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287 results on '"Koch, W J"'

5. Royal academy of medicine in Ireland section of surgery: Proceedings of registrar’s prize meeting held 22nd January, 1996

Author

Kennedy, J. G., Kasser, J. R., Glimcher, M. J., Salih, E. J., Neary, P., Condon, C., Kilbaugh, T., Redmond, H. P., Bouchier-Hayes, D., Fulton, G. J., Davies, M. G., Svendsen, E., Lefkowitz, R. J., Koch, W. J., Hagen, P -O., Fulton, G. J., Davies, M. G., Svendsen, E., Lefkowitz, R. J., Koch, W. J., Hagen, P -O., Johnston, S. M., Lipsett, P. A., Fox-Talbot, K. M., Lillemoe, K. D., Pitt, H. A., Kelly, J. L., O’Suilleabhain, C. B., Horgan, A. F., O’Riordain, M. G., Mannick, J. A., Rodrick, M. L., Chen, G., Kelly, C., Leahy, A., Bouchier-Hayes, D. J., Kelly, P., O’Riordain, D. S., Horgan, P. G., Keane, F. B., Tanner, W. A., Rothwell, J. F., Staunton, M. J., O’Mahony, L., Gaffney, E. F., Mealy, K., Hennessy, T. P. J., Coveney, E., Clary, B., Philip, R., and Kim Lyerly, H.

Published
1998
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9. Correction to :β adrenergic receptor kinase C-terminal peptide gene-therapy improves ß2-adrenergic receptor-dependent neoangiogenesis after hindlimb ischemia (Journal of Pharmacology and Experimental Therapeutics (2016) 356:2 (503–513) DOI: 10.1124/jpet.115.228411)

Author

Cannavo A., Liccardo D., Lymperopoulos A., Gambino G., D'Amico M. L., Rengo F., Koch W. J., Leosco D., Ferrara N., Rengo G., Cannavo, A., Liccardo, D., Lymperopoulos, A., Gambino, G., D'Amico, M. L., Rengo, F., Koch, W. J., Leosco, D., Ferrara, N., and Rengo, G.

Abstract

In the above article [Cannavo A, Liccardo D, Lymperopoulos A, Gambino G, D’Amico ML, Rengo F, Koch WJ, Leosco D, Ferrara N, and Rengo G (2016) J Pharmacol Exp Ther 356(2): 503–513; DOI: 10.1124/jpet.115.228411], the following funding information was omitted: This work was funded by the National Institutes of Health [Grant R37 HL061690]. The authors regret this error and any inconvenience it may have caused.

Published
2019

14. Connective tissue growth factor inhibition enhances cardiac repair and limits fibrosis after myocardial infarction

Author

Vainio, L. E. (Laura E.), Szabó, Z. (Zoltán), Lin, R. (Ruizhu), Ulvila, J. (Johanna), Yrjölä, R. (Raisa), Alakoski, T. (Tarja), Piuhola, J. (Jarkko), Koch, W. J. (Walter J.), Ruskoaho, H. (Heikki), Fouse, S. D. (Shaun D.), Seeley, T. W. (Todd W.), Gao, E. (Erhe), Signore, P. (Pierre), Lipson, K. E. (Kenneth E.), Magga, J. (Johanna), Kerkelä, R. (Risto), Vainio, L. E. (Laura E.), Szabó, Z. (Zoltán), Lin, R. (Ruizhu), Ulvila, J. (Johanna), Yrjölä, R. (Raisa), Alakoski, T. (Tarja), Piuhola, J. (Jarkko), Koch, W. J. (Walter J.), Ruskoaho, H. (Heikki), Fouse, S. D. (Shaun D.), Seeley, T. W. (Todd W.), Gao, E. (Erhe), Signore, P. (Pierre), Lipson, K. E. (Kenneth E.), Magga, J. (Johanna), and Kerkelä, R. (Risto)

Abstract

Summary Myocardial infarction (MI)−induced cardiac fibrosis attenuates cardiac contractile function, and predisposes to arrhythmias and sudden cardiac death. Expression of connective tissue growth factor (CTGF) is elevated in affected organs in virtually every fibrotic disorder and in the diseased human myocardium. Mice were subjected to treatment with a CTGF monoclonal antibody (mAb) during infarct repair, post-MI left ventricular (LV) remodeling, or acute ischemia−reperfusion injury. CTGF mAb therapy during infarct repair improved survival and reduced LV dysfunction, and reduced post-MI LV hypertrophy and fibrosis. Mechanistically, CTGF mAb therapy induced expression of cardiac developmental and/or repair genes and attenuated expression of inflammatory and/or fibrotic genes.

Published
2019

15. Design, analysis, and fabrication of a pressure box test fixture for tension damage tolerance testing of curved fuselage panels

Author

Smith, P. J, Bodine, J. B, Preuss, C. H, and Koch, W. J

Subjects
Composite Materials
Abstract

A pressure box test fixture was designed and fabricated to evaluate the effects of internal pressure, biaxial tension loads, curvature, and damage on the fracture response of composite fuselage structure. Previous work in composite fuselage tension damage tolerance, performed during NASA contract NAS1-17740, evaluated the above effects on unstiffened panels only. This work extends the tension damage tolerance testing to curved stiffened fuselage crown structure that contains longitudinal stringers and circumferential frame elements. The pressure box fixture was designed to apply internal pressure up to 20 psi, and axial tension loads up to 5000 lb/in, either separately or simultaneously. A NASTRAN finite element model of the pressure box fixture and composite stiffened panel was used to help design the test fixture, and was compared to a finite element model of a full composite stiffened fuselage shell. This was done to ensure that the test panel was loaded in a similar way to a panel in the full fuselage shell, and that the fixture and its attachment plates did not adversely affect the panel.

Published
1993

17. beta-2 adrenergic receptors overexpression promotes angiogenesis in the falling myocardium through activation of vascular endothelial growth factor/Akt transduction pathway

Author

C. Zincarelli, Marchese M., Fomminella D., Avallone E., Koch W. J., RENGO, GIUSEPPE, GOLINO, LUCA, ALTOBELLI, GIOVANNA GIUSEPPINA, CIMINI, VINCENZO, FORTUNATO, FRANCESCA, RENGO, ALESSANDRO, RENGO, FRANCO, LEOSCO, DARIO, C., Zincarelli, Rengo, Giuseppe, Golino, Luca, Altobelli, GIOVANNA GIUSEPPINA, Cimini, Vincenzo, Fortunato, Francesca, Marchese, M., Fomminella, D., Avallone, E., Rengo, Alessandro, Rengo, Franco, Koch, W. J., and Leosco, Dario

Subjects
Akt, angiogenesi, heart failure
Abstract

Possible role of VEGF/Akt transduction pathway after beta-2 adrenergic receptors overexpression-promoted angiogenesis.

Published
2008

18. Exercise stimulates angiogenesis and improves beta-adrenergic receptor signaling in the failing heart

Author

GOLINO, LUCA, LEOSCO, DARIO, IACCARINO, GUIDO, RENGO, GIUSEPPE, CIMINI, VINCENZO, ALTOBELLI, GIOVANNA GIUSEPPINA, MATRONE, GIOVANNA, FORTUNATO, FRANCESCA, RENGO, FRANCO, Sanzari E., Zincarelli C., Sorriento D., Koch W. J., Golino, Luca, Leosco, Dario, Iaccarino, Guido, Rengo, Giuseppe, Cimini, Vincenzo, Altobelli, GIOVANNA GIUSEPPINA, Sanzari, E., Matrone, Giovanna, Fortunato, Francesca, Zincarelli, C., Sorriento, D., Koch, W. J., and Rengo, Franco

Subjects
exercise, angiogenesi, heart failure, beta-adrenergic receptor
Abstract

Exercise upregulates beta-adrenergic receptor expression and this, in turn, stimulates angiogenesis.

Published
2006

19. beta-1 adrenergic receptor blokade and cardiac beta-2 overexpression stimulates angiogenesis in the failing heart

Author

RENGO, GIUSEPPE, LEOSCO, DARIO, IACCARINO, GUIDO, GOLINO, LUCA, CIMINI, VINCENZO, ALTOBELLI, GIOVANNA GIUSEPPINA, Sanzari E., Matrone G., Zincarelli C., Fortunato F., Koch W. J., RENGO, FRANCO, Rengo, Giuseppe, Leosco, Dario, Iaccarino, Guido, Golino, Luca, Cimini, Vincenzo, Altobelli, GIOVANNA GIUSEPPINA, Sanzari, E., Matrone, G., Zincarelli, C., Fortunato, F., Koch, W. J., and Rengo, Franco

Subjects
angiogenesis, adrenergi receptor, heart failure
Abstract

Angiogenesis in the failing heart is stimulated beta-1 AR blockade and cardiac beta-2 AR overexpression.

Published
2006

22. Impaired neoangiogenesis in β₂-adrenoceptor gene-deficient mice: restoration by intravascular human β₂-adrenoceptor gene transfer and role of NFκB and CREB transcription factors

Author

Ciccarelli, Michele, Sorriento, D., Cipolletta, E., Santulli, G., Fusco, A., Zhou, R., Eckhart, A. D., Peppel, K., Koch, W. J., Trimarco, B., and Iaccarino, Guido

Subjects
Male, Vascular Endothelial Growth Factor A, Cells, Knockout, Genetic Vectors, Neovascularization, Physiologic, beta-2, Transfection, Mice, Radioligand Assay, Ischemia, Receptors, deficiency/genetics/metabolism, Animals, Humans, Physiologic, Luciferases, Neovascularization, Cells, Cultured, Mice, Knockout, Cultured, Animal, NF-kappa B, Endothelial Cells, Gene Therapy, Genetic Therapy, CREB-Binding Protein, Research Papers, Disease Models, Animal, Adrenergic, Animals, Blood Flow Velocity, CREB-Binding Protein, metabolism, Cells, Cultured, Disease Models, Animal, Endothelial Cells, metabolism, Gene Therapy, Genetic Vectors, Humans, Ischemia, physiopathology/therapy, Luciferases, metabolism, Male, Mice, Mice, Knockout, NF-kappa B, metabolism, Neovascularization, Physiologic, Radioligand Assay, Receptors, deficiency/genetics/metabolism, Signal Transduction, Transfection, Vascular Endothelial Growth Factor A, metabolism, Disease Models, physiopathology/therapy, Receptors, Adrenergic, beta-2, Blood Flow Velocity, Signal Transduction
Abstract

There is much evidence supporting the role of β₂-adrenoceptors (β₂AR) in angiogenesis but the mechanisms underlying their effects have not been elucidated. Hence, we studied post-ischaemic angiogenesis in the hindlimb (HL) of β₂AR knock-out mice (β₂AR-/-) in vivo and explored possible molecular mechanisms in vitro.Femoral artery resection (FAR) was performed in wild-type and β₂AR-/- mice and adaptive responses to chronic HL ischaemia were explored; blood flow was measured by ultrasound and perfusion of dyed beads, bone rarefaction, muscle fibrosis and skin thickness were evaluated by immunoflourescence and morphometric analysis. Intrafemoral delivery of an adenovirus encoding the human β₂AR (ADβ₂AR) was used to reinstate β₂ARs in β₂AR-/- mice. Molecular mechanisms were investigated in mouse-derived aortic endothelial cells (EC) in vitro, focusing on NFκB activation and transcriptional activity.Angiogenesis was severely impaired in β₂AR-/- mice subjected to FAR, but was restored by gene therapy with ADβ₂AR. The proangiogenic responses to a variety of stimuli were impaired in β₂AR-/- EC in vitro. Moreover, removal of β₂ARs impaired the activation of NFκB, a transcription factor that promotes angiogenesis; neither isoprenaline (stimulates βARs) nor TNFα induced NFκB activation in β₂AR(-/-) EC. Interestingly, cAMP response element binding protein (CREB), a transcription factor that counter regulates NFκB, was constitutively increased in β₂AR(-/-) ECs. ADβ₂AR administration restored β₂AR membrane density, reduced CREB activity and reinstated the NFκB response to isoprenaline and TNFα.Our results suggest that β₂ARs control angiogenesis through the tight regulation of nuclear transcriptional activity.

Published
2010

27. Regulation of myocardial betaARK1 expression in catecholamine-induced cardiac hypertrophy in transgenic mice overexpressing alpha1B-adrenergic receptors

Author

Iaccarino, Guido, Keys, J. R., Rapacciuolo, A., Shotwell, K. F., Lefkowitz, R. J., Rockman, H. A., and Koch, W. J.

Subjects
chemically induced/complications/enzymology, Cardiomyopathy, etiology, Messenger, genetics/metabolism, Muscle Proteins, Cardiomegaly, biosynthesis/genetics, Transgenic, Mice, Phenylephrine, Adrenergic alpha-Agonists, Animals, Body Weight, Dilated, Cyclic AMP-Dependent Protein Kinases, metabolism, Myocardium, enzymology/pathology, Neuropeptide Y, Organ Size, RNA, biosynthesis, Receptors, Adrenergic, alpha-1, beta, Signal Transduction, beta-Adrenergic Receptor Kinases
Published
2001

28. Expression of a -adrenergic receptor kinase 1 inhibitor prevents the development of myocardial failure in gene-targeted mice

Author

Hunter, J. J., Lefkowitz, R. J., Choi, D.-J., Chien, K. R., Rockman, H. A., Ross, J., Iaccarino, G., and Koch, W. J.

Abstract

Heart failure is accompanied by severely impaired β-adrenergic receptor (βAR) function, which includes loss of βAR density and functional uncoupling of remaining receptors. An important mechanism for the rapid desensitization of βAR function is agonist-stimulated receptor phosphorylation by the βAR kinase (βARK1), an enzyme known to be elevated in failing human heart tissue. To investigate whether alterations in βAR function contribute to the development of myocardial failure, transgenic mice with cardiac-restricted overexpression of either a peptide inhibitor of βARK1 or the β2AR were mated into a genetic model of murine heart failure (MLP−/−). In vivo cardiac function was assessed by echocardiography and cardiac catheterization. Both MLP−/− and MLP−/−/β2AR mice had enlarged left ventricular (LV) chambers with significantly reduced fractional shortening and mean velocity of circumferential fiber shortening. In contrast, MLP−/−/βARKct mice had normal LV chamber size and function. Basal LV contractility in the MLP−/−/βARKct mice, as measured by LV dP/dtmax, was increased significantly compared with the MLP−/− mice but less than controls. Importantly, heightened βAR desensitization in the MLP−/− mice, measured in vivo (responsiveness to isoproterenol) and in vitro (isoproterenol-stimulated membrane adenylyl cyclase activity), was completely reversed with overexpression of the βARK1 inhibitor. We report here the striking finding that overexpression of this inhibitor prevents the development of cardiomyopathy in this murine model of heart failure. These findings implicate abnormal βAR-G protein coupling in the pathogenesis of the failing heart and point the way toward development of agents to inhibit βARK1 as a novel mode of therapy.

Published
1998
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29. Therapeutic safety of high myocardial expression levels of the molecular inotrope S100A1 in a preclinical heart failure model

Author

Weber, C, primary, Neacsu, I, additional, Krautz, B, additional, Schlegel, P, additional, Sauer, S, additional, Raake, P, additional, Ritterhoff, J, additional, Jungmann, A, additional, Remppis, A B, additional, Stangassinger, M, additional, Koch, W J, additional, Katus, H A, additional, Müller, O J, additional, Most, P, additional, and Pleger, S T, additional

Published
2013
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31. Poster session 3

Author

Nanka, O., primary, Krejci, E., additional, Pesevski, Z., additional, Sedmera, D., additional, Smart, N., additional, Rossdeutsch, A., additional, Dube, K. N., additional, Riegler, J., additional, Price, A. N., additional, Taylor, A., additional, Muthurangu, V., additional, Turner, M., additional, Lythgoe, M. F., additional, Riley, P. R., additional, Kryvorot, S., additional, Vladimirskaya, T., additional, Shved, I., additional, Schwarzl, M., additional, Seiler, S., additional, Huber, S., additional, Steendijk, P., additional, Maechler, H., additional, Truschnig-Wilders, M., additional, Pieske, B., additional, Post, H., additional, Caprio, C., additional, Baldini, A., additional, Chiavacci, E., additional, Dolfi, L., additional, Verduci, L., additional, Meghini, F., additional, Cremisi, F., additional, Pitto, L., additional, Kuan, T.-C., additional, Chen, M.-C., additional, Yang, T.-H., additional, Wu, W.-T., additional, Lin, C. S., additional, Rai, H., additional, Kumar, S., additional, Sharma, A. K., additional, Mastana, S., additional, Kapoor, A., additional, Pandey, C. M., additional, Agrawal, S., additional, Sinha, N., additional, Orlowska-Baranowska, E. H., additional, Placha, G., additional, Gora, J., additional, Baranowski, R., additional, Abramczuk, E., additional, Hryniewiecki, T., additional, Gaciong, Z., additional, Verschuren, J. J. W., additional, Wessels, J. A. M., additional, Trompet, S., additional, Stott, D. J., additional, Sattar, N., additional, Buckley, B., additional, Guchelaar, H. J., additional, Jukema, J. W., additional, Gharanei, M., additional, Hussain, A., additional, Mee, C. J., additional, Maddock, H. L., additional, Wijnen, W. J., additional, Van Den Oever, S., additional, Van Der Made, I., additional, Hiller, M., additional, Tijsen, A. J., additional, Pinto, Y. M., additional, Creemers, E. E., additional, Nikulina, S. U. Y., additional, Chernova, A., additional, Petry, A., additional, Rzymski, T., additional, Kracun, D., additional, Riess, F., additional, Pike, L., additional, Harris, A. L., additional, Gorlach, A., additional, Katare, R., additional, Oikawa, A., additional, Riu, F., additional, Beltrami, A. P., additional, Cesseli, D., additional, Emanueli, C., additional, Madeddu, P., additional, Zaglia, T., additional, Milan, G., additional, Franzoso, M., additional, Pesce, P., additional, Sarais, C., additional, Sandri, M., additional, Mongillo, M., additional, Butler, T. J., additional, Seymour, A.-M. L., additional, Ashford, D., additional, Jaffre, F., additional, Bussen, M., additional, Flohrschutz, I., additional, Martin, G. R., additional, Engelhardt, S., additional, Kararigas, G., additional, Nguyen, B. T., additional, Jarry, H., additional, Regitz-Zagrosek, V., additional, Van Bilsen, M., additional, Daniels, A., additional, Munts, C., additional, Janssen, B. J. A., additional, Van Der Vusse, G. J., additional, Van Nieuwenhoven, F. A., additional, Montalvo, C., additional, Villar, A. V., additional, Merino, D., additional, Garcia, R., additional, Llano, M., additional, Ares, M., additional, Hurle, M. A., additional, Nistal, J. F., additional, Dembinska-Kiec, A., additional, Beata Kiec-Wilk, B. K. W., additional, Anna Polus, A. P., additional, Urszula Czech, U. C., additional, Tatiana Konovaleva, T. K., additional, Gerd Schmitz, G. S., additional, Bertrand, L., additional, Balteau, M., additional, Timmermans, A., additional, Viollet, B., additional, Sakamoto, K., additional, Feron, O., additional, Horman, S., additional, Vanoverschelde, J. L., additional, Beauloye, C., additional, De Meester, C., additional, Martinez, E., additional, Martin, R., additional, Miana, M., additional, Jurado, R., additional, Gomez-Hurtado, N., additional, Bartolome, M. V., additional, San Roman, J. A., additional, Lahera, V., additional, Nieto, M. L., additional, Cachofeiro, V., additional, Rochais, F., additional, Sturny, R., additional, Mesbah, K., additional, Miquerol, L., additional, Kelly, R. G., additional, Messaoudi, S., additional, Gravez, B., additional, Tarjus, A., additional, Pelloux, V., additional, Samuel, J. L., additional, Delcayre, C., additional, Launay, J. M., additional, Clement, K., additional, Farman, N., additional, Jaisser, F., additional, Hadyanto, L., additional, Castellani, C., additional, Vescovo, G., additional, Ravara, B., additional, Tavano, R., additional, Pozzobon, M., additional, De Coppi, P., additional, Papini, E., additional, Vettor, R., additional, Thiene, G., additional, Angelini, A., additional, Meloni, M., additional, Caporali, A., additional, Cesselli, D., additional, Fortunato, O., additional, Avolio, E., additional, Schindler, R., additional, Simrick, S., additional, Brand, T., additional, Smart, N. S., additional, Herman, A., additional, Roura Ferrer, S., additional, Rodriguez Bago, J., additional, Soler-Botija, C., additional, Pujal, J. M., additional, Galvez-Monton, C., additional, Prat-Vidal, C., additional, Llucia-Valldeperas, A., additional, Blanco, J., additional, Bayes-Genis, A., additional, Foldes, G., additional, Maxime, M., additional, Ali, N. N., additional, Schneider, M. D., additional, Harding, S. E., additional, Reni, C., additional, Mangialardi, G., additional, De Pauw, A., additional, Sekkali, B., additional, Friart, A., additional, Ding, H., additional, Graffeuil, A., additional, Catalucci, D., additional, Balligand, J. L., additional, Azibani, F., additional, Tournoux, F., additional, Schlossarek, S., additional, Polidano, E., additional, Fazal, L., additional, Merval, R., additional, Carrier, L., additional, Chatziantoniou, C., additional, Buyandelger, B., additional, Linke, W., additional, Zou, P., additional, Kostin, S., additional, Ku, C., additional, Felkin, L., additional, Birks, E., additional, Barton, P., additional, Sattler, M., additional, Knoell, R., additional, Schroder, K., additional, Benkhoff, S., additional, Shimokawa, H., additional, Grisk, O., additional, Brandes, R. P., additional, Parepa, I. R., additional, Mazilu, L., additional, Suceveanu, A. I., additional, Suceveanu, A., additional, Rusali, L., additional, Cojocaru, L., additional, Matei, L., additional, Toringhibel, M., additional, Craiu, E., additional, Pires, A. L., additional, Pinho, M., additional, Pinho, S., additional, Sena, C., additional, Seica, R., additional, Leite-Moreira, A., additional, Dabroi, F., additional, Schiaffino, S., additional, Kiseleva, E., additional, Krukov, N., additional, Nikitin, O., additional, Ardatova, L., additional, Mourouzis, I., additional, Pantos, C., additional, Kokkinos, A. D., additional, Cokkinos, D. V., additional, Scoditti, E., additional, Massaro, M., additional, Carluccio, M. A., additional, Pellegrino, M., additional, Calabriso, N., additional, Gastaldelli, A., additional, Storelli, C., additional, De Caterina, R., additional, Lindner, D., additional, Zietsch, C., additional, Schultheiss, H.-P., additional, Tschope, C., additional, Westermann, D., additional, Everaert, B. R., additional, Nijenhuis, V. J., additional, Reith, F. C. M., additional, Hoymans, V. Y., additional, Timmermans, J. P., additional, Vrints, C. J., additional, Simova, I., additional, Mateev, H., additional, Katova, T., additional, Haralanov, L., additional, Dimitrov, N., additional, Mironov, N., additional, Golitsyn, S. P., additional, Sokolov, S. F., additional, Yuricheva, Y. U. A., additional, Maikov, E. B., additional, Shlevkov, N. B., additional, Rosenstraukh, L. V., additional, Chazov, E. I., additional, Radosinska, J., additional, Knezl, V., additional, Benova, T., additional, Slezak, J., additional, Urban, L., additional, Tribulova, N., additional, Virag, L., additional, Kristof, A., additional, Kohajda, Z. S., additional, Szel, T., additional, Husti, Z., additional, Baczko, I., additional, Jost, N., additional, Varro, A., additional, Sarusi, A., additional, Farkas, A. S., additional, Orosz, S. Z., additional, Forster, T., additional, Farkas, A., additional, Zakhrabova-Zwiauer, O. M., additional, Hardziyenka, M., additional, Nieuwland, R., additional, Tan, H. L., additional, Raaijmakers, A. J. A., additional, Bourgonje, V. J. A., additional, Kok, G. J. M., additional, Van Veen, A. A. B., additional, Anderson, M. E., additional, Vos, M. A., additional, Bierhuizen, M. F. A., additional, Benes, J., additional, Sebestova, B., additional, Ghouri, I. A., additional, Kemi, O. J., additional, Kelly, A., additional, Burton, F. L., additional, Smith, G. L., additional, Ozdemir, S., additional, Acsai, K., additional, Doisne, N., additional, Van Der Nagel, R., additional, Beekman, H. D. M., additional, Van Veen, T. A. B., additional, Sipido, K. R., additional, Antoons, G., additional, Harmer, S. C., additional, Mohal, J. S., additional, Kemp, D., additional, Tinker, A., additional, Beech, D., additional, Burley, D. S., additional, Cox, C. D., additional, Wann, K. T., additional, Baxter, G. F., additional, Wilders, R., additional, Verkerk, A., additional, Fragkiadaki, P., additional, Germanakis, G., additional, Tsarouchas, K., additional, Tsitsimpikou, C., additional, Tsardi, M., additional, George, D., additional, Tsatsakis, A., additional, Rodrigues, P., additional, Barros, C., additional, Najmi, A. K., additional, Khan, V., additional, Akhtar, M., additional, Pillai, K. K., additional, Mujeeb, M., additional, Aqil, M., additional, Bayliss, C. R., additional, Messer, A. E., additional, Leung, M.-C., additional, Ward, D., additional, Van Der Velden, J., additional, Poggesi, C., additional, Redwood, C. S., additional, Marston, S., additional, Vite, A., additional, Gandjbakhch, E., additional, Gary, F., additional, Fressart, V., additional, Leprince, P., additional, Fontaine, G., additional, Komajda, M., additional, Charron, P., additional, Villard, E., additional, Falcao-Pires, I., additional, Gavina, C., additional, Hamdani, N., additional, Stienen, G. J. M., additional, Niessens, H. W. M., additional, Leite-Moreira, A. F., additional, Paulus, W. J., additional, Memo, M., additional, Marston, S. B., additional, Vafiadaki, E., additional, Qian, J., additional, Arvanitis, D. A., additional, Sanoudou, D., additional, Kranias, E. G., additional, Elmstedt, N., additional, Lind, B., additional, Ferm-Widlund, K., additional, Westgren, M., additional, Brodin, L.-A., additional, Mansfield, C., additional, West, T., additional, Ferenczi, M., additional, Wijnker, P. J. M., additional, Foster, D. B., additional, Coulter, A., additional, Frazier, A., additional, Murphy, A. M., additional, Shah, M., additional, Sikkel, M. B., additional, Desplantez, T., additional, Collins, T. P., additional, O' Gara, P., additional, Lyon, A. R., additional, Macleod, K. T., additional, Ottesen, A. H., additional, Louch, W. E., additional, Carlson, C., additional, Landsverk, O. J. B., additional, Stridsberg, M., additional, Sjaastad, I., additional, Oie, E., additional, Omland, T., additional, Christensen, G., additional, Rosjo, H., additional, Cartledge, J., additional, Clark, L. A., additional, Ibrahim, M., additional, Siedlecka, U., additional, Navaratnarajah, M., additional, Yacoub, M. H., additional, Camelliti, P., additional, Terracciano, C. M., additional, Chester, A., additional, Gonzalez-Tendero, A., additional, Torre, I., additional, Garcia-Garcia, F., additional, Dopazo, J., additional, Gratacos, E., additional, Taylor, D., additional, Bhandari, S., additional, Seymour, A.-M., additional, Fliegner, D., additional, Jost, J., additional, Bugger, H., additional, Ventura-Clapier, R., additional, Carpi, A., additional, Campesan, M., additional, Canton, M., additional, Menabo, R., additional, Pelicci, P. G., additional, Giorgio, M., additional, Di Lisa, F., additional, Hancock, M., additional, Venturini, A., additional, Al-Shanti, N., additional, Stewart, C., additional, Ascione, R., additional, Angelini, G., additional, Suleiman, M.-S., additional, Kravchuk, E., additional, Grineva, E., additional, Galagudza, M., additional, Kostareva, A., additional, Bairamov, A., additional, Krychtiuk, K. A., additional, Watzke, L., additional, Kaun, C., additional, Demyanets, S., additional, Pisoni, J., additional, Kastl, S. P., additional, Huber, K., additional, Maurer, G., additional, Wojta, J., additional, Speidl, W. S., additional, Varga, Z. V., additional, Farago, N., additional, Zvara, A., additional, Kocsis, G. F., additional, Pipicz, M., additional, Csonka, C., additional, Csont, T., additional, Puskas, G. 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Published
2012
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32. AAV6. ARKct cardiac gene therapy ameliorates cardiac function and normalizes the catecholaminergic axis in a clinically relevant large animal heart failure model

Author

Raake, P. W. J., primary, Schlegel, P., additional, Ksienzyk, J., additional, Reinkober, J., additional, Barthelmes, J., additional, Schinkel, S., additional, Pleger, S., additional, Mier, W., additional, Haberkorn, U., additional, Koch, W. J., additional, Katus, H. A., additional, Most, P., additional, and Muller, O. J., additional

Published
2012
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34. Prior Exercise Improves Age-Dependent Vascular Endothelial Growth Factor Downregulation and Angiogenesis Responses to Hind-Limb Ischemia in Old Rats

Author

Leosco, D., primary, Rengo, G., additional, Iaccarino, G., additional, Sanzari, E., additional, Golino, L., additional, Lisa, G. D., additional, Zincarelli, C., additional, Fortunato, F., additional, Ciccarelli, M., additional, Cimini, V., additional, Altobelli, G. G., additional, Piscione, F., additional, Galasso, G., additional, Trimarco, B., additional, Koch, W. J., additional, and Rengo, F., additional

Published
2007
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