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1. Electron transport chain inhibition increases cellular dependence on purine transport and salvage

2. Comprehensive isotopomer analysis of glutamate and aspartate in small tissue samples

3. An interactive web application for exploring human plasma and fibroblast metabolomics data from patients with inborn errors of metabolism

4. The hexosamine biosynthesis pathway is a targetable liability in KRAS/LKB1 mutant lung cancer

5. Acetylation of the c-MYC oncoprotein is required for cooperation with the HTLV-1 p30(II) accessory protein and the induction of oncogenic cellular transformation by p30(II)/c-MYC.

6. Electron transport inhibition increases cellular dependence on purine transport and salvage

10. In vivo isotope tracing reveals a requirement for the electron transport chain in glucose and glutamine metabolism by tumors

11. Metabolic impact of pathogenic variants in the mitochondrial glutamyl‐tRNA synthetase EARS2

12. The hexosamine biosynthesis pathway is a targetable liability in lung cancers with concurrent KRAS and LKB1 mutations

13. Metabolic Diversity in Human Non-Small Cell Lung Cancer Cells

14. Functional Assessment of Lipoyltransferase-1 Deficiency in Cells, Mice, and Humans

16. Metabolic Heterogeneity in Human Lung Tumors

17. Metabolic changes upon GLS inhibition by CB-839 in glioma cell lines

18. Metabolic plasticity maintains proliferation in pyruvate dehydrogenase deficient cells

19. Acetylation of the c-MYC oncoprotein is required for cooperation with the HTLV-1 p30 II accessory protein and the induction of oncogenic cellular transformation by p30 II /c-MYC

20. Glutamine Oxidation Maintains the TCA Cycle and Cell Survival during Impaired Mitochondrial Pyruvate Transport

21. Acetylation of the c-MYC oncoprotein is required for cooperation with the HTLV-1 p30II accessory protein and the induction of oncogenic cellular transformation by p30II/c-MYC.

22. An interactive web application for exploring human plasma and fibroblast metabolomics data from patients with inborn errors of metabolism.

23. Electron transport chain inhibition increases cellular dependence on purine transport and salvage.

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