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3. Gut butyrate-producers confer post-infarction cardiac protection

Author

Chen, Hung-Chih, Liu, Yen-Wen, Chang, Kuan-Cheng, Wu, Yen-Wen, Chen, Yi-Ming, Chao, Yu-Kai, You, Min-Yi, Lundy, David J., Lin, Chen-Ju, Hsieh, Marvin L., Cheng, Yu-Che, Prajnamitra, Ray P., Lin, Po-Ju, Ruan, Shu-Chian, Chen, David Hsin-Kuang, Shih, Edward S. C., Chen, Ke-Wei, Chang, Shih-Sheng, Chang, Cindy M. C., Puntney, Riley, Moy, Amy Wu, Cheng, Yuan-Yuan, Chien, Hsin-Yuan, Lee, Jia-Jung, Wu, Deng-Chyang, Hwang, Ming-Jing, Coonen, Jennifer, Hacker, Timothy A., Yen, C-L. Eric, Rey, Federico E., Kamp, Timothy J., and Hsieh, Patrick C. H.

Published
2023
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5. Caveolin-3 and Caveolae regulate ventricular repolarization

Author

Markandeya, Yogananda S., Gregorich, Zachery R., Feng, Li, Ramchandran, Vignesh, O' Hara, Thomas, Vaidyanathan, Ravi, Mansfield, Catherine, Keefe, Alexis M., Beglinger, Carl J., Best, Jabe M., Kalscheur, Matthew M., Lea, Martin R., Hacker, Timothy A., Gorelik, Julia, Trayanova, Natalia A., Eckhardt, Lee L., Makielski, Jonathan C., Balijepalli, Ravi C., and Kamp, Timothy J.

Published
2023
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7. Combined Treatment of Human Induced Pluripotent Stem Cell–Derived Cardiomyocytes and Endothelial Cells Regenerate the Infarcted Heart in Mice and Non-Human Primates

Author

Cheng, Yu-Che, Hsieh, Marvin L., Lin, Chen-Ju, Chang, Cindy M.C., Huang, Ching-Ying, Puntney, Riley, Wu Moy, Amy, Ting, Chien-Yu, Herr Chan, Darien Zhing, Nicholson, Martin W., Lin, Po-Ju, Chen, Hung-Chih, Kim, Gina C., Zhang, Jianhua, Coonen, Jennifer, Basu, Puja, Simmons, Heather A., Liu, Yen-Wen, Hacker, Timothy A., Kamp, Timothy J., and Hsieh, Patrick C.H.

Published
2023
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8. Diminished Immune Cell Adhesion in Hypoimmune ICAM-1 Knockout Pluripotent Stem Cells

Author

Saha, Sayandeep, primary, Haynes, W. John, additional, Del Rio, Natalia, additional, Young, Elizabeth E, additional, Zhang, Jue, additional, Seo, Jiwon, additional, Huang, Liupei, additional, Holm, Alexis M, additional, Blashka, Wesley, additional, Murphy, Lydia, additional, Scholz, Merrick, additional, Henrichs, Abigale, additional, Suresh Babu, Jayalaxmi, additional, Steill, John, additional, Stewart, Ron E, additional, Kamp, Timothy J, additional, and Brown, Matthew E, additional

Published
2024
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9. Convergences of Life Sciences and Engineering in Understanding and Treating Heart Failure.

Author

Berry, Joel L, Zhu, Wuqiang, Tang, Yao Liang, Krishnamurthy, Prasanna, Ge, Ying, Cooke, John P, Chen, Yabing, Garry, Daniel J, Yang, Huang-Tian, Rajasekaran, Namakkal Soorapan, Koch, Walter J, Li, Song, Domae, Keitaro, Qin, Gangjian, Cheng, Ke, Kamp, Timothy J, Ye, Lei, Hu, Shijun, Ogle, Brenda M, Rogers, Jack M, Abel, E Dale, Davis, Michael E, Prabhu, Sumanth D, Liao, Ronglih, Pu, William T, Wang, Yibin, Ping, Peipei, Bursac, Nenad, Vunjak-Novakovic, Gordana, Wu, Joseph C, Bolli, Roberto, Menasché, Philippe, and Zhang, Jianyi

Subjects
Myocardium, Heart, Animals, Humans, Interdisciplinary Communication, Cooperative Behavior, Biomedical Engineering, Recovery of Function, Regeneration, Biomedical Research, Diffusion of Innovation, Heart Failure, Biological Science Disciplines, bioengineering, heart failure, myocardium, stem cells, tissue engineering, Cardiovascular, Heart Disease, Regenerative Medicine, Stem Cell Research, Stem Cell Research - Nonembryonic - Human, Bioengineering, Cardiorespiratory Medicine and Haematology, Clinical Sciences, Cardiovascular System & Hematology
Abstract

On March 1 and 2, 2018, the National Institutes of Health 2018 Progenitor Cell Translational Consortium, Cardiovascular Bioengineering Symposium, was held at the University of Alabama at Birmingham. Convergence of life sciences and engineering to advance the understanding and treatment of heart failure was the theme of the meeting. Over 150 attendees were present, and >40 scientists presented their latest work on engineering human functional myocardium for disease modeling, drug development, and heart failure research. The scientists, engineers, and physicians in the field of cardiovascular sciences met and discussed the most recent advances in their work and proposed future strategies for overcoming the major roadblocks of cardiovascular bioengineering and therapy. Particular emphasis was given for manipulation and using of stem/progenitor cells, biomaterials, and methods to provide molecular, chemical, and mechanical cues to cells to influence their identity and fate in vitro and in vivo. Collectively, these works are profoundly impacting and progressing toward deciphering the mechanisms and developing novel treatments for left ventricular dysfunction of failing hearts. Here, we present some important perspectives that emerged from this meeting.

Published
2019

10. Neutral sphingomyelinase regulates mechanotransduction in human engineered cardiac tissues and mouse hearts.

Author

Turner, Daniel G. P., De Lange, Willem J., Zhu, Yanlong, Coe, Christopher L., Simcox, Judith, Ge, Ying, Kamp, Timothy J., Ralphe, J. Carter, and Glukhov, Alexey V.

Subjects
SPHINGOMYELINASE, HYPERTENSION, PLURIPOTENT stem cells, CAVEOLAE, CERAMIDES
Abstract

Cardiovascular disease is the leading cause of death in the USA and is known to be exacerbated by elevated mechanical stress from hypertension. Caveolae are plasma membrane structures that buffer mechanical stress but have been found to be reduced in pathological conditions associated with chronically stretched myocardium. To explore the physiological implications of the loss of caveolae, we used human engineered cardiac tissue (ECT) constructs, composed of human induced pluripotent stem cell (hiPSC)‐derived cardiomyocytes and hiPSC‐derived cardiac fibroblasts, to develop a long‐term cyclic stretch protocol that recapitulates the effects of hypertension on caveolae expression, membrane tension, and the β‐adrenergic response. Leveraging this new stretch protocol, we identified neutral sphingomyelinases (nSMase) as mechanoregulated mediators of caveolae loss, ceramide production and the blunted β‐adrenergic response in this human cardiac model. Specifically, in our ECT model, nSMase inhibition via GW4869 prevented stretch‐induced loss of caveolae‐like structures, mitigated nSMase‐dependent ceramide production, and maintained the ECT contractile kinetic response to isoprenaline. These findings are correlated with a blood lipidomic analysis in middle‐aged and older adults, which revealed an increase of the circulating levels of ceramides in adults with hypertension. Furthermore, we found that conduction slowing from increased pressure loading in mouse left ventricle was abolished in the context of nSMase inhibition. Collectively, these findings identify nSMase as a potent drug target for mitigating stretch‐induced effects on cardiac function. Key points: We have developed a new stretch protocol for human engineered cardiac tissue that recapitulates changes in plasma membrane morphology observed in animal models of pressure/volume overload.Stretch of engineered cardiac tissue induces activation of neutral sphingomyelinase (nSMase), generation of ceramide, and disassembly of caveolae.Activation of nSMase blunts cardiac β‐adrenergic contractile kinetics and mediates stretch‐induced slowing of conduction and upstroke velocity.Circulating ceramides are increased in adults with hypertension, highlighting the clinical relevance of stretch‐induced nSMase activity. [ABSTRACT FROM AUTHOR]

Published
2024
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13. Distinct hypertrophic cardiomyopathy genotypes result in convergent sarcomeric proteoform profiles revealed by top-down proteomics

Author

Tucholski, Trisha, Cai, Wenxuan, Gregorich, Zachery R., Bayne, Elizabeth F., Mitchell, Stanford D., McIlwain, Sean J., de Lange, Willem J., Wrobbel, Max, Karp, Hannah, Hite, Zachary, Vikhorev, Petr G., Marston, Steven B., Lal, Sean, Li, Amy, dos Remedios, Cristobal, Kohmoto, Takushi, Hermsen, Joshua, Ralphe, J. Carter, Kamp, Timothy J., Moss, Richard L., and Ge, Ying

Published
2020

19. Human iPSC-derived Committed Cardiac Progenitors Generate Cardiac Tissue Grafts in a Swine Ischemic Cardiomyopathy Model without Triggering Ventricular Arrhythmias

Author

Raval, Amish N., primary, Schmuck, Eric G., additional, Roy, Sushmita, additional, Saito, Yukihiro, additional, Zhou, Tianhua, additional, Conklin, James, additional, Hacker, Timothy A., additional, Koonce, Chad, additional, Boyer, Meghan, additional, Stack, Kristin, additional, Hebron, Ellen, additional, Nagle, Scott K., additional, Hsieh, Patrick C.H., additional, and Kamp, Timothy J, additional

Published
2024
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23. Cardiac Repair in a Porcine Model of Acute Myocardial Infarction with Human Induced Pluripotent Stem Cell-Derived Cardiovascular Cells

Author

Ye, Lei, Chang, Ying-Hua, Xiong, Qiang, Zhang, Pengyuan, Zhang, Liying, Somasundaram, Porur, Lepley, Mike, Swingen, Cory, Su, Liping, Wendel, Jacqueline S, Guo, Jing, Jang, Albert, Rosenbush, Daniel, Greder, Lucas, Dutton, James R, Zhang, Jianhua, Kamp, Timothy J, Kaufman, Dan S, Ge, Ying, and Zhang, Jianyi

Subjects
Biological Sciences, Medical and Health Sciences, Developmental Biology
Published
2015

24. Cardiac Repair in a Porcine Model of Acute Myocardial Infarction with Human Induced Pluripotent Stem Cell-Derived Cardiovascular Cells

Author

Ye, Lei, Chang, Ying-Hua, Xiong, Qiang, Zhang, Pengyuan, Zhang, Liying, Somasundaram, Porur, Lepley, Mike, Swingen, Cory, Su, Liping, Wendel, Jacqueline S, Guo, Jing, Jang, Albert, Rosenbush, Daniel, Greder, Lucas, Dutton, James R, Zhang, Jianhua, Kamp, Timothy J, Kaufman, Dan S, Ge, Ying, and Zhang, Jianyi

Subjects
Heart Disease - Coronary Heart Disease, Stem Cell Research - Induced Pluripotent Stem Cell - Human, Heart Disease, Stem Cell Research, Transplantation, Cardiovascular, Stem Cell Research - Induced Pluripotent Stem Cell, Regenerative Medicine, Aetiology, Development of treatments and therapeutic interventions, 5.2 Cellular and gene therapies, 2.1 Biological and endogenous factors, Acute Disease, Animals, Apoptosis, Cell Differentiation, Cell Lineage, Cells, Cultured, Disease Models, Animal, Endothelial Cells, Fibrin, Heart Ventricles, Humans, Induced Pluripotent Stem Cells, Insulin-Like Growth Factor I, Microspheres, Myocardial Infarction, Myocardium, Myocytes, Cardiac, Myocytes, Smooth Muscle, Recovery of Function, Stem Cell Transplantation, Swine, Biological Sciences, Medical and Health Sciences, Developmental Biology
Abstract

Human induced pluripotent stem cells (hiPSCs) hold promise for myocardial repair following injury, but preclinical studies in large animal models are required to determine optimal cell preparation and delivery strategies to maximize functional benefits and to evaluate safety. Here, we utilized a porcine model of acute myocardial infarction (MI) to investigate the functional impact of intramyocardial transplantation of hiPSC-derived cardiomyocytes, endothelial cells, and smooth muscle cells, in combination with a 3D fibrin patch loaded with insulin growth factor (IGF)-encapsulated microspheres. hiPSC-derived cardiomyocytes integrated into host myocardium and generated organized sarcomeric structures, and endothelial and smooth muscle cells contributed to host vasculature. Trilineage cell transplantation significantly improved left ventricular function, myocardial metabolism, and arteriole density, while reducing infarct size, ventricular wall stress, and apoptosis without inducing ventricular arrhythmias. These findings in a large animal MI model highlight the potential of utilizing hiPSC-derived cells for cardiac repair.

Published
2014

25. Population-based high-throughput toxicity screen of human iPSC-derived cardiomyocytes and neurons

Author

Huang, Ching Ying, Nicholson, Martin W., Wang, Jyun Yuan, Ting, Chien Yu, Tsai, Ming Heng, Cheng, Yu Che, Liu, Chun Lin, Chan, Darien Z.H., Lee, Yi Chan, Hsu, Ching Chuan, Hsu, Yu Hung, Yang, Chiou Fong, Chang, Cindy M.C., Ruan, Shu Chian, Lin, Po Ju, Lin, Jen Hao, Chen, Li Lun, Hsieh, Marvin L., Cheng, Yuan Yuan, Hsu, Wan Tseng, Lin, Yi Ling, Chen, Chien Hsiun, Hsu, Yu Hsiang, Wu, Ying Ta, Hacker, Timothy A., Wu, Joseph C., Kamp, Timothy J., and Hsieh, Patrick C.H.

Published
2024
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28. Global Proteomic Analysis Reveals Alterations in Differentially Expressed Proteins between Cardiopathic Lamin A/C Mutations

Author

Anderson, Corey L., Brown, Kyle A., North, Ryan J., Walters, Janay K., Kaska, Sara T., Wolff, Mathew R., Kamp, Timothy J., Ge, Ying, and Eckhardt, Lee L.

Abstract

Lamin A/C (LMNA) is an important component of nuclear lamina. Mutations cause arrhythmia, heart failure, and sudden cardiac death. While LMNA-associated cardiomyopathy typically has an aggressive course that responds poorly to conventional heart failure therapies, there is variability in severity and age of penetrance between and even within specific mutations, which is poorly understood at the cellular level. Further, this heterogeneity has not previously been captured to mimic the heterozygous state, nor have the hundreds of clinical LMNA mutations been represented. Herein, we have overexpressed cardiopathic LMNA variants in HEK cells and utilized state-of-the-art quantitative proteomics to compare the global proteomic profiles of (1) aggregating Q353 K alone, (2) Q353 K coexpressed with WT, (3) aggregating N195 K coexpressed with WT, and (4) nonaggregating E317 K coexpressed with WT to help capture some of the heterogeneity between mutations. We analyzed each data set to obtain the differentially expressed proteins (DEPs) and applied gene ontology (GO) and KEGG pathway analyses. We found a range of 162 to 324 DEPs from over 6000 total protein IDs with differences in GO terms, KEGG pathways, and DEPs important in cardiac function, further highlighting the complexity of cardiac laminopathies. Pathways disrupted by LMNA mutations were validated with redox, autophagy, and apoptosis functional assays in both HEK 293 cells and in induced pluripotent stem cell derived cardiomyocytes (iPSC-CMs) for LMNA N195 K. These proteomic profiles expand our repertoire for mutation-specific downstream cellular effects that may become useful as druggable targets for personalized medicine approach for cardiac laminopathies.

Published
2024
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29. Calcium transients closely reflect prolonged action potentials in iPSC models of inherited cardiac arrhythmia.

Author

Spencer, C Ian, Baba, Shiro, Nakamura, Kenta, Hua, Ethan A, Sears, Marie AF, Fu, Chi-cheng, Zhang, Jianhua, Balijepalli, Sadguna, Tomoda, Kiichiro, Hayashi, Yohei, Lizarraga, Paweena, Wojciak, Julianne, Scheinman, Melvin M, Aalto-Setälä, Katriina, Makielski, Jonathan C, January, Craig T, Healy, Kevin E, Kamp, Timothy J, Yamanaka, Shinya, and Conklin, Bruce R

Subjects
Myocytes, Cardiac, Humans, Calcium, Nifedipine, Caffeine, Patch-Clamp Techniques, Cell Differentiation, Action Potentials, Genotype, Phenotype, Polymorphism, Single Nucleotide, Middle Aged, Infant, Newborn, Female, Ether-A-Go-Go Potassium Channels, Arrhythmias, Cardiac, Young Adult, Induced Pluripotent Stem Cells, NAV1.5 Voltage-Gated Sodium Channel, Myocytes, Cardiac, Polymorphism, Single Nucleotide, Infant, Newborn, Arrhythmias, Genetics, Stem Cell Research - Induced Pluripotent Stem Cell - Human, Heart Disease, Stem Cell Research, Stem Cell Research - Induced Pluripotent Stem Cell, Cardiovascular, 2.1 Biological and endogenous factors, Biochemistry and Cell Biology, Clinical Sciences
Abstract

Long-QT syndrome mutations can cause syncope and sudden death by prolonging the cardiac action potential (AP). Ion channels affected by mutations are various, and the influences of cellular calcium cycling on LQTS cardiac events are unknown. To better understand LQTS arrhythmias, we performed current-clamp and intracellular calcium ([Ca(2+)]i) measurements on cardiomyocytes differentiated from patient-derived induced pluripotent stem cells (iPS-CM). In myocytes carrying an LQT2 mutation (HERG-A422T), APs and [Ca(2+)]i transients were prolonged in parallel. APs were abbreviated by nifedipine exposure and further lengthened upon releasing intracellularly stored Ca(2+). Validating this model, control iPS-CM treated with HERG-blocking drugs recapitulated the LQT2 phenotype. In LQT3 iPS-CM, expressing NaV1.5-N406K, APs and [Ca(2+)]i transients were markedly prolonged. AP prolongation was sensitive to tetrodotoxin and to inhibiting Na(+)-Ca(2+) exchange. These results suggest that LQTS mutations act partly on cytosolic Ca(2+) cycling, potentially providing a basis for functionally targeted interventions regardless of the specific mutation site.

Published
2014

30. Lactate and Immunomagnetic-purified hiPSC-derived cardiomyocytes generate comparable engineered cardiac tissue constructs

Author

Rossler, Kalina J., primary, De Lange, Willem J., additional, Mann, Morgan W., additional, Aballo, Timothy J., additional, Melby, Jake A., additional, Zhang, Jianhua, additional, Kim, Gina, additional, Bayne, Elizabeth F., additional, Zhu, Yanlong, additional, Farrell, Emily T., additional, Kamp, Timothy J., additional, Ralphe, J. Carter, additional, and Ge, Ying, additional

Published
2023
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36. Copy number variant hotspots in Han Taiwanese population induced pluripotent stem cell lines - lessons from establishing the Taiwan human disease iPSC Consortium Bank

Author

Huang, Ching-Ying, Li, Ling-Hui, Hsu, Wan-Tseng, Cheng, Yu-Che, Nicholson, Martin W., Liu, Chun-Lin, Ting, Chien-Yu, Ko, Hui-Wen, Syu, Shih-Han, Wen, Cheng-Hao, Yan, Zhuge, Huang, Hsiang-Po, Su, Hong-Lin, Chiang, Po-Min, Shen, Chia-Ning, Chen, Hsin-Fu, Yen, B. Lin Ju, Lu, Huai-En, Hwang, Shiaw-Min, Chiou, Shih-Hwa, Ho, Hong-Nerng, Wu, Jer-Yuarn, Kamp, Timothy J., Wu, Joseph C., and Hsieh, Patrick C. H.

Published
2020
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37. Lactate and Immunomagnetic-purified iPSC-derived Cardiomyocytes Generate Comparable Engineered Cardiac Tissue Constructs

Author

Rossler, Kalina J, primary, de Lange, Willem, additional, Mann, Morgan W, additional, Aballo, Timothy J, additional, Melby, Jake A., additional, Zhang, Jianhua, additional, Kim, Gina, additional, Bayne, Elizabeth F, additional, Zhu, Yanlong, additional, Farrell, Emily T., additional, Kamp, Timothy J, additional, Ralphe, John Carter, additional, and Ge, Ying, additional

Published
2023
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40. Loss of Gut Microbiota Alters Immune System Composition and Cripples Postinfarction Cardiac Repair

Author

Tang, Tony W.H., Chen, Hung-Chih, Chen, Chen-Yun, Yen, Christopher Y.T., Lin, Chen-Ju, Prajnamitra, Ray P., Chen, Li-Lun, Ruan, Shu-Chian, Lin, Jen-Hao, Lin, Po-Ju, Lu, Hsueh-Han, Kuo, Chiung-Wen, Chang, Cindy M., Hall, Alexander D., Vivas, Eugenio I., Shui, Jr-Wen, Chen, Peilin, Hacker, Timothy A., Rey, Federico E., Kamp, Timothy J., and Hsieh, Patrick C.H.

Published
2019
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41. Contributors

Author

Aagaard, Philip, primary, Abrams, Dominic James, additional, Abriel, Hugues, additional, Adkisson, Wayne O., additional, Agullo-Pascual, Esperanza, additional, Alvarado, Francisco J., additional, Amin, Ahmad S., additional, Antzelevitch, Charles, additional, Anumonwo, Justus M.B., additional, Armaganijan, Luciana, additional, Arya, Arash, additional, Asirvatham, Samuel, additional, Atienza, Felipe, additional, Backx, Peter H., additional, Ballou, Lisa M., additional, Balse, Elise, additional, Balulad, Sujata, additional, Barbuti, Andrea, additional, Bardy, Gust H., additional, Bassil, Guillaume, additional, Benditt, David G., additional, Berenfeld, Omer, additional, Bers, Donald M., additional, Binah, Ofer, additional, Bogun, Frank, additional, Bongianino, Rossana, additional, Boyle, Noel G., additional, Boyle, Patrick M., additional, Breithardt, Günter, additional, Brini, Marisa, additional, Brink, Peter R., additional, Brugada, Pedro, additional, Buch, Eric, additional, Bukauskas, Feliksas F., additional, Calkins, Hugh, additional, Callans, David J., additional, Caples, Sean M., additional, Carafoli, Ernesto, additional, Catterall, William A., additional, Cerrone, Marina, additional, Chaumeil, Arnaud, additional, Chen, Caressa, additional, Chen, Lan S., additional, Chen, Peng-Sheng, additional, Cheng, Jianding, additional, Chiamvimonvat, Nipavan, additional, Christini, David J., additional, Chugh, Aman, additional, Climent, Andreu M., additional, Cohen, Ira S., additional, Connolly, Stuart J., additional, Cooper, Lebron, additional, Crespo, Eric M., additional, Crotti, Lia, additional, Csepe, Thomas A., additional, Cuoco, Frank, additional, Curtis, Anne B., additional, Damiano, Ralph J., additional, Darbar, Dawood, additional, Das, Mithilesh K., additional, d’Avila, Andre, additional, Delmar, Mario, additional, Delpón, Eva, additional, Denegri, Marco, additional, Denis, Arnaud, additional, Derval, Nicolas, additional, Deschênes, Isabelle, additional, Deshmukh, Abhishek, additional, Di Biase, Luigi, additional, Dickfeld, Timm M., additional, Dierckx, Hans, additional, Dinov, Borislav, additional, Dixit, Sanjay, additional, Dobrev, Dobromir, additional, Dubois, Remi, additional, Eckardt, Lars, additional, Edwards, Andrew G., additional, Ellenbogen, Kenneth A., additional, Ellinor, Patrick T., additional, Estes, N.A. Mark, additional, Fabritz, Larissa, additional, Fedorov, Vadim V., additional, Fernandez, Antonio B., additional, Teijeira Fernández, Elvis, additional, Filgueiras-Rama, David, additional, Fishbein, Michael C., additional, Fishman, Glenn I., additional, Frankel, David S., additional, Friedman, Paul, additional, Frontera, Antonio, additional, Gami, Apoor S., additional, Garabelli, Paul, additional, George, Alfred L., additional, Gerstenfeld, Edward P., additional, Gizurarson, Sigfus, additional, Gold, Michael R., additional, Goldberger, Jeffrey J., additional, Grace, Andrew, additional, Grassi, Guido, additional, Greenfield, Ruth Ann, additional, Gross, Wendy L., additional, Grubb, Blair P., additional, Guillem, María S., additional, Györke, Sándor, additional, Haïssaguerre, Michel, additional, Hake, Johan, additional, Halperin, Henry R., additional, Hansen, Brian J., additional, Hatem, Stéphane, additional, Hayes, David L., additional, Heijman, Jordi, additional, Herron, Todd J., additional, Hindricks, Gerhard, additional, Hocini, Mélèze, additional, Hohnloser, Stefan H., additional, Holmes, David R., additional, Hoshijima, Masahiko, additional, Hund, Thomas J., additional, Hutchinson, Mathew D., additional, Ilkhanoff, Leonard, additional, Ingles, Jodie, additional, Ip, James E., additional, Jackman, Warren M., additional, Jackson, Nicholas, additional, Jaïs, Pierre, additional, Jalife, José, additional, Jhun, Bong Sook, additional, John, Roy M., additional, Jongbloed, Monique, additional, Jordaens, Luc, additional, Kalman, Jonathan M., additional, Kamp, Timothy J., additional, Kanj, Mohamed H., additional, Kapa, Suraj, additional, Karabin, Beverly, additional, Karakikes, Ioannis, additional, Katritsis, Demosthenes G., additional, Kaur, Kuljeet, additional, Kirchhof, Paulus, additional, Kléber, André G., additional, Klein, George J., additional, Kohl, Peter, additional, Koneru, Jayanthi N., additional, Koruth, Jacob S., additional, Krahn, Andrew D., additional, Krogh-Madsen, Trine, additional, Kuck, Karl Heinz, additional, Kumar, Saurabh, additional, Kushnir, Alexander, additional, Lakdawala, Neal K., additional, Laksman, Zachary W.M., additional, Latchamsetty, Rakesh, additional, Lau, Dennis H., additional, Lerman, Bruce B., additional, Lin, Richard Z., additional, Lin, Shien-Fong, additional, Link, Mark S., additional, Liu, Bin, additional, Liu, Christopher F., additional, Lockwood, Deborah J., additional, Lopatin, Anatoli N., additional, Lubitz, Steven A., additional, Mahajan, Rajiv, additional, Makielski, Jonathan C., additional, Malik, Marek, additional, Marchlinski, Francis E., additional, Markowitz, Steven M., additional, Maron, Barry J., additional, Maron, Martin S., additional, Marx, Steven O., additional, Massé, Stéphane, additional, McCulloch, Andrew D., additional, McKelvie-Sebileau, Pippa, additional, Melby, Spencer J., additional, Metzner, Andreas, additional, Michailova, Anushka P., additional, Michaud, Gregory F., additional, Miller, John M., additional, Mishra, Jyotsna, additional, Mitrani, Raul D., additional, Mohler, Peter J., additional, Morady, Fred, additional, Myerburg, Robert J., additional, Nakagawa, Hiroshi, additional, Nalliah, Chrishan Joseph, additional, Nanthakumar, Kumaraswamy, additional, Napolitano, Carlo, additional, Narayan, Sanjiv M., additional, Natale, Andrea, additional, Nattel, Stanley, additional, Nazarian, Saman, additional, Nguyen, Thao P., additional, Nogami, Akihiko, additional, Noujaim, Sami F., additional, Nubret Le Coniat, Karine, additional, Olshansky, Brian, additional, O-Uchi, Jin, additional, Oudit, Gavin Y., additional, Ouyang, Feifan, additional, Ozcan, Cevher, additional, Packer, Douglas L., additional, Pandit, Sandeep V., additional, Panfilov, Alexander V., additional, Park, David S., additional, Patocskai, Bence, additional, Pauza, Dainius H., additional, Pauziene, Neringa, additional, Piccini, Jonathan P., additional, Pitt, Geoffrey S., additional, Po, Sunny S., additional, Prasad, Abhiram, additional, Priori, Silvia G., additional, Radwański, Przemysław B., additional, Rappel, Wouter-Jan, additional, Reiser, Michelle, additional, Restrepo, Alejandro Jimenez, additional, Robinson, Richard B., additional, Roden, Dan M., additional, Rosen, Michael R., additional, Rosso, Raphael, additional, Rudy, Yoram, additional, Rysevaite-Kyguoliene, Kristina, additional, Sabbah, Hani N., additional, Sacher, Frederic, additional, Sachse, Frank B., additional, Saguner, Ardan M., additional, Sanders, Prashanthan, additional, Sanguinetti, Michael C., additional, Santangeli, Pasquale, additional, Sarraf, Mohammad, additional, Satin, Jonathan, additional, Schalij, Martin Jan, additional, Scherlag, Benjamin J., additional, Schill, Matthew R., additional, Schleifer, J. William, additional, Schuessler, Richard B., additional, Schwartz, Peter J., additional, Seeger, Timon, additional, Semsarian, Christopher, additional, Seravalle, Gino, additional, Shah, Ashok J., additional, Shaw, Robin M., additional, Shen, Mark J., additional, Shen, Win–Kuang, additional, Sheu, Shey-Shing, additional, Shivkumar, Kalyanam, additional, Silva, Jennifer N.A., additional, Skanes, Allan C., additional, Soejima, Kyoko, additional, Somers, Virend K., additional, Sorajja, Dan, additional, Stavrakis, Stavros, additional, Steinberg, Christian, additional, Stevenson, Lynne Warner, additional, Stevenson, William G., additional, Sweeney, Michael O., additional, Swerdlow, Charles, additional, Takigawa, Masateru, additional, Tamargo, Juan, additional, Tandri, Harikrishna, additional, Tedrow, Usha B., additional, Thompson, Nathaniel, additional, Thompson, Paul D., additional, Tomaselli, Gordon F., additional, Towbin, Jeffrey A., additional, Trayanova, Natalia A., additional, Tristani-Firouzi, Martin, additional, Tseng, Zian H., additional, Ueda, Akiko, additional, Valdivia, Héctor H., additional, Valiunas, Virginijus, additional, van der Werf, Christian, additional, Van Hare, George F., additional, Vidmar, David, additional, Viskin, Sami, additional, Voigt, Niels, additional, Walsh, Edward P., additional, Wang, Paul J., additional, Wehrens, Xander H.T., additional, Weiss, Mark S., additional, Wilde, Arthur A.M., additional, Wilkoff, Bruce L., additional, Woo, Y. Joseph, additional, Wu, Joseph C., additional, Yee, Raymond, additional, Zaman, Junaid A.B., additional, Zarzoso, Manuel, additional, Zeitler, Emily P., additional, Zeppenfeld, Katja, additional, Zghaib, Tarek, additional, Zhang, Xiao-Dong, additional, and Zipes, Douglas P., additional

Published
2018
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44. Combined Treatment of Human Induced Pluripotent Stem Cell--Derived Cardiomyocytes and Endothelial Cells Regenerate the Infarcted Heart in Mice and Non-Human Primates.

Author

Yu-Che Cheng, Hsieh, Marvin L., Chen-Ju Lin, Chang, Cindy M. C., Ching-Ying Huang, Puntney, Riley, Wu Moy, Amy, Chien-Yu Ting, Zhing Herr Chan, Darien, Nicholson, Martin W., Po-Ju Lin, Hung-Chih Chen, Kim, Gina C., Jianhua Zhang, Coonen, Jennifer, Puja Basu, Simmons, Heather A., Yen-Wen Liu, Hacker, Timothy A., and Kamp, Timothy J.

Published
2023
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