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3. AMFR dysfunction causes autosomal recessive spastic paraplegia in human that is amenable to statin treatment in a preclinical model

4. The phenotypic spectrum of PTCD3 deficiency.

5. The phenotypic spectrum of PTCD3 deficiency

6. Influence of b2 adrenergic receptor polymorphism (rs1042713 and rs1042714) on anthropometric, hormonal and lipid profiles in polycystic ovarian syndrome

7. Expanding the phenotypic and genotypic spectrum of GGPS1 related congenital muscular dystrophy

8. Truncating mutations in YIF1B cause a progressive encephalopathy with various degrees of mixed movement disorder, microcephaly, and epilepsy

9. Loss of UGP2 in brain leads to a severe epileptic encephalopathy, emphasizing that bi-allelic isoform-specific start-loss mutations of essential genes can cause genetic diseases

10. Expanding the phenotypic and genotypic spectrum of GGPS1 related congenital muscular dystrophy.

12. Polymorphic variations in VDR gene in Saudi women with and without polycystic ovary syndrome (PCOS) and significant influence of seven polymorphic sites on anthropometric and hormonal parameters

13. Novel UBE3Bmutations: report of eight patients with Kaufman oculocerebrofacial syndrome with additional clinical findings from a highly consanguineous population

18. Clinical, radiological, and genetic characterization of SLC13A5 variants in Saudi families: Genotype phenotype correlation and brief review of the literature

19. A molecular study of pediatric pilomyxoid and pilocytic astrocytomas: Genome-wide copy number screening, retrospective analysis of clinicopathological features and long-term clinical outcome

22. Global Transcriptional Profiling of Granulosa Cells from Polycystic Ovary Syndrome Patients: Comparative Analyses of Patients with or without History of Ovarian Hyperstimulation Syndrome Reveals Distinct Biomarkers and Pathways

23. A Novel Homozygous Founder Variant of RTN4IP1 in Two Consanguineous Saudi Families

24. Further delineation of Temtamy syndrome of corpus callosum and ocular abnormalities

25. Molecular and clinical spectra of FBXL4 deficiency

26. Clinical, genetic, and functional characterization of the glycine receptor β-subunit A455P variant in a family affected by hyperekplexia syndrome

27. Clinico-radiological features, molecular spectrum, and identification of prognostic factors in developmental and epileptic encephalopathy due to inosine triphosphate pyrophosphatase (ITPase) deficiency

29. Clinico‐radiological features, molecular spectrum, and identification of prognostic factors in developmental and epileptic encephalopathy due to inosine triphosphate pyrophosphatase (ITPase) deficiency

30. A Novel GEMIN4 Variant in a Consanguineous Family Leads to Neurodevelopmental Impairment with Severe Microcephaly, Spastic Quadriplegia, Epilepsy, and Cataracts

31. Exome Sequencing Reveals Novel TTN Variants in Saudi Patients with Congenital Titinopathies

32. Phenotypic Variability of MEGF10 Variants Causing Congenital Myopathy: Report of Two Unrelated Patients from a Highly Consanguineous Population

34. Genetics of ataxia telangiectasia in a highly consanguineous population

35. ISCA2 mutation causes infantile neurodegenerative mitochondrial disorder

39. Expanding the mutational landscape and clinical phenotype of the YIF1B related brain disorder

40. Bi-allelic variants in HOPS complex subunit VPS41 cause cerebellar ataxia and abnormal membrane trafficking

41. Expanding the mutational landscape and clinical phenotype of the YIF1B related brain disorder

44. METTL23, a transcriptional partner of GABPA, is essential for human cognition

46. SLC25A42 ‐associated mitochondrial encephalomyopathy: Report of additional founder cases and functional characterization of a novel deletion

47. Hematological findings associated with tubulin‐folding cofactors D‐related encephalopathy: Expanding the phenotype

48. Bi-allelic variants in HOPS complex subunit VPS41 cause cerebellar ataxia and abnormal membrane trafficking

50. Mutations in C2orf37, encoding a nucleolar protein, cause hypogonadism, alopecia, diabetes mellitus, mental retardation, and extrapyramidal syndrome

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