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2. Carbon source availability drives nutrient utilization in CD8+ T cells

3. 13 C metabolite tracing reveals glutamine and acetate as critical in vivo fuels for CD8 T cells

5. Cell Context is the third axis of synergy for the combination of ATR inhibition and cisplatin in Ewing sarcoma

7. Ketolysis drives CD8+ T cell effector function through effects on histone acetylation

9. 13C metabolite tracing reveals glutamine and acetate as critical in vivo fuels for CD8+T cells

10. LKB1 controls inflammatory potential through CRTC2-dependent epigenetic remodeling

12. Data from CDK9 Blockade Exploits Context-dependent Transcriptional Changes to Improve Activity and Limit Toxicity of Mithramycin for Ewing Sarcoma

15. Supplementary Table S1 from CDK9 Blockade Exploits Context-dependent Transcriptional Changes to Improve Activity and Limit Toxicity of Mithramycin for Ewing Sarcoma

17. Supplementary Data from CDK9 Blockade Exploits Context-dependent Transcriptional Changes to Improve Activity and Limit Toxicity of Mithramycin for Ewing Sarcoma

18. Supplementary Figure S5 from Lurbinectedin Inactivates the Ewing Sarcoma Oncoprotein EWS-FLI1 by Redistributing It within the Nucleus

19. Supplementary Table S2 from Lurbinectedin Inactivates the Ewing Sarcoma Oncoprotein EWS-FLI1 by Redistributing It within the Nucleus

20. Supplementary Data S1A-S9 from Trabectedin Inhibits EWS-FLI1 and Evicts SWI/SNF from Chromatin in a Schedule-dependent Manner

21. Supplemental Legends from Lurbinectedin Inactivates the Ewing Sarcoma Oncoprotein EWS-FLI1 by Redistributing It within the Nucleus

22. Data from Lurbinectedin Inactivates the Ewing Sarcoma Oncoprotein EWS-FLI1 by Redistributing It within the Nucleus

23. Endometrial hyperplasia with loss of APC in a novel population of Lyz2-expressing mouse endometrial epithelial cells

24. Ketolysis is a metabolic driver of CD8+ T cell effector function through histone acetylation

26. Endometrial hyperplasia with loss of APC in a novel population of Lyz2-expressing mouse endometrial epithelial cells.

27. Mithramycin induces promoter reprogramming and differentiation of rhabdoid tumor

28. Abstract A75: Mithramycin evicts SWI/SNF from chromatin to induce epigenetic reprogramming in rhabdoid tumor

29. CDK9 Blockade Exploits Context-dependent Transcriptional Changes to Improve Activity and Limit Toxicity of Mithramycin for Ewing Sarcoma

30. ACLY and ACSS2 link nutrient-dependent chromatin accessibility to CD8 T cell effector responses

31. Direct therapeutic targeting of SWI/SNF induces epigenetic reprogramming and durable tumor regression in rhabdoid tumor

33. Trabectedin Inhibits EWS-FLI1 and Evicts SWI/SNF from Chromatin in a Schedule-dependent Manner

35. Lurbinectedin Inactivates the Ewing Sarcoma Oncoprotein EWS-FLI1 by Redistributing It within the Nucleus

36. Erratum: Corrigendum: Innate control of actin nucleation determines two distinct migration behaviours in dendritic cells

37. Innate control of actin nucleation determines two distinct migration behaviours in dendritic cells

42. Innate control of actin nucleation determines two distinct migration behaviours in dendritic cells

43. Repression of LKB1 by miR-17∼92Sensitizes MYC-Dependent Lymphoma to Biguanide Treatment

44. 13C metabolite tracing reveals glutamine and acetate as critical in vivo fuels for CD8 T cells.

45. Corrigendum: Innate control of actin nucleation determines two distinct migration behaviours in dendritic cells

46. NRF2-dependent regulation of the prostacyclin receptor PTGIR drives CD8 T cell exhaustion.

47. Ketolysis drives CD8 + T cell effector function through effects on histone acetylation.

48. 13 C metabolite tracing reveals glutamine and acetate as critical in vivo fuels for CD8 + T cells.

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