction notice to "Inhibition of CaMKII in mitochondria preserves endothelial barrier function after irradiation" [Free Radic. Biol. Med. 146C (2020) 287-298].

17. Inhibition of CaMKII in mitochondria preserves endothelial barrier function after irradiation.

18. Loss of MCU prevents mitochondrial fusion in G 1 -S phase and blocks cell cycle progression and proliferation.

19. Defective protein repair under methionine sulfoxide A deletion drives autophagy and ARE-dependent gene transcription.

20. CaMKII (Ca 2+ /Calmodulin-Dependent Kinase II) in Mitochondria of Smooth Muscle Cells Controls Mitochondrial Mobility, Migration, and Neointima Formation.

21. Inhibition of the mitochondrial calcium uniporter prevents IL-13 and allergen-mediated airway epithelial apoptosis and loss of barrier function.

22. Endothelial CaMKII as a regulator of eNOS activity and NO-mediated vasoreactivity.

23. Mitochondrial CaMKII inhibition in airway epithelium protects against allergic asthma.

24. Role of CaMKII in Ang-II-dependent small artery remodeling.

25. Inhibition of MCU forces extramitochondrial adaptations governing physiological and pathological stress responses in heart.

27. The mitochondrial uniporter controls fight or flight heart rate increases.

28. Joiner et al. reply.

29. CaMKII and stress mix it up in mitochondria.

30. β(IV)-Spectrin regulates TREK-1 membrane targeting in the heart.

31. βIV-Spectrin and CaMKII facilitate Kir6.2 regulation in pancreatic beta cells.

32. Oxidized Ca(2+)/calmodulin-dependent protein kinase II triggers atrial fibrillation.

33. Differential control of calcium homeostasis and vascular reactivity by Ca2+/calmodulin-dependent kinase II.

34. CaMKII is essential for the proasthmatic effects of oxidation.

35. Genetic inhibition of Na+-Ca2+ exchanger current disables fight or flight sinoatrial node activity without affecting resting heart rate.

36. CaMKII determines mitochondrial stress responses in heart.

37. β2-Adrenergic receptor supports prolonged theta tetanus-induced LTP.

38. Oxidation of CaMKII determines the cardiotoxic effects of aldosterone.

39. A β(IV)-spectrin/CaMKII signaling complex is essential for membrane excitability in mice.

40. EH domain proteins regulate cardiac membrane protein targeting.

41. I(f) and SR Ca(2+) release both contribute to pacemaker activity in canine sinoatrial node cells.

42. CaV1.2 beta-subunit coordinates CaMKII-triggered cardiomyocyte death and afterdepolarizations.

43. Dual role of K ATP channel C-terminal motif in membrane targeting and metabolic regulation.

44. Calmodulin kinase II is required for fight or flight sinoatrial node physiology.

45. A role for the S0 transmembrane segment in voltage-dependent gating of BK channels.

46. The beta-subunit composition of nicotinic acetylcholine receptors in the neurons of the guinea pig inferior mesenteric ganglion.

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