195 results on '"Jiang, Jifu"'
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2. Inhibition of NK cell cytotoxicity by tubular epithelial cell expression of Clr-b and Clr-f
3. Supplemental hydrogen sulfide in models of renal transplantation after cardiac death
4. The CaMK Family Differentially Promotes Necroptosis and Mouse Cardiac Graft Injury and Rejection
5. Field Modulation and Torque Production Mechanism in Dual-PM Reluctance Machines with Harmonic Winding
6. Toll-like receptor 3 is an endogenous sensor of cell death and a potential target for induction of long-term cardiac transplant survival
7. Mitochondrial permeability regulates cardiac endothelial cell necroptosis and cardiac allograft rejection
8. Evaluating the Effects of Kidney Preservation at 10 °C with Hemopure and Sodium Thiosulfate in a Rat Model of Syngeneic Orthotopic Kidney Transplantation.
9. An Addition of U0126 Protecting Heart Grafts From Prolonged Cold Ischemia Reperfusion Injury in Heart Transplantation: A New Preservation Strategy
10. A Novel Parallel-Excited Dual-PM Reluctance Machine With Enhanced Torque and Efficiency Performance
11. Quantitative Analysis of Hybrid-Excited Doubly Salient Machine With Subslot Bottom PMs and Its Comparative Study
12. Torque Improvement of a Hybrid-Excited Vernier Reluctance Machine with High-order-harmonic Winding Design for Electric Vehicle Application
13. Slot-PM-Assisted Hybrid Reluctance Generator With Self-Excited DC Source for Stand-Alone Wind Power Generation
14. A Novel High-Order-Harmonic Winding Design in Hybrid-Excited Reluctance Machine for Electric Vehicle Application
15. A Novel Slot PM-Assisted Hybrid Magnet Memory Machine
16. A Novel Slot-PM-Assisted Consequent-Pole-PM Machine With Hybrid Magnets of Ferrite PMs and Rare Earth PMs
17. Necroptosis Is Involved in CD4+ T Cell-Mediated Microvascular Endothelial Cell Death and Chronic Cardiac Allograft Rejection
18. A Novel Winding Switching Control Strategy of a Consequent-Pole Ferrite-PM Hybrid-Excited Machine for Electric Vehicle Application
19. Slot-PM-Assisted Hybrid Reluctance Generator With Self-Excited DC Source for Stand-Alone Wind Power Generation
20. Cyclophilin D Regulates the Nuclear Translocation of AIF, Cardiac Endothelial Cell Necroptosis and Murine Cardiac Transplant Injury
21. EXOGENOUS HYDROGEN SULFIDE TREATMENT REDUCES RENAL FIBROSIS ASSOCIATED WITH CHRONIC URETERAL OBSTRUCTION BY ATTENUATING EPITHELIAL-MESENCHYMAL TRANSITION: MP58-18
22. HYDROGEN SULFIDE TREATMENT MITIGATES DONOR TISSUE NECROSIS DURING PROLONGED COLD STORAGE AND IMPROVES SURVIVAL FOLLOWING ALLOGENEIC RENAL TRANSPLANTATION.: MP29-17
23. HYDROGEN SULFIDE SUPPLEMENTATION MITIGATES EFFECTS OF ISCHEMIA REPERFUSION INJURY IN A MURINE MODEL OF DONATION AFTER CARDIAC DEATH RENAL TRANSPLANTATION: MP29-16
24. Natural Killer Cells Mediate Long-term Kidney Allograft Injury
25. A novel doubly‐fed doubly‐salient machine with DC‐saturation‐relieving structure for wind power generation
26. A Novel Winding Switching Control Strategy for AC/DC Hybrid-Excited Wind Power Generator
27. Reduction of chronic allograft nephropathy by inhibition of extracellular signal-regulated kinase 1 and 2 signaling
28. Supplementing preservation solution with mitochondria-targeted H2S donor AP39 protects cardiac grafts from prolonged cold ischemia–reperfusion injury in heart transplantation
29. Endothelin-1, endothelin-3 and their receptors (endothelinA and endothelinB) in chronic renal transplant rejection in rats1
30. Lack of Renal Dopamine D5 Receptors Promotes Hypertension
31. ESTABLISHMENT OF DUODENOJEJUNAL BYPASS SURGERY IN MICE: A MODEL DESIGNED FOR DIABETIC RESEARCH
32. Peritransplant treatment with cobalt protoporphyrin attenuates chronic renal allograft rejection
33. Renal Tubular Epithelial Cell Self-Injury Through Fas/Fas Ligand Interaction Promotes Renal Allograft Injury
34. Hydrogen Sulfide Mitigates the Effects of Ischemia-Reperfusion Injury in a Donation After Cardiac Death Model of Renal Transplantation
35. Loss of receptor interacting protein kinases 3 and caspase‐8 augments intrinsic apoptosis in tubular epithelial cell and promote kidney ischaemia‐reperfusion injury
36. Over-expression of growth differentiation factor 15 (GDF15) preventing cold ischemia reperfusion (I/R) injury in heart transplantation through Foxo3a signaling
37. GYY4137, a Slow-Releasing Hydrogen Sulfide Donor, Ameliorates Renal Damage Associated with Chronic Obstructive Uropathy
38. Prevention of allograft rejection in heart transplantation through concurrent gene silencing of TLR and Kinase signaling pathways
39. MP29-16 HYDROGEN SULFIDE SUPPLEMENTATION MITIGATES EFFECTS OF ISCHEMIA REPERFUSION INJURY IN A MURINE MODEL OF DONATION AFTER CARDIAC DEATH RENAL TRANSPLANTATION
40. MP58-18 EXOGENOUS HYDROGEN SULFIDE TREATMENT REDUCES RENAL FIBROSIS ASSOCIATED WITH CHRONIC URETERAL OBSTRUCTION BY ATTENUATING EPITHELIAL-MESENCHYMAL TRANSITION
41. MP29-17 HYDROGEN SULFIDE TREATMENT MITIGATES DONOR TISSUE NECROSIS DURING PROLONGED COLD STORAGE AND IMPROVES SURVIVAL FOLLOWING ALLOGENEIC RENAL TRANSPLANTATION.
42. Attenuating Ischemia-Reperfusion Injury in Kidney Transplantation by Perfusing Donor Organs With siRNA Cocktail Solution
43. Natural Killer Cells Play a Critical Role in Cardiac Allograft Vasculopathy in an Interleukin-6–Dependent Manner
44. NK cells mediate chronic kidney allograft injury (169.26)
45. Induction of Kidney Allograft Tolerance by Soluble CD83 Associated With Prevalence of Tolerogenic Dendritic Cells and Indoleamine 2,3-Dioxygenase
46. Immunosuppression Involving Soluble CD83 Induces Tolerogenic Dendritic Cells That Prevent Cardiac Allograft Rejection
47. Free Bone Graft Attenuates Acute Rejection and in Combination with Cyclosporin A Leads to Indefinite Cardiac Allograft Survival
48. Long-Term Limb Allograft Survival Using a Short Course of Anti-CD45RB Monoclonal Antibody, LF 15-0195, and Rapamycin in a Mouse Model
49. Induction of Indefinite Cardiac Allograft Survival Correlates With Toll-Like Receptor 2 and 4 Downregulation After Serine Protease Inhibitor-1 (Serp-1) Treatment
50. Double-Negative T Cells, Activated by Xenoantigen, Lyse Autologous B and T Cells Using a Perforin/Granzyme-Dependent, Fas-Fas Ligand-Independent Pathway
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