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ABSTRACTThe structural stability of the hepatitis C model with the proliferation of infected and uninfected hepatocytes is investigated in this paper. The model is clinically verified to accurately reflect the viral dynamics of hepatitis C. It is demonstrated that the limit transition from the hepatitis C model to the Riccati system coupled with the diffusive terms leads to the elimination of the quadratic terms. Such a novel effect leads to the introduction of the concept of the deformed order-1 solitary solutions. The generalized operator of differentiation is used to construct the deformed order-1 solitary solutions to the Riccati system coupled with the diffusive terms. Finally, it is demonstrated that the Riccati system coupled with the diffusive terms admits non-deformed order-1 solitary solutions, which proves the structural instability of the hepatitis C model with the proliferation of infected and uninfected hepatocytes.

A scheme for the integration of $ \, {}^{C} \mathit{\boldsymbol{{D}}}^{(1/n)} $-type fractional differential equations (FDEs) is presented in this paper. The approach is based on the expansion of solutions to FDEs via fractional power series. It is proven that $ \, {}^{C} \mathit{\boldsymbol{{D}}}^{(1/n)} $-type FDEs can be transformed into equivalent $ \left(\, {}^{C} \mathit{\boldsymbol{{D}}}^{(1/n)}\right)^n $-type FDEs via operator calculus techniques. The efficacy of the scheme is demonstrated by integrating the fractional Riccati differential equation.