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3. A call for a better understanding of the role of dietary amino acids and post-translational protein modifications of microbiome in the progression of CKD

Author

Koppe, Laetitia, Beddhu, Srinivasan, Chauveau, Philippe, Kovesdy, Csaba P, Mafra, Denise, Joshi, Shivam, Kalantar-Zadeh, Kamyar, and Fouque, Denis

Subjects
Biomedical and Clinical Sciences, Clinical Sciences, Amino Acids, Diet, Gastrointestinal Microbiome, Humans, Protein Processing, Post-Translational, Renal Insufficiency, Chronic, Urology & Nephrology, Clinical sciences
Published
2021

6. Kidney cachexia or protein‐energy wasting in chronic kidney disease: facts and numbers

Author

Koppe, Laetitia, Fouque, Denis, and Kalantar‐Zadeh, Kamyar

Subjects
Nutrition, Kidney Disease, Renal and urogenital, Zero Hunger, Cachexia, Energy Metabolism, Humans, Kidney, Nutritional Status, Renal Dialysis, Renal Insufficiency, Chronic, Wasting Syndrome, Weight Loss, Protein-energy wasting, Chronic kidney disease, Energy intake, Malnutrition, Physiology, Clinical Sciences, Human Movement and Sports Sciences
Abstract

BackgroundWeight loss and homeostatic disturbances of both energy and protein balances are characteristics of several illnesses including cancer, heart failure, and chronic kidney disease (CKD). Different definitions have been used to describe this deleterious process. The term protein-energy wasting (PEW) has been proposed for CKD patients by the International Society of Renal Nutrition and Metabolism.MethodsWe searched the publication in Medline from February 2008 to September 2018 using PEW or cachexia in their title.ResultsSince its inception, the term PEW has been exceptionally successful, highlighted by 327 original publications referenced in PubMed over 10 years. Using this classification, several studies have confirmed that PEW is among the strongest predictors of mortality in CKD patients [hazard ratio of 3.03; confidence interval of 1.69-5.26 in 1068 haemodialysis patients and 1.40 (1.04-1.89) in 1487 non-dialysed patients across PEW stages 0 to 4]. Based on this classification, prevalence of PEW is 28% to 54% among 16 434 adults undergoing maintenance dialysis. PEW prevalence increases when renal function declines, that is, from 5% is a mandatory criterion for cachexia but supportive for PEW.ConclusionsThe recent understanding of cachexia physiopathology during CKD progression suggests that PEW and cachexia are closely related and that PEW corresponds the initial state of a continuous process that leads to cachexia, implicating the same metabolic pathways as in other chronic diseases. Despite the success of the definition of PEW, using a more uniform term such as 'kidney disease cachexia' could be more helpful to design future research through collaborative groups of researchers with focus on cachexia.

Published
2019

10. A prospective observational study for justification, safety, and efficacy of a third dose of mRNA vaccine in patients receiving maintenance hemodialysis

Author

François, Chantrel, Mathilde, Reydit, Aurélien, Tiple, Clémence, Bechade, Abdelkader, Bemrah, Cécile, Vigneau, Bénédicte, Sautenent, Isabelle, Kazes, Cécile, Courivaud, Jean-Marc, Gabriel, Stéphane, Edet, Lucile, Mercadal, Olivier, Moranne, Fatouma, Toure, Emmanuelle, Laurain, Alex, Ranlin, Nathalie, Longlune, François, Glowacki, Jean-Michel, Tivollier, Philippe, Brunet, Fréderic, Lavainne, Etienne, Berard, Ayman, Sarraj, Marc, Bauwens, Pascale, Testevuide, Henri, Vacher Coponat, Roula, Galland, Nicole, Schauder, Louis -Rachid, Salmi, Damiano, Cerasuolo, Anaïs, Tendron-Franzin, Sahar, Bayat, Michel, Halimi Jean, Aurore, Wolak, Stéphanie, Gentile, Bénédicte, Devictor, Elisabeth, Monnet, Denis, Boucaut Maitre, Mathieu, Nacher, Véronique, Merle, Jean-Philippe, Jais, Jean-Pierre, Daures, Alain, Vergnenegre, Carole, Loos-Ayav, Sylvie, Merle, Marc, Hazzan, Shirley, Gervolino, Jean-Michel, Nguyen, Silvia, Iacobelli, Espi, Maxime, Charmetant, Xavier, Barba, Thomas, Mathieu, Cyrille, Pelletier, Caroline, Koppe, Laetitia, Chalencon, Elodie, Kalbacher, Emilie, Mathias, Virginie, Ovize, Anne, Cart-Tanneur, Emmanuelle, Bouz, Christine, Pellegrina, Laurence, Morelon, Emmanuel, Juillard, Laurent, Fouque, Denis, Couchoud, Cécile, and Thaunat, Olivier

Published
2022
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11. List of contributors

Author

Adamczak, Marcin, primary, Amabile, Maria Ida, additional, Apata, Ibironke W., additional, Apetrii, Mugurel, additional, Avesani, Carla Maria, additional, Bahner, Udo, additional, Bailey, James L., additional, Bansal, Anip, additional, Barba, Christophe, additional, Bellorin-Font, Ezequiel, additional, Bross, Rachelle, additional, Brown-Tortorici, Amanda, additional, Burnier, Michel, additional, Burrowes, Jerrilynn Denise, additional, Carrero, Juan Jesús, additional, Cervantes, MacKenzie K., additional, Chadban, Steven, additional, Chadha, Vimal, additional, Chan, Maria, additional, Chan, Winnie, additional, Chazot, Charles, additional, Chiang, Janet M., additional, Chonchol, Michel, additional, Chwastiak, Lydia, additional, Covic, Adrian, additional, Cuppari, Lilian, additional, Dahl, Neera K., additional, Di Iorio, Biagio, additional, Di Mario, Francesca, additional, Druml, Wilfred, additional, Dukkipati, Ramanath, additional, Fazeli, Gholamreza, additional, Fiaccadori, Enrico, additional, Finkelstein, Fredric, additional, Fouque, Denis, additional, Franch, Harold A., additional, Friedman, Allon N., additional, Garimella, Pranav S., additional, Glassock, Richard J., additional, Goldfarb, David S., additional, González-Ortiz, Ailema, additional, Goraya, Nimrit, additional, Gutiérrez, Orlando M., additional, Hanafusa, Norio, additional, Heidland, August, additional, Heimbürger, Olof, additional, Hirschberg, Raimund, additional, Ikizler, T. Alp, additional, Johansen, Kirsten, additional, Johnson, Richard J., additional, Joshi, Shivam, additional, Kalantar-Zadeh, Kamyar, additional, Kang, Duk-Hee, additional, Kaysen, George A., additional, Kim, Jun-Chul, additional, Kistler, Brandon, additional, Koppe, Laetitia, additional, Kopple, Joel D., additional, Kovesdy, Csaba P., additional, Kramer, Holly J., additional, Kurokawa, Kiyoshi, additional, Lindholm, Bengt, additional, Martin, Kevin J., additional, Martino, Steve, additional, Marzocco, Stefania, additional, Massry, Shaul G., additional, Massy, Ziad A., additional, Mitch, William E., additional, Miyata, Toshio, additional, Molfino, Alessio, additional, Moradi, Hamid, additional, Nakagawa, Takahiko, additional, Narasaki, Yoko, additional, Puzziferri, Nancy, additional, Quinn, Noel, additional, Raj, Dominic S., additional, Raphael, Kalani L., additional, Regunathan-Shenk, Renu, additional, Rhee, Connie M., additional, Ritz, Eberhard, additional, Sabatino, Alice, additional, Sarnak, Mark J., additional, Scialla, Julia, additional, Seefried, Lothar, additional, Sellinger, John, additional, Shah, Anuja, additional, Shah, Neal B., additional, Shah, Sudhir V., additional, Singh, Manisha, additional, Spatola, Leonardo, additional, Stanton, Robert C., additional, Steiber, Alison L., additional, Stenvinkel, Peter, additional, St-Jules, David E., additional, Storer, Thomas W., additional, Sumida, Keiichi, additional, Sussman-Dabach, Elizabeth J., additional, Swaminathan, Sundararaman, additional, Sy, John, additional, Tantisattamo, Ekamol, additional, Vaziri, Nosratola D., additional, Voinescu, Alexandra, additional, Wang, Angela Yee-Moon, additional, Warady, Bradley A., additional, Weiner, Daniel E., additional, Wesson, Donald E., additional, Wiecek, Andrzej, additional, Williams, Mark E., additional, Wolfe, Bruce M., additional, Workeneh, Biruh T., additional, and Zhao, Ying-Yong, additional

Published
2022
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13. The ROMANOV study found impaired humoral and cellular immune responses to SARSCov-2 mRNA vaccine in virus unexposed patients receiving maintenance hemodialysis.

Author

Espi, Maxime, Charmetant, Xavier, Barba, Thomas, Koppe, Laetitia, Pelletier, Caroline, Kalbacher, Emilie, Chalencon, Elodie, Mathias, Virginie, Ovize, Anne, Cart-Tanneur, Emmanuelle, Bouz, Christine, Pellegrina, Laurence, Morelon, Emmanuel, Fouque, Denis, Juillard, Laurent, and Thaunat, Olivier

Published
2021
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14. Accumulation of natriuretic peptides is associated with protein energy wasting and activation of browning in white adipose tissue in chronic kidney disease

Author

Luce, Mathilde, Barba, Christophe, Yi, Dan, Mey, Anne, Roussel, Damien, Bres, Emilie, Benoit, Bérengère, Pastural, Myriam, Granjon, Samuel, Szelag, Jean Christophe, Laville, Maurice, Arkouche, Walid, Bouchara, Anais, Nyam, Elsa, Fouque, Denis, Soulage, Christophe O., and Koppe, Laetitia

Published
2020
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16. Role of altered intestinal microbiota in systemic inflammation and cardiovascular disease in chronic kidney disease

Author

Mafra, Denise, Lobo, Julie C, Barros, Amanda F, Koppe, Laetitia, Vaziri, Nosratola D, and Fouque, Denis

Subjects
Prevention, Kidney Disease, Digestive Diseases, Aetiology, 2.1 Biological and endogenous factors, Renal and urogenital, Cardiovascular, Oral and gastrointestinal, Good Health and Well Being, Animals, Carbohydrate Metabolism, Cardiovascular Diseases, Fats, Female, Humans, Inflammation, Intestinal Mucosa, Intestines, Lipopolysaccharides, Male, Microbiota, Prebiotics, Pregnancy, Probiotics, Proteins, Renal Insufficiency, Chronic, cardiovascular disease, chronic kidney disease, endotoxins, inflammation, intestinal microbiota, nutrients, oxidative stress, prebiotic, probiotics, uremic toxins, Microbiology, Medical Microbiology
Abstract

The normal intestinal microbiota plays a major role in the maintenance of health and disease prevention. In fact, the alteration of the intestinal microbiota has been shown to contribute to the pathogenesis of several pathological conditions, including obesity and insulin resistance, among others. Recent studies have revealed profound alterations of the gut microbial flora in patients and animals with chronic kidney disease (CKD). Alterations in the composition of the microbiome in CKD may contribute to the systemic inflammation and accumulation of gut-derived uremic toxins, which play a central role in the pathogenesis of accelerated cardiovascular disease and numerous other CKD-associated complications. This review is intended to provide a concise description of the potential role of the CKD-associated changes in the gut microbiome and its potential role the pathogenesis of inflammation and uremic toxicity. In addition, the potential efficacy of pre- and pro-biotics in the restoration of the microbiome is briefly described.

Published
2014

21. Protein-bound uremic toxins: putative modulators of calcineurin inhibitor exposure

Author

Carmen Lab, KOPPE Laetitia, Cardiovasculaire, métabolisme, diabétologie et nutrition (CarMeN), Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Hospices Civils de Lyon (HCL)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Institut National de Recherche pour l’Agriculture, l’Alimentation et l’Environnement (INRAE), Centre Hospitalier Lyon Sud [CHU - HCL] (CHLS), Hospices Civils de Lyon (HCL), and CarMeN, laboratoire

Subjects
[SDV] Life Sciences [q-bio], Transplantation, Nephrology, [SDV]Life Sciences [q-bio], Calcineurin Inhibitors, Humans, Uremic Toxins, Kidney Transplantation, Indican, Uremia
Abstract

International audience; No abstract available

Published
2022

22. Indoxyl sulfate inhibits muscle cell differentiation via Myf6/MRF4 and MYH2 downregulation.

Author

Bataille, Stanislas, McKay, Nathalie, Koppe, Laetitia, Beau, Alice, Benoit, Bérengère, Bartoli, Marc, Silva, Nathalie Da, Poitevin, Stéphane, Aniort, Julien, Chermiti, Rania, Burtey, Stéphane, and Dou, Laetitia

Subjects
MYOSIN, MUSCLE cells, REVERSE transcriptase polymerase chain reaction, CELL differentiation, GENE expression, ARYL hydrocarbon receptors
Abstract

Background Chronic kidney disease (CKD) is associated with a significant decrease in muscle strength and mass, possibly related to muscle cell damage by uremic toxins. Here, we studied in vitro and in vivo the effect of indoxyl sulfate (IS), an indolic uremic toxin, on myoblast proliferation, differentiation and expression of myogenic regulatory factors (MRF)—myoblast determination protein 1 (MyoD1), myogenin (Myog), Myogenic Factor 5 (Myf5) and myogenic regulatory factor 4 (Myf6 /MRF4)—and expression of myosin heavy chain, Myh2. Methods C2C12 myoblasts were cultured in vitro and differentiated in myotubes for 7 days in the presence of IS at a uremic concentration of 200 µM. Myocytes morphology and differentiation was analyzed after hematoxylin-eosin staining. MRF genes' expression was studied using reverse transcription polymerase chain reaction in myocytes and 5/6th nephrectomized mice muscle. Myf6 /MRF4 protein expression was studied using enzyme-linked immunosorbent assay; MYH2 protein expression was studied using western blotting. The role of Aryl Hydrocarbon Receptor (AHR)—the cell receptor of IS—was studied by adding an AHR inhibitor into the cell culture milieu. Results In the presence of IS, the myotubes obtained were narrower and had fewer nuclei than control myotubes. The presence of IS during differentiation did not modify the gene expression of the MRFs Myf5, MyoD1 and Myog, but induced a decrease in expression of Myf6 /MRF4 and MYH2 at the mRNA and the protein level. AHR inhibition by CH223191 did not reverse the decrease in Myf6/ MRF4 mRNA expression induced by IS, which rules out the implication of the ARH genomic pathway. In 5/6th nephrectomized mice, the Myf6 /MRF4 gene was down-regulated in striated muscles. Conclusion In conclusion, IS inhibits Myf6/ MRF4 and MYH2 expression during differentiation of muscle cells, which could lead to a defect in myotube structure. Through these new mechanisms, IS could participate in muscle atrophy observed in CKD. [ABSTRACT FROM AUTHOR]

Published
2024
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23. Rapid Waning of Immune Memory Against SARS-CoV-2 in Maintenance Hemodialysis Patients After mRNA Vaccination and Impact of a Booster Dose

Author

Espi, Maxime, primary, Charmetant, Xavier, additional, Mathieu, Cyrille, additional, Lalande, Alexandre, additional, Decimo, Didier, additional, Koppe, Laetitia, additional, Pelletier, Caroline, additional, Ovize, Anne, additional, Barbry, Alexia, additional, Morelon, Emmanuel, additional, Kalbacher, Emilie, additional, Fouque, Denis, additional, and Thaunat, Olivier, additional

Published
2023
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27. A call for a better understanding of the role of dietary amino acids and post-translational protein modifications of the microbiome in the progression of CKD.

Author

Koppe, Laetitia, Koppe, Laetitia, Beddhu, Srinivasan, Chauveau, Philippe, Kovesdy, Csaba P, Mafra, Denise, Joshi, Shivam, Kalantar-Zadeh, Kamyar, Fouque, Denis, Koppe, Laetitia, Koppe, Laetitia, Beddhu, Srinivasan, Chauveau, Philippe, Kovesdy, Csaba P, Mafra, Denise, Joshi, Shivam, Kalantar-Zadeh, Kamyar, and Fouque, Denis

Published
2021

29. Urea impairs β cell glycolysis and insulin secretion in chronic kidney disease

Author

Koppe, Laetitia, Nyam, Elsa, Vivot, Kevin, Fox, Jocelyn E. Manning, Dai, Xiao-Qing, Nguyen, Bich N., Trudel, Dominique, Attane, Camille, Moulle, Valentine S., MacDonald, Patrick E., Ghislain, Julien, and Poitout, Vincent

Subjects
Urea -- Health aspects, Chronic kidney failure -- Physiological aspects -- Care and treatment, Pancreatic beta cells -- Physiological aspects, Glycolysis -- Health aspects, Health care industry
Abstract

Disorders of glucose homeostasis are common in chronic kidney disease (CKD) and are associated with increased mortality, but the mechanisms of impaired insulin secretion in this disease remain unclear. Here, we tested the hypothesis that defective insulin secretion in CKD is caused by a direct effect of urea on pancreatic β cells. In a murine model in which CKD is induced by 5/6 nephrectomy (CKD mice), we observed defects in glucose-stimulated insulin secretion in vivo and in isolated islets. Similarly, insulin secretion was impaired in normal mouse and human islets that were cultured with disease-relevant concentrations of urea and in islets from normal mice treated orally with urea for 3 weeks. In CKD mouse islets as well as ureaexposed normal islets, we observed an increase in oxidative stress and protein O-GlcNAcylation. Protein O-GlcNAcylation was also observed in pancreatic sections from CKD patients. Impairment of insulin secretion in both CKD mouse and urea-exposed islets was associated with reduced glucose utilization and activity of phosphofructokinase 1 (PFK-1), which could be reversed by inhibiting O-GlcNAcylation. Inhibition of O- GlcNAcylation also restored insulin secretion in both mouse models. These results suggest that insulin secretory defects associated with CKD arise from elevated circulating levels of urea that increase islet protein O-GlcNAcylation and impair glycolysis., Introduction Disorders of glucose homeostasis affect approximately 50% of patients suffering from chronic kidney disease (CKD) (1-4) and play a major role in their mortality (5, 6). Glucose homeostasis relies [...]

Published
2016
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33. A prospective observational study for justification, safety, and efficacy of a third dose of mRNA vaccine in patients receiving maintenance hemodialysis

Author

Espi, Maxime, primary, Charmetant, Xavier, additional, Barba, Thomas, additional, Mathieu, Cyrille, additional, Pelletier, Caroline, additional, Koppe, Laetitia, additional, Chalencon, Elodie, additional, Kalbacher, Emilie, additional, Mathias, Virginie, additional, Ovize, Anne, additional, Cart-Tanneur, Emmanuelle, additional, Bouz, Christine, additional, Pellegrina, Laurence, additional, Morelon, Emmanuel, additional, Juillard, Laurent, additional, Fouque, Denis, additional, Couchoud, Cécile, additional, Thaunat, Olivier, additional, François, Chantrel, additional, Mathilde, Reydit, additional, Aurélien, Tiple, additional, Clémence, Bechade, additional, Abdelkader, Bemrah, additional, Cécile, Vigneau, additional, Bénédicte, Sautenent, additional, Isabelle, Kazes, additional, Cécile, Courivaud, additional, Jean-Marc, Gabriel, additional, Stéphane, Edet, additional, Lucile, Mercadal, additional, Olivier, Moranne, additional, Fatouma, Toure, additional, Emmanuelle, Laurain, additional, Alex, Ranlin, additional, Nathalie, Longlune, additional, François, Glowacki, additional, Jean-Michel, Tivollier, additional, Philippe, Brunet, additional, Fréderic, Lavainne, additional, Etienne, Berard, additional, Ayman, Sarraj, additional, Marc, Bauwens, additional, Pascale, Testevuide, additional, Henri, Vacher Coponat, additional, Roula, Galland, additional, Nicole, Schauder, additional, Louis -Rachid, Salmi, additional, Damiano, Cerasuolo, additional, Anaïs, Tendron-Franzin, additional, Sahar, Bayat, additional, Michel, Halimi Jean, additional, Aurore, Wolak, additional, Stéphanie, Gentile, additional, Bénédicte, Devictor, additional, Elisabeth, Monnet, additional, Denis, Boucaut Maitre, additional, Mathieu, Nacher, additional, Véronique, Merle, additional, Jean-Philippe, Jais, additional, Jean-Pierre, Daures, additional, Alain, Vergnenegre, additional, Carole, Loos-Ayav, additional, Sylvie, Merle, additional, Marc, Hazzan, additional, Shirley, Gervolino, additional, Jean-Michel, Nguyen, additional, and Silvia, Iacobelli, additional

Published
2022
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37. The impact of dietary nutrient intake on gut microbiota in the progression and complications of chronic kidney disease

Author

Koppe, Laetitia, Soulage, Christophe, Service de Néphrologie [Lyon], Hospices Civils de Lyon (HCL)-Centre Hospitalier Lyon Sud [CHU - HCL] (CHLS), Hospices Civils de Lyon (HCL), Cardiovasculaire, métabolisme, diabétologie et nutrition (CarMeN), Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Hospices Civils de Lyon (HCL)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Institut National de Recherche pour l’Agriculture, l’Alimentation et l’Environnement (INRAE), and ROSSI, Sabine

Subjects
Dietary Fiber, [SDV.MHEP] Life Sciences [q-bio]/Human health and pathology, microbiote intestinal, Sulfates, diète, Nutrients, maladie rénale chronique, Fatty Acids, Volatile, Diet, Gastrointestinal Microbiome, toxines urémiques, Eating, Nephrology, apport en protéines, Humans, Amino Acids, Renal Insufficiency, Chronic, Indican, [SDV.MHEP]Life Sciences [q-bio]/Human health and pathology, Ecosystem
Abstract

International audience; La maladie rénale chronique a été associée à des changements dans la fonction et la composition du microbiote intestinal. L'écosystème de l'intestin humain se compose de milliards de micro-organismes formant un authentique organe métaboliquement actif qui est alimenté par des nutriments pour produire des composés bioactifs. Ces métabolites dérivés du microbiote peuvent être protecteurs pour la fonction rénale (par exemple, les acides gras à chaîne courte issus de la fermentation des fibres alimentaires) ou délétères (par exemple, les toxines urémiques dérivées de l'intestin telles que le N-oxyde de triméthylamine, le sulfate de p-crésyle et le sulfate d'indoxyle de la fermentation des acides aminés). Bien que l'alimentation soit la pierre angulaire de la prise en charge du patient atteint d'insuffisance rénale chronique, elle reste une composante relativement sous-utilisée de l'arsenal thérapeutique du clinicien. Dans cette revue, nous décrivons les dernières avancées dans la compréhension de la diaphonie alimentation-microbiote dans le contexte urémique et comment cette communication pourrait contribuer à la progression et aux complications de la maladie rénale chronique. Nous discutons ensuite de la manière dont ces connaissances pourraient être exploitées pour des stratégies nutritionnelles personnalisées afin d'empêcher les patients atteints d'insuffisance rénale chronique de progresser vers l'insuffisance rénale et ses conséquences néfastes.

Published
2021

39. The ROMANOV study found impaired humoral and cellular immune responses to SARS-CoV-2 mRNA vaccine in virus-unexposed patients receiving maintenance hemodialysis

Author

Espi, Maxime, primary, Charmetant, Xavier, additional, Barba, Thomas, additional, Koppe, Laetitia, additional, Pelletier, Caroline, additional, Kalbacher, Emilie, additional, Chalencon, Elodie, additional, Mathias, Virginie, additional, Ovize, Anne, additional, Cart-Tanneur, Emmanuelle, additional, Bouz, Christine, additional, Pellegrina, Laurence, additional, Morelon, Emmanuel, additional, Fouque, Denis, additional, Juillard, Laurent, additional, and Thaunat, Olivier, additional

Published
2021
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43. Justification, safety, and efficacy of a third dose of mRNA vaccine in maintenance hemodialysis patients: a prospective observational study

Author

Espi, Maxime, primary, Charmetant, Xavier, additional, Barba, Thomas, additional, Pelletier, Caroline, additional, Koppe, Laetitia, additional, Chalencon, Elodie, additional, Kalbacher, Emilie, additional, Mathias, Virginie, additional, Ovize, Anne, additional, Cart-Tanneur, Emmanuelle, additional, Bouz, Christine, additional, Pellegrina, Laurence, additional, Morelon, Emmanuel, additional, Juillard, Laurent, additional, Fouque, Denis, additional, Couchoud, Cécile, additional, and Thaunat, Olivier, additional

Published
2021
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44. A Low Aromatic Amino-Acid Diet Improves Renal Function and Prevent Kidney Fibrosis in Mice with Chronic Kidney Disease

Author

Barba, Christophe, primary, Benoit, Bérengère, additional, Emilie, Bres, additional, Chanon, Stéphanie, additional, Vieille-Marchiset, Aurélie, additional, Pinteur, Claudie, additional, Pesenti, Sandra, additional, Glorieux, Griet, additional, Picard, Cécile, additional, Fouque, Denis, additional, Soulage, Christophe O., additional, and Koppe, Laetitia, additional

Published
2021
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49. COVID-19 vaccine acceptance among haemodialysis patients: a French survey

Author

Andrian, Titus, primary, Koppe, Laetitia, additional, Novel, Etienne, additional, Massat, Maeva, additional, Barba, Christophe, additional, Ioriatti, Donatella, additional, Barnel, Cecile, additional, Villar, Emmanuel, additional, Luce, Mathilde, additional, Guerraoui, Abdallah, additional, Mezaache, Sarah, additional, Kalbacher, Emilie, additional, Tournebize, Corentin, additional, Fouque, Denis, additional, and Chalencon, Elodie, additional

Published
2021
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