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1. ProstaMine: a bioinformatics tool for identifying subtype-specific co-alterations associated with aggressiveness in prostate cancer

2. SIX1 and EWS/FLI1 co-regulate an anti-metastatic gene network in Ewing Sarcoma

3. A harmonized resource of integrated prostate cancer clinical, -omic, and signature features

4. SOPHIE: Generative Neural Networks Separate Common and Specific Transcriptional Responses

5. Down syndrome is associated with altered frequency and functioning of tracheal multiciliated cells, and response to influenza virus infection

6. KDM3A/Ets1 epigenetic axis contributes to PAX3/FOXO1‐driven and independent disease‐promoting gene expression in fusion‐positive Rhabdomyosarcoma

7. High-Content Drug Discovery Targeting Molecular Bladder Cancer Subtypes

8. Crowdsourcing assessment of maternal blood multi-omics for predicting gestational age and preterm birth

9. Trisomy 21 activates the kynurenine pathway via increased dosage of interferon receptors

11. Allele-Specific DNA Methylation and Its Interplay with Repressive Histone Marks at Promoter-Mutant TERT Genes

12. Improved inference of chromosome conformation from images of labeled loci [version 3; peer review: 2 approved]

13. Multivalent Chromatin Engagement and Inter-domain Crosstalk Regulate MORC3 ATPase

14. DREAMTools: a Python package for scoring collaborative challenges [version 2; referees: 1 approved, 2 approved with reservations]

16. Microparticle-Delivered Cxcl9 Prolongs Braf Inhibitor Efficacy in Melanoma

17. Improved inference of chromosome conformation from images of labeled loci [version 2; peer review: 1 approved, 1 approved with reservations]

18. Non–muscle-invasive bladder cancer molecular subtypes predict differential response to intravesical Bacillus Calmette-Guérin

21. Data from Microparticle-Delivered Cxcl9 Prolongs Braf Inhibitor Efficacy in Melanoma

23. Data from Monocytic Subclones Confer Resistance to Venetoclax-Based Therapy in Patients with Acute Myeloid Leukemia

24. Supplementary Tables 1-9, 11, 13-15 from Loss of MAP3K7 Sensitizes Prostate Cancer Cells to CDK1/2 Inhibition and DNA Damage by Disrupting Homologous Recombination

25. Supplementary Figures S1-S5 from Monocytic Subclones Confer Resistance to Venetoclax-Based Therapy in Patients with Acute Myeloid Leukemia

26. Supplementary Figure 1 from RhoC Is an Unexpected Target of RhoGDI2 in Prevention of Lung Colonization of Bladder Cancer

28. Supplementary Figures 1-8 from Mesenchymal and MAPK Expression Signatures Associate with Telomerase Promoter Mutations in Multiple Cancers

30. Supplementary Tables S1-S6 from Monocytic Subclones Confer Resistance to Venetoclax-Based Therapy in Patients with Acute Myeloid Leukemia

33. Supplementary Figure 3 from RhoC Is an Unexpected Target of RhoGDI2 in Prevention of Lung Colonization of Bladder Cancer

34. Data from Loss of MAP3K7 Sensitizes Prostate Cancer Cells to CDK1/2 Inhibition and DNA Damage by Disrupting Homologous Recombination

36. Supplemental Figure Legends from Identification of a Small-Molecule Inhibitor That Disrupts the SIX1/EYA2 Complex, EMT, and Metastasis

37. Figure S6 from SIX2 Mediates Late-Stage Metastasis via Direct Regulation of SOX2 and Induction of a Cancer Stem Cell Program

39. Supplementary Information from The Capacity of the Ovarian Cancer Tumor Microenvironment to Integrate Inflammation Signaling Conveys a Shorter Disease-free Interval

40. Data from SIX2 Mediates Late-Stage Metastasis via Direct Regulation of SOX2 and Induction of a Cancer Stem Cell Program

44. Table S4 from The Capacity of the Ovarian Cancer Tumor Microenvironment to Integrate Inflammation Signaling Conveys a Shorter Disease-free Interval

45. Supplementary Table 1 from Identification of a Small-Molecule Inhibitor That Disrupts the SIX1/EYA2 Complex, EMT, and Metastasis

46. Data from The Capacity of the Ovarian Cancer Tumor Microenvironment to Integrate Inflammation Signaling Conveys a Shorter Disease-free Interval

47. Supplementary Methods from Identification of a Small-Molecule Inhibitor That Disrupts the SIX1/EYA2 Complex, EMT, and Metastasis

48. Data from GON4L Drives Cancer Growth through a YY1–Androgen Receptor–CD24 Axis

49. Supplementary Figures S1-7 from GON4L Drives Cancer Growth through a YY1–Androgen Receptor–CD24 Axis

50. Data from Concurrent Alterations in TERT, KDM6A, and the BRCA Pathway in Bladder Cancer

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