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2. Disease Progression of WHIM Syndrome in an International Cohort of 66 Pediatric and Adult Patients

3. Broad-spectrum antibodies against self-antigens and cytokines in RAG deficiency.

5. Expansion of immunoglobulin-secreting cells and defects in B cell tolerance in Rag-dependent immunodeficiency

7. Constitutive activation of WASp in X-linked neutropenia renders neutrophils hyperactive

9. Anti-Citrullinated Protein Antibody Reactivity towards Neutrophil-Derived Antigens : Clonal Diversity and Inter-Individual Variation

10. Generation of NK cells with chimeric-switch receptors to overcome PD1-mediated inhibition in cancer immunotherapy

11. A gain-of-function variant in the Wiskott-Aldrich syndrome gene is associated with a MYH9-related disease-like syndrome

12. IFNγ directly counteracts imatinib-induced apoptosis of primary human CD34+ CML stem/progenitor cells potentially through the upregulation of multiple key survival factors

13. P-020: PD1-based chimeric-switch receptor expressing NK cells recover from immune checkpoint inhibition in cancer

14. IFN gamma directly counteracts imatinib-induced apoptosis of primary human CD34+CML stem/progenitor cells potentially through the upregulation of multiple key survival factors

17. B cell–intrinsic deficiency of the Wiskott-Aldrich syndrome protein (WASp) causes severe abnormalities of the peripheral B-cell compartment in mice

18. Overactive WASp in X-linked neutropenia leads to aberrant B-cell division and accelerated plasma cell generation

19. Homozygous DNA Ligase IV R278H Mutation in Mice Leads to Leaky SCID and Represents a Model for Human LIG4 Syndrome

20. Constitutive activation of WASp in X-linked neutropenia renders neutrophils hyperactive

23. Constitutive activation of WASp leads to abnormal cytotoxic cells with increased granzyme B and degranulation response to target cells

30. Broad-spectrum antibodies against self-antigens and cytokines in RAG deficiency

31. Deletion of Wiskott-Aldrich syndrome protein triggers Rac2 activity and increased cross-presentation by dendritic cells

32. Congenital Defects in Neutrophil Dynamics

33. Deletion of Wiskott–Aldrich syndrome protein triggers Rac2 activity and increased cross-presentation by dendritic cells

34. An NK cell-independent role for slamf4 in controlling humoral Autoimmunity1

35. Deletion of WASp and N-WASp in B cells cripples the germinal center response and results in production of IgM autoantibodies

36. A novel isoform of the Ly108 gene ameliorates murine lupus

43. A novel isoform of the Ly108 gene ameliorates murine lupus

44. SLAM is a microbial sensor that regulates bacterial phagosome functions in macrophages

45. Defects of B Cell Tolerance and Expansion of Immunoglobulin Secreting Cells in Rag-dependent Immunodeficiency

49. Wiskott-Aldrich syndrome gene mutations modulate cancer susceptibility in the p53± murine model.

50. Β cell-intrinsic deficiency of the Wiskott-Aldrich syndrome protein (WASp) causes severe abnormalities of the peripheral B-cell compartment in mice.

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