166 results on '"Iekushi, Kazuma"'
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2. Relationship Between Left Ventricular Ejection Fraction and Treatment Characteristics in Hospitalized Patients With Heart Failure ― A Japanese Database Analysis ―
3. Predictive factors for bleeding during treatment with rivaroxaban and warfarin in Japanese patients with atrial fibrillation – Subgroup analysis of J-ROCKET AF
4. Twenty‐Four‐Hour Blood Pressure‐Lowering Efficacy of Sacubitril/Valsartan Versus Olmesartan in Japanese Patients With Essential Hypertension Based on Nocturnal Blood Pressure Dipping Status: A Post Hoc Analysis of Data From a Randomized, Double‐Blind Multicenter Study
5. Role of Serotonin in Angiogenesis in Diabetes
6. PS-C19-5: SAFETY PROFILES OF SACUBITRIL/VALSARTAN IN EARLY POST-MARKETING PHASE VIGILANCE FOR THE INDICATION OF CHRONIC HEART FAILURE AND HYPERTENSION
7. Achievement Rates for Low-Density Lipoprotein Cholesterol Goals in Patients at High Risk of Atherosclerotic Cardiovascular Disease in a Real-World Setting in Japan
8. Present Profiles of Novel Anticoagulant Use in Japanese Patients with Atrial Fibrillation: Insights from the Rivaroxaban Postmarketing Surveillance Registry
9. Net Clinical Benefit of Rivaroxaban versus Warfarin in Japanese Patients with Nonvalvular Atrial Fibrillation: A Subgroup Analysis of J-ROCKET AF
10. Rivaroxaban versus Warfarin in Japanese Patients with Nonvalvular Atrial Fibrillation in Relation to the CHADS2 Score: A Subgroup Analysis of the J-ROCKET AF Trial
11. Role of serotonin in angiogenesis: Induction of angiogenesis by sarpogrelate via endothelial 5-HT1B/Akt/eNOS pathway in diabetic mice
12. Selective Blockade of Periostin Exon 17 Preserves Cardiac Performance in Acute Myocardial Infarction
13. MicroRNA-34a regulates cardiac ageing and function
14. P147 EFFICACY AND SAFETY OF SACUBITRIL/VALSARTAN VERSUS AMLODIPINE IN JAPANESE PATIENTS WITH ESSENTIAL HYPERTENSION: A RANDOMIZED, OPEN-LABEL, MULTICENTER STUDY (PARASOL STUDY).
15. Acute myocardial infarction activates progenitor cells and increases Wnt signalling in the bone marrow
16. HGF as Angiogenic Factor and Therapeutic Approach
17. P1866TO INVESTIGATE THE EFFICACY AND SAFETY OF MOLIDUSTAT IN NON-DIALYSIS PATIENTS WITH RENAL ANEMIA WHO ARE NOT TREATED WITH ERYTHROPOIESIS-STIMULATING AGENTS: MIYABI ND-C
18. P1868TO INVESTIGATE THE EFFICACY AND SAFETY OF MOLIDUSTAT IN NON-DIALYSIS PATIENTS WITH RENAL ANEMIA WHO ARE TREATED WITH ERYTHROPOIESIS-STIMULATING AGENTS: MIYABI ND-M
19. Molidustat for the treatment of renal anaemia in patients with dialysis-dependent chronic kidney disease: design and rationale of three phase III studies
20. Molidustat for the treatment of renal anaemia in patients with non-dialysis-dependent chronic kidney disease: design and rationale of two phase III studies
21. Iron Regulation by Molidustat, a Daily Oral Hypoxia-Inducible Factor Prolyl Hydroxylase Inhibitor, in Patients with Chronic Kidney Disease
22. Long-Term Efficacy and Safety of Molidustat for Anemia in Chronic Kidney Disease: DIALOGUE Extension Studies
23. Erratum to: ‘Oral rivaroxaban for Japanese patients with symptomatic venous thromboembolism – the J-EINSTEIN DVT and PE program’
24. Inhibition of Lp(a)-induced functional impairment of endothelial cells and endothelial progenitor cells by hepatocyte growth factor
25. Oral rivaroxaban for Japanese patients with symptomatic venous thromboembolism – the J-EINSTEIN DVT and PE program
26. Clinical development of novel oral anticoagulant (NOAC), Rivaroxaban
27. Abstract 056: The Impact of Periostin as A Regulator of Cardiac Remodeling
28. Selective Blockade of Periostin Exon 17 Preserves Cardiac Performance in Acute Myocardial Infarction.
29. Novel mechanism of hepatocyte growth factor against prevention of inflammation and oxidative stress
30. Hepatocyte Growth Factor Inhibits Lipopolysaccharide-Induced Oxidative Stress via Epithelial Growth Factor Receptor Degradation
31. Elevated Levels of the Mediator of Catabolic Bone Remodeling RANKL in the Bone Marrow Environment Link Chronic Heart Failure with Osteoporosis
32. Therapeutic Option of Plasmid-DNA Based Gene Transfer
33. 97 VALSARTAN ATTENUATES LP(A)-INDUCED SEVERE NEOINTIMAL HYPERPLASIA
34. 741 CARDIAC AND RENAL PROTECTIVE EFFECTS OF HEPATOCYTE GROWTH FACTOR (HGF), INDUCED BY IRBESARTAN IN MOUSE MODEL OF SALT-SENSITIVE HYPERTENSION
35. Response to Hepatocyte Growth Factor and Cardiomyopathy in Dialysis Patients
36. 917 ROLE OF SEROTONIN IN ANGIOGENESIS
37. 940 COMBINED EFFECTS OF ANGIOTENSIN II RECEPTOR ANTAGONIST AND CALCIUM CHANNEL BLOCKER IN HYPERTENSIVE PATIENTS WITH TYPE 2 DIABETES-
38. Micro-RNA-34a Contributes to the Impaired Function of Bone Marrow-Derived Mononuclear Cells From Patients With Cardiovascular Disease
39. Hepatocyte Growth Factor Reduces Cardiac Fibrosis by Inhibiting Endothelial-Mesenchymal Transition
40. Regulation of Cardiac MicroRNAs by Bone Marrow Mononuclear Cell Therapy in Myocardial Infarction
41. The HGF/c-Met Receptor System Under Pathological Conditions
42. Comparison between Two Calcium Antagonists: Blood Pressure Reduction and Renal Effects in Hypertensive Patients with Type 2 Diabetes
43. Telmisartan Exerts Renoprotective Actions via Peroxisome Proliferator-Activated Receptor-γ/Hepatocyte Growth Factor Pathway Independent of Angiotensin II Type 1 Receptor Blockade
44. Hepatocyte Growth Factor Attenuates Transforming Growth Factor-β-Angiotensin II Crosstalk Through Inhibition of the PTEN/Akt Pathway
45. Hepatocyte growth factor attenuates renal fibrosis through TGF-β1 suppression by apoptosis of myofibroblasts
46. Hepatocyte Growth Factor Reduces Cardiac Fibrosis Through Inhibiting Endothelial-Mesenchymal Transition and Proliferation of Myofibroblast
47. Mechanism of Improved Cardiac Function After Bone Marrow Mononuclear Cell Therapy
48. Sox2 Transduction Enhances Cardiovascular Repair Capacity of Blood-Derived Mesoangioblasts
49. Preconditioning by toll-like receptor 2 agonist Pam3CSK4 reduces CXCL1-dependent leukocyte recruitment in murine myocardial ischemia/reperfusion injury*
50. Abstract 5122: Hepatocyte Growth Factor Attenuates Renal Fibrosis Through Myofibroblasts Anoikis Induced by FAK/MMP Activaton
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