29 results on '"Jang, Young‐Saeng"'
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2. APB-R3, a novel long-acting IL-18 binding protein, inhibits IL-18 activity, and thereby ameliorates NASH progress with reduced liver fibrosis
3. Combined Treatment With TGF-β1, Retinoic Acid, and Lactoferrin Robustly Generate Inducible Tregs (iTregs) Against High Affinity Ligand
4. Retinoic acid enhances lactoferrin-induced IgA responses by increasing betaglycan expression
5. Lactoferrin‐derived chimeric peptide (LFch) strongly boosts TGFβ1‐mediated inducible Treg differentiation possibly through downregulating TCR/CD28 signalling
6. Lactoferrin-derived Chimeric Peptide (LFch) boosts TGFβ1-induced Treg Differentiation by diminishing of TCR Signal intensity via TβRIII
7. Lactoferrin Potentiates Inducible Regulatory T Cell Differentiation through TGF-β Receptor III Binding and Activation of Membrane-Bound TGF-β
8. Lactoferrin‐derived chimeric peptide (LFch) strongly boosts TGFβ1‐mediated inducible Treg differentiation possibly through downregulating TCR/CD28 signalling.
9. IL-4 stimulates mouse macrophages to express APRIL through p38MAPK and two different downstream molecules, CREB and Stat6
10. TGF-β and BAFF derived from CD4+CD25+Foxp3+ T cells mediate mouse IgA isotype switching
11. TGF-β1 stimulates mouse macrophages to express APRIL through Smad and p38MAPK/CREB pathways
12. WED-427 - APB-R3, a novel long-acting IL-18 binding protein, inhibits IL-18 activity, and thereby ameliorates NASH progress with reduced liver fibrosis
13. Lactoferrin modulates BMDCs to be tolerogenic leading to enhancement of Foxp3+iTreg differentiation as well as suppression of T cell proliferation
14. Lactoferrin and TGF-β1 synergize to enhance differentiation of OVA-specific Foxp3+ iTregs
15. Lactoferrin Induces Tolerogenic Bone Marrow-Derived Dendritic Cells
16. Distinct Roles of TβRIII and LRP-1 in Lactoferrin-mediated Foxp3+ Inducible Tregs
17. Mechanism underlying the induction of Foxp3+ regulatory T cells by lactoferrin
18. Murine γδ T Cells Render B Cells Refractory to Commitment of IgA Isotype Switching
19. Mechanism underlying the suppressor activity of retinoic acid on IL4-induced IgE synthesis and its physiological implication
20. Unique Chemokine Profiles of Lung Tissues Distinguish Post-chemotherapeutic Persistent and Chronic Tuberculosis in a Mouse Model
21. Retinoic acid enhances lactoferrin-induced IgA responses by increasing betaglycan expression
22. Oral administration of fermented wild ginseng ameliorates DSS-induced acute colitis by inhibiting NF-κB signaling and protects intestinal epithelial barrier
23. Lactoferrin Combined with Retinoic Acid Stimulates B1 Cells to Express IgA Isotype and Gut-homing Molecules
24. Retinoic acid acts as a selective human IgA switch factor
25. Contrasting Roles of Different Endoglin Forms in Atherosclerosis
26. Retinoic acid, acting as a highly specific IgA isotype switch factor, cooperates with TGF-β1 to enhance the overall IgA response
27. Alum Directly Modulates Murine B Lymphocytes to Produce IgG1 Isotype
28. Lactoferrin Stimulates Mouse Macrophage to Express BAFF via Smad3 Pathway
29. Characterization of Mouse B Lymphoma Cells (CH12F3-2A) for the Study of IgA Isotype Switching
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