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5. Potential therapeutic targets discovery by transcriptome analysis of an in vitro human gastric signet ring carcinoma model

Author

Yamaguchi, Kyoko, Yoshihiro, Tomoyasu, Ariyama, Hiroshi, Ito, Mamoru, Nakano, Michitaka, Semba, Yuichiro, Nogami, Jumpei, Tsuchihashi, Kenji, Yamauchi, Takuji, Ueno, Shohei, Isobe, Taichi, Shindo, Koji, Moriyama, Taiki, Ohuchida, Kenoki, Nakamura, Masafumi, Nagao, Yoshihiro, Ikeda, Tetsuo, Hashizume, Makoto, Konomi, Hiroyuki, Torisu, Takehiro, Kitazono, Takanari, Kanayama, Tomohiro, Tomita, Hiroyuki, Oda, Yoshinao, Kusaba, Hitoshi, Maeda, Takahiro, Akashi, Koichi, and Baba, Eishi

Published
2022
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7. Possibility of Cell Block Specimens from Overnight-Stored Bile for Next-Generation Sequencing of Cholangiocarcinoma.

Author

Okuno, Mitsuru, Kanayama, Tomohiro, Iwata, Keisuke, Tanaka, Takuji, Tomita, Hiroyuki, Iwasa, Yuhei, Shirakami, Yohei, Watanabe, Naoki, Mukai, Tsuyoshi, Tomita, Eiichi, and Shimizu, Masahito

Subjects
*NUCLEOTIDE sequencing, *CHOLANGIOCARCINOMA, *GENETIC variation, *POSSIBILITY
Abstract

The identification of anticancer therapies using next-generation sequencing (NGS) is necessary for the treatment of cholangiocarcinoma. NGS can be easily performed when cell blocks (CB) are obtained from bile stored overnight. We compared NGS results of paired CB and surgically resected specimens (SRS) from the same cholangiocarcinoma cases. Of the prospectively collected 64 bile CBs from 2018 to 2023, NGS was performed for three cases of cholangiocarcinoma that could be compared with the SRS results. The median numbers of DNA and RNA reads were 95,077,806 [CB] vs. 93,161,788 [SRS] and 22,101,328 [CB] vs. 24,806,180 [SRS], respectively. We evaluated 588 genes and found that almost all genetic alterations were attributed to single-nucleotide variants, insertions/deletions, and multi-nucleotide variants. The coverage rate of variants in SRS by those found in CB was 97.9–99.2%, and the coverage rate of SRS genes by CB genes was 99.6–99.7%. The NGS results of CB fully covered the variants and genetic alterations observed in paired SRS samples. As bile CB is easy to prepare in general hospitals, our results suggest the potential use of bile CB as a novel method for NGS-based evaluation of cholangiocarcinoma. [ABSTRACT FROM AUTHOR]

Published
2024
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9. Time-course analysis of liver and serum galectin-3 in acute liver injury after alpha-galactosylceramide injection

Author

Matsuo, Mikiko, primary, Kanbe, Ayumu, additional, Noguchi, Kei, additional, Niwa, Ayumi, additional, Imaizumi, Yuko, additional, Kuroda, Takahito, additional, Ichihashi, Koki, additional, Okubo, Takafumi, additional, Mori, Kosuke, additional, Kanayama, Tomohiro, additional, Tomita, Hiroyuki, additional, and Hara, Akira, additional

Published
2024
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13. List of Contributors

Author

Aoki, Shigehisa, primary, Chen, Qiwen, additional, Chikada, Hiromi, additional, Ding, Jin, additional, Ge, Jian-Yun, additional, Guo, Ning-Ning, additional, Hara, Akira, additional, Hosaka, Kazunori, additional, Huang, Pengyu, additional, Isoda, Hiroko, additional, Kamiya, Akihide, additional, Kanayama, Tomohiro, additional, Katsuda, Takeshi, additional, Koike, Naoto, additional, Kojima, Nobuhiko, additional, Kurokawa, Tomohiro, additional, Li, Bin, additional, Li, Yu-Mei, additional, Liu, Li-Ping, additional, Matsuda, Michitaka, additional, Mihara, Hirotaka, additional, Mitaka, Toshihiro, additional, Miyamura, Norio, additional, Nishikawa, Yuji, additional, Nishina, Hiroshi, additional, Niwa, Ayumi, additional, Noguchi, Kei, additional, Ochiya, Takahiro, additional, Ohkohchi, Nobuhiro, additional, Su, Yulong, additional, Tanaka, Minoru, additional, Tanaka, Takuji, additional, Taniguchi, Hideki, additional, Tanimizu, Naoki, additional, Tao, Fumiya, additional, Terai, Shuji, additional, Tiyaboonchai, Amita, additional, Tomita, Hiroyuki, additional, Wang, Lu-Yuan, additional, Xie, Wei-Fen, additional, Xu, Yan, additional, Zhang, Qi, additional, and Zheng, Yun-Wen, additional

Published
2018
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14. Conditional ablation of heparan sulfate expression in stromal fibroblasts promotes tumor growth in vivo

Author

Niwa, Ayumi, primary, Taniguchi, Toshiaki, additional, Tomita, Hiroyuki, additional, Okada, Hideshi, additional, Kinoshita, Takamasa, additional, Mizutani, Chika, additional, Matsuo, Mikiko, additional, Imaizumi, Yuko, additional, Kuroda, Takahito, additional, Ichihashi, Koki, additional, Sugiyama, Takaaki, additional, Kanayama, Tomohiro, additional, Yamaguchi, Yu, additional, Sugie, Shigeyuki, additional, Matsuhashi, Nobuhisa, additional, and Hara, Akira, additional

Published
2023
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16. Assessment of type I interferon signatures in undifferentiated inflammatory diseases: A Japanese multicenter experience

Author

Miyamoto, Takayuki, primary, Honda, Yoshitaka, additional, Izawa, Kazushi, additional, Kanazawa, Nobuo, additional, Kadowaki, Saori, additional, Ohnishi, Hidenori, additional, Fujimoto, Masakazu, additional, Kambe, Naotomo, additional, Kase, Naoya, additional, Shiba, Takeshi, additional, Nakagishi, Yasuo, additional, Akizuki, Shuji, additional, Murakami, Kosaku, additional, Bamba, Masahiro, additional, Nishida, Yutaka, additional, Inui, Ayano, additional, Fujisawa, Tomoo, additional, Nishida, Daisuke, additional, Iwata, Naomi, additional, Otsubo, Yoshikazu, additional, Ishimori, Shingo, additional, Nishikori, Momoko, additional, Tanizawa, Kiminobu, additional, Nakamura, Tomoyuki, additional, Ueda, Takeshi, additional, Ohwada, Yoko, additional, Tsuyusaki, Yu, additional, Shimizu, Masaki, additional, Ebato, Takasuke, additional, Iwao, Kousho, additional, Kubo, Akiharu, additional, Kawai, Toshinao, additional, Matsubayashi, Tadashi, additional, Miyazaki, Tatsuhiko, additional, Kanayama, Tomohiro, additional, Nishitani-Isa, Masahiko, additional, Nihira, Hiroshi, additional, Abe, Junya, additional, Tanaka, Takayuki, additional, Hiejima, Eitaro, additional, Okada, Satoshi, additional, Ohara, Osamu, additional, Saito, Megumu K., additional, Takita, Junko, additional, Nishikomori, Ryuta, additional, and Yasumi, Takahiro, additional

Published
2022
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18. 糖質コルチコイドとシクロホスファミドによる寛解導入治療によって完全房室ブロックが改善した顕微鏡的多発血管炎の1例

Author

Tateyama, Shodai, primary, Kato, Ayaka, additional, Fuwa, Masayuki, additional, Asano, Motochika, additional, Taguchi, Koichiro, additional, Mori, Ichiro, additional, Ikeda, Takahide, additional, Kanayama, Tomohiro, additional, Miyazaki, Tatsuhiko, additional, and Morita, Hiroyuki, additional

Published
2022
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19. Metastatic Voyage of Ovarian Cancer Cells in Ascites with the Assistance of Various Cellular Components

Author

Uno, Kaname, primary, Iyoshi, Shohei, additional, Yoshihara, Masato, additional, Kitami, Kazuhisa, additional, Mogi, Kazumasa, additional, Fujimoto, Hiroki, additional, Sugiyama, Mai, additional, Koya, Yoshihiro, additional, Yamakita, Yoshihiko, additional, Nawa, Akihiro, additional, Kanayama, Tomohiro, additional, Tomita, Hiroyuki, additional, Enomoto, Atsushi, additional, and Kajiyama, Hiroaki, additional

Published
2022
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21. Neurosarcoidosis pathologically diagnosed via biopsy of a normal-sized inguinal lymph node with fluorodeoxyglucose accumulation on positron emission tomography/computed tomography in a patient with a history of brain Ewing’s sarcoma

Author

Fuwa, Masayuki, primary, Tateyama, Shodai, additional, Kato, Ayaka, additional, Asano, Motochika, additional, Taguchi, Koichiro, additional, Mori, Ichiro, additional, Ikeda, Takahide, additional, Kanayama, Tomohiro, additional, Shizui, Natsuko, additional, and Morita, Hiroyuki, additional

Published
2021
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22. Postmortem Diagnosis of Pulmonary Tumor Thrombotic Microangiopathy with Rapid Exacerbation in a Patient with Gastric Cancer: A Case Report

Author

Kamidani, Ryo, primary, Kumada, Keisuke, additional, Okada, Hideshi, additional, Yoshimura, Genki, additional, Kanayama, Tomohiro, additional, Tomita, Hiroyuki, additional, Miura, Tomotaka, additional, Oiwa, Hideaki, additional, Mizuno, Yosuke, additional, Kitagawa, Yuichiro, additional, Yasuda, Ryu, additional, Fukuta, Tetsuya, additional, Miyake, Takahito, additional, Doi, Tomoaki, additional, Yoshida, Takahiro, additional, Yoshida, Shozo, additional, Hara, Akira, additional, and Ogura, Shinji, additional

Published
2021
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23. ALDH1 and SALL4 Expression in Cell Block Samples from Patients with Lung Adenocarcinoma and Malignant Pleural Effusion

Author

Kanayama, Tomohiro, primary, Taniguchi, Toshiaki, additional, Tomita, Hiroyuki, additional, Niwa, Ayumi, additional, Noguchi, Kei, additional, Matsuo, Mikiko, additional, Imaizumi, Yuko, additional, Kuroda, Takahiro, additional, Hatano, Yuichiro, additional, Okazaki, Isao, additional, Kato, Tatsuo, additional, and Hara, Akira, additional

Published
2021
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26. Inhibition of FGF10-ERK signal activation suppresses intraductal papillary neoplasm of the bile duct and its associated carcinomas

Author

Tomita, Hiroyuki, primary, Tanaka, Kaori, additional, Hirata, Akihiro, additional, Okada, Hideshi, additional, Imai, Hisashi, additional, Shirakami, Yohei, additional, Ohnishi, Kotaro, additional, Sugie, Shigeyuki, additional, Aoki, Hitomi, additional, Hatano, Yuichiro, additional, Noguchi, Kei, additional, Kanayama, Tomohiro, additional, Niwa, Ayumi, additional, Suzui, Natsuko, additional, Miyazaki, Tatsuhiko, additional, Tanaka, Takuji, additional, Akiyama, Haruhiko, additional, Shimizu, Masahito, additional, Yoshida, Kazuhiro, additional, and Hara, Akira, additional

Published
2021
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27. Glucose transporter Glut1 controls diffuse invasion phenotype with perineuronal satellitosis in diffuse glioma microenvironment

Author

Miyai, Masafumi, primary, Kanayama, Tomohiro, additional, Hyodo, Fuminori, additional, Kinoshita, Takamasa, additional, Ishihara, Takuma, additional, Okada, Hideshi, additional, Suzuki, Hiroki, additional, Takashima, Shigeo, additional, Wu, Zhiliang, additional, Hatano, Yuichiro, additional, Egashira, Yusuke, additional, Enomoto, Yukiko, additional, Nakayama, Noriyuki, additional, Soeda, Akio, additional, Yano, Hirohito, additional, Hirata, Akihiro, additional, Niwa, Masayuki, additional, Sugie, Shigeyuki, additional, Mori, Takashi, additional, Maekawa, Yoichi, additional, Iwama, Toru, additional, Matsuo, Masayuki, additional, Hara, Akira, additional, and Tomita, Hiroyuki, additional

Published
2020
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30. Neurosarcoidosis pathologically diagnosed via biopsy of a normal-sized inguinal lymph node with fluorodeoxyglucose accumulation on positron emission tomography/computed tomography in a patient with a history of brain Ewing's sarcoma.

Author

Fuwa, Masayuki, Tateyama, Shodai, Kato, Ayaka, Asano, Motochika, Taguchi, Koichiro, Mori, Ichiro, Ikeda, Takahide, Kanayama, Tomohiro, Shizui, Natsuko, and Morita, Hiroyuki

Subjects
SARCOIDOSIS, EWING'S sarcoma, POSITRON emission tomography, LYMPH nodes, COMPUTED tomography, DIAGNOSIS
Published
2022
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31. Treatment Strategies Based on Histological Targets against Invasive and Resistant Glioblastoma

Author

Hara, Akira, Kanayama, Tomohiro, Noguchi, Kei, Niwa, Ayumi, Miyai, Masafumi, Kawaguchi, Masaya, Ishida, Kazuhisa, Hatano, Yuichiro, Niwa, Masayuki, and Tomita, Hiroyuki

Subjects
Article Subject, nervous system diseases
Abstract

Glioblastoma (GBM) is the most common and the most malignant primary brain tumor and is characterized by rapid proliferation, invasion into surrounding normal brain tissues, and consequent aberrant vascularization. In these characteristics of GBM, invasive properties are responsible for its recurrence after various therapies. The histomorphological patterns of glioma cell invasion have often been referred to as the “secondary structures of Scherer.” The “secondary structures of Scherer” can be classified mainly into four histological types as (i) perineuronal satellitosis, (ii) perivascular satellitosis, (iii) subpial spread, and (iv) invasion along the white matter tracts. In order to develop therapeutic interventions to mitigate glioma cell migration, it is important to understand the biological mechanism underlying the formation of these secondary structures. The main focus of this review is to examine new molecular pathways based on the histopathological evidence of GBM invasion as major prognostic factors for the high recurrence rate for GBMs. The histopathology-based pharmacological and biological targets for treatment strategies may improve the management of invasive and resistant GBMs.

Published
2019
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35. FGF10/FGFR2/ERK Signal Activation is Required for the Initiation, Maintenance and Progression of Intraductal Papillary Neoplasm of the Bile Duct

Author

Tomita, Hiroyuki, primary, Tanaka, Kaori, additional, Hirata, Akihiro, additional, Okada, Hideshi, additional, Imai, Hisashi, additional, Shirakami, Yohei, additional, Ohnishi, Kotaro, additional, Sugie, Shigeyuki, additional, Aoki, Hitomi, additional, Hatano, Yuichiro, additional, Noguchi, Kei, additional, Kanayama, Tomohiro, additional, Niwa, Ayumi, additional, Suzui, Natsuko, additional, Miyazaki, Tatsuhiko, additional, Tanaka, Takuji, additional, Akiyama, Haruhiko, additional, Shimizu, Masahito, additional, Yoshida, Kazuhiro, additional, and Hara, Akira, additional

Published
2019
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37. Correction: The different pathogenesis of sporadic adenoma and adenocarcinoma in non-ampullary lesions of the proximal and distal duodenum

Author

Niwa, Ayumi, primary, Kuwano, Seiya, additional, Tomita, Hiroyuki, additional, Kimura, Keita, additional, Orihara, Yukiya, additional, Kanayama, Tomohiro, additional, Noguchi, Kei, additional, Hisamatsu, Kenji, additional, Nakashima, Takayuki, additional, Hatano, Yuichiro, additional, Hirata, Akihiro, additional, Miyazaki, Tatsuhiko, additional, Kaneko, Kazuhiro, additional, Tanaka, Takuji, additional, and Hara, Akira, additional

Published
2018
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38. Promotion of cell proliferation by the proto-oncogeneDEKenhances oral squamous cell carcinogenesis through field cancerization

Author

Nakashima, Takayuki, primary, Tomita, Hiroyuki, additional, Hirata, Akihiro, additional, Ishida, Kazuhisa, additional, Hisamatsu, Kenji, additional, Hatano, Yuichiro, additional, Kanayama, Tomohiro, additional, Niwa, Ayumi, additional, Noguchi, Kei, additional, Kato, Keizo, additional, Miyazaki, Tatsuhiko, additional, Tanaka, Takuji, additional, Shibata, Toshiyuki, additional, and Hara, Akira, additional

Published
2017
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39. The different pathogenesis of sporadic adenoma and adenocarcinoma in non-ampullary lesions of the proximal and distal duodenum

Author

Niwa, Ayumi, primary, Kuwano, Seiya, additional, Tomita, Hiroyuki, additional, Kimura, Keita, additional, Orihara, Yukiya, additional, Kanayama, Tomohiro, additional, Noguchi, Kei, additional, Hisamatsu, Kenji, additional, Nakashima, Takayuki, additional, Hatano, Yuichiro, additional, Hirata, Akihiro, additional, Miyazaki, Tatsuhiko, additional, Kaneko, Kazuhiro, additional, Tanaka, Takuji, additional, and Hara, Akira, additional

Published
2017
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40. Distinctive crypt shape in a sessile serrated adenoma/polyp: Distribution of Ki67-, p16INK4a-, WNT5A-positive cells and intraepithelial lymphocytes

Author

Hisamatsu, Kenji, primary, Noguchi, Kei, additional, Tomita, Hiroyuki, additional, Muto, Aoi, additional, Yamada, Natsumi, additional, Kobayashi, Kazuhiro, additional, Hirata, Akihiro, additional, Kanayama, Tomohiro, additional, Niwa, Ayumi, additional, Ishida, Kazuhisa, additional, Nakashima, Takayuki, additional, Hatano, Yuichiro, additional, Suzui, Natsuko, additional, Miyazaki, Tatsuhiko, additional, and Hara, Akira, additional

Published
2017
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41. Specific Deletion of p16INK4awith Retention of p19ARFEnhances the Development of Invasive Oral Squamous Cell Carcinoma

Author

Ishida, Kazuhisa, Tomita, Hiroyuki, Kanayama, Tomohiro, Noguchi, Kei, Niwa, Ayumi, Kawaguchi, Masaya, Miyai, Masafumi, Matsuo, Mikiko, Imaizumi, Yuko, Kato, Keizo, Hatano, Yuichiro, Hirata, Akihiro, Okada, Hideshi, Shibata, Toshiyuki, and Hara, Akira

Abstract

The cyclin-dependent kinase inhibitor 2A (CDKN2A)/alternate reading frame (ARF) locus consists of two overlapping tumor suppressor genes, p16INK4aand p14ARF(p19ARFin mice), encoding two unrelated proteins in alternative reading frames. Previous reports suggest that p16INK4aand p14ARFalterations independently exhibit differential roles, and p16INK4ais more closely associated with a poor prognosis in oral cancer. However, the role of p16INK4a-specific loss in oral squamous cell carcinogenesis remains unclear. The authors assessed chemical carcinogen 4-nitroquinoline 1-oxide (4NQO)-induced multistep oral squamous cell carcinogenesis in mice carrying p16INK4a-specific loss with retention of the p19ARFgene (p16INK4a−/−). 4NQO-treated p16−/−mice exhibited a higher incidence and multiplicity of oral squamous cell carcinoma (OSCC) development relative to 4NQO-treated wild-type mice. 4NQO-treated p16INK4a−/−OSCC cells exhibited higher proliferation and up-regulation of Arf, transcription factor E2f1, tumor protein p63 (tp63), and oncogenic ΔNp63, an isoform p63, compared with observations in 4NQO-treated wild-type OSCC cells. Furthermore, the overexpression of oncogenic ΔNp63 was associated with human OSCC. In conclusion, these results in mice indicate the biological significance of p16INK4a-specific loss with retention of p19ARFin oral squamous cell carcinogenesis, and ΔNp63 may be a potential target for OSCC.

Published
2020
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42. Promotion of cell proliferation by the proto-oncogene DEK enhances oral squamous cell carcinogenesis through field cancerization.

Author

Nakashima, Takayuki, Tomita, Hiroyuki, Hirata, Akihiro, Ishida, Kazuhisa, Hisamatsu, Kenji, Hatano, Yuichiro, Kanayama, Tomohiro, Niwa, Ayumi, Noguchi, Kei, Kato, Keizo, Miyazaki, Tatsuhiko, Tanaka, Takuji, Shibata, Toshiyuki, and Hara, Akira

Subjects
SQUAMOUS cell carcinoma, CELL proliferation, DNA replication, LABORATORY mice, DOXYCYCLINE, PATIENTS
Abstract

Oral squamous cell carcinoma ( OSCC) develops through a multistep carcinogenic process involving field cancerization. The DEK gene is a proto-oncogene with functions in genetic and epigenetic modifications, and has oncogenic functions, including cellular proliferation, differentiation, and senescence. DEK overexpression is associated with malignancies; however, the functional roles of DEK overexpression are unclear. We demonstrated that DEK-expressing cells were significantly increased in human dysplasia/carcinoma in situ and OSCC. Furthermore, we generated ubiquitous and squamous cell-specific doxycycline ( DOX)-inducible Dek mice ( iDek and iDek-e mice respectively). Both DOX+ iDek and iDek-e mice did not show differences in the oral mucosa compared with DOX- mice. In the environment exposed to carcinogen, DOX-treated ( DOX+) iDek mice showed field cancerization and OSCC development. Microarray analysis revealed that DEK overexpression was mediated by the upregulation of DNA replication- and cell cycle-related genes, particularly those related to the G1/ S transition. Tongue tumors overexpressing DEK showed increased proliferating cell nuclear antigen and elongator complex protein 3 expression. Our data suggest that DEK overexpression enhanced carcinogenesis, including field cancerization, in OSCC by stimulating the G1/ S phase transition and promoting DNA replication, providing important insights into the potential applications of DEK as a target in the treatment and prevention of OSCC. [ABSTRACT FROM AUTHOR]

Published
2017
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43. In Situ Hybridization of Brain Slices.

Author

Kanayama T, Tomita H, and Hara A

Subjects
Humans, Brain, In Situ Hybridization, RNA, Messenger genetics, Brain Neoplasms, Neurosciences
Abstract

In situ hybridization (ISH) is an important technique for identifying gene expression at the cellular level in various organs, including brain slices. This approach hybridizes nucleic acid probes to cellular mRNA, allowing the detection of transcriptional products. Recent advances have enabled RNA preservation in formalin-fixed paraffin-embedded (FFPE) samples, making ISH applicable to brain tumor diagnosis and research. Here, we provide a concise overview of the standard application of chromogenic ISH in neuroscience research and neuropathology practice using FFPE blocks of brain slice sections., (© 2024. The Author(s), under exclusive license to Springer Science+Business Media, LLC, part of Springer Nature.)

Published
2024
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44. Immunohistochemistry of Brain Tissues.

Author

Hara A, Taniguchi T, Kanayama T, and Tomita H

Subjects
Immunohistochemistry, Coloring Agents, Fluorescein, Antibodies, Brain
Abstract

Immunohistochemistry (IHC) is the basis of histological or pathological analysis and is widely used to enable the detection and characterization of proteins in various organ tissues, including brain tissues. IHC is commonly performed on formalin-fixed paraffin-embedded (FFPE) tissues because of their easy storage and versatility. IHC is a key method for providing more accurate analysis of localization and function of neurons, neuroendocrine cells, and neural stem cells in the brain and other nervous systems. The related cells such as glial cells and neurovascular units have also been analyzed by IHC. Visualization of antibody-antigen interactions can be performed primarily in one of the following ways: chromogenically stained IHC and fluorescently stained IHC. In chromogenically stained IHC, an antibody is chemically conjugated to an enzyme, such as peroxidase, that can be reacted with a suitable substrate to give a colored product. In fluorescently stained IHC, the antibodies are finally tagged with fluorescent chemicals such as fluorescein isothiocyanate (FITC) or rhodamine. Here, we describe the standard methods of IHC applied to brain slice sections. Furthermore, an automated immunostainer is presented as another option for standardized immunohistochemistry., (© 2024. The Author(s), under exclusive license to Springer Science+Business Media, LLC, part of Springer Nature.)

Published
2024
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