666 results on '"Koo, Edward H"'
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2. Differential effects of familial Alzheimer’s disease-causing mutations on amyloid precursor protein (APP) trafficking, proteolytic conversion, and synaptogenic activity
3. Localized APP expression results in progressive network dysfunction by disorganizing spike timing
4. Machine learning classifies predictive kinematic features in a mouse model of neurodegeneration.
5. Evidence for a clathrin-independent endocytic pathway for APP internalization in the neuronal somatodendritic compartment
6. World-Wide FINGERS Network: A global approach to risk reduction and prevention of dementia.
7. Caspase Activation and Caspase-Mediated Cleavage of APP Is Associated with Amyloid β-Protein-Induced Synapse Loss in Alzheimer’s Disease
8. PERK-mediated induction of microRNA-483 disrupts cellular ATP homeostasis during the unfolded protein response
9. Extracellular Vesicle-Enriched miRNA-Biomarkers Show Improved Utility for Detecting Alzheimer’s Disease Dementia and Medial Temporal Atrophy
10. Enhanced cleavage of APP by co-expressed Bace1 alters the distribution of APP and its fragments in neuronal and non-neuronal cells
11. Tauopathy-associated PERK alleles are functional hypomorphs that increase neuronal vulnerability to ER stress
12. Chronic cerebral hypoperfusion alters the CypA-EMMPRIN-gelatinase pathway: Implications for vascular dementia
13. Cholestenoic acid, an endogenous cholesterol metabolite, is a potent γ-secretase modulator
14. Cerebral transcriptome analysis reveals age-dependent progression of neuroinflammation in P301S mutant tau transgenic male mice
15. Evidence that NLRC4 inflammasome mediates apoptotic and pyroptotic microglial death following ischemic stroke
16. Specific disruption of hippocampal mossy fiber synapses in a mouse model of familial Alzheimer's disease.
17. Localized APP expression results in progressive network dysfunction by disorganizing spike timing
18. Probing sporadic and familial Alzheimer’s disease using induced pluripotent stem cells
19. Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's disease
20. Biology and Pathophysiology of the Amyloid Precursor Protein
21. Transethnic genome-wide scan identifies novel Alzheimer's disease loci
22. The Aspartate-257 of Presenilin 1 is Indispensable for Mouse Development and Production of β-Amyloid Peptides through β-Catenin-Independent Mechanisms
23. Loss of Presenilin 1 is Associated with Enhanced β-Catenin Signaling and Skin Tumorigenesis
24. Presenilin 1 Negatively Regulates β-Catenin/T Cell Factor/Lymphoid Enhancer Factor-1 Signaling Independently of β-Amyloid Precursor Protein and Notch Processing
25. APP upregulation contributes to retinal ganglion cell degeneration via JNK3
26. Interaction between Amyloid Precursor Protein and Presenilins in Mammalian Cells: Implications for the Pathogenesis of Alzheimer Disease
27. The Utility of Brief Cognitive Tests for Patients With Type 2 Diabetes Mellitus: A Systematic Review
28. Modulation of amyloid β-protein clearance and Alzheimer’s disease susceptibility by the LDL receptor–related protein pathway
29. Chronic cerebral hypoperfusion alters the CypA-EMMPRIN-gelatinase pathway: Implications for vascular dementia
30. Differential effects of familial Alzheimer’s disease-causing mutations on amyloid precursor protein (APP) trafficking, proteolytic conversion, and synaptogenic activity
31. Localized APP pathology in the hippocampus is sufficient to result in progressive disorganization of the timing of neuronal firing patterns
32. Aβ-Induced Toxicity Mediated by Caspase Cleavage of the Amyloid Precursor Protein (APP)
33. APP Biology, Processing and Function
34. PS1 Interacts with and Facilitates β-Catenin Turnover
35. The multifaceted nature of amyloid precursor protein and its proteolytic fragments: friends and foes
36. sj-pdf-1-jcb-10.1177_0271678X221146401 - Supplemental material for Chronic cerebral hypoperfusion alters the CypA-EMMPRIN-gelatinase pathway: Implications for vascular dementia
37. And four equals one: Presenilin takes the γ-secretase role by itself
38. Amyloid precursor protein, Alzheimer’s disease, and Down’s syndrome
39. Neurodegenerative Diseases: What Is to Be Done?
40. Preclinical and Clinical Understanding: Major Gaps in Our Understanding and Capabilities
41. A
42. Rarity of the Alzheimer Disease–Protective APP A673T Variant in the United States
43. Polarized Sorting of β-Amyloid Precursor Protein and Its Proteolytic Products in MDCK Cells Is Regulated by Two Independent Signals
44. Trafficking of Cell Surface β-Amyloid Precursor Protein: Retrograde and Transcytotic Transport in Cultured Neurons
45. Precursor if Amyloid Protein in Alzheimer Disease Undergoes Fast Anterograde Axonal Transport
46. Polarized Secretion of β-Amyloid Precursor Protein and Amyloid β-Peptide in MDCK Cells
47. Secreted β-Amyloid Precursor Protein Stimulates Mitogen-Activated Protein Kinase and Enhances τ Phosphorylation
48. Amyloid Precursor Protein in Aged Nonhuman Primates
49. Increased Expression of β-Amyloid Precursor Protein During Neuronal Differentiation is Not Accompanied by Secretory Cleavage
50. Amyloid β-Protein as a Substrate Interacts with Extracellular Matrix to Promote Neurite Outgrowth
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