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3. A FOXO1-dependent transcription network is a targetable vulnerability of mantle cell lymphomas

5. Spatial Mapping of Human Hematopoiesis at Single Cell Resolution Reveals Aging-Associated Topographic Remodeling

10. SINGLE CELL RESOLUTION SPATIAL MAPPING OF HUMAN HEMATOPOIESIS REVEALS AGING-ASSOCIATED TOPOGRAPHIC REMODELING

11. Molecular profiling of the stroke-induced alterations in the cerebral microvasculature reveals promising therapeutic candidates

21. Comparison of Multiple Clinical Testing Modalities for Assessment of NPM1-Mutant AML

22. Age-Related Clonal Hematopoiesis Associated with Adverse Outcomes

23. TCR-dependent transformation of mature memory phenotype T cells in mice

25. Lysophospholipids--receptor revelations. (Viewpoint)

30. Clinical Laboratory Validation and Implementation of Quantitative, Real-Time PCR-based Detection of NPM1 Type A Mutation.

31. Endothelial sphingosine-1-phosphate receptor 1 deficiency exacerbates brain injury and blood brain barrier dysfunction upon subarachnoid hemorrhage in mice

32. A B Cell Regulome Links Notch to Downstream Oncogenic Pathways in Small B Cell Lymphomas

33. Notch-Regulated Enhancers in B-Cell Lymphoma Activate MYC and Potentiate B-Cell Receptor Signaling

36. An oncogenic MYB feedback loop drives alternate cell fates in adenoid cystic carcinoma

37. Myeloid neoplasm demonstrating a STAT5B-RARA rearrangement and genetic alterations associated with all-trans retinoic acid resistance identified by a custom next-generation sequencing assay

39. Molecular Classification of MYC-Driven B-Cell Lymphomas by Targeted Gene Expression Profiling of Fixed Biopsy Specimens

40. Cyclin C is a haploinsufficient tumour suppressor

41. NOTCH1 Mutations Occur Early during Cutaneous Squamous Cell Carcinogenesis

42. An epigenetic mechanism of resistance to targeted therapy in T cell acute lymphoblastic leukemia

45. Gauging NOTCH1 Activation in Cancer Using Immunohistochemistry

46. Abstract B39: MYC, a downstream target of BRD-NUT, is necessary and sufficient for the blockade of differentiation in NUT midline carcinoma

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