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9. Terminology for Red Cell Surface Antigens

Author

Daniels, G.L., primary, Anstee, D.J., additional, Cartron, J.P., additional, Dahr, W., additional, Garratty, G., additional, Henry, S., additional, Jorgensen, J., additional, Judd, W.J., additional, Kornstad, L., additional, Levene, C., additional, Lomas‐Francis, C., additional, Lubenko, A., additional, Moulds, J.J., additional, Moulds, J.M., additional, Moulds, M., additional, Overbeeke, M., additional, Reid, M.E., additional, Rouger, P., additional, Scott, M., additional, Seidl, S., additional, Sistonen, P., additional, Tani, Y., additional, Wendel, S., additional, and Zelinski, T., additional

Published
1999
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10. Terminology for Red Cell Surface Antigens. Makuhari Report

Author

Daniels, G.L., primary, Anstee, D.J., additional, Cartron, J. P., additional, Dahr, W., additional, Henry, S., additional, Issitt, P.D., additional, Jorgensen, J., additional, Judd, W.J., additional, Kornstad, L., additional, Levene, C., additional, Lomas-Francis, C., additional, Lubenko, A., additional, Mallory, D., additional, Moulds, J. M., additional, Moulds, J. J., additional, Okubo, Y., additional, Overbeeke, M., additional, Reid, M. E., additional, Rouger, P., additional, Seidl, S., additional, Sistonen, P., additional, Wendel, S., additional, and Zelinski, T., additional

Published
1996
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12. Blood Group Terminology 1995: ISBT Working Party on Terminology for Red Cell Surface Antigens

Author

Daniels, G. L., primary, Anstee, D. J., additional, Cartron, J. P., additional, Dahr, W., additional, Jørgensen, J., additional, Kornstad, L., additional, Levene, C., additional, Lomas-Francis, C., additional, Lubenko, A., additional, Mallory, D., additional, Moulds, J. J., additional, Okubo, Y., additional, Overbeeke, M., additional, Reid, M. E., additional, Rouger, P., additional, Seidl, S., additional, Sistonen, P., additional, Wendel, S., additional, Woodfield, G., additional, and Zelinski, T., additional

Published
1995
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13. What Is the Best Technique for the Detection of Red Cell Alloantibodies?

Author

Engelfriet, C.P., primary, Reesink, H.W., additional, Barbolla, L., additional, Carbonell, F., additional, Mitchell, R., additional, Contreras, M., additional, Knight, R., additional, Goldman, M., additional, Décary, F., additional, Garraty, G., additional, Gerber, H., additional, Hustings, H., additional, Kornstad, L., additional, Moulds, J.J., additional, Overbeeke, M.A.M., additional, Pirkola, A., additional, Voak, D., additional, and Ouwehand, W.H., additional

Published
1995
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14. ISBT Working Party on Terminology for Red Cell Surface Antigens: São Paulo Report

Author

Daniels, G. L., primary, Moulds, J. J., additional, Anstee, D. J., additional, Bird, G. W. G., additional, Brodheim, E., additional, Cartron, J.-P., additional, Dahr, W., additional, Engelfriet, C. P., additional, Issitt, P. D., additional, Jørgensen, J., additional, Kornstad, L., additional, Lewis, M., additional, Levene, C., additional, Lubenko, A., additional, Mallory, D., additional, Morel, P., additional, Nordhagen, R., additional, Okubo, Y., additional, Reid, M., additional, Rouger, Ph., additional, Salmon, Ch., additional, Seidl, S., additional, Sistonen, P., additional, Wendel, S., additional, Woodfield, G., additional, and Zelinski, T., additional

Published
1993
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17. INTERNATIONAL SOCIETY OF BLOOD TRANSFUSION SOCIÉTÉ INTERNATIONALE DE TRANSFUSION SANGUINE: ISBT Working Party on Terminology for Red Cell Surface Antigens: Los Angeles Report

Author

Lewis, M., primary, Anstee, D.J., additional, Bird, G. W. G., additional, Brodheim, E., additional, Cartron, J.-P., additional, Contreras, M., additional, Dahr, W., additional, Daniels, G. L., additional, Engelfriet, C. P., additional, Issitt, P. D., additional, Jørgensen, J., additional, Kornstad, L., additional, Lubenko, A., additional, McCreary, J., additional, Morel, P., additional, Moulds, J. J., additional, Nevanlinna, H., additional, Nordhagen, R., additional, Okubo, Y., additional, Pehta, J., additional, Rouger, Ph., additional, Rubinstein, P., additional, Salmon, Ch., additional, Seidl, S., additional, Sistonen, P., additional, Tippett, P., additional, Walker, R. H., additional, Woodfield, G., additional, Young, S., additional, and Zelinski, T., additional

Published
1991
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18. Blood Group Terminology 1990

Author

Lewis, M., primary, Anstee, D.J., additional, Bird, G.W.G., additional, Brodheim, E., additional, Cartron, J.-P., additional, Contreras, M., additional, Crookston, M.C., additional, Dahr, W., additional, Daniels, G.L., additional, Engelfriet, C.P., additional, Giles, C.M., additional, Issitt, P.D., additional, Jørgensen, J., additional, Kornstad, L., additional, Lubenko, A., additional, Marsh, W.L., additional, McCreary, J., additional, Moore, B.P.L, additional, Morel, P., additional, Moulds, J.J., additional, Nevanlinna, H., additional, Nordhagen, R., additional, Okubo, Y., additional, Rosenfield, R.E., additional, Rouger, Ph., additional, Rubinstein, P., additional, Salmon, Ch., additional, Seidl, S., additional, Sistonen, P., additional, Tippett, P., additional, Walker, R.H., additional, Woodfield, G., additional, and Young, S., additional

Published
1990
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19. IgG subclass distribution of anti-Rh, anti-Kell and anti-Duffy antibodies measured by sensitive haemagglutination assays.

Author

Michaelsen, T. E. and Kornstad, L.

Subjects
*IMMUNOGLOBULIN G, *IMMUNE serums, *ERYTHROCYTES, *IMMUNOGLOBULINS, *BLOOD products, *SERUM
Abstract

The IgG subclass distribution of anti Rh antibodies (anti-D, "anti-Da'. anti-c, anti-E), anti-Kell and anti-Duffy (anti-Fya) antibodies was measured by two haemagglutination techniques on microtitre plates. The first technique involved rabbit subclass specific antisera which were used to agglutinate red cells previously reacted with the patients' antibodies at high concentration. The second, which was more sensitive, had an additional step by introducing sheep anti-rabbit antibodies (sandwich technique). By the sensitive sandwich technique we revealed, for anti-D antibodies: IgGl 8/19, IgG3 1/19, IgG1/IgG3 8/19, IgGl/lgG2/IgG3/lgG4 1/19, IgG 1/IgG4 1/19; for the Da-reactive anti-D antibodies: IgGl 1/8, IgG1/IgG3 1/8, IgGl/IgG3/IgG4 6/8; for the anti-E antibodies: lgG1/IgG2/ lgG4 2/3,IgGl/IgG2/IgG3/IgG4 1/3: for the anti-c antibodies: IgGl 2/5, IgG3 1/5, IgG 1/ lgG3 1/5: for the anti-Kell antibodies: IgGl 9/20, lgGI/IgG3 1/20, IgGl/IgG4 8/20, IgGl/IgG3/IgG4 2/20; and for anti-Duffy antibodies: IgGl 1/8, IgGl/IgG4 7/8. These results are partly at variance with previously published results. [ABSTRACT FROM AUTHOR]

Published
1987

25. A transient monoclonal human IgGκ protein with anti-thyroglobulin activity: lack of cross-idiotypy with polyclonal anti-thyroglobulin antibodies.

Author

Michaelsen, Terje E., Gaarder, P. I., and Kornstad, L.

Subjects
IMMUNOGLOBULINS, GLYCOPROTEINS, SERUM, BLOOD proteins, PATIENTS, AUTOANTIBODIES
Abstract

A female patient in her early sixties had for a period of at least 4 months a monoclonal IgG1κ protein in her serum and at the same time a very high level of anti-thyroglobulin antibodies. Different isolation procedures showed that the M component was responsible for the high anti-thyroglobulin activity. Anti-idiotypic antibodies raised in a rabbit against the monoclonal protein inhibited the M component's anti-thyroglobulin activity, and purified thyroglobulin blocked the reaction between the M component and the anti-idiotypic antiserum. After the M component had disappeared, no cross-idiotypic immunoglobulins could be detected in the patient's serum, nor could the anti-idiotypic antiserum inhibit the residual anti-thyroglobulin activity present in the serum at this time. No reaction could be detected between the antiserum and the anti-thyroglobulin antibodies of sera of other patients. Idiotypic autoantibodies against the M component, which might have caused its disappearance, could not be demonstrated. [ABSTRACT FROM AUTHOR]

Published
1981

26. Presence of anti-c in the serum of 42 women giving birth to c positive babies: serological and clinical findings.

Author

Astrup, Jorunn, Kornstad, Leif, Astrup, J, and Kornstad, L

Abstract

During routine ante-natal testing during the period 1953-73 anti-c was found in the serum of 63 women. Twenty-four of them had received blood transfusion previously and of these 22 were multigravidae. In 39 women pregnancies alone were responsible for the immunization. The anti-c titres of the 42 women giving birth to c positive babies showed that the probability of developing haemolytic disease increases with higher titres. But low titres by no means exclude the existence of such disease. Among the 42 c positive babies 32 had a positive direct antiglobulin test. Some of the affected infants had a serious degree of haemolytic disease. [ABSTRACT FROM AUTHOR]

Published
1977
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35. The low-frequency MNS blood group antigens Ny a (MNS18) and Os a (MNS38) are associated with GPA amino acid substitutions

Author

Daniels, G.L., Bruce, L.J., Mawby, W.J., Green, C.A., Petty, A., Okubo, Y., Kornstad, L., and Tanner, M.J.A.

Abstract

BACKGROUND: Antigens of the MNS blood group system are located on two sialoglycoproteins, GPA and GPB, encoded by GYPA and GYPB . The molecular backgrounds of the low-frequency antigens Ny a and Os a are not known. STUDY DESIGN AND METHODS: Immunoblotting and a monoclonal antibody-specific immobilization of erythrocyte antigens (MAIEA) assay were used to analyze Os a . PCR-amplified products of the coding exons of GYPA were studied by single-strand conformation polymorphism analysis, and exon 3 was sequenced. Synthetic peptides were used in hemagglutination-inhibition tests. RESULTS: Sequencing of GYPA exon 3 of two unrelated Ny(a+) persons revealed heterozygosity for a T194A base change encoding an Asp27Glu substitution. Immunoblotting with anti-Os a and an MAIEA assay with MoAbs to GPA showed that Os a is on GPA. Sequencing exon 3 of an Os(a+) person from the only family with Os a revealed heterozygosity for a C273T base change encoding a Pro54Ser substitution. A synthetic peptide representing part of GPA with the Os a mutation (VRTVYP S EEETGE) completely inhibited anti-Os a , whereas the control peptide (VRTVYP P EEETGE) did not inhibit anti-Os a . CONCLUSION: Ny a and Os a are low-frequency antigens of the MNS blood group system that represent Asp27Glu and Pro54Ser substitutions in GPA, respectively.

Published
2000
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36. Possible effect of secretor locus on plasma concentration of factor VIII and von Willebrand factor

Author

Orstavik, KH, Kornstad, L, Reisner, H, and Berg, K

Abstract

A significant fraction (30%) of the genetically determined variance in plasma concentration of the von Willebrand factor antigen (vWf:Ag) has been shown to be related to ABH determinants. Individuals with blood group O, who have the highest amounts of blood group H substance, have the lowest concentration of vWf:Ag. The Lewis substances, Le(a) and Le(b), are biochemically closely related to the ABH substances as both can be produced from the same precursor substance. We studied the effect of the presence of the Lewis antigens on the plasma concentration of vWf:Ag and factor VIII antigen (VIII:Ag) in 323 individuals of different ABO groups from a series of twins and in 58 blood donors of blood group O. Among persons belonging to blood group O, those with the Le(a) antigen had a higher concentration of both vWf:Ag and VIII:Ag than individuals lacking Le(a). Le(a+b-) people are nonsecretors and Le(a-b+) people are secretors of ABH substance. Thus, the lowest concentration of vWf:Ag and VIII:Ag was found in group O secretors. The effect is most likely due to an effect of the secretor locus. This finding may be of importance for the detection of carriers of hemophilia A and for the diagnosis of type I von Willebrand disease.

Published
1989
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37. A Rare Blood Group Antigen, Rla (Rosenlund)

Author

Kornstad, L.

Abstract

A 'new' rare blood group antigen Rla (Rosenlund) is reported. The antigen is present in about 1 in 900 Norwegians. Nine unrelated Rl(a+) people were found. Studies of seven families, leading to the discovery of 20 additional Rl(a+) people, indicated the antigen is inherited as a Mendelian dominant character. Independent segregation was demonstrated between Rla and the ABO, MNSs, Rh, Lutheran, Duffy and Kidd blood group systems, the ABH secrctor genes, and sex. The Rla antigen is inactivated by papain treatment. The corresponding antibody, anti-Rla, has so far been found in three sera that also contain antibodies to other rare blood group antigens.

Published
1980
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39. Thyroid antibodies in blood donors: prevalence and clinical significance

Author

Bjøro, T., Gaarder, P. I., Smeland, E. B., and Kornstad, L.

Abstract

Abstract. In sera from 1643 randomly selected blood donors examined in 1979 thyroglobulin antibodies (TGA) were found in 3.4% and thyroid microsomal antibodies (TMA) in 7.0%. TMA, but not TGA, showed significant sex and age relationships. Eighty-two donors with TMA titres ≥ 1600 and/or TGA titres ≥ 128 were available for a follow-up study in 1982. In 69 of these with TMA there was a significantly increased incidence of pathological thyroid function-test values (T4, T3and TSH) as compared to age- and sexmatched donors without thyroid antibodies. In this group 9 individuals had overt and three latent hypothyroidism, two individuals were found with symptomless autoimmune thyroiditis and one with a non-toxic nodular goitre. Only 2 of these had been diagnosed before 1982, and none recognized before 1979. In addition, the group contained 9 individuals who had been treated because of hyperthyroidism, all except one before 1979. The likelihood of detecting a previously unrecognized hypothyroidism increased with increasing TMA titre. In contrast, the 24 donors with TGA did not show an increased frequency of pathological thyroid function-test- values in 1982.

Published
1984
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40. A Rare Blood Group Antigen, Rla (Rosenlund)

Author

Kornstad, L.

Abstract

A 'new' rare blood group antigen Rla (Rosenlund) is reported. The antigen is present in about 1 in 900 Norwegians. Nine unrelated Rl(a+) people were found. Studies of seven families, leading to the discovery of 20 additional Rl(a+) people, indicated the antigen is inherited as a Mendelian dominant character. Independent segregation was demonstrated between RZa and the ABO, MNSs, Rh, Lutheran, Duffy and Kidd blood group systems, the ABU secretor genes, and sex. The Rla antigen is inactivated by papain treatment. The corresponding antibody, anti-Rla, has so far been found in three sera that also contain antibodies to other rare blood group antigens.

Published
1981
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41. IgG subclass distribution of anti-Rh, anti-Kell and anti-Duffy antibodies measured by sensitive haemagglutination assays

Author

Michaelsen, T E and Kornstad, L

Subjects
Rh-Hr Blood-Group System, Sheep, Kell Blood-Group System, Immune Sera, Hemagglutination Tests, complex mixtures, Isoantibodies, Immunoglobulin G, parasitic diseases, Blood Group Antigens, Animals, Humans, Rabbits, Duffy Blood-Group System, Research Article
Abstract

The IgG subclass distribution of anti Rh antibodies (anti-D, 'anti-Du', anti-c, anti-E), anti-Kell and anti-Duffy (anti-Fya) antibodies was measured by two haemagglutination techniques on microtitre plates. The first technique involved rabbit subclass specific antisera which were used to agglutinate red cells previously reacted with the patients' antibodies at high concentration. The second, which was more sensitive, had an additional step by introducing sheep anti-rabbit antibodies (sandwich technique). By the sensitive sandwich technique we revealed, for anti-D antibodies: IgG1 8/19, IgG3 1/19, IgG1/IgG3 8/19, IgG1/IgG2/IgG3/IgG 41/19, IgG1/IgG4 1/19; for the Du reactive anti-D antibodies: IgG1 1/8, IgG1/IgG3 1/8, IgG1/IgG3/IgG4 6/8; for the anti-E antibodies: IgG1/IgG2/IgG4 2/3, IgG1/IgG2/IgG3/IgG4 1/3; for the anti-c antibodies: IgG1 2/5, IgG3 1/5, IgG1/IgG3 1/5; for the anti-Kell antibodies: IgG1 9/20, IgG1/IgG3 1/20, IgG1/IgG4 8/20, IgG1/IgG3/IgG4 2/20; and for anti-Duffy antibodies: IgG1 1/8, IgG1/IgG4 7/8. These results are partly at variance with previously published results.

Published
1987

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