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1. Phosphorylation of phase‐separated p62 bodies by ULK1 activates a redox‐independent stress response

3. p62/SQSTM1-droplet serves as a platform for autophagosome formation and anti-oxidative stress response

6. Autophagy regulates lipid metabolism through selective turnover of NCoR1

7. Phosphorylation of phase-separated p62 bodies by ULK1 activates a redox-independent stress response

10. Crucial role for autophagy in degranulation of mast cells

11. Development of P62-Keap1 Protein-Protein Interaction Inhibitors as Doxorubicin-Sensitizers Against Non-Small Cell Lung Cancer

20. Atg8, a Ubiquitin-like Protein Required for Autophagosome Formation, Mediates Membrane Tethering and Hemifusion

21. Autophagy Protects Integrity of Tumor Suppressors From Replication Stress

24. Inhibitors of the protein–protein interaction between phosphorylated p62 and Keap1 attenuate chemoresistance in a human hepatocellular carcinoma cell line

25. A ubiquitin-like system mediates protein lipidation

27. Biallelic UFM1 and UFC1 mutations expand the essential role of ufmylation in brain development

32. Development of Novel Inhibitors for Keap1-Nrf2 and Keap1-P62 Protein-Protein Interaction

33. Autophagy linked FYVE (Alfy/WDFY3) is required for establishing neuronal connectivity in the mammalian brain

34. Biallelic Variants in UBA5 Link Dysfunctional UFM1 Ubiquitin-like Modifier Pathway to Severe Infantile-Onset Encephalopathy

35. Author response: Autophagy linked FYVE (Alfy/WDFY3) is required for establishing neuronal connectivity in the mammalian brain

36. p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming

37. Structural and Functional Analysis of a Novel Interaction Motif within UFM1-activating Enzyme 5 (UBA5) Required for Binding to Ubiquitin-like Proteins and Ufmylation

38. Structural determinants in GABARAP required for the selective binding and recruitment of ALFY to LC 3B‐positive structures

40. ASC1/RAS2 Suppresses the Growth Defect on Glycerol Caused by the atp1–2 Mutation in the Yeast Saccharomyces cerevisiae

42. Phosphorylation of p62 Activates the Keap1-Nrf2 Pathway during Selective Autophagy

45. The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1

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