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31 results on '"Jo, Hyang Jeong"'

13. The Effect of Autophagy to Cell Death in Nutrient-Deprived H460 Cells

Author

Jang, Hye Yeon, primary, Jo, Hyang Jeong, additional, Hwhang, Ki Eun, additional, Kim, So Young, additional, Lee, Kang Kyoo, additional, Moon, Sun Rock, additional, Shin, Jeong Hyun, additional, Cho, Kyung Hwa, additional, Lee, Mi Kung, additional, Lee, Sam Youn, additional, Park, Soon Ah, additional, Park, Jong Kun, additional, Kim, Hui Jung, additional, and Yang, Sei Hoon, additional

Published
2010
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14. Induction of Autophagy by Low Dose of Cisplatin in H460 Lung Cancer Cells

Author

Shin, Jeong Hyun, primary, Jang, Hye Yeon, additional, Chung, Jin Soo, additional, Cho, Kyung Hwa, additional, Hwang, Ki Eun, additional, Kim, So Young, additional, Kim, Hui Jung, additional, Lee, Sam Youn, additional, Lee, Mi Kung, additional, Park, Soon Ah, additional, Moon, Sun Rock, additional, Lee, Kang Kyu, additional, Jo, Hyang Jeong, additional, and Yang, Sei Hoon, additional

Published
2010
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17. The Role and Significance of Biomarker for Plasma G-CSF in Patients with Primary Lung Cancer

Author

Song, Jung Sub, primary, Kim, So Young, additional, Jo, Hyang Jeong, additional, Lee, Kang Kyoo, additional, Shin, Jeong Hyun, additional, Shin, Seong Nam, additional, Kim, Dong, additional, Park, Seong Hoon, additional, Lee, Young Jin, additional, Ko, Chang Bo, additional, Lee, Mi Kung, additional, Choi, Soon Ho, additional, Jeong, Jong Hoon, additional, Park, Jung Hyun, additional, Kim, Hui Jung, additional, Kim, Hak Ryul, additional, Jeong, Eun Taik, additional, and Yang, Sei Hoon, additional

Published
2009
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20. The Clinical and Histopathologic Features according to Loss of LKB1 Protein Expression on Primary Lung Cancer

Author

Hwang, Ki Eun, primary, Jo, Hyang-Jeong, additional, Lee, Kang Kyoo, additional, Shim, Hyeok, additional, Song, Jung Sub, additional, Shin, Jeong Hyun, additional, Shin, Seong Nam, additional, Park, Seong-Hoon, additional, Hong, Kyeong-Man, additional, Park, Jung-Hyun, additional, Jeong, Jong-Hoon, additional, Kim, Hui Jung, additional, Kim, Hak-Ryul, additional, Yang, Sei-Hoon, additional, and Jeong, Eun-Taik, additional

Published
2008
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21. P2-054: Combination treatment with arsenic trioxide and sulindac induces apoptosis of NCI-H157 human lung carcinoma cells via activation of mitogen-activated protein kinases

Author

Yang, Sei-Hoon, primary, Kim, Hak-Ryul, additional, Kim, Eun Jung, additional, Jang, Hye Yeon, additional, Jo, Hyang-Jeong, additional, Shim, Hyeok, additional, Lee, Kang Kyoo, additional, Park, Seong-Hoon, additional, Park, Jung-Hyun, additional, and Jeong, Eun-Taik, additional

Published
2007
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27. Acute cholecystitis associated with Clonorchis sinensis infection.

Author

Oh JT, Kang DB, and Jo HJ

Abstract

Clonorchis sinensis is one of the most common causes of trematodiasis that is caused by the ingestion of raw fish contaminated with infective cysts. The adult flukes are predominantly present in the intrahepatic bile ducts, but occasionally they may be found in the pancreatic duct and extrahepatic bile ducts. The clinical manifestations depend on the number of flukes, the period of infestation, and complications such as pericholangitic abscess, cholangitis, bile duct stones, and cholangiocarcinoma. However, primary acute cholecystitis associated with C. sinensis infection is extremely rare. Herein, we report on a case of primary acute cholecystitis associated with C. sinensis infection.

Published
2014
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28. The role of autophagy induced by pemetrexed in lung adenocarcinoma cells.

Author

Park JH, Lee YR, So HS, Lee KK, Lee SY, Moon SR, Jo HJ, Lee S, Jeong K, Kwon KB, and Yang SH

Subjects
Adenine analogs & derivatives, Adenine pharmacology, Adenocarcinoma of Lung, Annexin A5 biosynthesis, Antimetabolites, Antineoplastic pharmacology, Cell Line, Tumor, Cell Survival, Guanine pharmacology, Humans, Pemetrexed, Poly(ADP-ribose) Polymerases metabolism, Thymidylate Synthase antagonists & inhibitors, Adenocarcinoma drug therapy, Adenocarcinoma pathology, Apoptosis drug effects, Autophagy drug effects, Glutamates pharmacology, Guanine analogs & derivatives, Lung Neoplasms drug therapy, Lung Neoplasms pathology
Abstract

Autophagy is known as an important regulatory mediator for cell survival or death and its important role in cancer. Pemetrexed (PTX) has been used in chemotherapy for lung cancer. However, the underlying molecular mechanisms have not been fully clarified. To investigate the role of autophagy induced by PTX in A549 cells, we performed MTT assay, acridine orange staining, western blotting, Annexin V/PI by using the 3-MA autophagy inhibitor. PTX induced autophagy after 48 h treatment in A549 cells. Furthermore, PTX showed acidic vesicular organelles (AVOs) and expressed LC3-II in A549 cells. The induction of autophagy by PTX was inhibited by 3-MA which was confirmed by reduced AVOs. When the autophagy was inhibited, Annexin V was increased. In addition, PARP cleavage was increased as shown by western blotting. Taken together, PTX induced autophagy in A549 cells and these cellular events possibly cause the apoptotic and/or necrotic cell death of A549 cells.

Published
2014
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29. [A case of actinomycosis of gallbladder presenting as acute cholecystitis].

Author

Lee JH, Hwang ET, Kim KH, Jo HJ, Kim TH, Choi SC, and Choi CS

Subjects
Actinomycosis drug therapy, Actinomycosis pathology, Cholecystectomy, Cholecystitis, Acute surgery, Gallbladder Diseases drug therapy, Gallbladder Diseases pathology, Humans, Male, Middle Aged, Penicillins administration & dosage, Tomography, X-Ray Computed, Actinomycosis diagnosis, Cholecystitis, Acute diagnosis, Gallbladder Diseases diagnosis
Abstract

Actinomycosis is a chronic suppurative and granulomatous disease, characterized by the formation of abscess, draining sinuses, abundant granulation, and dense fibrous tissue. Actinomycosis of the gallbladder is extremely rare. We report a case of an 56-years old man who abruptly presented with right upper quadrant abdominal pain. Abdominal CT showed that the gallbladder had 2 cm sized stone and an edematous thick wall. Our preoperative diagnosis was acute calculous cholecystitis. After the management of acute cholecystitis, laparoscopic cholecystectomy was performed but converted to open surgery due to severe adhesion to liver and greater omentum. Partial cholecystectomy was performed. Histologic section of the gallbladder showed sulfur granule with gram-positive branching bacilli compatible with actinomyces. After cholecystectomy, the patient received intravenous penicillin G for 2 weeks, followed by oral penicillin for 3 months.

Published
2009
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30. Combination treatment with arsenic trioxide and sulindac enhances apoptotic cell death in lung cancer cells via activation of oxidative stress and mitogen-activated protein kinases.

Author

Park JH, Kim EJ, Jang HY, Shim H, Lee KK, Jo HJ, Kim HJ, Yang SH, Jeong ET, and Kim HR

Subjects
Arsenic Trioxide, Arsenicals administration & dosage, Blotting, Western, DNA Damage drug effects, Enzyme Inhibitors pharmacology, Flow Cytometry, G1 Phase drug effects, Heme Oxygenase-1 metabolism, Histones metabolism, Humans, Hydrogen Peroxide metabolism, JNK Mitogen-Activated Protein Kinases antagonists & inhibitors, JNK Mitogen-Activated Protein Kinases metabolism, Lung Neoplasms drug therapy, Lung Neoplasms metabolism, Mitogen-Activated Protein Kinase 1 antagonists & inhibitors, Mitogen-Activated Protein Kinase 1 metabolism, Mitogen-Activated Protein Kinase 3 antagonists & inhibitors, Mitogen-Activated Protein Kinase 3 metabolism, Oxides administration & dosage, Phosphorylation drug effects, Reactive Oxygen Species metabolism, Sulindac administration & dosage, Tumor Cells, Cultured, p38 Mitogen-Activated Protein Kinases antagonists & inhibitors, p38 Mitogen-Activated Protein Kinases metabolism, Antineoplastic Combined Chemotherapy Protocols pharmacology, Apoptosis drug effects, Lung Neoplasms pathology, Mitogen-Activated Protein Kinases metabolism, Oxidative Stress drug effects
Abstract

Arsenic trioxide (As2O3) has been introduced to the treatment of acute promyelocytic leukemia (APL), and has also been shown to induce apoptosis in a variety of solid tumor cell lines, including non-small cell lung cancer. However, the prohibitively high concentration required for the induction of apoptotic cell death in many solid tumor cells is unacceptable for clinical utilization due to the excessive toxicity associated with this dose. Sulindac is known to enhance the cellular responsiveness of tumors toward chemotherapeutic drugs. Herein, we demonstrated that combination treatment with As2O3 and sulindac resulted in a synergistic augmentation of cytotoxicity in H157 lung cancer cells, which was revealed by apoptotic induction as demonstrated by an increase in the sub-G0/G1 fraction. In addition, combination treatment with As2O3 and sulindac increased reactive oxygen species (ROS) and oxidative stress, as evidenced by the heme oxygenase-1 (HO-1) expression and mitogen-activated protein kinase (MAPK) phosphorylation. MAPK inhibitors blocked the induction of HO-1 by combination treatment. Inhibitors of p38 and JNK partially inhibited the augmented cell death whereas the ERK inhibitor showed poor inhibition. Combination treatment with As2O3 and sulindac induced oxidative DNA damage in a time-dependent fashion, which was evaluated by H2AX phosphorylation along with HO-1 induction.

Published
2008

31. [Primary duodenal choriocarcinoma presenting as massive intestinal bleeding and metastasis to brain].

Author

Cho EY, Choi CS, Kim JW, Kweon JH, Kim TH, Seo GS, Jo HJ, Choi SC, and Nah YH

Subjects
Choriocarcinoma secondary, Duodenal Neoplasms pathology, Female, Humans, Middle Aged, Brain Neoplasms secondary, Choriocarcinoma diagnosis, Duodenal Neoplasms diagnosis, Gastrointestinal Hemorrhage diagnosis
Abstract

Duodenal choriocarcinoma, either primary or metastatic, is very rare. Early diagnosis and prompt initiation of chemotherapy improve the prognosis of this neoplasm. We herein present, together with the referred literatures, a case of a 47-year-old female patient who visited to our hospital with upper intestinal bleeding. She was diagnosed as duodenal choriocarcinoma by operation. Brain metastasis was found soon after the operation and combination chemotherapy was done.

Published
2006

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