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2. Natural history of ENPP1 deficiency: Nationwide Turkish Cohort Study of autosomal‐recessive hypophosphataemic rickets type 2.

Author

Dursun, Fatma, Turan, İhsan, Bitkin, Eda Çelebi, Bayramoğlu, Elvan, Çayır, Atilla, Erdeve, Şenay Savaş, Çakır, Esra Deniz Papatya, Çamtosun, Emine, Dilek, Semine Ozdemir, Kırmızıbekmez, Heves, Eser, Metin, Türkyılmaz, Ayberk, and Karagüzel, Gülay

Subjects
ARTERIAL calcification, SHORT stature, NATURAL history, HEARING disorders, RARE diseases
Abstract

Objective: Autosomal‐recessive hypophosphataemic rickets type 2 (ARHR2) is a rare disease that is reported in survivors of generalized arterial calcification of infancy (GACI). Design, Patients and Measurement: The objective of this study was to characterize a multicenter paediatric cohort with ARHR2 due to ectonucleotide pyrophosphatase/phosphodiesterase family member 1 (ENPP1) deficiency and with a diagnosis of GACI or GACI‐related findings. The clinical, biochemical and genetic characteristics of the patients were retrospectively retrieved. Results: We identified 18 patients from 13 families diagnosed with ARHR2. Fifteen of the patients had an ENPP1 variation confirmed with genetic analyses, and three were siblings of one of these patients, who had clinically diagnosed hypophosphataemic rickets (HRs) with the same presentation. From nine centres, 18 patients, of whom 12 (66.7%) were females, were included in the study. The mean age at diagnosis was 4.2 ± 2.2 (1.6–9) years. The most frequently reported clinical findings on admission were limb deformities (66.6%) and short stature (44.4%). At diagnosis, the mean height SD was −2.2 ± 1.3. Five of the patients were diagnosed with GACI in the neonatal period and treated with bisphosphonates. Other patients were initially diagnosed with ARHR2, but after the detection of a biallelic variant in the ENPP1 gene, it was understood that they previously had clinical findings associated with GACI. Three patients had hearing loss, and two had cervical fusion. After the treatment of HRs, one patient developed calcification, and one developed intimal proliferation. Conclusion: ARHR2 represents one manifestation of ENPP1 deficiency that usually manifests later in life than GACI. The history of calcifications or comorbidities that might be associated with GACI will facilitate the diagnosis in patients with ARHR2, and patients receiving calcitriol and phosphate medication should be carefully monitored for signs of calcification or intimal proliferation. [ABSTRACT FROM AUTHOR]

Published
2024
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6. Preservation of C-Peptide Levels in Children with New-Onset Type 1 Diabetes: A Comparison Based on Body Mass Index

Author

CİMBEK, Emine Ayça, ÖZTÜRK, Mehmet Aykut, and KARAGÜZEL, Gülay

Subjects
Health Care Sciences and Services, Sağlık Bilimleri ve Hizmetleri, Tip 1 diyabet,obezite,fazla kilo,C-peptid,çocuk, Type 1 diabetes,Obesity,Overweight,C-peptide,Children
Abstract

Aim: The contemporaneous increase in type 1 diabetes (T1D) and obesity prevalence in children has led to conflicting reports regardingthe effects of weight excess on beta-cell function and clinical manifestation of T1D. We aimed to analyze the association between bodymass index (BMI) and beta-cell function in a large cohort of new-onset T1D youth.Material and Methods: This study included 204 consecutive children with T1D aged 1-18 years. We retrospectively reviewed themedical data. The preservation of C-peptide was defined as a C-peptide level ≥0.6 ng/mL. Comparisons of variables between groupswere made using appropriate statistical procedures.Results: Eighteen percent of children were overweight or obese. Overweight/obesity was associated with significantly higher C-peptidelevels at onset [0.57 (0.05-2.99) vs 0.41 (0.05-2.58) ng/ml, p=0.01]. Preservation of C-peptide levels was observed in 67% of patients.Patients with preserved C-peptide levels were older at onset [10.4 (1.9-16.5) vs 7.5 (1.1-17.3) yr, p, Amaç: Çocuklarda tip 1 diyabet (T1D) ve obezite prevalansındaki eşzamanlı artış, aşırı kilonun beta hücre fonksiyonu ve T1D'nin ortayaçıkışı üzerine etkilerine dair çelişkili raporlara yol açmıştır. Yeni tanı T1D’li çocuklardan oluşan geniş bir kohortta vücut kütle indeksi(VKİ) ile beta hücre fonksiyonu arasındaki ilişkiyi incelemeyi amaçladık.Gereç ve Yöntemler: Bu çalışmaya 1-18 yaş arası T1D tanılı 204 ardışık çocuk dahil edildi. Tıbbi verileri geriye dönük olarak inceledik.Korunmuş C-peptid düzeyi ≥0.6 ng/mL olarak tanımlandı. Değişkenlerin gruplar arası karşılaştırmaları uygun istatistiksel prosedürlerkullanılarak yapıldı.Bulgular: Çocukların %18'i aşırı kilolu veya obezdi. Fazla kilo/obezite, tanı anında önemli ölçüde daha yüksek C-peptid seviyeleri ileilişkiliydi [0.57 (0.05-2.99)'ye karşı 0.41 (0.05-2.58) ng/ml, p=0.01]. Hastaların %67'sinde C-peptid düzeylerinin korunduğu gözlendi.Korunmuş C-peptid düzeyi grubundaki hastalar tanı anında daha ileri yaştaydı [10.4 (1.9-16.5)'e karşı 7.5 (1.1-17.3) yıl, p

Published
2022
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12. Risk Factors and Clinical Characteristics of Metabolic Bone Disease of Prematurity.

Author

Mutlu, Mehmet, Aktürk-Acar, Filiz, Kader, Şebnem, Aslan, Yakup, and Karagüzel, Gülay

Subjects
PREMATURE infant diseases, NEONATAL necrotizing enterocolitis, ANTICONVULSANTS, LENGTH of stay in hospitals, ADRENOCORTICAL hormones, CONFIDENCE intervals, OSTEOPENIA, MECHANICAL ventilators, DEXAMETHASONE, MULTIPLE regression analysis, PATIENTS, RETROSPECTIVE studies, CASE-control method, VERY low birth weight, RISK assessment, ARTIFICIAL respiration, SYMPTOMS, MEDICAL records, CAFFEINE, DESCRIPTIVE statistics, PARENTERAL feeding, ODDS ratio, BONE fractures, SMALL for gestational age, DISEASE risk factors
Abstract

Objective The study aimed to analyze the risk factors and clinical features of metabolic bone disease of prematurity (MBDP) in premature infants compared with infants of similar gestational age and birth weight without MBDP. Study Design This retrospective case–control study was performed by comparing 81 cases of MBDP with 63 controls to identify potential risk factors. Premature infants with a gestational age ≤33 weeks and birth weight <1,500 g were included. Medical records were examined in terms of maternal conditions, potential risk factors, and clinical characteristics. Results Bone fractures and invasive ventilator dependence were the most common clinical features of MBDP. Duration of invasive ventilation and total mechanical ventilation days, necrotizing enterocolitis, corticosteroid use, anticonvulsive drug use, duration of dexamethasone and caffeine use, total parenteral nutrition, and length of hospitalization were significantly higher in neonates with MBDP (p < 0.05). Breastfed neonates and those receiving human milk fortifier had a lower incidence of MBDP than those premature formula or mixed feeding (p < 0.05). Anticonvulsive drug use (odds ratio: 2.935; 95% confidence interval: 1.265–6.810) was identified as a risk factor for MBDP at multiple regression analysis. Conclusion Our results show that anticonvulsive drug use is a significant risk factor for the development of MBDP. If long-term use is not required, anticonvulsive drugs should be stopped as soon as possible. Further studies involving patients with MBDP are required to determine the risk factors and clinical features. Key Points MBDP is a multifactorial disorder. Anticonvulsive drug use is an important risk factor for the development of MBDP. Bone fractures and invasive ventilator dependence are the most common clinical features of MBDP. [ABSTRACT FROM AUTHOR]

Published
2023
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14. COVID-19 pandemisi öncesi ve pandemi döneminde çocuk yoğun bakım ünitelerine yatan diyabetik ketoasidoz hastalarının klinik ve laboratuvar özelliklerinin değerlendirilmesi

Author

YILMAZ, RESUL, SARI, MEHMET YUSUF, AZAPAĞASI, EBRU, KIRAL, EYLEM, DİNLEYİCİ, ENER ÇAĞRI, ŞİMŞEK, ENVER, BİLGİN, MUZAFFER, KIREL, BİRGÜL, HAVAN, MERVE, KESKİN, MEHMET, KARAOĞLAN, MURAT, YILDIRIM, AHMET, KANĞIN, MURAT, KAHVECİ, FEVZİ, ARSLANOĞLU, MEHMET ÖZGÜR, TÜTÜNCÜLER KÖKENLİ, FİLİZ, YAZICI ÖZKAYA, PINAR, TURANLI, EDA, ÖZÇİFÇİ, GÖKÇEN, DURSUN, OĞUZ, ÜLGEN TEKEREK, NAZAN, TALAY, MEHMET NUR, TEKGÜÇ, HAKAN, ALAÇAKIR, NURİ, ÖZTÜRK, MAKBULE NİLÜFER, Girgin İnceköy, Feyza, ÖZDEMİR, SEMİNE, ALBAYRAK, HATİCE, YILDIZDAŞ, RIZA DİNÇER, AĞIN, HASAN, SOYDAN, EKİN, YENER, NAZİK, GÜVEN, AYLA, HANCILI, SUNA, ŞEVKETOĞLU, ESRA, AKÇAY, NİHAL, BOYRAZ, MERVE, DUYU, MUHTEREM, KOÇOĞLU BARLAS, ÜLKEM, KARAGÜZEL, GÜLAY, YEŞİLBAŞ, OSMAN, BİNGÖL, İBRAHİM, ŞIK, SARE GÜNTÜLÜ, UYSAL YAZICI, MUTLU, ÇITAK, AGOP, DURAK, CANSU, AYGÜN, FATİH, BUĞRUL, FUAT, ÖZTÜRK, ZEYNEL ABİDİN, KENDİRLİ, TANIL, YETİMAKMAN, AYŞE FİLİZ, ÜRÜN, TUBA, ALTUĞ, ÜMÜT, KESİCİ, SELMAN, TUFAN, EREN NUR, KAÇMAZ, EBRU, and BOZAN, GÜRKAN

Published
2021

15. Anthropometric findings from birth to adulthood and their relation with karyotpye distribution in Turkish girls with Turner syndrome

Author

Sari, Erkan, Bereket, Abdullah, Yeşilkaya, Ediz, Baş, Firdevs, Bundak, Rüveyde, Aydn, Banu Küçükemre, Darcan, Şükran, Dündar, Bumin, Büyükinan, Muammer, Kara, Cengiz, Adal, Erdal, Aknc, Ayşehan, Atabek, Mehmet Emre, Demirel, Fatma, Çelik, Nurullah, Özkan, Behzat, Özhan, Bayram, Orbak, Zerrin, Ersoy, Betül, Doğan, Murat, Ataş, Ali, Turan, Serap, Gökşen, Damla, Tarm, Ömer, Yüksel, Bilgin, Ercan, Oya, Hatun, Şükrü, Şimşek, Enver, Ökten, Ayşenur, Abac, Ayhan, Döneray, Hakan, Özbek, Mehmet Nuri, Keskin, Mehmet, Önal, Hasan, Akyürek, Nesibe, Bulan, Kezban, Tepe, Derya, Emeksiz, Hamdi Cihan, Demir, Korcan, Kzlay, Deniz, Topaloğlu, Ali Kemal, Eren, Erdal, Özen, Samim, Demirbilek, Hüseyin, Abal, Saygn, Akn, Leyla, Eklioğlu, Beray Selver, Kaba, Sultan, Ank, Ahmet, Baş, Serpil, Unuvar, Tolga, Sağlam, Halil, Bolu, Semih, Özgen, Tolga, Doğan, Durmuş, Çakr, Esra Deniz, Şen, Yaşar, Andran, Nesibe, Çizmecioğlu, Filiz, Evliyaoğlu, Olcay, Karagüzel, Gülay, Pirgon, Özgür, Çatl, Gönül, Can, Hatice Dilek, Gürbüz, Fatih, Binay, Çiğdem, Baş, Veysel Nijat, Fidanc, Kürşat, Gül, Davut, Polat, Adem, Ackel, Cengizhan, Cinaz, Peyami, and Darendeliler, Feyza

Published
2016
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17. Molecular Diagnosis of Monogenic Diabetes and Clinical/Laboratory Features in Turkish Children

Author

Gökşen, Damla, primary, Yeşilkaya, Ediz, additional, Özen, Samim, additional, Kor, Yılmaz, additional, Eren, Erdal, additional, Korkmaz, Özlem, additional, Berberoğlu, Merih, additional, Karagüzel, Gülay, additional, Er, Eren, additional, Abacı, Ayhan, additional, Evliyaoğlu, Olcay, additional, Akbaş, Emine Demet, additional, Ünal, Edip, additional, Bolu, Semih, additional, Nalbantoğlu, Özlem, additional, Anık, Ahmet, additional, Tayfun, Meltem, additional, Büyükinan, Muammer, additional, Abalı, Saygın, additional, Can Yılmaz, Gülay, additional, Kör, Deniz, additional, Söbü, Elif, additional, Şıklar, Zeynep, additional, Polat, Recep, additional, and Darcan, Şükran, additional

Published
2021
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28. Neonatal Screening for Congenital Adrenal Hyperplasia in Turkey: Outcomes of Extended Pilot Study in 241,083 Infants

Author

Güran, Tülay, primary, Tezel, Başak, additional, Çakır, Meltem, additional, Akıncı, Ayşehan, additional, Orbak, Zerrin, additional, Keskin, Mehmet, additional, Selver Eklioğlu, Beray, additional, Ozon, Alev, additional, Özbek, Mehmet Nuri, additional, Karagüzel, Gülay, additional, Hatipoğlu, Nihal, additional, Gürbüz, Fatih, additional, Çizmecioğlu, Filiz Mine, additional, Kara, Cengiz, additional, Şimşek, Enver, additional, Baş, Firdevs, additional, Aydın, Murat, additional, and Darendeliler, Feyza, additional

Published
2020
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30. Amitriptyline Intoxication in Children

Author

Kamasak, Tülay, primary, Kader, Şebnem, additional, Mutlu, Mehmet, additional, Özkaya, Kagan, additional, Erduran, Erol, additional, Orhan, Fazıl, additional, Dilber, Embiya, additional, Kalyoncu, Mukaddes, additional, Karagüzel, Gülay, additional, Cakır, Murat, additional, Bahat, Elif, additional, and Cansu, Ali, additional

Published
2020
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33. Reference intervals for serum 17α-hydroxyprogesterone and ultrasonographic adrenal gland sizes in healthy newborns.

Author

Karagüzel, Gülay, Eyüboğlu, İlker, Özdem, Sebahat, Kader, Şebnem, Kaya, Serpil, and Beyhun, Nazim Ercument

Subjects
*ADRENAL glands, *ADRENAL diseases, *SERUM, *HYDROXYPROGESTERONE, *AGE groups, *CONGENITAL disorders
Abstract

Background and objectives: Serum 17α- hydroxyprogesterone (17OHP) and bilateral adrenal sizes are pivotal for clinical practice in both diagnosis and treatment of congenital adrenal disorders during the first month of life. Our aims were to determine the reference ranges for serum 17OHP and bilateral adrenal gland sizes according to sex and age groups in healthy term newborns. Materials and methods: A total of 156 healthy newborns, aged 4–7 days (Group 1) or 26–30 days old (Group 2) were included in the study. Serum 17OHP concentration was measured in the morning by radioimmunoassay. The right and left adrenal glands' width, length, and depth were measured with ultrasonography by the same radiologist and the volumes were calculated. Results: The clinical characteristics and serum 17OHP concentrations were similar in male and female newborns. Percentiles for serum 17OHP concentration and the volume of adrenal glands according to age groups and sexes were obtained. Mean 17OHP concentration was 4.67 ± 2.6 ng/ml and 4.49 ± 2.7 ng/ml at the first and fourth week of life, respectively (p >.05). There was a significant decrease in adrenal sizes during the fourth week of life. There was no significant correlation between serum 17OHP concentration and adrenal gland sizes. Conclusions: We have determined reference intervals for serum 17OHP concentration and bilateral adrenal gland sizes for healthy newborns. Although serum concentrations of 17OHP did not change significantly through the first month of life, our reference intervals for serum 17OHP concentration and adrenal sizes may improve clinical approach toward newborns who are suspected of adrenal disorder. We conclude that our reference intervals can guide for congenital adrenal screening regarding serum 17OHP concentration besides diagnosis of adrenal hypoplasia or hyperplasia with ultrasonographic adrenal gland sizes. [ABSTRACT FROM AUTHOR]

Published
2020
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36. Assessment of Anthropometric and Metabolic Follow-Up Characteristics of Patients with Type 1 Diabetes Mellitus

Author

KOCABAŞ, Abdullah, KOCABAŞ, Bilge Aldemir, KARAGÜZEL, Gülay, and AKÇURİN, Sema

Subjects
Antropometrik,Metabolik kontrol,Tip 1 diyabetes mellitus, Anthropometric,Metabolic control,Type 1 diabetes mellitus
Abstract

Amaç: Tip 1 diyabetes mellitus (T1DM), pankreatik beta hücrelerinin otoimmün yıkımı sonucunda insülin üretimindeki yetersizlik gelişmesi ile karakterize bir hastalıktır. Bu çalışmada, T1DM tanısı ile izlenen hastalarımızın antropometrik ve metabolik izlem parametrelerini değerlendirmeyi amaçladık. Gereç ve Yöntemler: Tip 1 DM tanısı ile takip ve tedavi edilen hastalara ait klinik ve laboratuvar verileri hasta kayıtlarından geriye dönük olarak değerlendirildi. Bulgular: Toplam 89 hastanın (45 erkek, 44 kız) verileri incelendi. Hastaların ortalama yaşı 12 ± 3.7 desimal yıl (DY); T1DM tanısı alma yaş ortalamaları ise 7.5 ± 3.9 DY (0.3-16.2) olarak hesaplandı. Cinsiyet dağılımı eşit olan hastaların ortalama izlem süresi 4.5 ± 3.4 yıl (0.1-16.2) bulundu. Olguların başvurudaki boy standart sapma ortalamaları (HSDS) -0.07± 1 iken final HSDS ortalamaları -0.37 ± 1 bulundu (p = 0.002). İzlemde 46 hastada (%51.6) remisyon gözlendi. Hastaların 12’si (%13.5) iyi metabolik kontrol, 32’si (%35.9) orta metabolik kontrol grubunda, 45 hasta (%50.6) kötü metabolik kontrol sınıfında yer alıyordu. Obezitenin insülin ihtiyacında artışa etkisini değerlendirmek amacıyla yapılan incelemede hastaların vücut kitle indeksleri ve ortalama insülin dozları arasında hafif derecede pozitif korelasyon saptandı (r=0.20, P 0.05). İyi-orta metabolik kontrollü ve kötü metabolik kontrollü hastalar arasında yaş, tanı yaşı, puberte durumu ve insülin ihtiyacı bakımından farklılık saptanmazken (p > 0.05), kötü metabolik kontrollü hastalarda son HSDS düzeyinin anlamlı derecede düşük olduğu görüldü (ortalama (-0.2 ± 0.9’a karşı -0.8 ± 1.1; p = 0.04). Sonuç: Yeni gelişen tedavi seçenekleri ve rejimlerine rağmen T1DM’li hastaların büyümesinde etkilenme olmaktadır. Olguların büyüme hızı, metabolik kontrol durumu ve vücut kitle indeksleri yakından izlenmelidir., Objective: Type 1 diabetes mellitus (T1DM) is characterized by inability to produce insulin due to the autoimmune destruction of the beta cells in the pancreas. In this study we aimed to evaluate anthropometric and metabolic parameters of patients diagnosed with T1DM.Material and Methods: Clinical and laboratory data of patients with T1DM were analyzed from medical records, retrospectively.Results: A total of 89 patients (45 male, 44 female) were evaluated in this study. The mean age of the patients was 12 ± 3.7 decimal years (DY) and age at diagnosis was 7.5 ± 3.9 DY (0.3-16.2 DY). Gender ratio was equal in patients and the mean follow-up time was 4.5 ± 3.4 years (0.1 – 16.2 years). While the mean height in standard deviation score (HSDS) of the patients at diagnosis was -0.07 ± 1, mean final HSDS was found to be -0.37 ± 1 (p = 0.002). Remission was achieved in 46 (51.6%) patients. 12 patients (13.5%) were classified in ‘good’; 32 patients (35.9%) in ‘moderate’ and 45 (50.6%) patients in ‘bad’ metabolic control groups, but four patients could not be classified in a metabolic control group. There was a slight correlation between body mass indexes and the mean insulin doses of the patients (r=0.20, P0.05) or daily insulin dose requirement (p>0.05) and delta-HSDS. While there was no significant difference in terms of age, age at diagnosis, pubertal stage and insulin requirement between the patients with goodmoderate metabolic control and bad metabolic control, final HSDS was significantly lower in patients with bad metabolic control (-0.2 ± 0.9 vs. -0.8 ± 1.1; p = 0.04). Conclusion: Growth is affected in children with T1DM despite new treatment options and regimens. Thus, growth velocity, metabolic control parameters and body mass index of these patients should be monitored regularly

Published
2013

37. Growth curves for Turkish Girls with Turner Syndrome: Results of the Turkish Turner Syndrome Study Group

Author

Darendeliler, Feyza, primary, Yeşilkaya, Ediz, additional, Bereket, Abdullah, additional, Baş, Firdevs, additional, Bundak, Rüveyde, additional, Sarı, Erkan, additional, Küçükemre Aydın, Banu, additional, Darcan, Şükran, additional, Dündar, Bumin, additional, Büyükinan, Muammer, additional, Kara, Cengiz, additional, Mazıcıoğlu, Mümtaz M., additional, Adal, Erdal, additional, Akıncı, Ayşehan, additional, Atabek, Mehmet Emre, additional, Demirel, Fatma, additional, Çelik, Nurullah, additional, Özkan, Behzat, additional, Özhan, Bayram, additional, Orbak, Zerrin, additional, Ersoy, Betül, additional, Doğan, Murat, additional, Ataş, Ali, additional, Turan, Serap, additional, Gökşen, Damla, additional, Tarım, Ömer, additional, Yüksel, Bilgin, additional, Ercan, Oya, additional, Hatun, Şükrü, additional, Şimşek, Enver, additional, Ökten, Ayşenur, additional, Abacı, Ayhan, additional, Döneray, Hakan, additional, Özbek, Mehmet Nuri, additional, Keskin, Mehmet, additional, Önal, Hasan, additional, Akyürek, Nesibe, additional, Bulan, Kezban, additional, Tepe, Derya, additional, Emeksiz, Hamdi Cihan, additional, Demir, Korcan, additional, Kızılay, Deniz, additional, Topaloğlu, Ali Kemal, additional, Eren, Erdal, additional, Özen, Samim, additional, Demirbilek, Hüseyin, additional, Abalı, Saygın, additional, Akın, Leyla, additional, Eklioğlu, Beray Selver, additional, Kaba, Sultan, additional, Anık, Ahmet, additional, Baş, Serpil, additional, Ünüvar, Tolga, additional, Sağlam, Halil, additional, Bolu, Semih, additional, Özgen, Tolga, additional, Doğan, Durmuş, additional, Çakır, Esra Deniz, additional, Şen, Yaşar, additional, Andıran, Nesibe, additional, Çizmecioğlu, Filiz, additional, Evliyaoğlu, Olcay, additional, Karagüzel, Gülay, additional, Pirgon, Özgür, additional, Çatlı, Gönül, additional, Can, Hatice Dilek, additional, Gürbüz, Fatih, additional, Binay, Çiğdem, additional, Baş, Veysel Nijat, additional, Sağlam, Celal, additional, Gül, Davut, additional, Polat, Adem, additional, Açıkel, Cengizhan, additional, and Cinaz, Peyami, additional

Published
2015
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38. Turner Syndrome and Associated Problems in Turkish Children: A Multicenter Study

Author

Yeşilkaya, Ediz, primary, Bereket, Abdullah, additional, Darendeliler, Feyza, additional, Baş, Firdevs, additional, Poyrazoğlu, Şükran, additional, Aydın, Banu Küçükemre, additional, Darcan, Şükran, additional, Dündar, Bumin, additional, Büyükinan, Muammer, additional, Kara, Cengiz, additional, Sarı, Erkan, additional, Adal, Erdal, additional, Akıncı, Ayşehan, additional, Atabek, Mehmet Emre, additional, Demirel, Fatma, additional, Çelik, Nurullah, additional, Özkan, Behzat, additional, Özhan, Bayram, additional, Orbak, Zerrin, additional, Ersoy, Betül, additional, Doğan, Murat, additional, Ataş, Ali, additional, Turan, Serap, additional, Gökşen, Damla, additional, Tarım, Ömer, additional, Yüksel, Bilgin, additional, Ercan, Oya, additional, Hatun, Şükrü, additional, Şimşek, Enver, additional, Ökten, Ayşenur, additional, Abacı, Ayhan, additional, Döneray, Hakan, additional, Özbek, Mehmet Nuri, additional, Keskin, Mehmet, additional, Önal, Hasan, additional, Akyürek, Nesibe, additional, Bulan, Kezban, additional, Tepe, Derya, additional, Emeksiz, Hamdi Cihan, additional, Demir, Korcan, additional, Kızılay, Deniz, additional, Topaloğlu, Ali Kemal, additional, Eren, Erdal, additional, Özen, Samim, additional, Abalı, Saygın, additional, Akın, Leyla, additional, Eklioğlu, Beray Selver, additional, Kaba, Sultan, additional, Anık, Ahmet, additional, Baş, Serpil, additional, Ünüvar, Tolga, additional, Sağlam, Halil, additional, Bolu, Semih, additional, Özgen, Tolga, additional, Doğan, Durmuş, additional, Çakır, Esra Deniz, additional, Şen, Yaşar, additional, Andıran, Nesibe, additional, Çizmecioğlu, Filiz, additional, Evliyaoğlu, Olcay, additional, Karagüzel, Gülay, additional, Pirgon, Özgür, additional, Çatlı, Gönül, additional, Can, Hatice Dilek, additional, Gürbüz, Fatih, additional, Binay, Çiğdem, additional, Baş, Veysel Nijat, additional, Fidancı, Kürşat, additional, Polat, Adem, additional, Gül, Davut, additional, Açıkel, Cengizhan, additional, Demirbilek, Hüseyin, additional, Cinaz, Peyami, additional, and Bondy, Carolyn, additional

Published
2015
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45. Cleidocranial Dysplasia: A Case Report

Author

Karagüzel, Gülay, primary, Aktürk, Filiz Acar, additional, Okur, Emelgül, additional, Gümele, Halit Reşit, additional, Gedik, Yusuf, additional, and Ökten, Ayşenur, additional

Published
2010
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48. Prevalence of Generalized Joint Hypermobility and Fibromyalgia Syndrome in the Children Population of Trabzon: A Turkish Study.

Author

BARÇAK, Ömer Faruk, KARKUCAK, Murat, ÇAPKIN, Erhan, KARAGÜZEL, Gülay, DİLBER, Beril, and DEDEOĞLU, Süleyman Semih

Abstract

Copyright of Turkish Journal of Physical Medicine & Rehabilitation / Turkiye Fiziksel Tip ve Rehabilitasyon Dergisi is the property of Turkish Society of Physical Medicine & Rehabilitation and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Published
2015
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