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255 results on '"Johansson, Mats W"'

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1. The Impact of Insulin Resistance on Loss of Lung Function and Response to Treatment in Asthma.

2. Utility of eosinophil peroxidase as a biomarker of eosinophilic inflammation in asthma

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3. COVID-19 Related Genes in Sputum Cells in Asthma: Relationship to Demographic Features and Corticosteroids

4. Extracellular DNA, Neutrophil Extracellular Traps, and Inflammasome Activation in Severe Asthma

5. Refractory airway type 2 inflammation in a large subgroup of asthmatic patients treated with inhaled corticosteroids

6. Eosinophil-Epithelial Cell Interactions in Asthma.

7. ALX receptor ligands define a biochemical endotype for severe asthma

8. Natural killer cell–mediated inflammation resolution is disabled in severe asthma

9. Biologic therapy in rare eosinophil-associated disorders: remaining questions and translational research opportunities.

10. Plasma interleukin-6 concentrations, metabolic dysfunction, and asthma severity: a cross-sectional analysis of two cohorts

12. A novel DNase assay reveals low DNase activity in severe asthma.

13. Asthma severity and corticosteroid response depend on variable type 1 and type 2 inflammation in the airway

14. Decreased plasma cartilage acidic protein 1 in COVID‐19

16. Biomarkers of the involvement of mast cells, basophils and eosinophils in asthma and allergic diseases

17. Molecular anatomy of eosinophil activation by IL5 and IL33

33. Plasma P-Selectin Is Inversely Associated with Lung Function and Corticosteroid Responsiveness in Asthma

37. Altered localization and cytoplasmic domain-binding properties of tyrosine-phosphorylated beta1 integrin

39. Eosinophil cytolysis on Immunoglobulin G is associated with microtubule formation and suppression of rho‐associated protein kinase signalling

44. Eosinophil cytolysis on Immunoglobulin G is associated with microtubule formation and suppression of rho‐associated protein kinase signalling.

48. ALX receptor ligands define a biochemical endotype for severe asthma

49. Sputum Mast Cells and Eosinophils Identify Asthmatics with Lower Lung Function and Proneness to Exacerbations