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257 results on '"Katsumata S"'

2. P1.25-01 Real-World Data of Completely Resected Lung Adenocarcinoma Harboring Common EGFR Mutation in Japan (CReGYT-01 EGFR study)

Author

Katsumata, S., primary, Hamada, A., additional, Haratake, N., additional, Takamori, S., additional, Takanashi, Y., additional, Hayasaka, K., additional, Nomura, K., additional, Toda, M., additional, Kaiho, T., additional, Hoshino, H., additional, Ozawa, H., additional, Matsuo, S., additional, Suzuki, J., additional, Nakahashi, K., additional, Fujino, K., additional, Takegahara, K., additional, Kinoshita, I., additional, Takada, K., additional, Yoshikawa, M., additional, Tomioka, Y., additional, Koki, Y., additional, Shimokawa, M., additional, Soh, J., additional, Shiono, S., additional, and Ohde, Y., additional

Published
2023
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7. EP.06C.03 Clinicopathological Analysis of Each EGFR Status in Surgically Resected Lung Adenocarcinoma: A Real-World Study (CReGYT-01 EGFR)

Author

HOSHINO, H., Katsumata, S., Hamada, A., Haratake, N., Nomura, K., Fujino, K., Yoshikawa, M., Suzawa, K., Shien, K., Suda, K., Ohara, S., Fukuda, S., Kinoshita, I., Hayasaka, K., Notsuda, H., Takamori, S., Muto, S., Takanashi, Y., Mizuno, K., Kawase, A., Hayakawa, T., Sekihara, K., Toda, M., Matsuo, S., Takegahara, K., Hashimoto, M., Nakahashi, K., Endo, M., Ozawa, H., Fujikawa, R., Tomioka, Y., Namba, K., Matsubara, T., Suzuki, J., Watanabe, H., Takada, K., Toyoda, T., Nakasone, S., Kawasaki, H., Shimokawa, M., Kouki, Y., Shiono, S., Soh, J., and Ohde, Y.

Published
2024
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8. P2.07A.04 Clinical Relevance of PD-L1 Expression Levels in Surgically Resected EGFR-Mutant Lung Adenocarcinoma Patients (CReGYT-01 Study)

Author

Kinoshita, F., Nomura, K., Takada, K., Muto, S., Matsubara, T., Kouki, Y., Katsumata, S., Hamada, A., Haratake, N., Fujino, K., Yoshikawa, M., Suzawa, K., Shien, K., Suda, K., Ohara, S., Fukuda, S., Suzuki, H., Okamoto, T., Hirai, F., Aokage, K., Shiono, S., Soh, J., Tsuboi, M., Shimokawa, M., Ohde, Y., Takenaka, T., and Yoshizumi, T.

Published
2024
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9. P2.07A.03 Prognostic Analysis of Completely Resected Lung Adenocarcinoma with Uncommon EGFR Mutations: CReGYT-01 EGFR Study

Author

Hayasaka, K., Haratake, N., Notsuda, H., Katsumata, S., Hamada, A., Nomura, K., Fujino, K., Yoshikawa, M., Suzawa, K., Shien, K., Suda, K., Ohara, S., Fukuda, S., Kinoshita, I., Takamori, S., Muto, S., Takanashi, Y., Mizuno, K., Kawase, A., Hayakawa, T., Sekihara, K., Toda, M., Matsuo, S., Takegahara, K., Hashimoto, M., Nakahashi, K., Endo, M., Ozawa, H., Fujikawa, R., Tomioka, Y., Namba, K., Matsubara, T., Suzuki, J., Watanabe, H., Takada, K., Hoshino, H., Toyoda, T., Koki, Y., Shiono, S., Shimokawa, M., Soh, J., Ohde, Y., and Okada, Y.

Published
2024
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14. A concrete treatment of efficient continuous group key agreement via multi-recipient PKEs

Author

Hashimoto, K. (Keitaro), Katsumata, S. (Shuichi), Postlethwaite, E.W. (Eamonn), Prest, T. (Thomas), Westerbaan, B. (Bas), Hashimoto, K. (Keitaro), Katsumata, S. (Shuichi), Postlethwaite, E.W. (Eamonn), Prest, T. (Thomas), and Westerbaan, B. (Bas)

Abstract

Continuous group key agreements (CGKAs) are a class of protocols that can provide strong security guarantees to secure group messaging protocols such as Signal and MLS. Protection against device compromise is provided by commit messages: at a regular rate, each group member may refresh their key material by uploading a commit message, which is then downloaded and processed by all the other members. In practice, propagating commit messages dominates the bandwidth consumption of existing CGKAs. We propose Chained CmPKE, a CGKA with an asymmetric bandwidth cost: in a group of N members, a commit message costs O(N) to upload and O(1) to download, for a total bandwidth cost of O(N). In contrast, TreeKEM costs (log N) in both directions, for a total cost (N log N). Our protocol relies on generic primitives, and is therefore readily post-quantum. We go one step further and propose post-quantum primitives that are tailored to \Chained CmPKE, which allows us to cut the growth rate of uploaded commit messages by two or three orders of magnitude compared to naive instantiations. Finally, we realize a software implementation of Chained CmPKE. Our experiments show that even for groups with a size as large as N = 2^10, commit messages can be computed and processed in less than 100 ms.

Published
2021
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15. Ultrafast transient reflectivity measurements of optimally doped Bi2+xSr2-xCaCu2O8+delta with disorder

Author

1000000313106, Toda, Y., Tsuchiya, S., Oda, M., Kurosawa, T., Katsumata, S., Naseska, M., Mertelj, T., Mihailovic, D., 1000000313106, Toda, Y., Tsuchiya, S., Oda, M., Kurosawa, T., Katsumata, S., Naseska, M., Mertelj, T., and Mihailovic, D.

Abstract

The effect of disorder in high-T-c superconductors is investigated from the viewpoint of photoinduced quasipartick (QP) dynamics, obtained through time-resolved pump-probe spectroscopy. We perform the measurements on an optimally doped (OPD) Bi2+xSr2-xCaCu2O8+delta (Bi2212) with an out-of-plane disorder induced by Bi-Sr substitution (x), which is present in standard Bi2212 but is not usually controlled. Based on the systematic change of the disorder with controlled x, we identify the changes in the dynamics of QP relaxation and gap formation in superconducting (SC) and pseudogap (PG) states. The onset temperature T* of the PG response increases with an increase in x, which is more significant than the slight decrease in T-c. These properties are equivalently reflected in the destruction fluences of the SC and PG states. Bi-Sr substitution also accelerates relaxation times for SC and PG QPs, which can be due to the increased phonon scattering probability caused by the disorder. In contrast, the SC gap recovery observed in the strongly excited condition shows an identical fluence dependence for various x, implying that the disorder does not significantly contribute to coherent gap formation.

Published
2021

16. Ultrafast transient reflectivity measurements of optimally doped Bi2+xSr2-xCaCu2O8+delta with disorder

Author

Toda, Y., Tsuchiya, S., Oda, M., Kurosawa, T., Katsumata, S., Naseska, M., Mertelj, T., Mihailovic, D., Toda, Y., Tsuchiya, S., Oda, M., Kurosawa, T., Katsumata, S., Naseska, M., Mertelj, T., and Mihailovic, D.

Abstract

The effect of disorder in high-T-c superconductors is investigated from the viewpoint of photoinduced quasipartick (QP) dynamics, obtained through time-resolved pump-probe spectroscopy. We perform the measurements on an optimally doped (OPD) Bi2+xSr2-xCaCu2O8+delta (Bi2212) with an out-of-plane disorder induced by Bi-Sr substitution (x), which is present in standard Bi2212 but is not usually controlled. Based on the systematic change of the disorder with controlled x, we identify the changes in the dynamics of QP relaxation and gap formation in superconducting (SC) and pseudogap (PG) states. The onset temperature T* of the PG response increases with an increase in x, which is more significant than the slight decrease in T-c. These properties are equivalently reflected in the destruction fluences of the SC and PG states. Bi-Sr substitution also accelerates relaxation times for SC and PG QPs, which can be due to the increased phonon scattering probability caused by the disorder. In contrast, the SC gap recovery observed in the strongly excited condition shows an identical fluence dependence for various x, implying that the disorder does not significantly contribute to coherent gap formation.

Published
2021

40. A single residue, Lys108, of the delta-opioid receptor prevents the mu-opioid-selective ligand [D-Ala2,N-MePhe4,Gly-ol5]enkephalin from binding to the delta-opioid receptor.

Author

Minami, M, Nakagawa, T, Seki, T, Onogi, T, Aoki, Y, Katao, Y, Katsumata, S, and Satoh, M

Abstract

Previously, we found that replacement of the region around the first extracellular loop of the delta-opioid receptor (OPR) with the corresponding region of the mu-OPR gives the high affinity for [D-Ala2,N-MePhe4,Gly-ol5]enkephalin (DAMGO), a mu-opioid-selective ligand, to the resultant chimeric receptor, DMDD, suggesting that the difference in the amino acid sequence within this region between the mu- and delta-OPRs is critical for the discrimination between these receptors by DAMGO. In the current study, we carried out systematic replacements of seven non-conserved residues in this region of the delta-OPR with the corresponding amino acid found in the mu-OPR. Among the seven mutant receptors, only one mutant receptor, delta K108N, showed high affinity (Ki = 18.68 +/- 5.27 nM) for DAMGO, which was comparable to that of the DMDD receptor (Ki = 23.77 +/- 4.27 nM) and 75-fold higher than that of the wild-type delta-OPR (Ki = 1405 +/- 161 nM). Lys108 in the delta-OPR was systematically replaced with 19 kinds of amino acids other than lysine. Among the resultant mutant receptors, 14 mutants bound DAMGO with Ki values comparable to those of the DMDD receptor, ranging from 4.20 to 43.38 nM. These findings suggest that Lys108 of the delta-OPR prevents DAMGO from binding to the delta-OPR rather than that the asparagine residue at the corresponding position in the mu-OPR is necessary for DAMGO binding. In addition, the replacement of Lys108 of the delta-OPR with asparagine dramatically increased the affinity for other peptidic mu receptor-selective ligands, such as dermorphin and D-Pen-Cys-Tyr-D-Trp-Orn-Thr-Pen-Thr-NH2.

Published
1996

48. Two cases of colonic epithelial tumors associating with von Recklinghausen's disease.

Author

Chen, P., primary, Osone, T., additional, Sugiura, H., additional, Takahashi, H., additional, Sugita, K., additional, Shiratori, Y., additional, Kinoshita, T., additional, Kubota, Y., additional, Katsumata, S., additional, Horimukai, F., additional, Takeshita, T., additional, Miyaoka, M., additional, Matsumoto, E., additional, Saito, T., additional, and Ashizawa, S., additional

Published
1986
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