17 results on '"Karim, Muhammad Zia"'
Search Results
2. Notochordal cell-derived conditioned medium protects human nucleus pulposus cells from stress-induced apoptosis
3. NTG-101: A Novel Molecular Therapy that Halts the Progression of Degenerative Disc Disease
4. A Single Injection of NTG-101 Reduces the Expression of Pain-Related Neurotrophins in a Canine Model of Degenerative Disc Disease
5. 158. The effects of a single injection of NTG-101 upon neurotrophin expression in a canine model of degenerative disc disease
6. 213. Study comparing NTG-101, a growth factor-based therapy with mesenchymal stem cell (MSC) based treatment of degenerative disc disease (DDD) in pre-clinical rodent model
7. 122. Cartilage derived stem cells: a novel cellular replacement therapy to treat degenerative disc disease
8. Thursday, September 27, 2018 2:05 PM–3:05 PM Section on Biologics and Basic Research Abstract Presentations
9. Notochordal Cell-Based Therapeutics Can Regenerate the Degenerative Disc
10. Notochordal Cell-Based Therapeutics Regenerate the Degenerative Disc: Evidence in a Preclinical Animal Model of Disc Disease
11. The biological basis of degenerative disc disease: proteomic and biomechanical analysis of the canine intervertebral disc
12. A Quantitative Assessment of the Changes in the Molecular Phenotype of the Rat Tail Intervertebral Disc Induced by Stereotactic, Image-Guided Needle Puncture
13. 117 - Notochordal Cell-Based Therapeutics Can Regenerate the Degenerative Disc
14. A Quantitative Proteomic Analysis of the Non-Chondrodystrophic and Chondrodystrophic Canine Nucleus Pulposus
15. Canine Notochordal Cell-Secreted Factors Protect Murine and Human Nucleus Pulposus Cells from Apoptosis by Inhibition of Activated Caspases -9 and -3/7
16. Intervertebral Disc-Derived Stem Cells
17. The Equilibrative Nucleoside Transporter (ENT1) can be phosphorylated at multiple sites by PKC and PKA
Catalog
Books, media, physical & digital resources
Discovery Service for Jio Institute Digital Library
For full access to our library's resources, please sign in.