16 results on '"Ke, Sujie"'
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2. Mito-TEMPO, a Mitochondria-Targeted Antioxidant, Improves Cognitive Dysfunction due to Hypoglycemia: an Association with Reduced Pericyte Loss and Blood-Brain Barrier Leakage
3. Thyroid hormone receptor alpha sumoylation modulates white adipose tissue stores
4. Altered Caffeine Metabolism Is Associated With Recurrent Hypoglycemia in Type 2 Diabetes Mellitus: A UPLC–MS-Based Untargeted Metabolomics Study
5. Lifestyle is associated with thyroid function in subclinical hypothyroidism: a cross-sectional study
6. Mito-TEMPO, a Mitochondria-Targeted Antioxidant, Improves Cognitive Dysfunction due to Hypoglycemia: an Association with Reduced Pericyte Loss and Blood-Brain Barrier Leakage
7. Subcutaneous administration of liraglutide ameliorates learning and memory impairment by modulating tau hyperphosphorylation via the glycogen synthase kinase-3β pathway in an amyloid β protein induced alzheimer disease mouse model
8. Thyroid hormone receptor β sumoylation is required for thyrotropin regulation and thyroid hormone production
9. Additional file 1 of Lifestyle is associated with thyroid function in subclinical hypothyroidism: a cross-sectional study
10. Correlation between Thyroid Homeostasis and Obesity in Subclinical Hypothyroidism: Community-Based Cross-Sectional Research
11. Recurrent non-severe hypoglycemia aggravates cognitive decline in diabetes and induces mitochondrial dysfunction in cultured astrocytes
12. Insufficient nocturnal sleep was associated with a higher risk of fibrosis in patients with diabetes with metabolic associated fatty liver disease
13. Exenatide alleviates mitochondrial dysfunction and cognitive impairment in the 5×FAD mouse model of Alzheimer’s disease
14. Hypertension outcomes of adrenalectomy in patients with primary aldosteronism: a systematic review and meta-analysis
15. Thyroid hormone treatment activates protective pathways in both in vivo and in vitro models of neuronal injury
16. [Exendin-4 ameliorates high glucose- and TNF-α-induced vascular endothelial cell damage by inhibiting p38 MAPK and NF-κB p65 translocation].
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