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191 results on '"Infusino, F"'

13. Myval versus alternative balloon- and self-expandable transcatheter heart valves: A central core lab analysis of conduction disturbances

Author

Santos-Martinez, S., Halim, J., Castro-Mejia, A., De Marco, F., Trani, Carlo, Martin, P., Infusino, Fabio, Ancona, M., Moreno, R., den Heijer, P., Nombela-Franco, L., Bedogni, Francesco, Sardella, G., Montorfano, M., Revilla-Orodea, A., Delgado-Arana, J. R., Barrero, A., Gomez-Salvador, I., Ijsselmuiden, A. J. J., Redondo, A., Gutierrez, H., Serrador, A., Serruys, P. W., Roman, J. A. S., Amat-Santos, I. J., Trani C. (ORCID:0000-0001-9777-013X), Infusino F., Bedogni F., Santos-Martinez, S., Halim, J., Castro-Mejia, A., De Marco, F., Trani, Carlo, Martin, P., Infusino, Fabio, Ancona, M., Moreno, R., den Heijer, P., Nombela-Franco, L., Bedogni, Francesco, Sardella, G., Montorfano, M., Revilla-Orodea, A., Delgado-Arana, J. R., Barrero, A., Gomez-Salvador, I., Ijsselmuiden, A. J. J., Redondo, A., Gutierrez, H., Serrador, A., Serruys, P. W., Roman, J. A. S., Amat-Santos, I. J., Trani C. (ORCID:0000-0001-9777-013X), Infusino F., and Bedogni F.

Abstract

Background: Several studies have compared surface electrocardiographic changes following different self-expandable (SE) (Evolut (Medtronic, USA); Acurate (Boston Scientific, USA); Portico (Abbott, USA); and Allegra (NVT, Germany)) and balloon-expandable (BE) Sapien-3 (Edwards Lifesciences, USA) transcatheter heart valves. We aimed to compare these prosthesis with the novel Myval BE prosthesis (Meril Life, India). Methods: Academic European registry of consecutive patients with severe aortic stenosis who received any of the 6 aforementioned valves. Baseline, post-procedural, and discharge 12‐leads electrocardiograms (ECG) were centrally analyzed and compared. Results: A total of 1131 patients were included: 135 Myval (11.9%), 290 Sapien-3 (25.6%), 298 Evolut (26.3%), 180 Acurate (15.9%), 125 Portico (11.1%), and 103 Allegra (9.1%). There were no baseline differences in intraventricular conduction disturbances rate. Compared to the novel BE Myval, there were similar procedural and in-hospital outcomes. Similar rates of early new permanent pacemaker implant (PPI) were observed amongst Myval (7.4%), Sapien-3 (13.4%), and Acurate (9.1%), but Evolut, Portico, and Allegra presented significantly higher rates (18.5%, p = 0.003; 29.5% p < 0.001 and 22%, p = 0.001, respectively). Central analysis of ECGs, unraveled significant prolongation of the PR segment with Evolut, Portico and Allegra whereas Evolut, Acurate, and Portico showed significant QRS widening compared to Myval. However, at discharge no differences in PR segment duration were observed while, Evolut, and Portico– but not Acurate, Allegra or Sapien-3 – still presented significant widening of QRS segment compared to Myval. Conclusions: After blinded central ECG analysis, the novel Myval balloon-expandable prosthesis was associated with a low rate of early conduction disturbances.

Published
2022

14. Fatal Takotsubo syndrome in critical COVID-19 related pneumonia

Author

Titi, L., Magnanimi, E., Mancone, M., Infusino, F., Coppola, G., Del Nonno, F., Colombo, D., Nardacci, R., Falasca, L., d&apos, Amati, G., Tarsitano, M. G., Merlino, L., Fedele, F., and Pugliese, F.

Subjects
0301 basic medicine, medicine.medical_specialty, Resuscitation, Autopsy, 030204 cardiovascular system & hematology, Myocardial rupture, Article, Pathology and Forensic Medicine, 03 medical and health sciences, 0302 clinical medicine, Hematoma, autopsy, Internal medicine, Edema, Medicine, business.industry, Takotsubo Syndrome, Contraction band necrosis, COVID-19, COVID-19 related pneumonia, Stress cardiomyopathy, Aged, 80 and over, Biopsy, COVID-19 Nucleic Acid Testing, Fatal Outcome, Humans, Male, Myocardium, Takotsubo Cardiomyopathy, General Medicine, medicine.disease, Pneumonia, 030104 developmental biology, Coagulative necrosis, Cardiology, stress cardiomyopathy, medicine.symptom, business, Cardiology and Cardiovascular Medicine, COVID-19 related pneumonia, COVID-19
Abstract

Highlights • Takotsubo Syndrome (TTS) is increasingly reported in COVID-19 patients • The pathogenic mechanisms underlying TTS in this particular setting are still unknown • Autopsy findings in fatal cases may contribute to understand the possible role of SARS-CoV-2 in the onset of TTS, COVID-19 can involve several organs and systems, often with indirect and poorly clarified mechanisms. Different presentations of myocardial injury have been reported, with variable degrees of severity, often impacting on the prognosis of COVID-19 patients. The pathogenic mechanisms underlying cardiac damage in SARS-CoV-2 infection are under active investigation. We report the clinical and autopsy findings of a fatal case of Takotsubo Syndrome occurring in an 83-year-old patient with COVID-19 pneumonia. The patient was admitted to Emergency Department with dyspnea, fever and diarrhea. A naso-pharyngeal swab test for SARS-CoV-2 was positive. In the following week his conditions worsened, requiring intubation and deep sedation. While in the ICU, the patient suddenly showed ST segment elevation. Left ventricular angiography showed decreased with hypercontractile ventricular bases and mid-apical ballooning, consistent with diagnosis of Takotsubo syndrome (TTS). Shortly after the patient was pulseless. After extensive resuscitation maneuvers, the patient was declared dead. Autopsy revealed a subepicardial hematoma, in absence of myocardial rupture. On histology, the myocardium showed diffuse edema, multiple foci of contraction band necrosis in both ventricles and occasional coagulative necrosis of single cardiac myocytes. Abundant macrophages CD68+ were detected in the myocardial interstitium. The finding of diffuse contraction band necrosis supports the pathogenic role of increased catecholamine levels; the presence of a significant interstitial inflammatory infiltrate, made up by macrophages, remains of uncertain significance.

Published
2021
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15. Antiinflammatory Therapy with Canakinumab for Atherosclerotic Disease

Author

Ridker PM, Everett BM, Thuren T, MacFadyen JG, Chang WH, Ballantyne C, Fonseca F, Nicolau J, Koenig W, Anker SD, Kastelein JJP, Cornel JH, Pais P, Pella D, Genest J, Cifkova R, Lorenzatti A, Forster T, Kobalava Z, Vida-Simiti L, Flather M, Shimokawa H, Ogawa H, Dellborg M, Rossi PRF, Troquay RPT, Libby P, Glynn RJ, Novo S, Krum H, Varigos J, Siostrzonek P, Sinnaeve P, Gotcheva N, Yong H, Urina-Triana M, Milicic D, Vettus R, Manolis AJ, Wyss F, Sigurdsson A, Fucili A, Veze I, Petrauskiene B, Salvador L, Klemsdal TO, Medina F, Budaj A, Otasevic P, Lainscak M, Seung KB, Commerford P, Donath M, Hwang JJ, Kultursay H, Bilazarian S, East C, Forgosh L, Harris B, Ligueros M, Bohula E, Charmarthi B, Cheng S, Chou S, Danik J, McMahon G, Maron B, Ning M, Olenchock B, Pande R, Perlstein T, Pradhan A, Rost N, Singhal A, Taqueti V, Wei N, Burris H, Cioffi A, Dalseg AM, Ghosh N, Gralow J, Mayer T, Rugo H, Fowler V, Limaye AP, Cosgrove S, Levine D, Lopes R, Scott J, Hilkert R, Tamesby G, Mickel C, Manning B, Woelcke J, Tan M, Manfreda S, Ponce T, Kam J, Saini R, Banker K, Salko T, Nandy P, Tawfik R, O’Neil G, Manne S, Jirvankar P, Lal S, Nema D, Jose J, Collins R, Bailey K, Blumenthal R, Colhoun H, Gersh B, Abreu M, Actis MV, Aiub J, Aiub F, Albisu J, Alvarisqueta A, Avalos V, Barreto M, Berli MA, Blumberg C, Bocanera M, Botta C, Bowen L, Budassi N, Buhlman S, Westberg JC, Carabajal T, Caruso G, Casala J, Cendali G, Coloma G, Berra FC, Cuneo C, Degennaro N, Dellasa M, Diaz M, Dos Santos P, Espinosa V, Facello A, Facello M, Farias E, Fernandez AA, Ferrari V, Pacora FF, Flores GS, Franco M, Gabito A, Viola HG, Garcia F, Garcia Duran R, Garcia Pinna J, Glenny J, Godoy Sanchez M, Grosse A, Guzman P, Hasbani E, Hominal M, Ibañez J, Jure H, Jure D, Vico ML, Liniado G, Luciardi H, Luquez H, Maehara G, Maffei L, Majul C, Mallagray M, Marinaro S, Martinez J, Massaccesi R, De Los Milagros Had M, Azize GM, Montana O, Montenegro E, Morell Y, Muntaner J, Navarrete S, Olmedo M, Paganini M, Paz S, Perez Manghi F, Piskorz D, Polato C, Recoaro R, Romano A, Salinger M, Sanchez A, Saravia MA, Sarjanovich R, Scaro G, Schiavi LB, Soler J, Tinnirello V, Tomassi A, Valle M, Vallejo MA, Venturini C, Marcela Wenetz LM, Yossen M, Zaidman C, Zalazar L, Zangroniz P, Amerena J, Brady L, Colquhoun D, Eccleston D, Ferreira-Jardim A, French J, Jayasinghe R, Mcintosh C, Ord M, Plotz M, Purnell P, Roberts-Thomson P, Schultz C, Shanahan T, Tan R, Taverner P, Turner F, Vibert J, Vorster M, William M, Youssef G, Bergler-Klein J, Brath H, Brodmann M, Fliesser-Goerzer E, Haider K, Heeren G, Hiden C, Mandic L, Paulweber B, Ploechl A, Prenner A, Steringer-Mascherbauer R, Strohner-Kaestenbauer H, Barbato E, Bouvy C, Briké C, Charlier F, Cools F, De Knijf K, De Wolf L, Delforge M, Deweerdt N, Gits F, Goffinet C, Hermans K, Hollanders G, Mestdagh I, Pirenne B, Servaes V, Simons N, Tahon S, Theunissen E, Van Genechten G, Vervoort G, Vissers C, Vranckx P, Vrolix M, Abib E Jr, Abrantes J, Araujo Fonseca M, Barbosa E, Barroso W, Barroso A, Bodanese L, Botelho R, Costa Amorim R, Da Costa F, Da Silva A, Da Silva O Jr, Da Silva D Jr, Ferreira Dos Santos T, Dos Santos F, Dos Santos A, Duda N, Feitosa G, Felario Junior GA, Ferraz R, Filho P, Fonseca A, Wanderley FF, Freitas E, Fucci F, Marengo Garcia De Carvalho L, Hernandez M, Hettwer Magedanz E, Julião K, Kormann A, Lameira A, Lima F, Lino E, Maia L, Manenti E, Marchi AL, Fischer SM, Michalaros Y, Moraes J Jr, Moreira L, Pagnan M, Pesce F, Pinheiro L, Rassi S, Reis G, Reis H, Resende I, Roel A, Ruschel K, Saporito W, Saraiva JF, Seroqui M, Silva R, Unterkircher B, Vicente C, Vieira N, Xavier JP, Zucchetti C, Angelova I, Dimitrov G, Genova D, Gospodinov K, Goudev A, Grigorova V, Hristova K, Makedonska JJ, Katova T, Kostov K, Lazov P, Manov E, Manukov I, Manukov D, Milanova M, Kabakchieva VM, Petrov D, Petrusheva T, Pramatarova I, Raev D, Runev N, Sirakova-Taseva A, Tisheva-Gospodinova S, Todorova A, Tzekova M, Yakovova S, Yanev T, Abulencia K, Arora S, Baker A, Bata IR, Beaudry M, Belle Isle J, Bilodeau N, Boivin MC, Bolduc H, Bourgeois S, Brons S, Cantor W, Chaussé I, Chhabra A, Chouinard G, Cleveland T, Dattani D, Deslongchamps F, Diodati J, Drouin K, Duchesne L, Fontaine S, d'Amours DG, Gervais B, Gosselin G, Graham J, Grover A, Gupta A, Haldane H, Hartleib M, Hickey L, Huynh T, Johnston J, Julien VE, Lachance P, Lake J, Lamontagne C, Lauzon C, Lepage S, Maheux K, Manyari D, Martin E, McPherson C, Mehta S, Michaud N, Kouz SM, Murphy G, OKeefe D, Otis R, Ouimet F, Pandey S, Peck C, Perkins L, Richert L, Robbins K, Robinson S, Cabau JR, Ross B, Roy C, Roy M, Roy A, Rupka D, Affaki GS, Saunders K, Savard D, Soucy D, St Amour E, Thiessen S, Vertes G, Vezina M, Vincelli G, Weisnagel SJ, Zadra R, Chen J, Chen Y, Dong X, Feng Y, Feng Z, Fu G, Han B, Hao Y, He Y, He Z, Hong T, Jia Z, Jiang T, Jiang J, Jiang X, Ke Y, Li Y, Li Z, Li W, Li X, Liu P, Liu Y, Liu B, Liu S, Liu L, Lu Z, Lv Y, Ma C, Ma G, Peng L, Qing L, Ren L, Sang X, Song M, Sun Z, Wang J, Wang Y, Wei J, Wu W, Wu J, Xu H, Yan J, Yang P, Yang K, Yao Z, Yaoqing H, Yuan Z, Zhai Z, Zhang J, Zhang Y, Zhao R, Zhou H, Accini Mendoza JL, Aparicio CV, Castillo T, Chaverra I, Conrado Y, Coronel J, Cotes C, Cuentas I, Cuervo A, Dussan MA, Echeverria L, Hernandez E, Ibarra J, Isaza D, Jimenez D, Lopez P, Manzur F, Mejia I, Mendoza Y, Molina DI, Patino JM, Rodriguez D, Rodriguez LM, Rodriguez SM, Sanchez Vallejo G, Luz Serrano H, Sotomayor A, Urina M, Vesga B, Yupanqui H, Akrap B, Busic N, Ciglenecki N, Cmrecnjak J, Fucak E, Gabor M, Jeric M, Jutrisa N, Kordic K, Planinc I, Popovic Z, Radeljic V, Sesto I, Sutalo K, Tusek S, Belohlavek J, Budkova J, Busak L, Capova L, Cech V, Cermak O, Coufalova Z, Cyprian R, Dedek V, Dedkova S, Ferkl R, Hanak P, Hanustiakova A, Homza M, Horackova K, Houra M, Iveta H, Kaiserova L, Kala P, Karel I, Kellnerova I, Koleckar P, Kreckova M, Krupicka J, Lorenc Z, Machova V, Malik J, Masarikova L, Matyasek I, Mikus M, Mikusova T, Ondrasik J, Otava M, Palubova L, Pavlickova L, Peterka M, Petrova I, Pokorna B, Povolny P, Radvan M, Reznakova S, Rickova Z, Roszkowska P, Rotreklova M, Samkova D, Skalicka H, Slechticka A, Sternthal P, Telekes P, Tesak M, Vesely P, Vesely J, Vins P, Vitovec M, Vodnansky P, Zidova M, Keba E, Laane E, Pool T, Randvee L, Ratnik E, Reimand M, Reinmets S, Rivis L, Siemann M, Stern M, Toom M, Vahula V, Apel T, Axthelm C, Ayasse D, Ayasse M, Baar M, Baeumer A, Bagi ES, Becker B, Binder A, Blankenberg S, Braun P, Johansen BB, Contzen C, Delfonso F, Denecke C, Dengler T, Donaubauer T, Eichinger G, Englmann E, Erhard M, Faghih M, Foerster A, Frankenstein L, Fuchs R, Furch G, Gaeb-Strasas B, Germann H, Giese C, Goette A, Gravenhorst-Muenter U, Haege R, Haenel T, Hagemann D, Hagenow A, Hanefeld M, Heider J, Heisters J, Hennig D, Hielscher S, Himpel-Boenninghoff A, Holscher A, Hornig M, Jeserich M, Kaczmarek N, Kanitz S, Kara YD, Khariouzov A, Kiefer R, Kiroglu K, Klamm M, Klein C, Korth-Wiemann B, Krapivsky A, Kuenzler J, Kuntzsch A, Landers B, Lappo M, Laube S, Leggewie S, Lehmann D, Lepp H, Lierse T, Lindner C, Luecke-Uzar M, Luedemann J, Marschke T, Maruzzo S, Mauersberger K, Maus O, Meinrich M, Meissner A, Moehring B, Muehlhaus J, Mueller S, Muenter KC, Muenzel T, Naumann R, Nebel J, Neumann J, Nuding S, Overhoff U, Papke B, Pencz I, Peter Y, Peukert AM, Radde I, Rau T, Regner S, Reichenbach D, Reimer D, Rinke A, Roettges R, Romanski A, Rummel R, Samer H, Sanuri M, Sarnighausen HE, Schäfer B, Scheibner T, Schermaul KH, Schindler A, Schlundt C, Schmidt E, Schmidt K, Schnabel A, Schoen N, Schorn K, Schroeder T, Schulenburg D, Schulz M, Schulze U, Schulze J, Schumacher M, Schwerin G, Schwerin M, Stadelmeier S, Stahl HD, Stahl A, Stockhausen J, Stockhausen G, Stoessel J, Stolze K, Stratmann M, Szymanowski N, Teschner AB, Teske A, Uecker C, Veit S, Voeller H, Walter I, Walter J, Walther I, Weber HG, Weimer J, Wichterich K, Wiebusch A, Willmerdinger M, Willner C, Winkelmann B, Winkler J, Wistuba T, Woehrle J, Wohnlich T, Wolf S, Woyczak D, Wrage P, Zirlik A, Anadiotis A, Chachalis G, Dermitzakis A, Kafarakis P, Kaldara E, Kolokathis F, Kostakou P, Lekakis J, Manolis A, Mantas I, Megalou A, Milkas A, Nanas J, Olympios CD, Patsilinakos S, Perperis A, Poulimenos L, Saloustros I, Tsioufis K, Tsorbatzoglou K, Vardas P, Zarifis I, Aguilar M, Arango JL, Borrayo NA, Corona V, Guerrero A, Guzman I, Haase F, De Krumbach L, Montenegro P, Munoz R, Munoz N, Paniagua A, Solares A, Vogel M, Anita S, Blazsek Z, Decsi K, Fulop T, Hangyal T, Hegedus V, Kalina A, Karakai H, Katona A, Kiss RG, Kovacs A, Laszlo Z, Lupkovics G, Medvegy M, Merkely B, Mihaly N, Nagy AC, Dékány JN, Nikoletta P, Noori E, Penzes J, Poor F, Sarszegi Z, Simay A, Simon J, Szakal I, Szatmarine V, Szocs A, Zilahi Z, Karsai XZ, Andersen K, Sigurdadottir E, Skuladottir F, Abdullakutty J, Abhaichand R, Abhyankar A, Agarwal DK, Aggarwal RK, Ahire N, Awasthi AK, Babu R, Bai A, Bali HK, Banker D, Bhadade S, Bisne V, Bohra P, Raghu C, Chauhan D, Chauhan H, Chavada J, Chaware G, Chella S, Chintala P, Dash D, Desai D, Devasia T, Dhanak R, Dobariya H, Dudhatra N, Duhan S, Fulwani M, Ghondale N, Ghosh S, Gohel P, Govindaraj D, Goyal B, Goyal S, Gundala AK, Gupta M, Hardas S, Iby M, Jagtap P, Jain A, Joshi U, Karpuram M, Kaur H, Khan A, Khan R, Kodem DR, Koeitti P, Kulkarni L, Kullal P, Kumar KS, Kumbla M, Latheef K, Lohkare M, Santosh MJ, Makhe B, Mandati M, Mehta A, Minocha G, Mittal A, Modi R, Mohan K, Oomman A, Pai R, Pai V, Palaniswami N, Pansheriya A, Parekh N, Patel J, Patel R, Patole T, Praveen M, Radhakrishnan V, Rajan B A, Rajasekhar D, Rao M, Rao MB, Rao NM, Rathnavel S, Rathore A, Rathore SRS, Rawat S, Reddy NC, Sarma R, Sathe S, Shah J, Shaikh P, Sharma K, Sharma S, Sharma T, Shetty P, Sidhu G, Singh V, Sohi GS, Srinath VS, Raju SS, Taran A, Thakkar B, Velusamy K, Vijan V, Vora V, Vuriya AK, Agosta GF, Antonicelli R, Ardissino D, Argiolas G, Baldin MG, Benedetti G, Berti S, Bevilacqua MT, Bolognesi MG, Dessalvi CC, Calabrese A, Campanale EG, Candusso R, Caso P, Cosmi F, Crea F, Crocamo A, De Caterina R, De Rosa S, Destro M, Di Biase M, Dognini GP, Eleuteri E, Fedele F, Ferrario M, Gabrielli D, Gamba C, Ganau A, Gravellone M, Iannopollo G, Indolfi C, Infusino F, Invitti C, Landolfi A, Lembo G, Liberato NL, Mannucci E, Marino P, Mariottoni B, Marziali A, Mercuro G, Monti L, Mos L, Mureddu V, Musumeci MB, Panzarino C, Parente A, Perotti M, Filardi PP, Petrillo C, Piatti P, Priori S, Racca V, Ragghianti B, Renda G, Righini V, Sarcone M, Senni M, Soro E, Tamburrini P, Vallone L, Villani GQ, Volpe M, Ajioka M, Akai Y, Ashino K, Baden M, Doi M, Eki Y, Endo T, Fukuike C, Hagiwara Y, Hasegawa K, Higuchi Y, Higuchi T, Hioki M, Hirayama A, Hiroma J, Hosokawa S, Ichisawa M, Iijima T, Inada T, Inagaki M, Ito K, Kaigawa K, Kajihara S, Kamiya H, Kamiya J, Kaneno Y, Katahira K, Kataoka M, Kawai M, Kawasaki T, Kojima E, Komura Y, Kuramochi T, Kuruma T, Kyo E, Mani H, Miyamoto T, Morii I, Morinaga Y, Morisawa T, Nagai Y, Naka T, Nakamura Y, Nakamura S, Nakayoshi K, Nishibe A, Ogawa M, Okada Y, Okawa M, Sakamoto Y, Sakurada M, Sasaki S, Seki S, Shimomura H, Shinozaki T, Sugimoto N, Suzuki A, Taguchi S, Takahashi J, Takase S, Tanabe K, Tanaka A, Tani S, Tomioka J, Tsuboi H, Tsuji M, Tsujita K, Tsujiyama S, Umesu A, Yamada T, Yamaguchi E, Yamamoto H, Yamamoto T, Yamane M, Yanase T, Yasuoka S, Yasutake M, Yokoyama M, Yoshida M, Yoshimoto E, Yunoki C, Balode A, Dormidontova G, Flaksa I, Nagele-Luse I, Rancane G, Sime I, Bartuseviciene S, Cepinskiene L, Dobilas V, Grigaraviciene I, Marcinkeviciene J, Mazutavicius R, Miliuniene R, Motiejuniene R, Norkiene S, Norkute-Macijauske U, Rudys A, Slapikas R, Stonkute K, Strazdiene D, Tijuneliene E, Urbonas G, Vanagiene S, Viezelis M, Arenas Leon JL, Bayram E, Carrillo J, Davalos C, De Los Rios M, Delgadillo T, Hernández N, Leon S, Mendoza N, Muñoz W, Ramos G, Anneveldt A, Bakker H, Brouwer M, Bunschoten P, De Boer P, De Jong C, De Vos A, Den Hartog F, Doesborg L, Dommerholt R, Drost I, Ellenbroek D, Engelen W, Folkeringa RJ, Hamer BJB, Herrman JP, Hoogslag PAM, Jansen M, Jerzewski A, Joosten C, Kalkman C, Kietselaer B, Kok M, Kooiman E, Kose V, Lardinois R, Lenderink T, Lok DJA, Lousberg A, Meijlis P, Mulder R, Singerling M, Smeele F, Stroes E, Swart HP, Ten Holt W, Van Der Wal M, Van Der Zwaan C, Van Kempen WW, Van Maarseveen M, Van Stein I, Viergever EP, Visseren FLJ, Voors C, Nugteren SKZ, Ata B, Berulfsen A, Rønnevik TD, Dickstein K, Furuseth B, Grundtvig M, Hansen H, Hofsoey K, Høivik HO, Bøen RH, Hurtig U, Pettersen KI, Johansen E, Kleve R, Kolleroy C, Moen S, Nilsen V, Norin V, Otterstad JE, Risberg K, Rønnevik P, Sirnes PA, Skjelvan G, Strand S, Szacinski G, Vegsundvåg J, Alcalde JM, Gomez Sanchez J, Rodriguez J, Rodriguez A, Zena N, Baszak J, Cymerman K, Czerski T, Fratczak M, Jaguszewska G, Kawka-Urbanek T, Koba M, Kopaczewski J, Kopczyńska M, Laniec M, Lysek R, Sciborski R, Szpajer M, 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M., Everett, B. M., Thuren, T., Macfadyen, J. G., Chang, W. H., Ballantyne, C., FONSECA E PIRES, CARLOS EDUARDO, Nicolau, J., Koenig, W., Anker, S. D., Kastelein, J. J. P., Cornel, J. H., Pais, P., Pella, D., Genest, J., Cifkova, R., Lorenzatti, A., Forster, T., Kobalava, Z., Vida-Simiti, L., Flather, M., Shimokawa, H., Ogawa, H., Dellborg, M., Rossi, P. R. F., Troquay, R. P. T., Libby, P., Glynn R., J, CANTOS Trial, Group, Perrone, Filardi, P, ACS - Amsterdam Cardiovascular Sciences, Vascular Medicine, ACS - Pulmonary hypertension & thrombosis, and ACS - Atherosclerosis & ischemic syndromes

Subjects
0301 basic medicine, 030204 cardiovascular system & hematology, law.invention, 0302 clinical medicine, c-reactive protein, Randomized controlled trial, law, Cardiovascular Disease, middle aged, double-blind method, antibodies, Myocardial infarction, humans, Stroke, interleukin-1beta, biology, Antibodies, Monoclonal, drug, General Medicine, Lipid, Aged, anti-inflammatory agents, monoclonal, humanized, atherosclerosis, cardiovascular diseases, dose-response relationship, female, incidence, infections, lipids, male, myocardial infarction, neutropenia, secondary prevention, stroke, Anti-Inflammatory Agent, aged, Editorial, Atherosclerosi, Monoclonal, Human, medicine.drug, medicine.medical_specialty, Neutropenia, Antibodies, Monoclonal, Humanized, Infections, Placebo, antibodies, monoclonal, dose-response relationship, drug, infection, medicine (all), 03 medical and health sciences, Internal medicine, medicine, Dose-Response Relationship, Drug, business.industry, Antiinflammatory Therapy, Canakinumab, for Atherosclerotic Disease, C-reactive protein, medicine.disease, Surgery, Canakinumab, 030104 developmental biology, biology.protein, business
Abstract

Background: Experimental and clinical data suggest that reducing inflammation without affecting lipid levels may reduce the risk of cardiovascular disease. Yet, the inflammatory hypothesis of atherothrombosis has remained unproved. Methods: We conducted a randomized, double-blind trial of canakinumab, a therapeutic monoclonal antibody targeting interleukin-1β, involving 10,061 patients with previous myocardial infarction and a high-sensitivity C-reactive protein level of 2 mg or more per liter. The trial compared three doses of canakinumab (50 mg, 150 mg, and 300 mg, administered subcutaneously every 3 months) with placebo. The primary efficacy end point was nonfatal myocardial infarction, nonfatal stroke, or cardiovascular death. RESULTS: At 48 months, the median reduction from baseline in the high-sensitivity C-reactive protein level was 26 percentage points greater in the group that received the 50-mg dose of canakinumab, 37 percentage points greater in the 150-mg group, and 41 percentage points greater in the 300-mg group than in the placebo group. Canakinumab did not reduce lipid levels from baseline. At a median follow-up of 3.7 years, the incidence rate for the primary end point was 4.50 events per 100 person-years in the placebo group, 4.11 events per 100 person-years in the 50-mg group, 3.86 events per 100 person-years in the 150-mg group, and 3.90 events per 100 person-years in the 300-mg group. The hazard ratios as compared with placebo were as follows: in the 50-mg group, 0.93 (95% confidence interval [CI], 0.80 to 1.07; P = 0.30); in the 150-mg group, 0.85 (95% CI, 0.74 to 0.98; P = 0.021); and in the 300-mg group, 0.86 (95% CI, 0.75 to 0.99; P = 0.031). The 150-mg dose, but not the other doses, met the prespecified multiplicity-adjusted threshold for statistical significance for the primary end point and the secondary end point that additionally included hospitalization for unstable angina that led to urgent revascularization (hazard ratio vs. placebo, 0.83; 95% CI, 0.73 to 0.95; P = 0.005). Canakinumab was associated with a higher incidence of fatal infection than was placebo. There was no significant difference in all-cause mortality (hazard ratio for all canakinumab doses vs. placebo, 0.94; 95% CI, 0.83 to 1.06; P = 0.31). Conclusions: Antiinflammatory therapy targeting the interleukin-1β innate immunity pathway with canakinumab at a dose of 150 mg every 3 months led to a significantly lower rate of recurrent cardiovascular events than placebo, independent of lipid-level lowering. (Funded by Novartis; CANTOS ClinicalTrials.gov number, NCT01327846.)

Published
2017

16. Coronary Artery Aneurysms Presenting as Acute Coronary Syndrome: An Unusual Case of IgG4-Related Disease Vascular Involvement

Author

Ruggio, A, Iaconelli, A, Panaioli, E, Bernardini, F, Tinelli, G, Savino, G, Infusino, F, Leccisotti, L, Manna, R, Crea, F., Ruggio A, Iaconelli A, Panaioli, Elena, Bernardini, Federica, Tinelli G (ORCID:0000-0002-2212-3226), Savino G, Infusino F, Leccisotti L (ORCID:0000-0002-6000-2898), Manna R (ORCID:0000-0003-1560-3907), Crea F. (ORCID:0000-0001-9404-8846), Ruggio, A, Iaconelli, A, Panaioli, E, Bernardini, F, Tinelli, G, Savino, G, Infusino, F, Leccisotti, L, Manna, R, Crea, F., Ruggio A, Iaconelli A, Panaioli, Elena, Bernardini, Federica, Tinelli G (ORCID:0000-0002-2212-3226), Savino G, Infusino F, Leccisotti L (ORCID:0000-0002-6000-2898), Manna R (ORCID:0000-0003-1560-3907), and Crea F. (ORCID:0000-0001-9404-8846)

Abstract

We report the case of a 65-year-old man referred to our department because of a complex coronary aneurysmal disease presenting as subacute ST-segment elevation myocardial infarction due to intraluminal thromboembolism. The patient's past medical history (recurrent episodes of lymphadenopathies, parotid swelling, prostatitis, and dacryoadenitis) raised the suspicion of a unifying systemic disorder with coronary involvement. An extensive infectious and autoimmune screening was performed, leading to the final diagnosis of IgG4-related disease. Our case highlights the importance to include this rare and recently described disease in the diagnostic workup of acquired coronary aneurysms., Nous rapportons le cas d'un homme âgé de 65 ans qui a été orienté vers notre service en raison d'une maladie coronarienne anévrismale complexe caractérisée par un infarctus subaigu du myocarde avec élévation du segment ST imputable à une thrombose intraluminale. Les antécédents médicaux de ce patient (épisodes récurrents de lymphadénopathie, d'enflure de la glande parotide, de prostatite et de dacryoadénite) ont amené à soupçonner une maladie systémique unificatrice avec atteinte des coronaires. Un dépistage exhaustif des maladies infectieuses et auto-immunes a été effectué et a conduit au diagnostic final de maladie associée aux IgG4. Le cas que nous rapportons met en lumière l'importance d'inclure cette maladie rare et de description récente dans l'analyse diagnostique des anévrismes acquis des coronaires.

Published
2018

17. Successful Transcatheter Treatment of Left Pulmonary Artery to Left Atrium Communication Diagnosed in Adulthood

Author

Lillo, R., Graziani, Francesca, Burzotta, Francesco, Locorotondo, G., Aurigemma, Cristina, Romagnoli, Elisa, Bibiana Delogu, A., Grandinetti, M., Panaioli, E., Scacciavillani, Roberto, Infusino, Fabio, Savino, G., Massetti, Massimo, Trani, Carlo, Graziani F. (ORCID:0000-0002-4520-5689), Burzotta F. (ORCID:0000-0002-6569-9401), Aurigemma C., Romagnoli E., Scacciavillani R., Infusino F., Massetti M. (ORCID:0000-0002-7100-8478), Trani C. (ORCID:0000-0001-9777-013X), Lillo, R., Graziani, Francesca, Burzotta, Francesco, Locorotondo, G., Aurigemma, Cristina, Romagnoli, Elisa, Bibiana Delogu, A., Grandinetti, M., Panaioli, E., Scacciavillani, Roberto, Infusino, Fabio, Savino, G., Massetti, Massimo, Trani, Carlo, Graziani F. (ORCID:0000-0002-4520-5689), Burzotta F. (ORCID:0000-0002-6569-9401), Aurigemma C., Romagnoli E., Scacciavillani R., Infusino F., Massetti M. (ORCID:0000-0002-7100-8478), and Trani C. (ORCID:0000-0001-9777-013X)

Abstract

Successful Transcatheter Treatment of Left Pulmonary Artery to Left Atrium Communication Diagnosed in Adulthood

Published
2020

22. Micro and macro models of granular computing induced by the indiscernibility relation

Author

Bisi, C, Chiaselotti, G, Ciucci, D, Gentile, T, Infusino, F, Infusino, F., CIUCCI, DAVIDE ELIO, GENTILE, TOMMASO, Bisi, C, Chiaselotti, G, Ciucci, D, Gentile, T, Infusino, F, Infusino, F., CIUCCI, DAVIDE ELIO, and GENTILE, TOMMASO

Abstract

In rough set theory (RST), and more generally in granular computing on information tables (GRC-IT), a central tool is the Pawlak's indiscernibility relation between objects of a universe set with respect to a fixed attribute subset. Let us observe that Pawlak's relation induces in a natural way an equivalence relation ≈ on the attribute power set that identifies two attribute subsets yielding the same indiscernibility partition. We call indistinguishability relation of a given information table I the equivalence relation ≈, that can be considered as a kind of global indiscernibility. In this paper we investigate the mathematical foundations of indistinguishability relation through the introduction of two new structures that are, respectively, a complete lattice and an abstract simplicial complex. We show that these structures can be studied at both a micro granular and a macro granular level and that are naturally related to the core and the reducts of I. We first discuss the role of these structures in GrC-IT by providing some interpretations, then we prove several mathematical results concerning the fundamental properties of such structures.

Published
2017

23. Locally finite complexes, modules and generalized information systems.

Author

Chiaselotti, G. and Infusino, F.

Subjects
*ROUGH sets, *INFORMATION storage & retrieval systems, *FINITE, The, *COMPUTATIONAL mathematics
Abstract

Simplicial complexes (here briefly complexes) are set systems on an arbitrary set which are object of study in many areas of both mathematics and theoretical computer science. Usually, they are investigated over finite sets. However, in general, when we consider an arbitrary set Ω (not necessarily finite) and a complex on Ω , the most natural property related to finiteness is the following: for any subset X of Ω , if F ∈ for all finite subsets F of X , then X ∈ . We call locally finite any complex having such a property. Bearing in mind some motivations and constructions derived from the analysis of information systems in rough set theory, in this paper we associate with any locally finite complex a corresponding pre-closure operator σ and, through it, we establish several properties of . Next, we investigate the main features of the specific sub-class of locally finite complexes for which σ is a closure operator. We call these complexes closable and exhibit a particular family of closable locally finite complexes using left-modules on rings with identity. Finally, we establish a representation result according to which we can associate a pairing structure with any closable locally finite complex. [ABSTRACT FROM AUTHOR]

Published
2022
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25. Prevention of Cardiovascular Disease: Screening for Magnesium Deficiency

Author

Severino, P., Netti, L., Mariani, M. V., Maraone, A., D'Amato, A., Scarpati, R., Infusino, F., Pucci, M., Lavalle, C., Maestrini, V., Mancone, M., Fedele, F., and Salzano, A.

Subjects
lcsh:Diseases of the circulatory (Cardiovascular) system, ischemia, Disease, Review Article, magnesium, 030204 cardiovascular system & hematology, Bioinformatics, prevention, cardiovascular disease, Sudden cardiac death, Coronary artery disease, 03 medical and health sciences, 0302 clinical medicine, Diabetes mellitus, Magnesium deficiency (medicine), medicine, Endothelial dysfunction, 030304 developmental biology, 0303 health sciences, business.industry, medicine.disease, lcsh:RC666-701, Metabolic syndrome, Cardiology and Cardiovascular Medicine, business, Dyslipidemia
Abstract

Magnesium is an essential mineral naturally present in the human body, where it acts as cofactor in several enzymatic reactions. Magnesium is a key cardiovascular regulator, which maintains electrical, metabolic, and vascular homeostasis. Moreover, magnesium participates in inflammation and oxidative processes. In fact, magnesium deficiency is involved in the pathophysiology of arterial hypertension, diabetes mellitus, dyslipidemia, metabolic syndrome, endothelial dysfunction, coronary artery disease, cardiac arrhythmias, and sudden cardiac death. In consideration of the great public-health impact of cardiovascular disease, the recognition of the negative effects of magnesium deficiency suggests the possible role of hypomagnesaemia as cardiovascular risk factor and the use of serum magnesium level for the screening and prevention of cardiovascular risk factors and cardiovascular diseases. Moreover, it might help with the identification of new therapeutical strategies for the management of cardiovascular disease through magnesium supplementation.

Published
2019

26. Myocardial ischemia in women when genetic susceptibility matters

Author

Severino, P, D’Amato, A, Pucci, M, Mariani, Mv, Netti, L, Infusino, F, Mancone, M, and Fedele, F

Subjects
coronary microvascular dysfunction, gender, ischemic heart disease, coronary artery disease, ion channels, KATP, females
Published
2019

28. The granular partition lattice of an information table

Author

Chiaselotti, G, Ciucci, D, Gentile, T, Infusino, F, Infusino, F., CIUCCI, DAVIDE ELIO, GENTILE, TOMMASO, Chiaselotti, G, Ciucci, D, Gentile, T, Infusino, F, Infusino, F., CIUCCI, DAVIDE ELIO, and GENTILE, TOMMASO

Abstract

In this paper we study the lattice of all indiscernibility partitions induced from attribute subsets of a knowledge representation system (information table in the finite case). This lattice, that we here call granular partition lattice, is a very well studied order structure in granular computing and data base theory and it provides a complete hierarchical classification of the knowledge obtained from all possible choices of attribute subsets. We show that it has a lattice structure also in the infinite case and we provide several isomorphic characterizations for this lattice. We discuss the potentiality of this order structure from both a micro-granular and a macro-granular perspective. Furthermore, the sub-poset of all the indiscernibility closures needed to determine when an arbitrary partition is an indiscernibility one is studied. Finally, we show the monotonic behaviour of the granular partition lattice with respect to entropy of partitions and attribute dependency in decision tables.

Published
2016

29. Generalizations of rough set tools inspired by graph theory

Author

Chiaselotti, G, Ciucci, D, Gentile, T, Infusino, F, Infusino, F., CIUCCI, DAVIDE ELIO, GENTILE, TOMMASO, Chiaselotti, G, Ciucci, D, Gentile, T, Infusino, F, Infusino, F., CIUCCI, DAVIDE ELIO, and GENTILE, TOMMASO

Abstract

We introduce and study new generalizations of some rough set tools. Namely, the extended core, the generalized discernibility function, the discernibility space and the maximum partitioner. All these concepts where firstly introduced during the application of rough set theory to graphs, here we show that they have an interesting and useful interpretation also in the general setting. Indeed, among other results, we prove that reducts can be computed in incremental polynomial time, we give some conditions in order that a partition coincides with an indiscernibility partition of a given information table and we give the conditions such that a discernibility matrix corresponds to an information table.

Published
2016

33. Rough set theory applied to simple undirected graphs

Author

Ciucci, D, Wang, G, Mitra, S., Wu, WZ, Chiaselotti, G, Gentile, T, Infusino, F, Infusino, F., CIUCCI, DAVIDE ELIO, Ciucci, D, Wang, G, Mitra, S., Wu, WZ, Chiaselotti, G, Gentile, T, Infusino, F, Infusino, F., and CIUCCI, DAVIDE ELIO

Abstract

The incidence matrix of a simple undirected graph is used as an information table. Then, rough set notions are applied to it: approximations, membership function, positive region and discernibility matrix. The particular cases of complete and bipartite graphs are analyzed. The symmetry induced in graphs by the indiscernibility relation is studied and a new concept of generalized discernibility matrix is introduced.

Published
2015

34. Preclusivity and simple graphs: The n–cycle and n–path cases

Author

Yao,Y, Hu, Q, Yu, H, Grzymala-Busse, JW, Chiaselotti, G, Ciucci, D, Gentile, T, Infusino, F, CIUCCI, DAVIDE ELIO, Infusino, F., Yao,Y, Hu, Q, Yu, H, Grzymala-Busse, JW, Chiaselotti, G, Ciucci, D, Gentile, T, Infusino, F, CIUCCI, DAVIDE ELIO, and Infusino, F.

Abstract

Two classes of graphs, the n–cycles and n–paths, are interpreted as preclusivity spaces. In this way, it is possible to define two pairs of approximations on them: one based on a preclusive relation and another one based on a similarity relation. Further, two relations can be defined among the set of vertices and they define two different graphs, which are here studied.

Published
2015

35. Preclusivity and simple graphs

Author

Yao, Y, Hu, Q, Yu, H, GrzymalaBusse, JW, Chiaselotti, G, Ciucci, D, Gentile, T, Infusino, F, CIUCCI, DAVIDE ELIO, Infusino, F., Yao, Y, Hu, Q, Yu, H, GrzymalaBusse, JW, Chiaselotti, G, Ciucci, D, Gentile, T, Infusino, F, CIUCCI, DAVIDE ELIO, and Infusino, F.

Abstract

The adjacency relation of a simple undirected graph is a preclusive (irreflexive and symmetric) relation. Hence, it originates a preclusive space enabling us to define the lower and upper preclusive approximations of graphs and two orthogonality graphs. Further, the possibility of defining the similarity lower and upper approximations and the sufficiency operator on graphs will be investigated, with particular attention to complete and bipartite graphs. All these mappings will be put in relation with Formal Concept Analysis and the theory of opposition.

Published
2015

36. Some classifications of graphs with respect to a set adjacency relation.

Author

Chiaselotti, G., Gentile, T., and Infusino, F. G.

Subjects
BIPARTITE graphs, UNDIRECTED graphs, TENSOR products, COMPLETE graphs, PATHS & cycles in graph theory, COMPUTATIONAL complexity
Abstract

For any finite simple undirected graph G = (V (G) , E (G)) , we consider the binary relation ← G on the powerset 𝒫 (V (G)) of its vertex set given by B ← G A if ⋂ v ∈ B N G (v) ⊇ ⋂ v ∈ A N G (v) , where N G (v) denotes the neighborhood of a vertex v. We call the above relation set adiacence dependency (sa)-dependency of G. With the relation ← G we associate an intersection-closed family C L s a (G) of vertex subsets and the corresponding induced lattice ℭ (G) : = (C L s a (G) , ⊆) , which we call sa-lattice of G. Through the equality of sa-lattices, we introduce an equivalence relation ≡ s a between graphs and propose three different classifications of graphs based on such a relation. Furthermore, we determine the sa-lattice for various graph families, such as complete graphs, complete bipartite graphs, cycles and paths and, next, we study such a lattice in relation to the Cartesian and the tensor product of graphs, verifying that in most cases it is a graded lattice. Finally, we provide two algorithms, namely, the T-DI ALGORITHM and the O-F ALGORITHM, in order to provide two different computational ways to construct the sa-lattice of a graph. For the O-F ALGORITHM we also determine its computational complexity. [ABSTRACT FROM AUTHOR]

Published
2021
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40. Rough set theory and digraphs

Author

Chiaselotti, G, Ciucci, D, Gentile, T, Infusino, F, Chiaselotti, G, Ciucci, D, Gentile, T, and Infusino, F

Abstract

In this paper we apply rough set theory to information tables induced from finite directed graphs without loops and multiples arcs (digraphs). Specifically, we use the adjacency matrix of a digraph as a particular type of information table. In this way, we are able to explore on digraphs the notions of indiscernibility partitions, lower and upper approximations, generalized core, reducts and discernibility matrix. All these ideas will be exemplified on standard digraph families as well on examples from social networks and patterns of flight routes between airports.

Published
2017

43. SYMMETRY GEOMETRY BY PAIRINGS.

Author

CHIASELOTTI, G., GENTILE, T., and INFUSINO, F.

Subjects
MATHEMATICAL symmetry, GEOMETRY, CLOSURE operators, SET theory, PARTITIONS (Mathematics)
Abstract

In this paper, we introduce a symmetry geometry for all those mathematical structures which can be characterized by means of a generalization (which we call pairing) of a finite rectangular table. In more detail, let $\unicode[STIX]{x1D6FA}$ be a given set. A pairing $\mathfrak{P}$ on $\unicode[STIX]{x1D6FA}$ is a triple $\mathfrak{P}:=(U,F,\unicode[STIX]{x1D6EC})$ , where $U$ and $\unicode[STIX]{x1D6EC}$ are nonempty sets and $F:U\times \unicode[STIX]{x1D6FA}\rightarrow \unicode[STIX]{x1D6EC}$ is a map having domain $U\times \unicode[STIX]{x1D6FA}$ and codomain $\unicode[STIX]{x1D6EC}$. Through this notion, we introduce a local symmetry relation on $U$ and a global symmetry relation on the power set ${\mathcal{P}}(\unicode[STIX]{x1D6FA})$. Based on these two relations, we establish the basic properties of our symmetry geometry induced by $\mathfrak{P}$. The basic tool of our study is a closure operator $M_{\mathfrak{P}}$ , by means of which (in the finite case) we can represent any closure operator. We relate the study of such a closure operator to several types of others set operators and set systems which refine the notion of an abstract simplicial complex. [ABSTRACT FROM AUTHOR]

Published
2019
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50. Endothelial and Platelet Function in Children With Previous Kawasaki Disease

Author

Laurito, M, Stazi, A, Delogu, Angelica Bibiana, Milo, M, Battipaglia, I, Scalone, G, Infusino, F, Villano, A, Russo, G, Iannotta, R, Saracino, A, Parrinello, R, Figliozzi, S, Sestito, A, Romagnoli, Costantino, Lanza, Ga, Crea, F., Delogu, Angelica Bibiana (ORCID:0000-0002-2283-3180), Romagnoli, Costantino (ORCID:0000-0003-1176-2943), Laurito, M, Stazi, A, Delogu, Angelica Bibiana, Milo, M, Battipaglia, I, Scalone, G, Infusino, F, Villano, A, Russo, G, Iannotta, R, Saracino, A, Parrinello, R, Figliozzi, S, Sestito, A, Romagnoli, Costantino, Lanza, Ga, Crea, F., Delogu, Angelica Bibiana (ORCID:0000-0002-2283-3180), and Romagnoli, Costantino (ORCID:0000-0003-1176-2943)

Abstract

We investigated whether children with a previous Kawasaki disease (KD) have evidence of abnormal vascular and/or platelet function. We included 14 patients with previous KD and 14 matched controls. We assessed endothelial function by flow-mediated dilation (FMD), carotid intima-media thickness (cIMT), coronary microvascular function by coronary blood flow response (CBFR) to cold pressor test, and platelet reactivity by measuring monocyte-platelet aggregates (MPAs) and CD41-platelet expression by flow cytometry. No differences were found between the groups in FMD, cIMT, or CBFR to cold pressor test. The MPAs were similar in patients with KD and controls. CD41-platelet expression, however, was significantly increased in patients with KD compared with controls, both at rest (14.3 ± 1.9 vs 12.4 ± 1.9 mean fluorescence intensity [mfi], P = .01) and after adenosine diphosphate stimulation (19.3 ± 1.3 vs 17 ± 1.7 mfi, P < .001). In conclusion, children with a previous episode of KD showed increased platelet activation, compared with healthy participants despite no apparent vascular abnormality at follow-up.

Published
2013

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