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2. Supp Figure 4 from Involvement of Epigenetics and EMT-Related miRNA in Arsenic-Induced Neoplastic Transformation and Their Potential Clinical Use

3. Supp Figure 1 from Involvement of Epigenetics and EMT-Related miRNA in Arsenic-Induced Neoplastic Transformation and Their Potential Clinical Use

4. Supp Figure 3 from Involvement of Epigenetics and EMT-Related miRNA in Arsenic-Induced Neoplastic Transformation and Their Potential Clinical Use

5. Supp Figure legends from Involvement of Epigenetics and EMT-Related miRNA in Arsenic-Induced Neoplastic Transformation and Their Potential Clinical Use

6. Supp Figure 3 continued from Involvement of Epigenetics and EMT-Related miRNA in Arsenic-Induced Neoplastic Transformation and Their Potential Clinical Use

7. Supp Figure 2A from Involvement of Epigenetics and EMT-Related miRNA in Arsenic-Induced Neoplastic Transformation and Their Potential Clinical Use

8. Supp Table 1, Supp Table 2 from Involvement of Epigenetics and EMT-Related miRNA in Arsenic-Induced Neoplastic Transformation and Their Potential Clinical Use

9. Data from Involvement of Epigenetics and EMT-Related miRNA in Arsenic-Induced Neoplastic Transformation and Their Potential Clinical Use

10. Supplementary Table S4 from A Panel of Novel Detection and Prognostic Methylated DNA Markers in Primary Non–Small Cell Lung Cancer and Serum DNA

11. Supplementary Table 2 from Association between Lifestyle Factors and CpG Island Methylation in a Cancer-Free Population

12. Supplementary information from A Panel of Novel Detection and Prognostic Methylated DNA Markers in Primary Non–Small Cell Lung Cancer and Serum DNA

13. Data from Changes in CpG Islands Promoter Methylation Patterns during Ductal Breast Carcinoma Progression

16. Supplementary Tables 1-4 from Changes in CpG Islands Promoter Methylation Patterns during Ductal Breast Carcinoma Progression

17. Supplementary Table 1 from Association between Lifestyle Factors and CpG Island Methylation in a Cancer-Free Population

20. Supplementary figures 1-4 from Changes in CpG Islands Promoter Methylation Patterns during Ductal Breast Carcinoma Progression

23. Data from Genome-Wide Promoter Analysis Uncovers Portions of the Cancer Methylome

24. Supplementary Table 6 from Genome-Wide Promoter Analysis Uncovers Portions of the Cancer Methylome

25. Supplementary Figure 1 from N-Methyl-d-Aspartate Receptor Type 2B Is Epigenetically Inactivated and Exhibits Tumor-Suppressive Activity in Human Esophageal Cancer

27. Supplementary Table 8 from Genome-Wide Promoter Analysis Uncovers Portions of the Cancer Methylome

28. Supplementary Figure 2 from N-Methyl-d-Aspartate Receptor Type 2B Is Epigenetically Inactivated and Exhibits Tumor-Suppressive Activity in Human Esophageal Cancer

30. Supplementary Table 3 from Genome-Wide Promoter Analysis Uncovers Portions of the Cancer Methylome

31. Supplementary Table 5 from Genome-Wide Promoter Analysis Uncovers Portions of the Cancer Methylome

32. Data from N-Methyl-d-Aspartate Receptor Type 2B Is Epigenetically Inactivated and Exhibits Tumor-Suppressive Activity in Human Esophageal Cancer

34. Supplementary Table 7 from Genome-Wide Promoter Analysis Uncovers Portions of the Cancer Methylome

35. Supplementary Table 4 from Genome-Wide Promoter Analysis Uncovers Portions of the Cancer Methylome

36. Supplementary Figure 3 from N-Methyl-d-Aspartate Receptor Type 2B Is Epigenetically Inactivated and Exhibits Tumor-Suppressive Activity in Human Esophageal Cancer

38. Supplementary Table 1 from Genome-Wide Promoter Analysis Uncovers Portions of the Cancer Methylome

39. Supplementary Table 2 from Genome-Wide Promoter Analysis Uncovers Portions of the Cancer Methylome

45. CDC91L1 (PIG-U) is a newly discovered oncogene in human bladder cancer

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