Search

Your search keyword '"Ing, Alvin J."' showing total 34 results
Start Over Author "Ing, Alvin J."
34 results on '"Ing, Alvin J."'

4. Reversal of collateral ventilation using endoscopic polymer foam in COPD patients undergoing endoscopic lung volume reduction with endobronchial valves: A controlled parallel group trial

Author

Ing, Alvin J., primary, Jayapadman, Anand, additional, Kim, Woo‐Veen, additional, Ly, Chelsea, additional, Ho‐Shon, Kevin, additional, Lilburn, Paul, additional, Carew, Alan, additional, Ng, Benjamin J. H., additional, Saghaie, Tajalli, additional, and Williamson, Jonathan P., additional

Published
2022
Full Text
View/download PDF

10. The effect of chronic obstructive pulmonary disease on laryngopharyngeal sensitivity

Author

Clayton, Nicola A., Carnaby-Mann, Giselle D., Peters, Matthew J., and Ing, Alvin J.

Subjects
Pharynx -- Research -- Physiological aspects, Sensory discrimination -- Research -- Analysis, Lung diseases, Obstructive -- Health aspects -- Research, Throat -- Research -- Physiological aspects, Health
Abstract

Abstract Patients with chronic obstructive pulmonary disease (COPD) may be at increased risk of aspiration secondary to impaired swallow function. One possible cause of this impairment is a reduction in [...]

Published
2012

11. Bronchial Rheoplasty for Treatment of Chronic Bronchitis. Twelve-Month Results from a Multicenter Clinical Trial

Author

Valipour, Arschang, primary, Fernandez-Bussy, Sebastian, additional, Ing, Alvin J., additional, Steinfort, Daniel P., additional, Snell, Gregory I., additional, Williamson, Jonathan P., additional, Saghaie, Tajalli, additional, Irving, Louis B., additional, Dabscheck, Eli J., additional, Krimsky, William S., additional, and Waldstreicher, Jonathan, additional

Published
2020
Full Text
View/download PDF

23. Managing cough as a defense mechanism and as a symptom: a consensus report of the American College of Chest Physicians

Author

Irwin, Richard S., Boulet, Louis-Philipe, Cloutier, Michelle M., Fuller, Richard, Gold, Philip M., Hoffstein, Victor, Ing, Alvin J., McCool, F. Dennis, O'Byrne, Paul, Poe, Robert H., Prakash, Udaya B.S., Pratter, Melvin R., and Rubin, Bruce K.

Subjects
American College of Chest Physicians -- Reports, Cough -- Analysis -- Reports, Health, Analysis, Reports
Abstract

SUMMARY AND RECOMMENDATIONS(†) 1. Cough can (a) be an important defense mechanism to help clear excessive secretions and foreign material from airways; (b) be an important factor in the spread [...]

Published
1998

24. Cough and gastroesophageal reflux

Author

Ing, Alvin J.

Subjects
Gastroesophageal reflux -- Complications, Cough -- Causes of, Health, Health care industry
Published
1997

25. The patient with chronic cough

Author

Ing, Alvin J. and Breslin, Antony B. X.

Subjects
Cough -- Care and treatment, Medical history taking -- Health aspects, Health
Abstract

Chronic cough may have underlying causes, such as asthma, gastrointestinal reflux, and chronic sinusitis. The importance of accurate history taking is stressed.

Published
1997

26. Chronic persistent cough and clearance of esophageal acid

Author

Ing, Alvin J., Ngu, Meng C., and Breslin, Antony B.X.

Subjects
Cough -- Causes of -- Physiological aspects, Esophagus -- Injuries -- Physiological aspects, Gastroesophageal reflux -- Physiological aspects, Health, Physiological aspects, Injuries, Causes of
Abstract

Unexplained chronic persistent cough has been shown to be associated with increased episodes of otherwise asymptomatic gastroesophageal reflux; however, normal subjects without cough also exhibit some reflux. We postulate that the prompt clearance of refluxed acid from the esophagus may play an important role in the prevention of cough, and we sought to determine if patients with chronic cough have impaired clearance. Thirty patients with unexplained chronic cough underwent 24-h ambulatory esophageal pH monitoring. Compared to 12 matched control subjects, patients experienced significantly more episodes (all values expressed as median [range]) of reflux per 24 h (88.3 [5.0 to 338.0] vs 5.7 [0 to 13.0]; p, We have previously shown in a controlled study, using 24-h ambulatory esophageal pH monitoring, that chronic persistent cough which remains unexplained after a standard diagnostic evaluation is associated with increased [...]

Published
1992

31. Autosomal dominant hereditary sensory neuropathy with chronic cough and gastro-oesophageal reflux: clinical features in two families linked to chromosome 3p22-p24

Author

Spring, Penelope J, Kok, Cindy, Nicholson, Garth A, Ing, Alvin J, Spies, Judith M, Bassett, Mark, Cameron, John, Kerlin, Paul, Bowler, Simon, Pollard, John H, Tuck, Roger, Spring, Penelope J, Kok, Cindy, Nicholson, Garth A, Ing, Alvin J, Spies, Judith M, Bassett, Mark, Cameron, John, Kerlin, Paul, Bowler, Simon, Pollard, John H, and Tuck, Roger

Abstract

Autosomal dominant hereditary sensory neuropathy (HSN I) is a clinically and genetically heterogeneous group of disorders, and in some families it is due to mutations in the serine palmitoyltransferase (SPTLC1) gene. We have characterized two families with HSN I associated with cough and gastro-oesophageal reflux (GOR). From a large Australian family, 27 individuals and from a smaller family, 11 individuals provided clinical information and blood for genetic analysis. Affected individuals had an adult onset of paroxysmal cough, GOR and distal sensory loss. Cough could be triggered by noxious odours or by pressure in the external auditory canal (Arnold's ear-cough reflex). Other features included throat clearing, hoarse voice, cough syncope and sensorineural hearing loss. Neurophysiological and pathological studies demonstrated a sensory axonal neuropathy. Gastric emptying studies were normal, and autonomic function and sweat tests were either normal or showed distal hypohidrosis. Cough was likely to be due to a combination of denervation hypersensitivity of the upper airways and oesophagus, and prominent GOR. Most affected individuals were shown on 24 h ambulatory oesophageal pH monitoring to have multiple episodes of GOR, closely temporally associated with coughing. Hoarse voice was probably attributable to acid-induced laryngeal damage, and there was no evidence of vocal cord palsy. No other cause for cough was found on most respiratory or otorhinological studies. Linkage to chromosome 3p22-p24 has been found in both families, with no evidence of linkage to loci for known HSN I, autosomal dominant hereditary motor and sensory neuropathy, hereditary GOR or triple A syndrome. These families represent a genetically novel variant of HSN I, with a distinctive cough owing to involvement of the upper aerodigestive tract.

Published
2005

Catalog

Books, media, physical & digital resources
See catalog results