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1. POS-534 TRAJECTORIES OF CLINICAL AND LABORATORY CHARACTERISTICS ASSOCIATED WITH COVID-19 IN HEMODIALYSIS PATIENTS BY SURVIVAL

3. Processing of Non-Conjugative Resistance Plasmids by Conjugation Nicking Enzyme of Staphylococci.

5. Biochemical and immunological characterization of serum biotinidase in profound biotinidase deficiency

12. Dosing patterns, drug costs, and hematologic outcome in anemic patients with chronic kidney disease switching from darbepoetin alfa to epoetin alfa.

13. 2004 annual report of the American Chiropractic Association.

14. Human biotinidase isn't just for recycling biotin.

17. Delayed-onset profound biotinidase deficiency

23. Editor's Choice - Challenges of Predicting Arteriovenous Access Survival Prior to Conversion from Catheter.

24. A phase 3b, multicenter, open-label, single-arm study of roxadustat within a US dialysis organization: The DENALI study.

25. Effectiveness of cold HD for the prevention of HD hypotension and mortality in the general HD population.

26. Virtual Screening for the Discovery of Microbiome β-Glucuronidase Inhibitors to Alleviate Cancer Drug Toxicity.

27. Trajectories of clinical and laboratory characteristics associated with COVID-19 in hemodialysis patients by survival.

28. Transmission of SARS-CoV-2 considering shared chairs in outpatient dialysis: a real-world case-control study.

29. Real-time prediction of intradialytic relative blood volume: a proof-of-concept for integrated cloud computing infrastructure.

30. Hepatitis B Vaccination Response in Hemodialysis Patients: The Impact of Dialysis Shift.

31. Switching from Epoetin Alfa (Epogen®) to Epoetin Alfa-Epbx (RetacritTM) Using a Specified Dosing Algorithm: A Randomized, Non-Inferiority Study in Adults on Hemodialysis.

33. Conserved S-Layer-Associated Proteins Revealed by Exoproteomic Survey of S-Layer-Forming Lactobacilli.

35. Associates of cardiopulmonary arrest in the perihemodialytic period.

36. Sodium thiosulfate therapy for calcific uremic arteriolopathy.

37. Identification of alternatively spliced human biotinidase mRNAs and putative localization of endogenous biotinidase.

38. Mutations in BTD causing biotinidase deficiency.

39. Examination of the signal peptide region of human biotinidase using a baculovirus expression system.

40. Mutations causing profound biotinidase deficiency in children ascertained by newborn screening in the United States occur at different frequencies than in symptomatic children.

41. Mutation in a putative glycosylation site (N489T) of biotinidase in the only known Japanese child with biotinidase deficiency.

42. Double mutation (A171T and D444H) is a common cause of profound biotinidase deficiency in children ascertained by newborn screening the the United States. Mutations in brief no. 128. Online.

43. Mutations in the human biotinidase gene that cause profound biotinidase deficiency in symptomatic children: molecular, biochemical, and clinical analysis.

44. Profound biotinidase deficiency in two asymptomatic adults.

45. Mutation (Q456H) is the most common cause of profound biotinidase deficiency in children ascertained by newborn screening in the United States.

46. Arg538 to Cys mutation in a CpG dinucleotide of the human biotinidase gene is the second most common cause of profound biotinidase deficiency in symptomatic children.

48. Biochemical and immunologic characterization of serum biotinidase in partial biotinidase deficiency.

49. Isoforms of human serum biotinidase.

50. Biotinidase.

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