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2. Three‐dimensional phenotyping of peach tree‐crown architecture utilizing terrestrial laser scanning

Author

Jordan Knapp‐Wilson, Rafael Bohn Reckziegel, Srijana Thapa Magar, Alexander Bucksch, and Dario J. Chavez

Subjects
Plant culture, SB1-1110
Abstract

Abstract Tree training systems for temperate fruit have been developed throughout history by pomologists to improve light interception, fruit yield, and fruit quality. These training systems direct crown and branch growth to specific configurations. Quantifying crown architecture could aid the selection of trees that require less pruning or that naturally excel in specific growing/training system conditions. Regarding peaches [Prunus persica (L.) Batsch], access tools such as branching indices have been developed to characterize tree‐crown architecture. However, the required branching data (BD) to develop these indices are difficult to collect. Traditionally, BD have been collected manually, but this process is tedious, time‐consuming, and prone to human error. These barriers can be circumnavigated by utilizing terrestrial laser scanning (TLS) to obtain a digital twin of the real tree. TLS generates three‐dimensional (3D) point clouds of the tree crown, wherein every point contains 3D coordinates (x, y, z). To facilitate the use of these tools for peach, we selected 16 young peach trees scanned in 2021 and 2022. These 16 trees were then modeled and quantified using the open‐source software TreeQSM. As a result, “in silico” branching and biometric data for the young peach trees were calculated to demonstrate the capabilities of TLS phenotyping of peach tree‐crown architecture. The comparison and analysis of field measurements (in situ) and in silico BD, biometric data, and quantitative structural model branch uncertainty data were utilized to determine the reconstructive model's reliability as a source substitute for field measurements. Mean average deviation when comparing young tree (YT) height was approx. 5.93%, with crown volume was approx. 13.26% across both 2021 and 2022. All point clouds of the YTs in 2022 showed residuals lower than 12 mm to cylinders fitted to all branches, and mean surface coverage greater than 40% for both the trunk and primary branching orders.

Published
2023
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3. The impact of respiratory complex dysfunction in Caenorhabditis elegans

Author

Knapp-Wilson, Amber L. and Collinson, Ian

Abstract

Mitochondria are essential organelles of eukaryotic cells, responsible for the generation of the majority of the energy carrier molecule ATP. This essential process of energy production is achieved by oxidative phosphorylation (OXPHOS), facilitated by a set of proteins collectively named the electron transport chain (ETC). Loss of mitochondrial function has devastating effects on the cell and is usually associated with a disease state. The model organism Caenorhabditis elegans (C. elegans) is genetically tractable, has large progeny broods and provides information on a whole animal level. In recent years, it has become an established model organism for identifying connections between mitochondrial dysfunction and lifespan. Using bioinformatics combined with protein modelling, the C. elegans B0491.5 protein has been identified as the homologue of NDUFA11, a key mammalian component of the electron transport chain complex I accessory subunit. NDUFA11 is linked to complex assembly and stabilisation; loss of NDUFA11 results in a reduction of complex I and metabolic perturbation. The research presented in my thesis investigates the impact of reducing B0491.5 activity on whole animal physiology and mitochondrial biochemistry. I have shown through a genetic knockout that b0419.5 is an essential gene, and using RNAi to suppress b0491.5 expression, that in its absence animal growth is delayed and reproductive capabilities severely reduced. Depleting B0491.5 also has a significant impact on mitochondrial physiology, disrupting respiration activity and seemingly limiting animals to achieve only minimum energy demands. Mitochondrial biochemistry establishes B0491.5 enrichment in mitochondria, in addition to validating the protein's association with complex I. Furthermore, evidence is presented suggesting B0491.5 participates in the formation of complex I containing supercomplexes. When B0491.5 is reduced, there is disruption to supercomplex formation and decreased levels of complex I, proteome wide changes to cytosolic and mitochondrial proteins, as well as morphological and physiology mitochondrial perturbation. These changes are thought to be due to atypical cellular respiration, linked to a disruption of complex organisation within the ETC, due to the role NDUFA11 has to play within complex I and the respirasome.

Published
2019

8. Three-dimensional phenotyping of peach tree crown architecture utilizing terrestrial laser scanning

Author

Jordan Knapp-Wilson, Rafael Bohn Reckziegel, Srijana Thapa Magar, Alexander Bucksch, and Dario Chavez

Abstract

Tree training systems for temperate fruit have been developed throughout history by pomologists to improve light interception, fruit yield, and fruit quality. These training systems direct crown and branch growth to specific configurations. Quantifying crown architecture could aid the selection of trees that require less pruning or that naturally excel in specific growing/training system conditions. Regarding peaches [Prunus persica (L.) Batsch], access tools such as branching indices (BIs) have been developed to characterize tree crown architecture. However, the required branching data to develop these indices are difficult to collect. Traditionally, branching data have been collected manually, but this process is tedious, time-consuming, and prone to human error. These barriers can be circumnavigated by utilizing terrestrial LiDAR (TLS) to obtain a digital twin of the real tree. TLS generates three-dimensional (3D) point clouds of the tree crown, wherein every point contains 3D coordinates (x, y, z). To facilitate the use of these tools for peach, we selected four young peach trees scanned in 2021 and 2022. These four young trees were then modeled and quantified using the open-source software TreeQSM. As a result, “in silico” branching and biometric data for the young peach trees were calculated to demonstrate the capabilities of TLS phenotyping of peach tree-crown architecture. The comparison and analysis of field measurements (in situ) and in silico branching data (BD), biometric data, and residual ground truth data were utilized to determine the reconstructive model’s reliability as a source substitute for field measurements. Mean average deviation (MAD) when comparing young tree height was approx. 8.2%, with crown volume (crV) was approx. 7.6% across both 2021 and 2022. All point clouds of the young trees in 2022 showed residuals < 10mm to cylinders fitted to all branches, and mean surface coverage >50% across all branching orders.

Published
2023
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12. The impact of the COVID-19 pandemic on UK medical education. A nationwide student survey

Author

Tekkis, Nicholas Pari, Rafi, Damir, Brown, Sam, Courtney, Alona, Kawka, Michal, Howell, Ann-Marie, McLean, Kenneth, Gardiner, Matthew, Mavroveli, Stella, Hutchinson, Peter, Tekkis, Paris, Wilkinson, Paul, Sam, Amir H, Savva, N, Kontovounisios, Christos, Tekkis, N, Rafi, D, Brown, S, Courtney, A, Kawka, M, Howell, A, McLean, K, Gardiner, M, Mavroveli, S, Hutchinson, P, Tekkis, P, Wilkinson, P, Sam, A H, Kontovounisios, C, Mclean, K, Singal, A, Chia, C, Chia, W, Ganesananthan, S, Ooi, S Z Y, Pengelly, S, Wellington, J, Mak, S, Subbiah Ponniah, H, Heyes, A, Aberman, I, Ahmed, T, Al-Shamaa, S, Appleton, L, Arshad, A, Awan, H, Baig, Q, Benedict, K, Berkes, S, Citeroni, N L, Damani, A, de Sancha, A, Fisayo, T, Gupta, S, Haq, M, Heer, B, Jones, A, Khan, H, Kim, H, Meiyalagan, N, Miller, G, Minta, N, Mirza, L, Mohamed, F, Ramjan, F, Read, P, Soni, L, Tailor, V, Tas, R N, Vorona, M, Walker, M, Winkler, T, Bardon, A, Acquaah, J, Ball, T, Bani, W, Elmasry, A, Hussein, F, Kolluri, M, Lusta, H, Newman, J, Nott, M, Perwaiz, M I, Rayner, R, Shah, A, Shaw, I, Yu, K, Cairns, M, Clough, R, Gaier, S, Hirani, D, Jeyapalan, T, Li, Y, Patel, C R, Shabir, H, Wang, Y A, Weatherhead, A, Dhiran, A, Renney, O, Wells, P, Ferguson, S, Joyce, A, Mergo, A, Adebayo, O, Ahmad, J, Akande, O, Ang, G, Aniereobi, E, Awasthi, S, Banjoko, A, Bates, J, Chibada, C, Clarke, N, Craner, I, Desai, D D, Dixon, K, Duffaydar, H I, Kuti, M, Mughal, A Z, Nair, D, Pham, M C, Preest, G G, Reid, R, Sachdeva, G S, Selvaratnam, K, Sheikh, J, Soran, V, Stoney, N, Wheatle, M, Howarth, K, Knapp-Wilson, A, Lee, K S, Mampitiya, N, Masson, C, McAlinden, J J, McGowan, N, Parmar, S C, Robinson, B, Wahid, S, Willis, L, Risquet, R, Adebayo, A, Dhingra, L, Kathiravelupillai, S, Narayanan, R, Soni, J, Ghafourian, P, Hounat, A, Lennon, K A, Abdi Mohamud, M, Chou, W, Chong, L, Graham, C J, Piya, S, Riad, A M, Vennard, S, Wang, J, Kawar, L, Maseland, C, Myatt, R, Tengku Saifudin, T N S, Yong, S Q, Douglas, F, Ogbechie, C, Sharma, K, Zafar, L, Bajomo, M O, Byrne, M H V, Obi, C, Oluyomi, D I, Patsalides, M A, Rajananthanan, A, Richardson, G, Clarke, A, Roxas, A, Adeboye, W, Argus, L, McSweeney, J, Rahman-Chowdhury, M, Hettiarachchi, D S, Masood, M T, Antypas, A, Thomas, M, de Andres Crespo, M, Zimmerman, M, Dhillon, A, Abraha, S, Burton, O, Jalal, A H B, Bailey, B, Casey, A, Kathiravelupillai, A, Missir, E, Boult, H, Campen, D, Collins, J M, Dulai, S, Elhassan, M, Foster, Z, Horton, E, Jones, E, Mahapatra, S, Nancarrow, T, Nyamapfene, T, Rimmer, A, Robberstad, M, Robson-Brown, S, Saeed, A, Sarwar, Y, Taylor, C, Vetere, G, Whelan, M K, Williams, J, Zahid, D, Chand, C, and Matthews, M

Subjects
Crises, Courses (Education), Medical sciences, United Kingdom
Published
2022

14. The impact of the COVID-19 pandemic on UK medical education. A nationwide student survey

Author

Nicholas Pari, Tekkis, Damir, Rafi, Sam, Brown, Alona, Courtney, Michal, Kawka, Ann-Marie, Howell, Kenneth, McLean, Matthew, Gardiner, Stella, Mavroveli, Peter, Hutchinson, Paris, Tekkis, Paul, Wilkinson, Amir H, Sam, Nicos, Savva, Christos, Kontovounisios, N, Tekkis, D, Rafi, S, Brown, A, Courtney, M, Kawka, A, Howell, K, McLean, M, Gardiner, S, Mavroveli, P, Hutchinson, P, Tekkis, P, Wilkinson, A H, Sam, N, Savva, C, Kontovounisios, K, Mclean, A, Singal, C, Chia, W, Chia, S, Ganesananthan, S Z Y, Ooi, S, Pengelly, J, Wellington, S, Mak, H, Subbiah Ponniah, A, Heyes, I, Aberman, T, Ahmed, S, Al-Shamaa, L, Appleton, A, Arshad, H, Awan, Q, Baig, K, Benedict, S, Berkes, N L, Citeroni, A, Damani, A, de Sancha, T, Fisayo, S, Gupta, M, Haq, B, Heer, A, Jones, H, Khan, H, Kim, N, Meiyalagan, G, Miller, N, Minta, L, Mirza, F, Mohamed, F, Ramjan, P, Read, L, Soni, V, Tailor, R N, Tas, M, Vorona, M, Walker, T, Winkler, A, Bardon, J, Acquaah, T, Ball, W, Bani, A, Elmasry, F, Hussein, M, Kolluri, H, Lusta, J, Newman, M, Nott, M I, Perwaiz, R, Rayner, A, Shah, I, Shaw, K, Yu, M, Cairns, R, Clough, S, Gaier, D, Hirani, T, Jeyapalan, Y, Li, C R, Patel, H, Shabir, Y A, Wang, A, Weatherhead, A, Dhiran, O, Renney, P, Wells, S, Ferguson, A, Joyce, A, Mergo, O, Adebayo, J, Ahmad, O, Akande, G, Ang, E, Aniereobi, S, Awasthi, A, Banjoko, J, Bates, C, Chibada, N, Clarke, I, Craner, D D, Desai, K, Dixon, H I, Duffaydar, M, Kuti, A Z, Mughal, D, Nair, M C, Pham, G G, Preest, R, Reid, G S, Sachdeva, K, Selvaratnam, J, Sheikh, V, Soran, N, Stoney, M, Wheatle, K, Howarth, A, Knapp-Wilson, K S, Lee, N, Mampitiya, C, Masson, J J, McAlinden, N, McGowan, S C, Parmar, B, Robinson, S, Wahid, L, Willis, R, Risquet, A, Adebayo, L, Dhingra, S, Kathiravelupillai, R, Narayanan, J, Soni, P, Ghafourian, A, Hounat, K A, Lennon, M, Abdi Mohamud, W, Chou, L, Chong, C J, Graham, S, Piya, A M, Riad, S, Vennard, J, Wang, L, Kawar, C, Maseland, R, Myatt, T N S, Tengku Saifudin, S Q, Yong, F, Douglas, C, Ogbechie, K, Sharma, L, Zafar, M O, Bajomo, M H V, Byrne, C, Obi, D I, Oluyomi, M A, Patsalides, A, Rajananthanan, G, Richardson, A, Clarke, A, Roxas, W, Adeboye, L, Argus, J, McSweeney, M, Rahman-Chowdhury, D S, Hettiarachchi, M T, Masood, A, Antypas, M, Thomas, M, de Andres Crespo, M, Zimmerman, A, Dhillon, S, Abraha, O, Burton, A H B, Jalal, B, Bailey, A, Casey, A, Kathiravelupillai, E, Missir, H, Boult, D, Campen, J M, Collins, S, Dulai, M, Elhassan, Z, Foster, E, Horton, E, Jones, S, Mahapatra, T, Nancarrow, T, Nyamapfene, A, Rimmer, M, Robberstad, S, Robson-Brown, A, Saeed, Y, Sarwar, C, Taylor, G, Vetere, M K, Whelan, J, Williams, D, Zahid, C, Chand, and M, Matthews

Subjects
2019-20 coronavirus outbreak, Medical education, Students, Medical, Coronavirus disease 2019 (COVID-19), Education, Medical, Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), COVID-19, General Medicine, United Kingdom, Education, Pandemic, Humans, Students, Pandemics
Abstract

Dear EditorThe COVID-19 pandemic had a significant impact on the delivery of medical education across the UK, with the majority of face-to-face teaching cancelled during the first wave, and medical...

Published
2021

15. Maintenance of complex I and its supercomplexes by NDUF-11 is essential for mitochondrial structure, function and health

Author

Vicki A. M. Gold, Holly C Ford, Christopher R. Neal, Andrew P. Halestrap, Paul Verkade, Ian Collinson, Gonçalo C. Pereira, Robin A. Corey, Emma Buzzard, Amber Knapp-Wilson, Andrew P. Richardson, and Patricia E. Kuwabara

Subjects
0301 basic medicine, electron-transfer chain, Respirasome, Mitochondrial disease, Protein subunit, Worm, Supercomplexes, Mitochondrion, Biology, super-complexes, Electron Transport, electron cryo-tomography, 03 medical and health sciences, 0302 clinical medicine, Electron transfer chain, worm, medicine, Animals, Humans, Respiratory function, Caenorhabditis elegans, Cryo-electron tomography, respirasome, Electron Transport Complex I, Respiration, Cell Biology, medicine.disease, biology.organism_classification, NDUF-11, Mitochondria, Cell biology, mitochondria, 030104 developmental biology, Coenzyme Q – cytochrome c reductase, Mitochondrial Membranes, mitochondrial ultrastructure, Reactive Oxygen Species, Intermembrane space, Oxidation-Reduction, Mitochondrial ultrastructure, 030217 neurology & neurosurgery, respiration, Research Article
Abstract

Mitochondrial supercomplexes form around a conserved core of monomeric complex I and dimeric complex III; wherein a subunit of the former, NDUFA11, is conspicuously situated at the interface. We identified nduf-11 (B0491.5) as encoding the Caenorhabditis elegans homologue of NDUFA11. Animals homozygous for a CRISPR-Cas9-generated knockout allele of nduf-11 arrested at the second larval (L2) development stage. Reducing (but not eliminating) expression using RNAi allowed development to adulthood, enabling characterisation of the consequences: destabilisation of complex I and its supercomplexes and perturbation of respiratory function. The loss of NADH dehydrogenase activity was compensated by enhanced complex II activity, with the potential for detrimental reactive oxygen species (ROS) production. Cryo-electron tomography highlighted aberrant morphology of cristae and widening of both cristae junctions and the intermembrane space. The requirement of NDUF-11 for balanced respiration, mitochondrial morphology and development presumably arises due to its involvement in complex I and supercomplex maintenance. This highlights the importance of respiratory complex integrity for health and the potential for its perturbation to cause mitochondrial disease. This article has an associated First Person interview with Amber Knapp-Wilson, joint first author of the paper., Summary: Destabilisation of complex I and its supercomplexes by removal of the assembly factor NDUF-11 severely affects balanced respiration, mitochondrial morphology and, ultimately, whole animal physiology.

Published
2021
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17. Maintenance of Complex I and respiratory super-complexes by NDUF-11 is essential for respiratory function, mitochondrial structure and health inC. elegans

Author

Paul Verkade, Christopher R. Neal, Vicki A. M. Gold, Andrew P. Halestrap, Emma Buzzard, Gonçalo C. Pereira, Amber Knapp-Wilson, Robin A. Corey, Ian Collinson, Patricia E. Kuwabara, and Andrew P. Richardson

Subjects
Chemistry, RNA interference, Coenzyme Q – cytochrome c reductase, Protein subunit, Respiration, Respiratory function, Destabilisation, Allele, Respiratory system, Cell biology
Abstract

Mitochondrial super-complexes form around a conserved core of monomeric complex I and dimeric complex III; wherein subunit NDUFA11, of the former, is conspicuously situated at the interface. We identifiedB0491.5(NDUF-11) as theC. eleganshomologue, of which animals homozygous for a CRISPR-Cas9 generated knockout allele arrested at the L2 development stage. Reducing expression by RNAi allowed development to the adult stage, enabling characterisation of the consequences: destabilisation of complex I and its super-complexes, and perturbation of respiratory function. The loss of NADH-dehydrogenase activity is compensated by enhanced complex II activity, resulting in excessive detrimental ROS production. Meanwhile, electron cryo-tomography highlight aberrant cristae morphology and widening of the inter-membrane space and cristae junctions. The requirement of NDUF-11 for balanced respiration, mitochondrial morphology and development highlights the importance of complex I/ super-complex maintenance. Their perturbation by this, or other means, is likely to be the cause of metabolic stress and disease.

Published
2021
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18. Maintenance of complex I and its supercomplexes by NDUF-11 is essential for mitochondrial structure, function and health

Author

Knapp-Wilson, Amber, primary, Pereira, Gonçalo C., additional, Buzzard, Emma, additional, Ford, Holly C., additional, Richardson, Andrew, additional, Corey, Robin A., additional, Neal, Chris, additional, Verkade, Paul, additional, Halestrap, Andrew P., additional, Gold, Vicki A. M., additional, Kuwabara, Patricia E., additional, and Collinson, Ian, additional

Published
2021
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19. Maintenance of Complex I and respiratory super-complexes by NDUF-11 is essential for respiratory function, mitochondrial structure and health inC. elegans

Author

Knapp-Wilson, Amber, primary, Pereira, Gonçalo C., additional, Buzzard, Emma, additional, Richardson, Andrew, additional, Corey, Robin A., additional, Neal, Chris, additional, Verkade, Paul, additional, Halestrap, Andrew P., additional, Gold, Vicki A.M., additional, Kuwabara, Patricia, additional, and Collinson, Ian, additional

Published
2021
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20. The impact of respiratory complex dysfunction in Caenorhabditis elegans

Author

Knapp-Wilson, Amber L and Knapp-Wilson, Amber L

Abstract

Mitochondria are essential organelles of eukaryotic cells, responsible for the generation of the majority of the energy carrier molecule ATP. This essential process of energy production is achieved by oxidative phosphorylation (OXPHOS), facilitated by a set of proteins collectively named the electron transport chain (ETC). Loss of mitochondrial function has devastating effects on the cell and is usually associated with a disease state. The model organism Caenorhabditis elegans (C. elegans) is genetically tractable, has large progeny broods and provides information on a whole animal level. In recent years, it has become an established model organism for identifying connections between mitochondrial dysfunction and lifespan. Using bioinformatics combined with protein modelling, the C. elegans B0491.5 protein has been identified as the homologue of NDUFA11, a key mammalian component of the electron transport chain complex I accessory subunit. NDUFA11 is linked to complex assembly and stabilisation; loss of NDUFA11 results in a reduction of complex I and metabolic perturbation. The research presented in my thesis investigates the impact of reducing B0491.5 activity on whole animal physiology and mitochondrial biochemistry. I have shown through a genetic knockout that b0419.5 is an essential gene, and using RNAi to suppress b0491.5 expression, that in its absence animal growth is delayed and reproductive capabilities severely reduced. Depleting B0491.5 also has a significant impact on mitochondrial physiology, disrupting respiration activity and seemingly limiting animals to achieve only minimum energy demands. Mitochondrial biochemistry establishes B0491.5 enrichment in mitochondria, in addition to validating the protein’s association with complex I. Furthermore, evidence is presented suggesting B0491.5 participates in the formation of complex I containing supercomplexes. When B0491.5 is reduced, there is disruption to supercomplex formation and decreased levels

Published
2019

21. The impact of the COVID-19 pandemic on UK medical education. A nationwide student survey.

Author

Tekkis NP, Rafi D, Brown S, Courtney A, Kawka M, Howell AM, McLean K, Gardiner M, Mavroveli S, Hutchinson P, Tekkis P, Wilkinson P, Sam AH, Savva N, Kontovounisios C, Tekkis N, Rafi D, Brown S, Courtney A, Kawka M, Howell A, McLean K, Gardiner M, Mavroveli S, Hutchinson P, Tekkis P, Wilkinson P, Sam AH, Savva N, Kontovounisios C, Tekkis N, Rafi D, Brown S, Courtney A, Kawka M, Howell A, McLean K, Gardiner M, Mavroveli S, Hutchinson P, Tekkis P, Wilkinson P, Sam AH, Savva N, Kontovounisios C, Tekkis N, Brown S, Kawka M, Mclean K, Savva N, Wilkinson P, Sam AH, Singal A, Chia C, Chia W, Ganesananthan S, Ooi SZY, Pengelly S, Wellington J, Mak S, Subbiah Ponniah H, Heyes A, Aberman I, Ahmed T, Al-Shamaa S, Appleton L, Arshad A, Awan H, Baig Q, Benedict K, Berkes S, Citeroni NL, Damani A, de Sancha A, Fisayo T, Gupta S, Haq M, Heer B, Jones A, Khan H, Kim H, Meiyalagan N, Miller G, Minta N, Mirza L, Mohamed F, Ramjan F, Read P, Soni L, Tailor V, Tas RN, Vorona M, Walker M, Winkler T, Bardon A, Acquaah J, Ball T, Bani W, Elmasry A, Hussein F, Kolluri M, Lusta H, Newman J, Nott M, Perwaiz MI, Rayner R, Shah A, Shaw I, Yu K, Cairns M, Clough R, Gaier S, Hirani D, Jeyapalan T, Li Y, Patel CR, Shabir H, Wang YA, Weatherhead A, Dhiran A, Renney O, Wells P, Ferguson S, Joyce A, Mergo A, Adebayo O, Ahmad J, Akande O, Ang G, Aniereobi E, Awasthi S, Banjoko A, Bates J, Chibada C, Clarke N, Craner I, Desai DD, Dixon K, Duffaydar HI, Kuti M, Mughal AZ, Nair D, Pham MC, Preest GG, Reid R, Sachdeva GS, Selvaratnam K, Sheikh J, Soran V, Stoney N, Wheatle M, Howarth K, Knapp-Wilson A, Lee KS, Mampitiya N, Masson C, McAlinden JJ, McGowan N, Parmar SC, Robinson B, Wahid S, Willis L, Risquet R, Adebayo A, Dhingra L, Kathiravelupillai S, Narayanan R, Soni J, Ghafourian P, Hounat A, Lennon KA, Abdi Mohamud M, Chou W, Chong L, Graham CJ, Piya S, Riad AM, Vennard S, Wang J, Kawar L, Maseland C, Myatt R, Tengku Saifudin TNS, Yong SQ, Douglas F, Ogbechie C, Sharma K, Zafar L, Bajomo MO, Byrne MHV, Obi C, Oluyomi DI, Patsalides MA, Rajananthanan A, Richardson G, Clarke A, Roxas A, Adeboye W, Argus L, McSweeney J, Rahman-Chowdhury M, Hettiarachchi DS, Masood MT, Antypas A, Thomas M, de Andres Crespo M, Zimmerman M, Dhillon A, Abraha S, Burton O, Jalal AHB, Bailey B, Casey A, Kathiravelupillai A, Missir E, Boult H, Campen D, Collins JM, Dulai S, Elhassan M, Foster Z, Horton E, Jones E, Mahapatra S, Nancarrow T, Nyamapfene T, Rimmer A, Robberstad M, Robson-Brown S, Saeed A, Sarwar Y, Taylor C, Vetere G, Whelan MK, Williams J, Zahid D, Chand C, and Matthews M

Subjects
Humans, Pandemics, Students, United Kingdom epidemiology, COVID-19 epidemiology, Education, Medical, Students, Medical
Published
2022
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