17 results on '"Hong, Huiqi"'
Search Results
2. SIRT3 prevents angiotensin II-induced renal tubular epithelial-mesenchymal transition by ameliorating oxidative stress and mitochondrial dysfunction
3. Protocatechuic aldehyde protects against isoproterenol-induced cardiac hypertrophy via inhibition of the JAK2/STAT3 signaling pathway
4. Author Correction: Systematic evaluation and optimization of the experimental steps in RNA G-quadruplex structure sequencing
5. Cis- and trans-regulations of pre-mRNA splicing by RNA editing enzymes influence cancer development
6. SIRT6 suppresses isoproterenol-induced cardiac hypertrophy through activation of autophagy
7. Systematic evaluation and optimization of the experimental steps in RNA G-quadruplex structure sequencing
8. MicroRNA-34c-5p provokes isoprenaline-induced cardiac hypertrophy by modulating autophagy via targeting ATG4B
9. RNA editing mediates the functional switch of COPA in a novel mechanism of hepatocarcinogenesis
10. Suppression of adenosine-to-inosine (A-to-I) RNA editome by death associated protein 3 (DAP3) promotes cancer progression
11. Chrysophanol attenuated isoproterenol‐induced cardiac hypertrophy by inhibiting Janus kinase 2/signal transducer and activator of transcription 3 signaling pathway
12. Bidirectional regulation of adenosine-to-inosine (A-to-I) RNA editing by DEAH box helicase 9 (DHX9) in cancer
13. An RNA editing/dsRNA binding-independent gene regulatory mechanism of ADARs and its clinical implication in cancer
14. The poly(ADP-ribosyl)ation of FoxO3 mediated by PARP1 participates in isoproterenol-induced cardiac hypertrophy
15. ADAR-Mediated RNA Editing Predicts Progression and Prognosis of Gastric Cancer
16. An RNA editing/binding-independent gene regulatory mechanism of ADARs and its clinical implication in cancer
17. Regulatory factors governing adenosine-to-inosine (A-to-I) RNA editing
Catalog
Books, media, physical & digital resources
Discovery Service for Jio Institute Digital Library
For full access to our library's resources, please sign in.