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1. Aryl amino acetamides prevent Plasmodium falciparum ring development via targeting the lipid-transfer protein PfSTART1

2. Erythrocyte membrane protein 3 (EMAP3) is exposed on the surface of thePlasmodium bergheiinfected red blood cell

4. A scalable CRISPR-Cas9 gene editing system facilitates CRISPR screens in the malaria parasitePlasmodium berghei

6. Aryl amino acetamides prevent the development ofPlasmodium falciparumrings via inhibition of the lipid transfer protein PfSTART1

7. PTEX helps efficiently traffic haemoglobinases to the food vacuole in Plasmodium falciparum

8. Sulfonylpiperazine compounds prevent Plasmodium falciparum invasion of red blood cells through interference with actin-1/profilin dynamics

11. The sulfonylpiperazine MMV020291 prevents red blood cell invasion by the malaria parasite Plasmodium falciparum through interference with actin-1/profilin dynamics

12. The Plasmodium falciparum parasitophorous vacuole protein P113 interacts with the parasite protein export machinery and maintains normal vacuole architecture

13. A revised mechanism for how Plasmodium falciparum recruits and exports proteins into its erythrocytic host cell

14. A revised mechanism for how Plasmodium falciparum recruits and exports proteins into its erythrocytic host cell

15. Characterisation of complexes formed by parasite proteins exported into the host cell compartment ofPlasmodium falciparuminfected red blood cells

16. Characterisation of complexes formed by parasite proteins exported into the host cell compartment of Plasmodium falciparum infected red blood cells.

17. Spatial organization of protein export in malaria parasite blood stages

18. Plasmodium falciparum parasites deploy RhopH2 into the host erythrocyte to obtain nutrients, grow and replicate

19. Plasmodium falciparum parasites deploy RhopH2 into the host erythrocyte to obtain nutrients, grow and replicate

20. Author response: Plasmodium falciparum parasites deploy RhopH2 into the host erythrocyte to obtain nutrients, grow and replicate

21. Author Correction: Aryl amino acetamides prevent Plasmodium falciparum ring development via targeting the lipid-transfer protein PfSTART1.

22. Pf ATP4 inhibitors in the Medicines for Malaria Venture Malaria Box and Pathogen Box block the schizont-to-ring transition by inhibiting egress rather than invasion.

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