137 results on '"Kasper, Lydia"'
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2. Toll-like receptor 4 and CD11b expressed on microglia coordinate eradication of Candida albicans cerebral mycosis
3. Candida albicans translocation through the intestinal epithelial barrier is promoted by fungal zinc acquisition and limited by NFκB-mediated barrier protection
4. I want to break free – macrophage strategies to recognize and kill Candida albicans, and fungal counter-strategies to escape
5. "We've got to get out"—Strategies of human pathogenic fungi to escape from phagocytes.
6. Metabolic adaptation of intracellular bacteria and fungi to macrophages
7. “Under Pressure” – How fungi evade, exploit, and modulate cells of the innate immune system
8. From intestinal colonization to systemic infections: Candida albicans translocation and dissemination
9. The fungal peptide toxin Candidalysin activates the NLRP3 inflammasome and causes cytolysis in mononuclear phagocytes
10. Candidalysin is a fungal peptide toxin critical for mucosal infection
11. Transkingdom mechanism of MAMP generation by chitotriosidase (CHIT1) feeds oligomeric chitin from fungal pathogens and allergens into TLR2-mediated innate immune sensing
12. Cover Image: The fungivorous amoeba Protostelium aurantium targets redox homeostasis and cell wall integrity during intracellular killing of Candida parapsilosis (Cellular Microbiology 11/2021)
13. The fungivorous amoeba Protostelium aurantium targets redox homeostasis and cell wall integrity during intracellular killing of Candida parapsilosis
14. Thriving within the host: Candida spp. interactions with phagocytic cells
15. Lysosome Fusion Maintains Phagosome Integrity during Fungal Infection
16. ATRP1-marker-based system for gene complementation, overexpression, reporter gene expression and gene modification inCandida glabrata
17. The Dual Function of the Fungal Toxin Candidalysin during Candida albicans—Macrophage Interaction and Virulence
18. Fungal biotin homeostasis is essential for immune evasion after macrophage phagocytosis and virulence
19. RNAi as a Tool to Study Virulence in the Pathogenic Yeast Candida glabrata
20. RNAi as a Tool to Study Virulence in the Pathogenic Yeast Candida glabrata
21. A TRP1-marker-based system for gene complementation, overexpression, reporter gene expression and gene modification in Candida glabrata.
22. Candida albicans-Induced Epithelial Damage Mediates Translocation through Intestinal Barriers
23. Immunoproteomic Analysis of Antibody Responses to Extracellular Proteins of Candida albicans Revealing the Importance of Glycosylation for Antigen Recognition
24. Metals in fungal virulence
25. The Fungal Pathogen Candida glabrata Does Not Depend on Surface Ferric Reductases for Iron Acquisition
26. A Novel Hybrid Iron Regulation Network Combines Features from Pathogenic and Nonpathogenic Yeasts
27. Aspartyl proteinases of Candida albicans induce caspase-11: implication in promoting inflammatory response
28. Virulence factors in fungal pathogens of man
29. Immunoproteomic Analysis of Antibody Responses to Extracellular Proteins of Candida albicans Revealing the Importance of Glycosylation for Antigen Recognition
30. In vivoinduction of neutrophil chemotaxis by secretory aspartyl proteinases ofCandida albicans
31. Metals in fungal virulence.
32. Divergent Evolution of the Transcriptional Network Controlled by Snf1-Interacting Protein Sip4 in Budding Yeasts
33. Antifungal activity of clotrimazole against Candida albicans depends on carbon sources, growth phase and morphology
34. Secretory Aspartyl Proteinases Cause Vaginitis and Can Mediate Vaginitis Caused by Candida albicans in Mice
35. Intracellular survival ofCandida glabratain macrophages: immune evasion and persistence
36. Of mice, flies – and men? Comparing fungal infection models for large-scale screening efforts
37. Induction of Caspase-11 by Aspartyl Proteinases of Candida albicans and Implication in Promoting Inflammatory Response
38. One Small Step for a Yeast - Microevolution within Macrophages Renders Candida glabrata Hypervirulent Due to a Single Point Mutation
39. Identification of Candida glabrata Genes Involved in pH Modulation and Modification of the Phagosomal Environment in Macrophages
40. A family of glutathione peroxidases contributes to oxidative stress resistance in Candida albicans
41. Immune Evasion, Stress Resistance, and Efficient Nutrient Acquisition Are Crucial for Intracellular Survival of Candida glabrata within Macrophages
42. Secreted aspartic proteases ofCandida albicansactivate the NLRP3 inflammasome
43. In vivo induction of neutrophil chemotaxis by secretory aspartyl proteinases of Candida albicans.
44. Immunoproteomic Analysis of Antibody Responses to Extracellular Proteins of Candida albicans Revealing the Importance of Glycosylation for Antigen Recognition.
45. True posterior communicating artery aneurysms: are they more prone to rupture? A biomorphometric analysis
46. Predicting aneurysm rupture probabilities through the application of a computed tomography angiography–derived binary logistic regression model
47. Intracellular survival of Candida glabrata in macrophages: immune evasion and persistence.
48. Toll-like receptor 4 and CD11b expressed on microglia coordinate eradication of Candida albicanscerebral mycosis
49. From intestinal colonization to systemic infections: Candida albicanstranslocation and dissemination
50. In vivoinduction of neutrophil chemotaxis by secretory aspartyl proteinases of Candida albicans
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