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1. Generation of nanobodies from transgenic ‘LamaMice’ lacking an endogenous immunoglobulin repertoire

3. The classical pathway triggers pathogenic complement activation in membranous nephropathy

8. Nanobody‐based heavy chain antibodies and chimeric antibodies.

10. Generation and characterization of antagonistic anti-human CD39 nanobodies

11. Time-resolved role of P2X4 and P2X7 during CD8+ T cell activation

12. The TRPM2 ion channel regulates metabolic and thermogenic adaptations in adipose tissue of cold-exposed mice

13. An antigen-specific chimeric autoantibody receptor NK cell strategy for the elimination of anti-PLA2R1 and anti-THSD7A antibody-secreting cells

15. Development of Antibody and Nanobody Tools for P2X7

18. Auto-aggressive CXCR6+ CD8 T cells cause liver immune pathology in NASH

19. Increased surface P2X4 receptor regulates anxiety and memory in P2X4 internalization-defective knock-in mice

20. Different localization of P2X4 and P2X7 receptors in native mouse lung - lack of evidence for a direct P2X4-P2X7 receptor interaction.

25. Generation and characterization of antagonistic anti-human CD39 nanobodies.

26. Time-resolved role of P2X4 and P2X7 during CD8+ T cell activation.

27. ADP-Ribosylation of P2X7: A Matter of Life and Death for Regulatory T Cells and Natural Killer T Cells

32. IRF4 is required for migration of CD4+ T cells to the intestine but not for Th2 and Th17 cell maintenance

35. ZBP1 subcellular localization and association with stress granules is controlled by its Z-DNA binding domains

36. Data from Nanobody Targeting of Epidermal Growth Factor Receptor (EGFR) Ectodomain Variants Overcomes Resistance to Therapeutic EGFR Antibodies

37. Supplementary Figures 1-3 and Table1 from Nanobody Targeting of Epidermal Growth Factor Receptor (EGFR) Ectodomain Variants Overcomes Resistance to Therapeutic EGFR Antibodies

38. Author Correction: Auto-aggressive CXCR6+ CD8 T cells cause liver immune pathology in NASH

39. Macrophages and glia are the dominant P2X7-expressing cell types in the gut nervous system—No evidence for the role of neuronal P2X7 receptors in colitis

40. A transplant “immunome” screening platform defines a targetable epitope fingerprint of multiple myeloma

43. P2X7 receptor induces mitochondrial failure in monocytes and compromises NLRP3 inflammasome activation during sepsis

44. Macrophages and glia are the dominant P2X7-expressing cell types in the gut nervous system – no evidence for a role of neuronal P2X7 receptors in colitis

48. A simple, sensitive, and low‐cost FACS assay for detecting antibodies against the native SARS‐CoV‐2 spike protein

50. P2X7 is expressed on human innate‐like T lymphocytes and mediates susceptibility to ATP‐induced cell death

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