11 results on '"I. Losano"'
Search Results
2. Extracorporeal septoplasty with internal nasal valve stabilisation
- Author
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I. Tasca, G. Ceroni Compadretti, T. I. Losano, Y. Lijdens, and C. Boccio
- Subjects
Otorhinolaryngology ,RF1-547 - Published
- 2018
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3. Poster session Wednesday 11 December all day display: 11/12/2013, 09:30-16:00 * Location: Poster area
- Author
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S. Bosi, K. Wierzbowska-Drabik, W. Mullens, C. Goh, M. Abdel Ghany, J. Monmeneu, M. Perez Guillen, B. Zaborska, A. Di Lenarda, H. Mueller, M. Dluzniewski, R. Del Pozo Contreras, E. Laraudogoitia Zaldumbide, S. Yurdakul, O. Bech-Hanssen, M. Fernandez Garcia, R. Ippolito, C. Torromeo, B. Popescu, M. Cameli, P. Gaudron, M. Salvetti, R. Amano, E. Osto, P. Cabeza Lainez, G. Generati, C.H. Attenhofer Jost, J. Rueda-Soriano, F. Negri, T. Zielinski, M. El Serafi, Y. Agmon, I. Losano, Y. Qin, I. Castiglione, G. Santambrogio, A. Farhati, P. Menasche, K. Wdowiak-Okrojek, R. Juneja, G. Di Sciascio, N. Gaibazzi, D. Shin, F. Romeo, O. Huttin, P. Puddu, I. Ikonomidis, T. Baran, G. Tinica, A. Bel Minguez, E. Erdogan, M. Herruzo Rojas, I. Ter Horst, J. Suarez De Lezo, P. Bertrand, B. Putnikovic, O. Kretschmar, M. Gigli, F. Scholz, M. Lainscak, O. Rifaie, E. Tahirovic, A. Svanadze, G. Makavos, L. Iliuta, L. G. T. Zacharias, M. Baldelli, A. Porto, C. Di Nora, O. Asghar, A. Ramalli, W. Krol, M. Ahmed Abdel-Rahman, R. Autschbach, R. Tripodi, A. Budaj, V. Velagic, J. Kurcz, J. Aguilar, V. V. Kochmasheva, O. Enescu, H. Triantafyllidi, J. Diago, J. Park, J. Breur, F. Tona, M. Cikes, C. Maurea, T. Edvardsen, B. Igual Munoz, B. Michalski, J. Separovic Hanzevacki, A. Hagege, L. Gullestad, P. Sogaard, T. Fritz-Hansen, M. Rosca, A. Kuch-Wocial, C. Gonzalez Canovas, H. Uyarel, M. Guazzi, E. Pigatto, M. Carminati, R. Soyka, P. Tortoli, D. Djordjevic-Radojkovic, D. De Palma, L. Yuan, E. Mazzotta, M. Henein, D. Botezatu, J. Feng, L. Casteilla, C. Vignati, I. Burazor, M. Krestjyaninov, E. Zhdanova, D. Milicic, H. Mahfoudhi, A. Aziz, P. Trivilou, R. Hoffmann, A. Mysiak, C. Martini, K. Haugaa, F J V M Francisco Jose Valera Martinez, J. Lessick, M. Maccherini, C. Olympios, D. Mutlak, B. Haugen, M. Martin, A. Santoro, A. Orda, B. Skoric, S. Mihaila, M. Jung, G. Leenders, A. Bozkurt, M. Greco, M. Muratori, A. Subinas Elorriaga, Z. Radunovic, A. Osa-Saez, Z. Suciu, M. Alloni, F. Alamanni, J. Choi, J. Schwartz, M. Mericskay, M. Gurzun, D. Leone, P. Omede, J. Sawicki, D. Saura Espin, E. W. Remme, M. Bando, M. Varoudi, J. Gonzalez Carrillo, G. F. Gjerdalen, S. Aakhus, T. Bombardini, F. Veglio, L. Baduena, A. Calin, B. Austin, C. Viacroze, S. Aytekin, C. Santoro, I. Benedek, S. Comenale Pinto, F. Verbrugge, G. Styczynski, M. Sunbul, N. Pandian, T. Forster, J. Hisdal, S. Mondillo, M. Mourali, L. Magda, A. Quesada-Carmona, E. Caiani, G. Pavlidis, S. Ojeda, W. Ding, S. Ramakrishnan, L. Stefanczyk, A. Voumbourakis, A. M. Maceira Gonzalez, B. Igual, C. Selton-Suty, O. B. Kerbikov, B. Karolko, P. Lipiec, F. Meijboom, T. Andersen, M. Pellegrino, M. Lopez-Lereu, J. Kasprzak, Y. Zhao, R. Lang, M. Valdés Chávarri, J. Muir, A. Goetzenich, J. Hooper, M. Driessen, M. Greutmann, S. Casablanca, V. Curci, P. Szymanski, M. Cramer, F. Tosello, C. Gronlund, M. Chiavarelli, A. Cuvelier, P. Mogutova, F. Bandera, G. Greil, P. Fernandez Garcia, E.R. Valsangiacomo Buechel, M. Sobczynska, M. Kennedy, S. Boitard, D. Voilliot, H. Bellsham-Revell, A. Casacalenda, M. Sata, P. J. Sanchez Millan, S. Nishio, C. Chrisochoou, S. Mirfeizi, C. Beladan, K. Steine, M. Lisi, N. Krylova, A. Vlahovic-Stipac, S. Carerj, A. Oxenius, B. Geloneze, R. Calabro, E. Occhetta, P. Caso, R. Massey, B. Cengiz, M. Palencia-Perez, X. Xu, S. Brili, A. Evangelista, D. Mesa, S. Abadi, V. Reskovic Luksic, G. De La Morena Valenzuela, M. Anzini, S. Iliceto, A. Saxena, D. Vinereanu, G. Ussia, M. Sikora-Frac, S. Censi, R. Razavi, T. Wakatsuki, M. Romero, L. Punzi, C. Stefanadis, M. Pepi, E. Chueca Gonzalez, D. Rea, R. Chistol, I. Michalowska, N. Hayes, J. D'hoge, H. Aloui, D. Verhaert, I. Lekuona Goya, O. Sklyanna, K. Taamallah, S. Urheim, B. Natali, G. Sieswerda, M. Casartelli, D. Czarnecka, K. Lagerstrand, T. Chamova, E. Solberg, L. Sabia, M. Vatankulu, M. Obremska, D. Stolfo, H. Haouala, G. Bajraktari, G. Oria Gonzalez, I. Tournev, N. Olsen, O. R. Coelho, F. Spano, J. Yip, M. Anastasiou-Nana, A. Montero Argudo, S. Poli, J.-M. Sellal, P. Kulakowski, K. Kawecka-Jaszcz, O. Sonmez, M. Merlo, A. Chiru, A. Moreo, A. Colombo, R. Dahmani, W. Fehri, V. Rameev, D. Liu, A. Olszanecka, G. Placha, N. Kouris, A. Zaroui, J. Ljubas, G. Famoso, A. Massoni, S. Gao, M. Delgado, I Rodriguez Sanchez, R. Vazquez Garcia, D. Peluso, V. Planat-Benard, J. Cosin-Sales, E. Avenatti, V. Karidas, G. Sinagra, B. Jako, E. Alfonzetti, C. Hernandez Acuna, H. Farouk, D. Foley, M. Chmiela, P. Gripari, G. Patti, J. C. Pareja, Y. Hwang, C. Polte, D. Damaskos, D. Aronson, T. Rechcinski, T. Soeki, D. Simon, D. Anderson, N. Maurea, A. Brunet, C. Florescu, M. Marchei, A. Safarova, F. Cozzi, A. Neskovic, S. Mega, V. Miro-Palau, K. Darahim, B. Bednarz, A. Bitto, F. De Stefano, E. Kostarska-Srokosz, A. Nemes, G. Vizzari, T. Leiner, N. E. Hasselberg, P. Maffei, F. Mezni, Z. Bogdanovic, S. Kul, W. Kosmala, M. Rivero-Ayerza, G. Piscopo, M. Schiariti, V. Cammalleri, V. Kostopoulos, S. Storve, S. Stoerk, I. Planinc, B. Mutlu, J. R. M. Souza, J. J. Onaindia Gandarias, V. Donghi, H. Hamdi, G. Bagadur, A. Mabrouk Salem Omar, M. Floria, A. Klisiewicz, G. Barbati, A. Akhundova, A. Cacicedo, M. Annabi, D. Domingo-Valero, J. Simpson, J. Suarez De Lezo Herreros De Tejada, F. Cesana, D. Sergi, G. Alongi, M. Coppola, L. Grieten, G. Woo, L. Badano, G. Ertl, L. Caballero Jimenez, E. Donal, A. Kalapos, A. Anna Klisiewicz, H. Duengen, F. Mazuelos, U. Aguirre Larracoechea, N. Hasselberg, P. Domsik, L. Fusini, Z. Rezine, M. Misailidou, M. Rodriguez Serrano, D. Waterhouse, K. Keramida, F. Procaccio, G. Dell'era, N. Popova, F. Musumeci, D. Presutti, S F de Marchi, J. Van 'T Sant, S. Moisseyev, K. Paraskevopoulos, L. Molano, J. Estornell Erill, M. Gaspari, Z. Kobalava, I. Jedrzejewska, M. Galderisi, S. Neubauer, Piotr Hoffman, U. Cucchini, O. Miller, W. Kong, A. Swiatowiec, M. Vrolix, C. Grattoni, K. Broch, P. Ibrahimi, M. Garcia Navarro, R. Sheahan, P. Hoffman, M. Boratynska, J. Castillo Ortiz, R. Jankovic-Tomasevic, S. Wijers, P. Lindqvist, C. Tiu, V De Francesco, C. Goffredo, P. Agostoni, H. Yamada, V. Varano, T. Al-Maimoony, P. Wester, P. Schoof, J. Son, P. Piotr Szymanski, F. Righini, O. Agbulut, P. Nardinocchi, A. Aljalloud, I. Stankovic, O. A. Smiseth, L. Halmai, A. Bacaksiz, S. Rayasamudra, D. Filipiak, D. Muraru, D. Zysko, S. Muscoli, O. Goktekin, M. Przewlocka-Kosmala, S. Ryu, Z. Baricevic, M. Meine, J. Monmeneu Menadas, L. Gheorghe, A. Cremonesi, M. Lipczynska, A. Chaim, M. K. Smedsrud, M.D. Espinosa García, S. Mbarki, I. Stamatopoulos, L. Ling, F. Jashari, Y. Juilliere, D. Lahidheb, B. Mcadam, B. Bijnens, B. Pezo Nikolic, V. Guergueltcheva, J.-P. Vallee, O. Erdogan, R. Muscariiello, R. Mincu, M. Deljanin Ilic, C. Coppola, F. Arenga, J. Walker, M. Bono, J. Segura, R. Mechmeche, H. Uppal, S. Hayashi, A. Alhadad, M. Klinger, S. Herrmann, S. Snare, J. Estornell, M. Grecu, L. Lukasz Mazurkiewicz, J. Hotchi, C. Cipresso, E. Esposito, T. Marwick, N. Poteshkina, C. Zito, A. Squeri, V. Razin, I. Paraskevaidis, M. Jemaa, R. Marcun, R. Potluri, A. Anton Ladislao, F. Buendia-Fuentes, M. Pavlovic, S. Salinger-Martinovic, B. Igual-Munoz, T. Seoane, K. Mischke, G. Tamborini, G. Kim, A. Kardos, G. Pizzino, C. Matei, N. Hatam, V. P. Dityatev, H. Torp, A. Degiovanni, F. Rigo, M. Janulewicz, M. Gospodinova, M. Pan, P. Vallerio, F. Gaita, X. Jin, M. Akkaya, B. Pinamonti, A. Javanbakht, B. Lamia, N. A. Yaroshchuk, L. Musial-Bright, W. J. Nadruz, I. Papadakis, G. Kunszt, Y. Hirata, A. Shim, P. Maciejewski, M. Oliva Sandoval, S. Kadivec, E. Pilichowska-Paszkiet, F. Ranocchi, H. Neametalla, K. Hu, I. Sari, F. Carrasco, R. Ancona, R. Weber, R. Ivanova, A. Bartorelli, K. Eskesen, L. Teresi, P. Lopez Lereu, A. Holmgren, M. Kosnik, M. Turfan, M. Sobieszczanska-Malek, E. Kongsgaard, A. Bell, G. Hong, S. Denchev, A. Tasal, D. Mihalcea, F. Weidemann, G. De Caridi, A. Haggui, N. Hajlaoui, P. Alonso-Fernandez, A. Quattrone, M. Massetti, W. Braksator, I. Lekakis, T. Sahin, S. Carasso, F. Dassie, C. Bucca, C. Ginghina, C. A. Szmigielski, J. Baran, Z. Li, E. Aliot, A. Milan, J. Farkas, C. Smeets, D. Stanojevic, H. Dalen, S. Apostolovic, C. Moretti, G. Bruno, X. Zhao, E. Christoforatou, C. Arra, H. Poorzand, J. Ruvira, R. Matasic, F. Maffessanti, T. Vaugrenard, E. Szymczyk, R. Gimaev, S. Tellatin, C. Magnino, S. Velasco Del Castillo, P. Vandervoort, P. Doevendans, A. Dragan, M. Florescu, D. Carballo, P. Marino, D. Lovric, J. Nilson, L. Tong, H. Khorshid, R. Enache, A. Ruck, T. Benedek, R. Winter, M. Ruiz Ortiz, and E. Johansson
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medicine.medical_specialty ,business.industry ,medicine ,Radiology, Nuclear Medicine and imaging ,Medical physics ,General Medicine ,Session (computer science) ,Cardiology and Cardiovascular Medicine ,business - Published
- 2013
4. Poster session: Dobutamine stress echo
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C. Vizza, X. Jeanrenaud, M. Satendra, L. Monti, A. Kovacs, D. Vanoli, V. Charles, A. Kardos, M. Chiarlo, C. Gruner, C. Monaco, A. Kuch-Wocial, K. Mizia-Stec, L. Sargento, D. Kautznerova, N. Resseguier, G. Tamborini, C. Cruz, P. Jurzak, A. Cogo, E. Lira, D. Al Mesned, Y. Aizawa, A. Chmiel, R. Corti, K. Kim, G. N. Elkilany, M. Haarman, R. Badagliacca, R. Weber, R. Bruno, L. Di Pino, I. Kaplanis, D. Kalimanovska-Ostric, D. Tsounis, M. Varoudi, H. Yoon, P. Goncalves, G. Mpitsios, I. Garcia-Lunar, P. Min, R. Mogelvang, K. Gieszczyk-Strozik, A. Blundo, A. Tarr, E. W. Remme, David Garcia-Dorado, V. Petronilli, A. Patel, C. Sousa, R. M. Lang, J. Mcghie, V. Monivas Palomero, C. Nomura, H. Yoshikawa, N. Lagopati, M. Gomberg-Maitland, R. Kalil, H. K. Jeon, K. Mrabet, A. Riberi, C. Zito, A. Khalatbari, D. Tarasov, L. Fusini, P. Marques, S. Hassantash, I. Zimbarra Cabrita, M. Francone, A. Germain, A. Theron, J. Sousa, A. Kantorova, F. Collart, C. J. Vrints, A. Forteza, C. Tamburino, D. Cerna, S. Buccheri, M. Taborsky, I. P. Monte, F. Elmkies, A. Castro Beiras, S. Ranjbar, A. Perpinia, O. A. Tolba, R. Pretre, T. Chua, F. Fedele, M. Calcagnino, D. Dragulescu, M. Greutmann, M. Pepi, M. Bartesaghi, S. Urheim, R. Muscariello, F. Ben Moussa, W. Saib, M. Thameur, J. Ternacle, V. Matzraki, M. Ghannouchi, G. Kocabay, A. Margulescu, R. Sicari, R. Ippolito, M. Kloeckner, A. Toth, J. Gonzalez Mirelis, K. Sugi, M. Geleijnse, T. Otsuka, A. Hervold, S. Benyoussef, B. Basnyat, H. Suomi, L. Gargani, M. Stosic, P. Monney, J. Segovia Cubero, M. Karvandi, P. Sousa, J. Gonzalez-Mirelis, P. Caso, M. Murata, M. Vieira, C. Fulcheri, M. Júlia Maciel, P. Garcia-Pavia, M. Bobbo, J. Sun, B. Nardi, V. Pyrgakis, J. W. Kim, F. Alamanni, D. Ozel, A. Cordovil, S. Cimino, S. Papa, A. Carro, E. Leiballi, S. Karakas, J. Cho, C. Mornos, H. Masai, M. D'angelo, S. Mingo Santos, J. Kang, N. Nishiyama, J. Brugada, W. Tsang, Y. Yoon, B. Herzog, F. Dominguez Rodriguez, G. Ertl, E.R. Valsangiacomo Buechel, H. Shin, M. Palinsky, P. Gaudron, O. Gaemperli, A. Bouzas Mosquera, R. Bogle, J. Rodriguez-Palomares, N. Liel-Cohen, J. Burrello, M. Henein, H. Yilmaz, M. Laine, C. Foucher, K. Tanimoto, P. Schiattarella, G. Teixido, V. Schiano Lomoriello, M. R. El-Shanshory, N. Lousada, T. Minarik, F. Machado, G. Hashimoto, Y. Ishikawa, P. Atkinson, I. Zairi, B. Lee, V. Lanska, T. Biering-Sorensen, D. Vinereanu, H. Dores, M. Nakamura, R. Kockova, A. Chenzbraun, A. Manrique, N. A. Garcia, C. Zimmermann, L. Carpinteiro, H. Youn, J. Guimaraes, P. Meimoun, M. Mohammed, A. Gaspar, G. Styczynski, M. Castella, R. Esposito, A. Karavidas, F. Tosello, J. Mills, J. E. Sanderson, Y. Lau, D. Lee, C. Chin, M. Dostanic, D. Liu, P. Lupinek, T. Sato, M. Lewis, M. Reali, E. Cervesato, A. Apor, D. Sharif, S. Leggio, T. Ono, S. Wos, S. Kadrabulatova, S. Miyoshi, B. Milakovic, M. Gonzalez-Alujas, Y. Y. Lam, W. Tietge, M. Tramarin, L. Balzarini, E.-S. El-Hawary, G. E. Nagib Elkilany, P. Lim, P. Lindqvist, F. Veronesi, G. Flahaut, M. Thomas, A. Redheuil, Y. Ahn, M. Galderisi, M. Cavero Gibanel, J. Roquette, G. D. Lenders, F. Cicogna, P. Nihoyannopoulos, S. Taddei, C. Shahla, O. Mirea, A. Aleixo, E. Altekin, A. Milan, J. Roncalli, V. Mor-Avi, P. Crapanzano, S. Wang, A. Rodrigues, D. De Palma, M. Sitges, J. Peteiro, G. Maldonado, A. Nagy, J. Wang, M. Miglioranza, M. J. Claeys, J. Kluin, R. H. Strasser, J. Masura, B. Pezzuto, S. Aakhus, M C De Knegt, F. Broullon, N. Bhave, Y. Kusunose, R. Domburg, S. Moral Torres, J. Song, G. Carlomagno, P. Carrilho-Ferreira, A. Mornos, K. Sedlacek, Y. Villain, S. Arapi, M. Segetova, T. Le Tourneau, M. Kucuk, H. Tsuruta, J.-L. Monin, L. Badano, C. Mueller, C. Jorge, J. Kautzner, U. Schubert, L. Zhong, B. Suran, J. Clerc, I. Demir, S. Chamuleau, P. Tittel, E. Boussabah, P. Punjabi, L. Guimaraes, C. Magnino, B. Delasalle, D. Leone, J. Gruenenfelder, H. Blafield, F. Thuny, J. Jensen, J. Silva Cardoso, S. Stoebe, S. Sioua, K. Fukuda, M. Nocioni, P. Linden, V. Sanchez, D. Silva, V. Sikula, F. Pizzino, L. Kryze, A. Lebreiro, M. Deljanin-Ilic, A. Arsenio, S. Takatsuki, M. Kaldararova, A. Sikora-Puz, M. Cinello, S. Naffati, M. Pirscova, V. Lisignoli, A. Hagendorff, T. Iwaki, M. Niemann, E. Rees, U. Rosenschein, V. Vrsanska, C. A. Szmigielski, G. L. Nicolosi, G. Di Bella, D. Pfeiffer, R. Giorgi, K. Korpi, E. Paucca, M. Sanchez Garcia, S. Kammoun, M. Rodolico, Arturo Evangelista, I. Baka, J. Lima, C. Yu, B. Hong, C. Fischer, P. Morera, F. C. Tanner, R. Manganaro, M. Mezzapesa, B. Seifert, A. Berruezo, H. Guterman, K. Sveric, U. Wiklund, R. Sant'anna, R. Piazza, L. Oreto, L. Mont, J. Rosso, B. P. Paelinck, S. Severino, J. Park, S. Morhy, S. Mingo, A. Ledakowicz-Polak, L. Arcari, E. E.-S. El-Hawary, E. Caiani, R. Fabregas Casal, A. Bensaid, N. A. E.-A. El-Shitany, F. Veglio, L. Gutierrez, R. Massey, R. Mimo, A. Yanikoglu, A. Al Akhfash, J. Rodriguez Garrido, S. Kovalova, N. Patrascu, M. Liu, B. Bijnens, J.-L. Dubois-Rande, M. Suzuki, I. Garcia Lunar, D. Muraru, S. Iwanaga, R. Borras, R. Karpov, T. Nastasovic, T. Gonzalez-Alujas, M. Jasinski, H. Marques, W. Voelker, D. Maan Hasson, K. Murbrach, J. Yoon, M. Cusma-Piccione, S. Carerj, E. Hopp, D. A. Rees, M. Zielinska, M. Forkmann, M. Sotiropoulos, I. Zegri, Y. Neuder, V. Hraska, R. Iengo, I. Losano, P. Gripari, J. Avierinos, I. Simkova, M. Yaacobi, F. Weidemann, C. Sordelli, H. Jeong, T. Osaki, M. Kubanek, R. Sharma, M. Yamamoto, D. Bettex, J. Sivertsen, G. Bruno, A E Van Den Bosch, P. Kracht, P. L. Van Herck, J. Roos-Hesselink, D. Cozma, E. Teiger, L. Said, B. Freed, A. Loimaala, T. Pinho, L. Pomidori, A. Mantovani, A. Santoro, R. Kadour, R. Calabro, S. Rim, L. Sim, B. Merkely, P. Gueret, R. Jansen, G. Curatolo, C.H. Attenhofer Jost, C. Gambardella, V. Jarvinen, P. Hol, D. Mihalcea, P. Sogaard, D. Peluso, O. Kretschmar, F. Fang, H. Cuellar, F. Maffessanti, R Palma Dos Reis, J. Grapsa, A. Sharif-Rasslan, H. Kwon, P. Novak, R. Gallet, C. Sportouch, O. Enescu, H. Chung, M. Valtonen, D. Dawson, A. G. Fraser, M. Lyra Georgosopoulou, Q. Shang, V. Leonelli, L. Agati, A. Khalil, G. Habib, M. Cavero, A. Ionac, M. Florescu, S. Pescariu, L. Ascione, M. Carmo, A. Marouen, A. D'Andrea, S. Champagne, S. Iliceto, J. P. Halcox, M. Mizia, Z. Gasior, M. Cramer, S F de Marchi, S. Goncalves, L. Dal Bianco, N. Cortez-Dias, U. Richter, I. Santos, U. Naslund, E. Gonzalez Lopez, M. Rover, H. Vago, A. E.-A. El-Shitany, G Teixido Tura, M. Sramko, J. Necas, S. Fennira, M. Gomez Bueno, L. Zakhama, L. Costanzo, H. Zemir, F. Dunstan, R. Pecoraro, R. Hocking, L. Gabrielli, R. Tan, J. Tintera, L. Pratali, V. Monivas, B. Bouzas Zubeldia, B. Segafredo, T. Leiria, R. Mincu, A. Kaczynska, L. Petrescu, J. M. Bosmans, A. Ben Yaala, A. Ploussi, K. Hu, Z. Frikha, L. De Luca, E. Choi, J. Yanez Wonenburger, I. Serbanoiu, C. Iacoboni, J. Trochu, S. Montserrat, X. Luo, E. Pavlukova, D. Martinez Ruiz, G. Lazaros, B. Tan, D. Hudziak, J. Petrovicova, S. Herrmann, P. Biaggi, E. Picano, I. E. Rodrigus, Y. Lam, M. Jeong, M. Fedorco, P. Beltran Correas, C. Felix, L. Polak, C. Wunderlich, S. Hohlfeld, S. Tripepi, M. Haberka, R. Poscia, L. Halmai, A. Luycx-Bore, K. Tunstall, D. Becker, H. Dave, P. Lemarchand, and M. Carvalho
- Subjects
Leading edge ,business.industry ,Reference values ,Healthy subjects ,Medicine ,Radiology, Nuclear Medicine and imaging ,Geometry ,General Medicine ,Edge (geometry) ,Cardiology and Cardiovascular Medicine ,business - Published
- 2012
5. Assessment of Anti-Hypertensive Drug Adherence by Serial Aldosterone-To-Renin Ratio Measurement.
- Author
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Buffolo F, Sconfienza E, Burrello J, Losano I, Mengozzi G, Priolo G, Avataneo V, D'Avolio A, Veglio F, Rabbia F, Mulatero P, and Monticone S
- Abstract
Reduced or absent compliance to anti-hypertensive treatment is a major obstacle to the achievement of blood pressure target in patients with arterial hypertension. Current available methods for therapeutic adherence assessment display low accuracy, limited applicability in clinical practice and/or high costs. We designed a prospective study to evaluate the accuracy of serial measurement of ARR to assess the therapeutic compliance to RAAS inhibitors. We prospectively enrolled 80 subjects: 40 patients with arterial hypertension and 40 normotensive controls. The ARR was evaluated at baseline and 2 and 8 week after initiation of a RAAS inhibitor in patients with hypertension, and at baseline and 2 weeks for the control group. Adherence to the prescribed therapy was confirmed by therapeutic drug monitoring. We observed a significant increase of renin levels and reduction of aldosterone levels after RAAS inhibitors initiation, with consequent reduction of ARR. Delta ARR (ΔARR), defined as relative change in ARR before and after treatment initiation, provided high accuracy for determination of therapeutic compliance, with an AUC of 0.900 at 2 weeks and 0.886 at 8 weeks. A cut-off of -48% of ΔARR provided 90% sensitivity and 75% specificity, at 2 and 8 weeks. In conclusion, the measurement of ΔARR is a powerful test, cheap and widely available to accurately identify the non-adherence to RAAS inhibitors treatment. Herein we propose the implementation of ΔARR in clinical practice through a multi-step flow-chart for the management of patients with uncontrolled blood pressure, with identification of those suspected of non-adherence, reserving therapeutic drug monitoring for non-adherence confirmation., Competing Interests: The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest., (Copyright © 2021 Buffolo, Sconfienza, Burrello, Losano, Mengozzi, Priolo, Avataneo, D’Avolio, Veglio, Rabbia, Mulatero and Monticone.)
- Published
- 2021
- Full Text
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6. Lung Ultrasonography for the Diagnosis of SARS-CoV-2 Pneumonia in the Emergency Department.
- Author
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Pivetta E, Goffi A, Tizzani M, Locatelli SM, Porrino G, Losano I, Leone D, Calzolari G, Vesan M, Steri F, Ardito A, Capuano M, Gelardi M, Silvestri G, Dutto S, Avolio M, Cavallo R, Bartalucci A, Paglieri C, Morello F, Richiardi L, Maule MM, and Lupia E
- Subjects
- Adult, Aged, COVID-19 diagnosis, COVID-19 Nucleic Acid Testing, False Negative Reactions, Female, Humans, Italy, Male, Middle Aged, Prospective Studies, SARS-CoV-2, Sensitivity and Specificity, Ultrasonography, COVID-19 diagnostic imaging, Emergency Service, Hospital, Lung diagnostic imaging
- Abstract
Study Objective: Accurate diagnostic testing to identify severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection is critical. Although highly specific, SARS-CoV-2 reverse transcriptase-polymerase chain reaction (RT-PCR) has been shown in clinical practice to be affected by a noninsignificant proportion of false-negative results. This study seeks to explore whether the integration of lung ultrasonography with clinical evaluation is associated with increased sensitivity for the diagnosis of coronavirus disease 2019 pneumonia, and therefore may facilitate the identification of false-negative SARS-CoV-2 RT-PCR results., Methods: This prospective cohort study enrolled consecutive adult patients with symptoms potentially related to SARS-CoV-2 infection who were admitted to the emergency department (ED) of an Italian academic hospital. Immediately after the initial assessment, a lung ultrasonographic evaluation was performed and the likelihood of SARS-CoV-2 infection, based on both clinical and lung ultrasonographic findings ("integrated" assessment), was recorded. RT-PCR SARS-CoV-2 detection was subsequently performed., Results: We enrolled 228 patients; 107 (46.9%) had SARS-CoV-2 infection. Sensitivity and negative predictive value of the clinical-lung ultrasonographic integrated assessment were higher than first RT-PCR result (94.4% [95% confidence interval {CI} 88.2% to 97.9%] versus 80.4% [95% CI 71.6% to 87.4%] and 95% [95% CI 89.5% to 98.2%] versus 85.2% [95% CI 78.3% to 90.6%], respectively). Among the 142 patients who initially had negative RT-PCR results, 21 tested positive at a subsequent molecular test performed within 72 hours. All these false-negative cases were correctly identified by the integrated assessment., Conclusion: This study suggests that, in patients presenting to the ED with symptoms commonly associated with SARS-CoV-2 infection, the integration of lung ultrasonography with clinical evaluation has high sensitivity and specificity for coronavirus disease 2019 pneumonia and it may help to identify false-negative results occurring with RT-PCR., (Copyright © 2020 American College of Emergency Physicians. Published by Elsevier Inc. All rights reserved.)
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- 2021
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7. The spectrum of low-renin hypertension.
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Buffolo F, Monticone S, Pecori A, Pieroni J, Losano I, Cavaglià G, Tetti M, Veglio F, and Mulatero P
- Subjects
- Aldosterone blood, Aldosterone metabolism, Humans, Hyperaldosteronism complications, Hyperaldosteronism genetics, Hyperaldosteronism metabolism, Hypertension blood, Hypertension genetics, Molecular Diagnostic Techniques, Hypertension diagnosis, Hypertension etiology, Renin blood
- Abstract
Low-renin hypertension (LRH) is a frequent condition in patients with arterial hypertension, accounting for 30% of patients. Monogenic forms can cause LRH in a minority of cases. However, in the large majority of patients, LRH is caused by the combined effects of congenital and acquired factors, comprising dietary habits. Several genetic variants have been proposed as co-factors in the pathogenesis of LRH with normal-low serum aldosterone. Emerging evidences support the hypothesis that a large proportion of LRH with normal-high serum aldosterone is associated with subclinical primary aldosteronism (PA). The recent identification of aldosterone-producing cell clusters (APCCs) as the possible cause of subclinical PA, further supported the concept of a continuous spectrum of autonomous aldosterone secretion, from subclinical forms towards overt PA. In this review we describe the main aspects of LRH, focusing on molecular basis, clinical risk profile and patients' management., Competing Interests: Declaration of Competing Interest Paolo Mulatero received fees for educational speech from DIASORIN., (Copyright © 2020 Elsevier Ltd. All rights reserved.)
- Published
- 2020
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8. Prevalence of Hypokalemia and Primary Aldosteronism in 5100 Patients Referred to a Tertiary Hypertension Unit.
- Author
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Burrello J, Monticone S, Losano I, Cavaglià G, Buffolo F, Tetti M, Covella M, Rabbia F, Veglio F, Pasini B, Williams TA, and Mulatero P
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- Adult, Aged, Comorbidity, Female, Humans, Hypertension therapy, Male, Mass Screening, Middle Aged, Prevalence, Referral and Consultation, Retrospective Studies, Risk, Hyperaldosteronism epidemiology, Hypertension epidemiology, Hypokalemia epidemiology
- Abstract
Primary aldosteronism (PA) was considered a rare disorder almost always associated with hypokalemia. The widespread screening of patients with hypertension unveiled an increased prevalence of PA with normokalemic hypertension the prevailing phenotype. Many studies have reported the prevalence of hypokalemia in patients with PA; conversely, the prevalence of PA in patients with hypokalemia is unknown. In this retrospective observational study, we define the prevalence of hypokalemia in referred patients with hypertension and the prevalence of PA in patients with hypokalemia and hypertension. Hypokalemia was present in 15.8% of 5100 patients with hypertension, whereas 76.9% were normokalemic, and 7.3% hyperkalemic. The prevalence of PA in patients with hypokalemia was 28.1% and increased with decreasing potassium concentrations up to 88.5% of patients with spontaneous hypokalemia and potassium concentrations <2.5 mmol/L. A multivariate regression analysis demonstrated the association of hypokalemia with the occurrence of cardiovascular events independent of PA diagnosis. An association of PA with the occurrence of cardiovascular events and target organ damage independent of hypokalemia was also demonstrated. In conclusion, our results confirm that PA is a frequent cause of secondary hypertension in patients with hypokalemia, and the presence of hypertension and spontaneous hypokalemia are strong indications for PA diagnosis. Finally, we show that PA and hypokalemia are associated with an increased risk of cardiovascular events.
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- 2020
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9. Diagnostic approach to low-renin hypertension.
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Monticone S, Losano I, Tetti M, Buffolo F, Veglio F, and Mulatero P
- Subjects
- Animals, Humans, Hyperaldosteronism blood, Hyperaldosteronism diagnosis, Hyperaldosteronism metabolism, Hypertension blood, Liddle Syndrome blood, Liddle Syndrome diagnosis, Liddle Syndrome metabolism, Renin blood, Hypertension diagnosis, Hypertension metabolism, Renin metabolism
- Abstract
Renin-angiotensin-aldosterone system (RAAS) plays a crucial role in maintaining water and electrolytes homoeostasis, and its deregulation contributes to the development of arterial hypertension. Since the historical description of the "classical" RAAS, a dramatic increase in our understanding of the molecular mechanisms underlying the development of both essential and secondary hypertension has occurred. Approximatively 25% of the patients affected by arterial hypertension display low-renin levels, a definition that is largely arbitrary and depends on the investigated population and the specific characteristics of the assay. Most often, low-renin levels are expression of a physiological response to sodium-volume overload, but also a significant number of secondary hereditary or acquired conditions falls within this category. In a context of suppressed renin status, the concomitant examination of plasma aldosterone levels (which can be inappropriately elevated, within the normal range or suppressed) and plasma potassium are essential to formulate a differential diagnosis. To distinguish between the different forms of low-renin hypertension is of fundamental importance to address the patient to the proper clinical management, as each subtype requires a specific and targeted therapy. The present review will discuss the differential diagnosis of the most common medical conditions manifesting with a clinical phenotype of low-renin hypertension, enlightening the novelties in genetics of the familial forms., (© 2018 John Wiley & Sons Ltd.)
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- 2018
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10. Arterial hypertension and cancer.
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Milan A, Puglisi E, Ferrari L, Bruno G, Losano I, and Veglio F
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- Antihypertensive Agents adverse effects, Antineoplastic Agents adverse effects, Humans, Hypertension chemically induced, Neoplasms chemically induced, Risk Assessment, Risk Factors, Antihypertensive Agents therapeutic use, Antineoplastic Agents therapeutic use, Hypertension drug therapy, Neoplasms drug therapy
- Abstract
Arterial hypertension and cancer are two of the most important causes of mortality in the world; correlations between these two clinical entities are complex and various. Cancer therapy using old (e.g., mitotic spindle poisons) as well as new (e.g., monoclonal antibody) drugs may cause arterial hypertension through different mechanisms; sometimes the increase of blood pressure levels may be responsible for chemotherapy withdrawal. Among newer cancer therapies, drugs interacting with the VEGF (vascular endothelial growth factors) pathways are the most frequently involved in hypertension development. However, many retrospective studies have suggested a relationship between antihypertensive treatment and risk of cancer, raising vast public concern. The purposes of this brief review have then been to analyse the role of chemotherapy in the pathogenesis of hypertension, to summarize the general rules of arterial hypertension management in this field and finally to evaluate the effects of antihypertensive therapy on cancer disease., (© 2013 UICC.)
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- 2014
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11. Left heart morphology and function in primary aldosteronism.
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Milan A, Magnino C, Fabbri A, Chiarlo M, Bruno G, Losano I, and Veglio F
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- Animals, Heart Atria diagnostic imaging, Heart Atria physiopathology, Hemodynamics, Humans, Hypertrophy, Left Ventricular diagnostic imaging, Hypertrophy, Left Ventricular physiopathology, Ultrasonography, Ventricular Dysfunction, Left diagnostic imaging, Ventricular Dysfunction, Left physiopathology, Hyperaldosteronism complications, Hypertrophy, Left Ventricular etiology, Ventricular Dysfunction, Left etiology, Ventricular Function, Left
- Abstract
Primary aldosteronism is the most frequent cause of secondary hypertension, accounting for up to 11% of cases in selected populations. Patients affected by primary aldosteronism have shown higher prevalence of cardiovascular and cerebrovascular events compared with patients with essential hypertension, despite similar blood pressure levels. Several studies have been performed over past years aiming to explain these data; many of these evaluated echocardiographic differences in hypertension-related cardiac organ damage between primary aldosteronism and essential hypertension. This article summarizes the present knowledge about structural and functional alteration of the human left heart in primary aldosteronism.
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- 2012
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