Search

Objectives: Helicobacter pylori (H.Pylori) is a Gram(-), microaerophilic bacteria and the etiological factor of chronic activegastritis and peptic ulcer. Some studies indicated that this bacterium found atoral cavity which is a potential reservoir for stomach. Several studies showedthat H. pylori may found in salivaand subgingival plaque of chronic periodontitis patients. However, there is nodata related to aggressive periodontitis patients. In this study, we aimed todetermine the prevalence of H. pyloriin subgingival plaque samples of chronic, aggressive periodontitis andgingivitis patients and to increase the awareness of the patients for gastricproblems. Materialsand Methods: This study included 155 patients (61 with gingivitis,60 with chronic periodontitis, and 34 with aggressive periodontitis) who didnot have gastric disease symptom and did not use antibiotics in the last 3months. The subgingival plaque samples were taken using sterile paper points.The existence of H. pylori, A. actinomycetemcomitans,and P. gingivaliswas detected by RT-PCR. Results: H. pylori was not detected in any groupsat the end of microbiological analysis.However, a high occurrence of A. actinomycetemcomitans (97.1%) and P. gingivalis(100%) was observed in the aggressive periodontitis group. However, A. actinomycetemcomitansand P. gingivalis were found in 30% and 21.7% of patients,respectively, with chronic periodontitis. A.actinomycetemcomitans and P. gingivaliswere found in 24.6% of patients in the gingivitis group.Conclusions: H. pylori were not detected in samples, indicating that subgingival plaque may notbe a primary reservoir for this bacterium., Amaç: Helicobacter pylori (H. Pylori),bir gram (-), mikroaerofilik bakteri olup, kronik aktif gastrit ve peptikülserin etyolojik faktörüdür. Bazı çalışmalar, bu bakterinin, oral kavitedebulunduğunda, mide için potansiyel rezervuar olabileceğini göstermiştir. Çeşitliçalışmalar, H. pylori'nin kronik periodontitisli hastaların tükrük vesubgingival plaklarında görülebileceğini göstermiştir. Bununla birlikte agresifperiodontitis hastaları ile ilgili herhangi bir veri yoktur. Bu çalışmada, kronik, agresifperiodontitis ve gingivitis hastalarının subgingival plak örneklerinde H.pylori prevalansını saptamayı ve hastaların gastrik problemler konusundabilinçlenmesini arttırmayı amaçladık.Gereçve Yöntem: Bu çalışma,gastrik hastalık semptomu olmayan ve son 3 ayda antibiyotik kullanmayan 155hasta (61 adet gingivitis, 60'ı kronik periodontitisli ve 34 agresifperiodontitisli) içermekteydi. Subgingivalplak örnekleri steril paper point kullanılarak alındı. H.pylori, A. actinomycetemcomitans ve P. gingivalis'in varlığı RT-PCR ile tespitedildi.Bulgular: Mikrobiyolojikanalizin sonunda herhangi bir grupta H. pylori tespit edilmedi. Bununla birlikte, agresif periodontit grubunda yüksekoranda A. actinomycetemcomitans (%97.1) ve P. gingivalis (%100) görülmüştür. Bununla birlikte, A.actinomycetemcomitans ve P. gingivalis, kronik periodontitisli hastalarınsırasıyla %30 ve %21.7'sinde bulunmuştur. A. actinomycetemcomitans ve P.gingivalis gingivitisli hastaların %24.6'sında bulundu. Sonuç: H. pylori, örneklerde saptanmamışolması, subgingival plağın bu bakteri için birincil rezervuar olmayabileceğinigösterdi.

Objective The aim of the study was to evaluate the effects of vitamin D deficiency on the mandibular bone structure by fractal analysis and panoramic morphometric indices. Methods Ninety participants were divided into three groups as 30 individuals with severe vitamin D deficiency, 30 individuals with vitamin D deficiency, and 30 individuals with vitamin D sufficiency. Fractal dimension analysis (FD), panoramic mandibular index (PMI), mandibular cortical index (MCI), and mandibular cortical thickness measurement (CTM) were evaluated on panoramic radiographs. Results FD values of the patients with vitamin D deficiency were found to be statistically lower than the patients with vitamin D sufficiency (p < 0.05). FD value of supracortical area above the angulus mandible (FD2) in patients with severe vitamin D deficiency was significantly lower than FD values (p = 0.002). There was no statistically significant difference between the groups in the CTM (p > 0.05). PMI was significantly lower in patients with severe vitamin D deficiency (p < 0.001). There was a significant difference in MCI values between the groups (p < 0.05). Conclusion Vitamin D deficiency causes a decrease in bone mineral density in the mandible, and an increase in alveolar porosity. FD analysis and radiomorphometric indices in panoramic radiographs can be used to assess osteoporotic changes in patients with vitamin D deficiency.