167 results on '"Keramidas, Angelo"'
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2. Anti-seizure mechanisms of midazolam and valproate at the β2(L51M) variant of the GABAA receptor
3. Pharmacological activation of ATF6 remodels the proteostasis network to rescue pathogenic GABAA receptors
4. A pain-causing and paralytic ant venom glycopeptide
5. Proteostasis Regulators Restore Function of Epilepsy-Associated GABAA Receptors
6. GRIN1 variants associated with neurodevelopmental disorders reveal channel gating pathomechanisms
7. Erythromelalgia caused by the missense mutation p.Arg220Pro in an alternatively spliced exon of SCN9A (NaV1.7).
8. Correlations of receptor desensitization of gain-of-function GABRB3 variants with clinical severity.
9. Erythromelalgia caused by the missense mutation p.Arg220Pro in an alternatively spliced exon of SCN9A (NaV1.7)
10. Correlations of receptor desensitization of gain-of-function GABRB3 variants with clinical severity
11. Potent neuroprotection after stroke afforded by a double-knot spider-venom peptide that inhibits acid-sensing ion channel 1a
12. Shisa7-Dependent Regulation of GABAAReceptor Single-Channel Gating Kinetics
13. Additional file 1 of Pharmacological activation of ATF6 remodels the proteostasis network to rescue pathogenic GABAA receptors
14. Shisa7-dependent regulation of GABAA receptor single-channel gating kinetics
15. Shisa7-Dependent Regulation of GABAA Receptor Single-Channel Gating Kinetics.
16. Regulation of NMDA receptor trafficking and gating by activity-dependent CaMKIIα phosphorylation of the GluN2A subunit
17. Zolpidem and eszopiclone prime α1β2γ2 GABAA receptors for longer duration of activity
18. Functional reconstitution of glycinergic synapses incorporating defined glycine receptor subunit combinations
19. An outline of desensitization in pentameric ligand-gated ion channel receptors
20. The activation mechanism of [[alpha].sub.1][[beta].sub.2][[gamma].sub.2s] and [[alpha].sub.3][[beta].sub.3][[gamma].sub.2s][GABA.sub.A] receptors
21. Pharmacological activation of ATF6 remodels the proteostasis network to rescue pathogenic GABAA receptors.
22. Decision letter: Unmasking coupling between channel gating and ion permeation in the muscle nicotinic receptor
23. Agonist-dependent single channel current and gating in [[alpha].sub.4][[beta].sub.2][delta] and [[alpha].sub.1][[beta].sub.2][[gamma].sub.2s] [GABA.sub.A] receptors
24. Single channel analysis of conductance and rectification in cation-selective, mutant glycine receptor channels
25. Cation-selective mutations in the M2 domain of the inhibitory glycine receptor channel reveal determinants of ion-charge selectivity
26. Effects of GluN2A and GluN2B gain-of-function epilepsy mutations on synaptic currents mediated by diheteromeric and triheteromeric NMDA receptors
27. A Novel Glycine Receptor Variant with Startle Disease Affects Syndapin I and Glycinergic Inhibition
28. The effects of insecticides on two splice variants of the glutamate‐gated chloride channel receptor of the major malaria vector,Anopheles gambiae
29. The pre-M1 segment of the α1 subunit is a transduction element in the activation of the GABAA receptor
30. GluClR-mediated inhibitory postsynaptic currents reveal targets for ivermectin and potential mechanisms of ivermectin resistance
31. Effects of glutamate and ivermectin on single glutamate-gated chloride channels of the parasitic nematode H. contortus
32. The effects of insecticides on two splice variants of the glutamate-gated chloride channel receptor of the major malaria vector, Anopheles gambiae.
33. SAHA (Vorinostat) Corrects Inhibitory Synaptic Deficits Caused by Missense Epilepsy Mutations to the GABAA Receptor γ2 Subunit
34. Structure/Function Studies of the α4 Subunit Reveal Evolutionary Loss of a GlyR Subtype Involved in Startle and Escape Responses
35. Inhibitory synapse deficits caused by familial α1 GABAA receptor mutations in epilepsy
36. Physiological and pharmacological properties of inhibitory postsynaptic currents mediated by α5β1γ2, α5β2γ2 and α5β3γ2 GABA A receptors
37. γ1-Containing GABA-A Receptors Cluster at Synapses Where they Mediate Slower Synaptic Currents than γ2-Containing GABA-A Receptors
38. Structure-Function Analysis of the GlyR α2 Subunit Autism Mutation p.R323L Reveals a Gain-of-Function
39. Multiple sodium channel isoforms mediate the pathological effects of Pacific ciguatoxin-1
40. Probing the Structural Mechanism of Partial Agonism in Glycine Receptors Using the Fluorescent Artificial Amino Acid, ANAP
41. Ivermectin-Activated, Cation-Permeable Glycine Receptors for the Chemogenetic Control of Neuronal Excitation
42. The Free Zinc Concentration in the Synaptic Cleft of Artificial Glycinergic Synapses Rises to At least 1 μM
43. Investigating the Mechanism by Which Gain-of-function Mutations to the α1 Glycine Receptor Cause Hyperekplexia
44. SAHA (Vorinostat) Corrects Inhibitory Synaptic Deficits Caused by Missense Epilepsy Mutations to the GABAA Receptor γ2 Subunit.
45. Physiological and pharmacological properties of inhibitory postsynaptic currents mediated by α5β1γ2, α5β2γ2 and α5β3γ2 GABAA receptors.
46. Zolpidem and eszopiclone prime α1β2γ2 GABAAreceptors for longer duration of activity
47. Correlating Structural and Energetic Changes in Glycine Receptor Activation
48. Structure-Function Analysis of the GlyR α2 Subunit Autism Mutation p.R323L Reveals a Gain-of-Function.
49. GABAA Receptor α and γ Subunits Shape Synaptic Currents via Different Mechanisms
50. New Hyperekplexia Mutations Provide Insight into Glycine Receptor Assembly, Trafficking, and Activation Mechanisms
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