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7 results on '"K. Koletsos"'

1. Posters

Author

H. Spapen, M. Diltoer, K. Delanghe, G. Ingels, L. Huyghens, C. Jones, R. D. Griffiths, R. R. Macmillan, C. Harris, S. T. Atherton, P. Nee, M. Köller, P. Wachtler, M. Senkal, M. Kemen, W. König, L. Kompan, M. M. Berger, R. Chioléro, A. Pannatier, C. Cayeux, L. Tappy, J. López Martínez, M. Sánchez Castilla, R. Díaz Abad, M. A. García Salazar, M. J. Jiménez Martín, F. Del Nogal Sáez, I. Leonard, B. Harte, J. Collier, S. Maguire, D. Phelan, Rong Xinzhou, Xiao Guangxia, Li Ao, Zhang Yaping, G. Briassoulis, S. Venkataraman, A. Thompson, R. Khorram, W. Kauhl, A. Heiden, R. Hettich, W. Behrendt, M. Bitzani, D. Matamis, K. Koletsos, N. Pondikidis, D. Botsios, D. Riggos, and A. Ye. Shestopalov

Subjects
Critical Care and Intensive Care Medicine
Published
1996

2. Comparison of two methods, the thermodilution method of Fick and the Douglas bag method, in estimating the resting energy expenditure (REE)

Author

D. Riggos, D. Matamis, D. Botsios, M. Bizani, K. Koletsos, N. Gerogianni, and D. Lagonidis

Subjects
Mechanical ventilation, Icu patients, medicine.medical_specialty, Measurement method, Nutrition and Dietetics, Critically ill, business.industry, Endocrinology, Diabetes and Metabolism, medicine.medical_treatment, Surgery, Endocrinology, Energy expenditure, Intensive care, Statistics, Basal metabolic rate, medicine, Resting energy expenditure, business
Abstract

Critically ill patients need appropriate nutritional support strictly correlated to their REE to avoid complications either from hypo or hyper alimentation. Resting Energy Expenditure (REE) in the I.C.U setting can be measured or calculated by several methods. The aim of our study was to compare two methods of indirect calorimetry. The REE calculated by the Fick equation (REET) and the traditional method of Douglas bag (REEi). Twelve ICU patients on mechanical ventilation were studied and the following results were obtained REET = 2295±552 Kcal/day and REEi = 2339±512 Kcal/day (mean±sd). A good correlation between the two methods showed by regression analysis. We conclude that the evaluation of REE using the thermodilution method of Fick is reliable in estimating the energy expenditure of critical ill patients.

Published
1995

3. Influence of continuous haemofiltration-related hypothermia on haemodynamic variables and gas exchange in septic patients

Author

D. Riggos, K. Koletsos, S. Bursztein, K. Mavromatidis, D. Matamis, K. Sombolos, and M. Tsagourias

Subjects
Adult, Male, Critical Illness, medicine.medical_treatment, Ultrafiltration, Hemodynamics, Hypothermia, Critical Care and Intensive Care Medicine, Body Temperature, Sepsis, Hemofiltration, medicine, Humans, Aged, Pulmonary Gas Exchange, business.industry, Acute Kidney Injury, Middle Aged, medicine.disease, Shock, Septic, Shock (circulatory), Concomitant, Anesthesia, Female, medicine.symptom, business, Complication
Abstract

To investigate the influence of continuous haemofiltration (CHF) on haemodynamics, gas exchange and core temperature in critically ill septic patients with acute renal failure. In 20 patients (17 male, 3 female) ultrafiltration rate, core temperature, gas exchange and haemodynamic variables were measured at regular intervals during the first 48 h of haemofiltration. Baseline data were compared to those obtained 30 min after initiating CHF and also to those during hypothermia (if observed). Haemodynamic variables remained remarkably constant throughout the study period. In patients with a relatively low ultrafiltration rate (855±278 ml/h) temperature did not change, while in patients with a high ultrafiltration rate (1468±293 ml/h) core temperature significantly decreased from 37.6±0.9°C to 34.8±0.8°C (p

Published
1994

4. Ventilatory post-stimulus potentiation in patients with brain damage

Author

Nicholas R. Anthonisen, K Koletsos, K Markopoulou, Ioanna Mitrouska, D Patakas, D Riggos, and Dimitris Georgopoulos

Subjects
Pulmonary and Respiratory Medicine, Adult, Male, Time Factors, Adolescent, Brain damage, Hyperoxia, Critical Care and Intensive Care Medicine, Central nervous system disease, Respiration, medicine, Humans, Hypoxia, Capnography, Analysis of Variance, medicine.diagnostic_test, business.industry, Apnea, Carbon Dioxide, medicine.disease, Pathophysiology, Breath Tests, Periodic breathing, Anesthesia, Brain Damage, Chronic, Female, medicine.symptom, business
Abstract

In normal humans when a brief hypoxic ventilatory stimulus is terminated abruptly by breathing 100% O2, ventilation during hyperoxia gradually declines to baseline prehypoxic levels without an undershoot. This has been interpreted as evidence of decay of short-term potentiation (STP), a mechanism located in the brainstem and not dependent upon higher center inputs. STP decay may be important in preventing periodic breathing by damping ventilatory responses to cyclic stimuli. Patients with brain damage commonly have periodic breathing that may be caused partly by impairment of STP activation. To test this 12 tracheostomized patients with severe brain damage (Glasgow score 9.9 +/- 0.6) were studied. Breathing stability was estimated by at least 6 h of capnography and from these records apnea index (AI, episodes/hour) and cyclic changes of end-tidal CO2 (c-PETCO2, cycles/hour) were derived. STP activation was examined by brief exposure to hypoxia (45 s, end-tidal O2 = 50 mm Hg) followed by hyperoxia. Forty-four hypoxic-hyperoxic runs were analyzed and compared with 19 normoxic-hyperoxic trials. At the end of the hypoxia ventilation (VI) increased 39.5 +/- 5.8% and PETCO2 decreased 2.7 +/- 0.6 mm Hg to 91.5 +/- 2.2% of baseline value. When hypoxia was terminated abruptly by hyperoxia VI dropped immediately to 63.2 +/- 7.2% of baseline, remaining for 35 s significantly lower than the corresponding values acquired during hyperoxia after normoxia. After hypoxia, apneas occurred in 19 of 44 hyperoxic runs. There was a negative relationship between nadir hyperoxic ventilation after hypoxia and both AI and c-PETCO2.(ABSTRACT TRUNCATED AT 250 WORDS)

Published
1995

5. Treatment of sepsis

Author

E. Racadot, G. Capellier, J. J. Lanoret, K. Koletsos, F. Fourrier, C. J. Fisher, J. F. Dhainaut, I. Kioumis, D. Matamis, M. Bittl, A. Grammenos, P. Nightingale, J. C. Sadoff, Mathias Haller, H. Forst, K. Peler, C. Caron, C. Garrard, P. Balvay, M. W. Bodmer, G. van der Linden, H. Mehdaoui, F. P. Lenhart, W. Kellermann, J. J. Lanore, E. A. Panacek, C. Chopin, A. Rime, R. L. Schein, H. Tissot, G. E. Foulke, A. Boulot, P. Herve, F. Pochard, E. L. Warren, C. Socolovski, J. Cohen, D. Rigos, Jean Louis Vincent, I. Runge, J. Wijdenes, S. Stephens, J. Goudemand, J. J. Huart, A. Cordier, F. Barale, Josef Briegel, J. Zimmerman, K. Sobolos, G. Park, and S. M. Opal

Subjects
Sepsis, medicine.medical_specialty, Mean arterial pressure, business.industry, Septic shock, Pain medicine, Anesthesiology, Emergency medicine, medicine, Sepsis syndrome, Critical Care and Intensive Care Medicine, business, medicine.disease
Published
1992

6. Ventilatory post-stimulus potentiation in patients with brain damage.

Author

Georgopoulos D, Mitrouska I, Koletsos K, Markopoulou K, Riggos D, Patakas D, and Anthonisen NR

Subjects
Adolescent, Adult, Analysis of Variance, Breath Tests, Carbon Dioxide analysis, Carbon Dioxide physiology, Female, Humans, Hyperoxia physiopathology, Hypoxia physiopathology, Male, Time Factors, Brain Damage, Chronic physiopathology, Respiration physiology
Abstract

In normal humans when a brief hypoxic ventilatory stimulus is terminated abruptly by breathing 100% O2, ventilation during hyperoxia gradually declines to baseline prehypoxic levels without an undershoot. This has been interpreted as evidence of decay of short-term potentiation (STP), a mechanism located in the brainstem and not dependent upon higher center inputs. STP decay may be important in preventing periodic breathing by damping ventilatory responses to cyclic stimuli. Patients with brain damage commonly have periodic breathing that may be caused partly by impairment of STP activation. To test this 12 tracheostomized patients with severe brain damage (Glasgow score 9.9 +/- 0.6) were studied. Breathing stability was estimated by at least 6 h of capnography and from these records apnea index (AI, episodes/hour) and cyclic changes of end-tidal CO2 (c-PETCO2, cycles/hour) were derived. STP activation was examined by brief exposure to hypoxia (45 s, end-tidal O2 = 50 mm Hg) followed by hyperoxia. Forty-four hypoxic-hyperoxic runs were analyzed and compared with 19 normoxic-hyperoxic trials. At the end of the hypoxia ventilation (VI) increased 39.5 +/- 5.8% and PETCO2 decreased 2.7 +/- 0.6 mm Hg to 91.5 +/- 2.2% of baseline value. When hypoxia was terminated abruptly by hyperoxia VI dropped immediately to 63.2 +/- 7.2% of baseline, remaining for 35 s significantly lower than the corresponding values acquired during hyperoxia after normoxia. After hypoxia, apneas occurred in 19 of 44 hyperoxic runs. There was a negative relationship between nadir hyperoxic ventilation after hypoxia and both AI and c-PETCO2.(ABSTRACT TRUNCATED AT 250 WORDS)

Published
1995
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7. Influence of continuous haemofiltration-related hypothermia on haemodynamic variables and gas exchange in septic patients.

Author

Matamis D, Tsagourias M, Koletsos K, Riggos D, Mavromatidis K, Sombolos K, and Bursztein S

Subjects
Acute Kidney Injury etiology, Acute Kidney Injury physiopathology, Adult, Aged, Body Temperature, Critical Illness, Female, Humans, Hypothermia physiopathology, Male, Middle Aged, Shock, Septic physiopathology, Acute Kidney Injury therapy, Hemodynamics, Hemofiltration adverse effects, Hypothermia etiology, Pulmonary Gas Exchange, Shock, Septic complications
Abstract

Objective: To investigate the influence of continuous haemofiltration (CHF) on haemodynamics, gas exchange and core temperature in critically ill septic patients with acute renal failure., Patients and Methods: In 20 patients (17 male, 3 female) ultrafiltration rate, core temperature, gas exchange and haemodynamic variables were measured at regular intervals during the first 48 h of haemofiltration. Baseline data were compared to those obtained 30 min after initiating CHF and also to those during hypothermia (if observed)., Main Results: Haemodynamic variables remained remarkably constant throughout the study period. In patients with a relatively low ultrafiltration rate (855 +/- 278 ml/h) temperature did not change, while in patients with a high ultrafiltration rate (1468 +/- 293 ml/h) core temperature significantly decreased from 37.6 +/- 0.9 degrees C to 34.8 +/- 0.8 degrees C (p < 0.001). There was a statistically significant correlation between temperature decrease and ultrafiltration rate (r = -0.68, Y = 1.8-0.003 X, p < 0.01). Hypothermic patients also showed a mean decrease in VO2 from 141 +/- 22 ml/min/m2 to 112 +/- 22 ml/min/m2 (p < 0.01) with a concomitant increase in PaO2 from 103 +/- 37 mmHg to 140 +/- 42 mmHg (p < 0.001) and in PvO2 from 35 +/- 4 mmHg to 41 +/- 5 mmHg (p < 0.001)., Conclusions: 1) Continuous haemofiltration does not cause significant alternations in haemodynamic variables. 2) Hypothermia frequently occurs in patients undergoing continuous haemofiltration with high ultrafiltration rates. These hypothermic patients show a reduction in VO2 leading to an increase in PvO2 and PaO2. This mild hypothermia in these circumstances has no evident deleterious effects.

Published
1994
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