312 results on '"Juin, Philippe"'
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2. Low BCL-xL expression in triple-negative breast cancer cells favors chemotherapy efficacy, and this effect is limited by cancer-associated fibroblasts
3. Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018
4. Intérêt de l’outil web bc-GenExMiner en oncologie
5. Targeting of MCL-1 in breast cancer-associated fibroblasts reverses their myofibroblastic phenotype and pro-invasive properties
6. Development of an absolute assignment predictor for triple-negative breast cancer subtyping using machine learning approaches
7. Using mammary organoids to study cilia
8. Cancer associated fibroblasts mitigate the efficacy of the combination of chemotherapy and BCL-xL targeting in triple negative breast cancer cells
9. Targeting PUMA/Bcl-xL interaction by new specific compounds to unleash apoptotic process in cancer cells
10. Structure-guided design of pyridoclax derivatives based on Noxa / Mcl-1 interaction mode
11. EMT and primary ciliogenesis: For better or worse in sickness and in health
12. NOXA/MCL-1 axis determines cell-death decision between apoptosis and pyroptosis and the inflammatory secretome of breast cancer cells treated with anti-mitotics.
13. Cancer associated fibroblasts mitigate the efficacy of the combination of chemotherapy and BCL-xL targeting in triple negative breast cancer cells
14. Synthesis and cytotoxicity studies of newly designed benzyl-hydroquinone derivatives
15. The oncogenic tyrosine kinase Lyn impairs the pro-apoptotic function of Bim
16. ChemR23 activation reprograms macrophages toward a less inflammatory phenotype and dampens carcinoma progression
17. Novel 1,6-naphthyridin-2(1H)-ones as potential anticancer agents targeting Hsp90
18. STING-dependent paracriny shapes apoptotic priming of breast tumors in response to anti-mitotic treatment
19. EMT and primary ciliogenesis: For better or worse in sickness and in health.
20. A combination of in silico and SAR studies to identify binding hot spots of Bcl-xL inhibitors
21. E2F1 interacts with BCL‐xL and regulates its subcellular localization dynamics to trigger cell death
22. Chapter 11 - Using mammary organoids to study cilia
23. Synthesis of new mixed phenol/heterocyclic derivatives and studies of their activity as inhibitors of Bax/Bcl-xL interaction
24. Survivin contributes to DNA repair by homologous recombination in breast cancer cells
25. Échappement tumoral aux inhibiteurs de HER2 : théorie de la sédimentation
26. Keeping Cell Death Alive: An Introduction into the French Cell Death Research Network
27. Bax Activation by the BH3-Only Protein Puma Promotes Cell Dependence on Antiapoptotic Bcl-2 Family Members
28. An EMT–primary cilium–GLIS2 signaling axis regulates mammogenesis and claudin-low breast tumorigenesis
29. Escape from p21-mediated Oncogene-induced Senescence Leads to Cell Dedifferentiation and Dependence on Anti-apoptotic Bcl-xL and MCL1 Proteins
30. Resistance to HER2 inhibitors: Is addition better than substitution? Rationale for the hypothetical concept of drug sedimentation
31. Synthesis and biological studies of new piperidino-1,2,3-triazole hybrids with 3-aryl isoxazole side chains
32. TCTP contains a BH3-like domain, which instead of inhibiting, activates Bcl-xL
33. Lien entre reprogrammation métabolique et résistance à l’apoptose induites par le stroma dans les cancers du sein triple négatif
34. Decoding and unlocking the BCL-2 dependency of cancer cells
35. An EMT–primary cilium–GLIS2 signaling axis regulates mammogenesis and claudin-low breast tumorigenesis
36. Functional and physical interaction between Bcl‐XL and a BH3‐like domain in Beclin‐1
37. Bax activation and mitochondrial insertion during apoptosis
38. Mitochondria as the target of the pro-apoptotic protein Bax
39. Random forest of perfect trees: concept, performance, applications and perspectives
40. bc-GenExMiner 4.5: new mining module computes breast cancer differential gene expression analyses
41. Mitochondria at Center of Exchanges between Cancer Cells and Cancer-Associated Fibroblasts during Tumor Progression
42. Targeting of BCL-2 Family Members during Anticancer Treatment: A Necessary Compromise between Individual Cell and Ecosystemic Responses?
43. Development of an absolute assignment predictor for triple-negative breast cancer subtyping using machine learning approaches
44. Identification of paclitaxel resistance through a new statistical approach based on a random forest of perfect trees classifcation.
45. RE: Immune Checkpoint Profiles in Luminal B Breast Cancer (Alliance)
46. Mitotic stress-induced secretome primes cancer cells to apoptosis and maximizes paclitaxel response in breast tumors when combined with BCL-xL-targeting BH3 mimetics
47. c-Myc-induced sensitization to apoptosis is mediated through cytochrome c release
48. Differentiation-dependent Sensitivity to Apoptogenic Factors in PC12 Cells
49. The p18 Truncated Form of Bax Behaves Like a Bcl-2 Homology Domain 3-only Protein
50. The First α Helix of Bax Plays a Necessary Role in Its Ligand-Induced Activation by the BH3-Only Proteins Bid and PUMA
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