43 results on '"Jha, Jay C."'
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2. Endothelial NOX5 Obliterates the Reno-Protective Effect of Nox4 Deletion by Promoting Renal Fibrosis via Activation of EMT and ROS-Sensitive Pathways in Diabetes
3. Modulation of Oxidative Stress in Cardiovascular Diseases
4. Differential sympathetic response to lesion-induced chronic kidney disease in rabbits
5. Empagliflozin modulates renal sympathetic and heart rate baroreflexes in a rabbit model of diabetes
6. Nox (NADPH Oxidase) 1, Nox4, and Nox5 Promote Vascular Permeability and Neovascularization in Retinopathy
7. Endothelial or vascular smooth muscle cell-specific expression of human NOX5 exacerbates renal inflammation, fibrosis and albuminuria in the Akita mouse
8. Contribution of the Renal Nerves to Hypertension in a Rabbit Model of Chronic Kidney Disease
9. Combined NOX1/4 inhibition with GKT137831 in mice provides dose-dependent reno- and atheroprotection even in established micro- and macrovascular disease
10. New Insights Into the Use of Biomarkers of Diabetic Nephropathy
11. Podocyte-specific Nox4 deletion affords renoprotection in a mouse model of diabetic nephropathy
12. 437-P: NOX5 in Human Peripheral Blood Mononuclear Cells Is a Potential Biomarker for Unstable Diabetic Vascular Disease
13. Independent of Renox, NOX5 Promotes Renal Inflammation and Fibrosis in Diabetes by Activating ROS-sensitive Pathways
14. Adverse renal effects of NLRP3 inflammasome inhibition by MCC950 in an interventional model of diabetic kidney disease
15. Dyslipidemia, Diabetes and Atherosclerosis: Role of Inflammation and ROS-Redox-Sensitive Factors
16. Identifying and interpreting novel targets that address more than one diabetic complication: a strategy for optimal end organ protection in diabetes
17. Oxidative Stress, Nox Isoforms and Complications of Diabetes—Potential Targets for Novel Therapies
18. NAD(P)H oxidase isoforms as therapeutic targets for diabetic complications
19. Independent of Renox, NOX5 Promotes Renal Inflammation and Fibrosis in Diabetes by Activating ROS-Sensitive Pathways.
20. MODULATION OF SYMPATHETIC NERVE ACTIVITY BY SGLT2 INHIBITOR EMPAGLIFLOZIN IN DIABETIC RABBITS
21. Oxidative Stress and Inflammation in Renal and Cardiovascular Complications of Diabetes
22. NADPH Oxidase Inhibition: Preclinical and Clinical Studies in Diabetic Complications
23. 479-P: Inhibition of the NLRP3 Inflammasome by MCC950 Accelerates Renal Injury in Diabetes Model
24. 478-P: Nox5 Aggravates Renal Injury Independent of Nox4 Expression in Diabetes
25. Treatment with SGLT2 Inhibitor Empagliflozin Modulates Renal Sympathetic Nerve Activity in Diabetic Rabbits
26. The role of Nox5 in diabetic complications
27. A causal link between oxidative stress and inflammation in cardiovascular and renal complications of diabetes
28. NADPH Oxidase Nox5 Aggravates Renal Injury in Akita Mouse Model of Diabetic Nephropathy
29. Genetic Targeting or Pharmacologic Inhibition of NADPH Oxidase Nox4 Provides Renoprotection in Long-Term Diabetic Nephropathy
30. NADPH Oxidase Nox5 Accelerates Renal Injury in Diabetic Nephropathy
31. The emerging role of NADPH oxidase NOX5 in vascular disease
32. Elucidating NOX5 as a Potential Therapeutic Target and a Biomarker of Kidney Disease in Diabetes
33. Diabetes and Kidney Disease: Role of Oxidative Stress
34. miR-21 promotes renal fibrosis in diabetic nephropathy by targeting PTEN and SMAD7
35. Podocyte-specific Nox4 deletion affords renoprotection in a mouse model of diabetic nephropathy
36. Are reactive oxygen species still the basis for diabetic complications?
37. Nox-4 deletion reduces oxidative stress and injury by PKC-α-associated mechanisms in diabetic nephropathy
38. Genetic Targeting or Pharmacologic Inhibition of NADPH Oxidase Nox4 Provides Renoprotection in Long-Term Diabetic Nephropathy
39. Transforming growth factor-β1-mediated renal fibrosis is dependent on the regulation of transforming growth factor receptor 1 expression by let-7b
40. Identifying and interpreting novel targets that address more than one diabetic complication: a strategy for optimal end organ protection in diabetes
41. Are reactive oxygen species still the basis for diabetic complications?
42. Oxidative Stress and Inflammation in Renal and Cardiovascular Complications of Diabetes.
43. 499-P: The Relative Roles of Pro-oxidant Enzymes Nox4 and Nox5 in Diabetic Kidney Disease.
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