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439 results on '"Kashiwagi N"'

16. Antigen Society #12 Report (Bw54, Bw55, Bw56 and Bw42)

Author

Sekiguchi, S., Neumeyer, H., Kashiwagi, N., Tsuji, K., Kobayashi, K., Konoeda, Y., Ohkubo, M., Atoh, M., Tokunaga, K., Yagita, A., Inoko, H., Fong, R., Mervart, H., Paik, Y., Reekers, P., Hammond, M., Du Toit, E., Call, E., and Dupont, Bo, editor

Published
1989
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19. Antigen Report: HLA-B51 and HLA-Bw52

Author

Kashiwagi, N., Taylor, C., Ting, A., Albert, Ekkehard D., editor, Baur, Max P., editor, Mayr, Wolfgang R., editor, Bertrams, J., editor, Goldmann, S., editor, Grosse-Wilde, H., editor, Opelz, G., editor, Rittner, C., editor, Schendel, D. J., editor, Scholz, S., editor, and Wank, R., editor

Published
1984
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28. Association between non-alcoholic hepatic steatosis and hyper reactive blood pressure response on the exercise treadmill test.

Author

Laurinavicius, A. G., Bittencourt, M. S., Blaha, M. J., Nary, F. C., Kashiwagi, N. M., Conceiçao, R. D., Meneghelo, R. S., Prado, R. R., Carvalho, J. A. M., Nasir, K., Blumenthal, R. S., and Santos, R. D.

Subjects
HYPERTENSION, CARDIOVASCULAR disease diagnosis, CARDIOVASCULAR diseases risk factors, FATTY degeneration, BLOOD pressure, TREADMILLS, ULTRASONIC imaging
Abstract

Aims: Non-alcoholic hepatic steatosis (HS) is associated with hypertension and increased cardiovascular risk. While Blood pressure hyper-reactive response (HRR) during peak exercise indicates an increased risk of incident hypertension and increased cardiovascular risk, no data on the association of non-alcoholic HS and HRR exists. In this study, we have evaluated the association of HS with HRR. Methods: We included 13 410 consecutive individuals with a mean age: 42.4 ± 8.9 years, 3561 (26.6%) female with normal resting blood pressure and without a previous diagnosis of hypertension, who underwent symptom limited exercise treadmill test, abdominal ultrasonography and clinical and laboratory evaluation. HS was detected by abdominal ultrasonography. HRR was defined by a peak exercise systolic blood pressure >220mmHg and/or elevation of 15mmHg or more in diastolic blood pressure from rest to peak exercise. Results: The prevalence of HS was 29.5% (n ¼ 3956). Overall, 4.6% (n = 619) of the study population presented a HRR. Subjects with HS had a higher prevalence of HRR (8.1 vs. 3.1%, odds ratio 2.8, 95% CI 2.4--3.3, P < 0.001). After adjustment for body mass index, waist circumference, fasting plasma glucose and low density lipoprotein cholesterol, HS (odds ratio 1.4, 95% CI 1.1-1.6, P = 0.002) remained independently associated with HRR. HS was additive to obesity markers in predicting exercise HRR. Conclusions: Non-alcoholic HS is independently associated with hyper-reactive exercise blood pressure response. [ABSTRACT FROM AUTHOR]

Published
2016
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