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1. Zfp697 is an RNA-binding protein that regulates skeletal muscle inflammation and regeneration.

3. Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition). Autophagy

4. Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition).

5. Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

6. The mitochondrial mRNA-stabilizing protein SLIRP regulates skeletal muscle mitochondrial structure and respiration by exercise-recoverable mechanisms.

7. The 24-hour molecular landscape after exercise in humans reveals MYC is sufficient for muscle growth.

8. Zfp697 is an RNA-binding protein that regulates skeletal muscle inflammation and remodeling.

9. Mustn1 is a smooth muscle cell-secreted microprotein that modulates skeletal muscle extracellular matrix composition.

10. The 24-Hour Time Course of Integrated Molecular Responses to Resistance Exercise in Human Skeletal Muscle Implicates MYC as a Hypertrophic Regulator That is Sufficient for Growth.

11. Sustained PGC-1α2 or PGC-1α3 expression induces astrocyte dysfunction and degeneration.

12. Aerobic exercise training mitigates tumor growth and cancer-induced splenomegaly through modulation of non-platelet platelet factor 4 expression.

14. Zfp697 is an RNA-binding protein that regulates skeletal muscle inflammation and regeneration.

15. Epiblast-like stem cells established by Wnt/β-catenin signaling manifest distinct features of formative pluripotency and germline competence.

17. Impaired oxygen-sensitive regulation of mitochondrial biogenesis within the von Hippel-Lindau syndrome.

18. SnapShot: Regulation and biology of PGC-1α.

19. Muscle-secreted neurturin couples myofiber oxidative metabolism and slow motor neuron identity.

20. Distinct subtypes of proprioceptive dorsal root ganglion neurons regulate adaptive proprioception in mice.

21. β 2 -Adrenergic Signaling Modulates Mitochondrial Function and Morphology in Skeletal Muscle in Response to Aerobic Exercise.

22. Comparative Analysis of Skeletal Muscle Transcriptional Signatures Associated With Aerobic Exercise Capacity or Response to Training in Humans and Rats.

23. Exercise training reverses cancer-induced oxidative stress and decrease in muscle COPS2/TRIP15/ALIEN.

24. PGC-1α isoforms coordinate to balance hepatic metabolism and apoptosis in inflammatory environments.

25. Resistance training in young men induces muscle transcriptome-wide changes associated with muscle structure and metabolism refining the response to exercise-induced stress.

26. Kynurenic Acid and Gpr35 Regulate Adipose Tissue Energy Homeostasis and Inflammation.

27. Effects of N-acetylcysteine on isolated skeletal muscle contractile properties after an acute bout of aerobic exercise.

28. Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure.

29. Exercise training decreases NADPH oxidase activity and restores skeletal muscle mass in heart failure rats.

30. Strength training prior to muscle injury potentiates low-level laser therapy (LLLT)-induced muscle regeneration.

32. Aerobic exercise training rescues cardiac protein quality control and blunts endoplasmic reticulum stress in heart failure rats.

33. Resistance training-induced changes in integrated myofibrillar protein synthesis are related to hypertrophy only after attenuation of muscle damage.

34. Peroxisome Proliferator-activated Receptor γ Coactivator-1 α Isoforms Selectively Regulate Multiple Splicing Events on Target Genes.

35. Akt/mTOR pathway contributes to skeletal muscle anti-atrophic effect of aerobic exercise training in heart failure mice.

37. Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition).

38. NADPH oxidase hyperactivity induces plantaris atrophy in heart failure rats.

39. Autophagy signaling in skeletal muscle of infarcted rats.

40. High- versus moderate-intensity aerobic exercise training effects on skeletal muscle of infarcted rats.

41. Exercise training prevents skeletal muscle damage in an experimental sepsis model.

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