Search

Your search keyword '"Hiroshima, Yuka"' showing total 164 results
Start Over Author "Hiroshima, Yuka"
164 results on '"Hiroshima, Yuka"'

3. Utility of a haemoglobin test of gingival crevicular fluid: A multicentre, observational study.

Author

Ito, Hiroshi, Numabe, Yukihiro, Hashimoto, Shuichi, Sekino, Satoshi, Murakashi, Etsuko, Ishiguro, Hitomi, Sasaki, Daisuke, Yaegashi, Takashi, Takai, Hideki, Mezawa, Masaru, Ogata, Yorimasa, Watanabe, Hisashi, Izumi, Yuichi, Kido, Jun‐Ichi, Hiroshima, Yuka, and Nagata, Toshihiko

Subjects
PERIODONTITIS treatment, PROTEIN analysis, PREDICTIVE tests, RESEARCH funding, GINGIVA, HEMOGLOBINS, SCIENTIFIC observation, GINGIVITIS, ASPARTATE aminotransferase, TREATMENT effectiveness, RESEARCH, DENTAL plaque, EXUDATES & transudates, PERIODONTITIS, EVALUATION
Abstract

Objective: The purpose of this study was to verify the accuracy and utility of clinical parameters (plaque index, gingival crevicular fluid volume, probing depth, clinical attachment level, bleeding on probing and gingival index) and biochemical parameters (aspartate aminotransferase, protein and haemoglobin) in a longitudinal analysis during the supportive periodontal therapy period. Subjects and Methods: A total of 279 test sites of 128 patients were investigated clinically and biochemically. After the first examination of clinical and biochemical parameters, periodontal support treatments were administered immediately and performed once every three months up to the second examination. Results: All of the clinical and biochemical parameters were significantly lower at the second examination than at the first, except for the plaque index and bleeding on probing. Of these parameters, in particular, aspartate aminotransferase and haemoglobin in the gingival crevicular fluid were significantly reduced compared to those of the first examination in both the ≤4 and ≥5 mm probing depth groups, and they clearly suggested that periodontitis tended to recover. Conclusion: Adding the haemoglobin test to the bleeding on probing test strongly improves the accuracy of measurement of clinical parameters after periodontal treatment. [ABSTRACT FROM AUTHOR]

Published
2024
Full Text
View/download PDF

5. Porphyromonas gingivalis outer membrane vesicles in cerebral ventricles activate microglia in mice.

Author

Yoshida, Kayo, Yoshida, Kaya, Seyama, Mariko, Hiroshima, Yuka, Mekata, Mana, Fujiwara, Natsumi, Kudo, Yasusei, and Ozaki, Kazumi

Subjects
BRAIN, DISEASE progression, BIOLOGICAL models, CYTOKINES, ALZHEIMER'S disease, NEURONS, CEREBRAL ventricles, CELL membranes, ANIMAL experimentation, IMMUNOHISTOCHEMISTRY, TAU proteins, WESTERN immunoblotting, INFLAMMATION, GRAM-negative anaerobic bacteria, GENE expression, NEUROINFLAMMATION, CELLS, RESEARCH funding, ABDOMEN, ENDOPEPTIDASES, POLYMERASE chain reaction, MICE, PHOSPHORYLATION
Abstract

Objective: Porphyromonas gingivalis (Pg) is thought to be involved in the progression of Alzheimer's disease (AD). Whether Pg or its contents can reach the brain and directly affect neuropathology is, however, unknown. Here, we investigated whether outer membrane vesicles (OMVs) of Pg translocate to the brain and induce the pathogenic features of AD. Material and Methods: Pg OMVs were injected into the abdominal cavity of mice for 12 weeks. Pg OMV translocation to the brain was detected by immunohistochemistry using an anti‐gingipain antibody. Tau protein and microglial activation in the mouse brain were examined by western blotting and immunohistochemistry. The effect of gingipains on inflammation was assessed by real‐time polymerase chain reaction using human microglial HMC3 cells. Results: Gingipains were detected in the region around cerebral ventricles, choroid plexus, and ventricular ependymal cells in Pg OMV‐administered mice. Tau and phosphorylated Tau protein increased and microglia were activated. Pg OMVs also increased the gene expression of proinflammatory cytokines in HMC3 cells in a gingipain‐dependent manner. Conclusion: Pg OMVs, including gingipains, can reach the cerebral ventricle and induce neuroinflammation by activating microglia. Pg OMVs may provide a better understanding of the implications of periodontal diseases in neurodegenerative conditions such as AD. [ABSTRACT FROM AUTHOR]

Published
2023
Full Text
View/download PDF

6. Utility of a haemoglobin test of gingival crevicular fluid: A multicentre, observational study

Author

Ito, Hiroshi, primary, Numabe, Yukihiro, additional, Hashimoto, Shuichi, additional, Sekino, Satoshi, additional, Murakashi, Etsuko, additional, Ishiguro, Hitomi, additional, Sasaki, Daisuke, additional, Yaegashi, Takashi, additional, Takai, Hideki, additional, Mezawa, Masaru, additional, Ogata, Yorimasa, additional, Watanabe, Hisashi, additional, Izumi, Yuichi, additional, Kido, Jun‐Ichi, additional, Hiroshima, Yuka, additional, and Nagata, Toshihiko, additional

Published
2023
Full Text
View/download PDF

11. Lipocalin 2, synthesized using a cell‐free protein synthesis system and encapsulated into liposomes, inhibits the adhesion of Porphyromonas gingivalis to human oral epithelial cells

Author

Kido, Jun‐ichi, primary, Hiroshima, Yuka, additional, Kido, Rie, additional, Yoshida, Kaya, additional, Inagaki, Yuji, additional, Naruishi, Koji, additional, Kajimoto, Kazuaki, additional, Kataoka, Masatoshi, additional, Shinohara, Yasuo, additional, and Yumoto, Hiromichi, additional

Published
2022
Full Text
View/download PDF

18. Imaging features of a myoepithelial carcinoma of the nasal cavity : A case report and literature review

Author

Shinya, Takayoshi, Matsushita, Tomoki, Hiroshima, Yuka, Otomi, Yoichi, Kanematsu, Yasuhisa, Bando, Yoshimi, Uehara, Hisanori, Kitamura, Yoshiaki, Harada, Masafumi, Shinya, Takayoshi, Matsushita, Tomoki, Hiroshima, Yuka, Otomi, Yoichi, Kanematsu, Yasuhisa, Bando, Yoshimi, Uehara, Hisanori, Kitamura, Yoshiaki, and Harada, Masafumi

Abstract

Myoepithelial carcinoma of the nasal cavity is extremely rare. We report the case of a 66-year-old man with myoepithelial carcinoma of the nasal cavity. Computed tomography (CT) and magnetic resonance imaging revealed a lobulated soft tissue mass with central necrosis and hemorrhage, as well as an invasion of the skull base and left orbit. The patient presented with continuous nasal congestion and heavy head and had no elevated level of squamous cell carcinoma-related antigen. CT, magnetic resonance imaging, or 18F-fluorodeoxyglucose (FDG) positron emission tomography/CT revealed no evidence of a metastatic lesion. 18F-FDG accumulation in the tumor was inhomogeneous and moderate. Histopathological examination of the resected specimen confirmed a well-circumscribed solid tumor with septa, a small area of hemorrhage, and necrosis. The subsequent diagnosis was a myoepithelial carcinoma of the left nasal cavity. This case shows that nasal myoepithelial carcinoma might appear as a well-defined lobulated mass with hemorrhagic necrosis and intense contrast enhancement in the solid component. We conjecture that hemorrhagic necrosis and intense enhancement values may be potential markers of nasal myoepithelial carcinoma.

Published
2022

19. Porphyromonas gingivalis outer membrane vesicles in cerebral ventricles activate microglia in mice

Author

Yoshida, Kayo, Yoshida, Kaya, Seyama, Mariko, Hiroshima, Yuka, Mekata, Mana, Fujiwara, Natsumi, Kudo, Yasusei, Ozaki, Kazumi, Yoshida, Kayo, Yoshida, Kaya, Seyama, Mariko, Hiroshima, Yuka, Mekata, Mana, Fujiwara, Natsumi, Kudo, Yasusei, and Ozaki, Kazumi

Abstract

Objective: Porphyromonas gingivalis (Pg) is thought to be involved in the progression of Alzheimer's disease (AD). Whether Pg or its contents can reach the brain and directly affect neuropathology is, however, unknown. Here, we investigated whether outer membrane vesicles (OMVs) of Pg translocate to the brain and induce the pathogenic features of AD. Material and Methods: Pg OMVs were injected into the abdominal cavity of mice for 12 weeks. Pg OMV translocation to the brain was detected by immunohistochemistry using an anti-gingipain antibody. Tau protein and microglial activation in the mouse brain were examined by western blotting and immunohistochemistry. The effect of gingipains on inflammation was assessed by real-time polymerase chain reaction using human microglial HMC3 cells. Results: Gingipains were detected in the region around cerebral ventricles, choroid plexus, and ventricular ependymal cells in Pg OMV-administered mice. Tau and phosphorylated Tau protein increased and microglia were activated. Pg OMVs also increased the gene expression of proinflammatory cytokines in HMC3 cells in a gingipain-dependent manner. Conclusion: Pg OMVs, including gingipains, can reach the cerebral ventricle and induce neuroinflammation by activating microglia. Pg OMVs may provide a better understanding of the implications of periodontal diseases in neurodegenerative conditions such as AD.

Published
2022

20. 最終糖化産物は口腔上皮細胞におけるLipocain2の発現を増加させる

Author

Kido, Rie, Hiroshima, Yuka, Kido, Jun-ichi, Ikuta, Takahisa, Sakamoto, Eijiro, Inagaki, Yuji, Naruishi, Koji, and Yumoto, Hiromichi

Subjects
human oral epithelial cells, diabetes mellitus, AGEs, lipocalin2, periodontitis
Abstract

Background and Objectives: Diabetes mellitus (DM), a risk factor of periodontal diseases, exacerbates the pathological condition of periodontitis. A major factor for DM complications is advanced glycation end-products (AGEs) that accumulate in periodontal tissues and cause inflammatory events. Lipocalin 2 (LCN2) is an antimicrobial peptide and inflammation-related factor, and LCN2 levels increase in DM. In the present study, the effects of AGEs and lipopolysaccharide of Porphyromonas gingivalis (P.g-LPS) on LCN2 expression in human oral epithelial cells (TR146 cells) and the role of secreted LCN2 in periodontitis with DM were investigated. Material and Methods: TR146 cells were cultured with AGEs (AGE2) and control BSA and cell viability was estimated, or with P.g-LPS. Conditioned medium and cell lysates were prepared from cultures of epithelial cells and used for western blotting and ELISA to analyze LCN2, RAGE, IL-6, MAPK and NF-κB. RNA was isolated from AGE-treated TR146 cells and differentiated HL-60 (D-HL-60) cells and used for quantitative real-time PCR to examine the expression of LCN2 and interleukin-6 (IL-6) mRNAs. RAGE- and LCN2-siRNAs (siRAGE, siLCN2) were transfected into epithelial cells, and AGE-induced LCN2 expression was investigated. D-HL-60 cells were co-cultured with TR146 cells that were transfected with siLCN2 and treated with AGEs, IL-6 mRNA expression in D-HL-60 cells and cell migration were investigated. Results: AGEs increased the expression levels of LCN2 and IL-6 in oral epithelial cells. siRAGE and a neutralizing antibody for RAGE inhibited AGE-induced LCN2 expression. AGEs stimulated the phosphorylation of ERK, p38 and NF-kB in epithelial cells, and their inhibitors suppressed AGE-induced LCN2 expression. In contrast, P.g-LPS did not show a significant increase on LCN2 level in TR146 cells that expressed toll-like receptor 2. In co-culture experiments, AGE-induced LCN2 inhibited IL-6 mRNA expression in D-HL-60 cells, and LCN2 knockdown in epithelial cells suppressed HL-60 cell migration. Conclusion: These results suggested that AGEs increase LCN2 expression via RAGE, MAPK, and NF-κB signaling pathways in oral epithelial cells, and secreted LCN2 may influence the pathological condition of periodontitis with DM.

Published
2020

22. Evaluation of bleeding on probing and gingival crevicular fluid enzyme activity for detection of periodontally active sites during supportive periodontal therapy

Author

Ito, Hiroshi, Numabe, Yukihiro, Sekino, Satoshi, Murakashi, Etsuko, Iguchi, Hitomi, Hashimoto, Shuichi, Sasaki, Daisuke, Yaegashi, Takashi, Kunimatsu, Kazushi, Takai, Hideki, Mezawa, Masaru, Ogata, Yorimasa, Watanabe, Hisashi, Hagiwara, Satsuki, Izumi, Yuichi, Hiroshima, Yuka, Kido, Jun-Ichi, and Nagata, Toshihiko

Published
2014
Full Text
View/download PDF

23. Axillary Lymph Node Uptake on 18F-FDG PET/CT after COVID-19 Vaccination: A Direct Comparison Study with Influenza Vaccination.

Author

Otomi, Yoichi, Shinya, Takayoshi, Kasai, Hiroto, Okada, Naoko, Matsushita, Tomoki, Higashi, Kohei, Matsuzaki, Saya, Hiroshima, Yuka, Kubo, Michiko, Otsuka, Hideki, and Harada, Masafumi

Subjects
COVID-19 vaccines, INFLUENZA vaccines, LYMPH nodes, POSITRON emission tomography, COVID-19
Abstract

Copyright of Molecular Imaging & Radionuclide Therapy is the property of Galenos Yayinevi Tic. LTD. STI and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Published
2023
Full Text
View/download PDF

24. Autoinducer Analogs Can Provide Bactericidal Activity to Macrolides in Pseudomonas aeruginosa through Antibiotic Tolerance Reduction

Author

Abe, Mizuki, Murakami, Keiji, Hiroshima, Yuka, Amoh, Takashi, Sebe, Mayu, Kataoka, Keiko, Fujii, Hideki, Abe, Mizuki, Murakami, Keiji, Hiroshima, Yuka, Amoh, Takashi, Sebe, Mayu, Kataoka, Keiko, and Fujii, Hideki

Abstract

Macrolide antibiotics are used in treating Pseudomonas aeruginosa chronic biofilm infections despite their unsatisfactory antibacterial activity, because they display several special activities, such as modulation of the bacterial quorum sensing and immunomodulatory effects on the host. In this study, we investigated the effects of the newly synthesized P. aeruginosa quorum-sensing autoinducer analogs (AIA-1, -2) on the activity of azithromycin and clarithromycin against P. aeruginosa. In the killing assay of planktonic cells, AIA-1 and -2 enhanced the bactericidal ability of macrolides against P. aeruginosa PAO1; however, they did not affect the minimum inhibitory concentrations of macrolides. In addition, AIA-1 and -2 considerably improved the killing activity of azithromycin and clarithromycin in biofilm cells. The results indicated that AIA-1 and -2 could affect antibiotic tolerance. Moreover, the results of hydrocarbon adherence and cell membrane permeability assays suggested that AIA-1 and -2 changed bacterial cell surface hydrophobicity and accelerated the outer membrane permeability of the hydrophobic antibiotics such as azithromycin and clarithromycin. Our study demonstrated that the new combination therapy of macrolides and AIA-1 and -2 may improve the therapeutic efficacy of macrolides in the treatment of chronic P. aeruginosa biofilm infections.

Published
2021

26. Roles of lysosomal proteolytic systems in AQP5 degradation in the submandibular gland of rats following chorda tympani parasympathetic denervation

Author

Azlina, Ahmad, Javkhlan, Purevjav, Hiroshima, Yuka, Hasegawa, Takahiro, Yao, Chenjuan, Akamatsu, Tetsuya, and Hosoi, Kazuo

Subjects
Proteolysis -- Research, Sympathectomy -- Health aspects, Apoptosis -- Research, Cathepsins -- Physiological aspects, Cathepsins -- Research, Biological sciences
Abstract

Chorda tympani denervation (CTD) of rats was earlier shown to result in loss of submandibular gland (SMG) weight (at only 1 wk) and in continued reduction in aquaporin 5 (AQP5) protein expression (until 4 wk), without affecting its mRNA synthesis (Li X, Azlina A, Karabasil MR, Purwanti N, Hasegawa T, Yao C, Akamatsu T, Hosoi K. Am J Physiol Gastrointest Liver Physiol 295:G112-G123, 2008). The present study indicated that despite elevation of bax, a proapoptosis protein, by CTD, the operation also increased the level of bcl-2, an antiapoptosis protein, in the SMG. Terminal deoxynucleotidyl transferase dUTP nick-end labeling (TUNEL assay) showed no increase in the number of apoptotic cells in the SMG. CTD, however, induced strongly and transiently (at 1-3 days) the protein expression of LC3B-II, a marker protein of autophagosomes, suggesting that the reduction in the gland weight was due to onset of autophagy by CTD. Upon CTD, Lamp2, a lysosomal marker, gradually increased in amount, reaching a peak at the 14th day. Immunohistochemical analysis revealed an increase in the number of lysosome-like structures positive for both AQP5 and Lamp2 in the acinar cells of the SMG after CTD; similar changes were observed also for AQP5 and LC3Bs. These data suggest that AQP5 in the SMG entered autophagosomes and/or lysosomes for degradation upon CTD. In vitro AQP5-degrading activity was found in the SMG extracts, and such activity was shown to be increased by CTD. Inhibitor experiments implied cathepsins B and L to be candidate enzymes for this degradation under normal and CTD conditions, respectively. protein degradation; autophagy; lysosomal proteolysis; LC3B; apoptosis doi: 10.1152/ajpgi.00194.2010.

Published
2010

31. 11C-Methionine PET/CT and 18F-FDG PET/CT in the Evaluation of Adult Alveolar Rhabdomyosarcoma.

Author

Hiroshima, Yuka, Otomi, Yoichi, Shinya, Takayoshi, Otsuka, Hideki, and Harada, Masafumi

Subjects
*METHIONINE, *MAGNETIC resonance imaging, *RHABDOMYOSARCOMA, *NASAL tumors, *LYMPHATIC metastasis, *PARANASAL sinuses, *POSITRON emission tomography
Abstract

A 70-year-old man with a tumor in the nasal and paranasal space, was pathologically diagnosed with an alveolar rhabdomyosarcoma, with right cervical lymph node metastasis. Magnetic resonance imaging revealed a primary tumor in the nasal and paranasal sinuses, with associated intracranial infiltration. 11C-methionine positron emission tomography/computed tomography (PET/CT) revealed increased uptake in the primary tumor and right cervical lymph node metastasis. 18F-fluorodeoxyglucose PET/CT also revealed increased uptake in the primary tumor and right cervical lymph node. However, the physiological brain uptake overlapped with the primary tumor uptake. Our case suggests the usefulness of 11C-methionine PET/CT for accurately assessing the extent of alveolar rhabdomyosarcoma, especially in cases with intracranial infiltration or those approximating the brain. [ABSTRACT FROM AUTHOR]

Published
2023
Full Text
View/download PDF

34. PKR induces the expression of NLRP3 by regulating the NF-κB pathway in Porphyromonas gingivalis-infected osteoblasts

Author

Yoshida, Kaya, Okamura, Hirohiko, Hiroshima, Yuka, Abe, Kaori, Kido, Jun-ichi, Shinohara, Yasuo, and Ozaki, Kazumi

Subjects
integumentary system, Double-stranded RNA-dependent kinase, viruses, NF-kappa B, osteoblasts, inflammasomes, Porphyromonas gingivalis
Abstract

The double-stranded RNA-dependent kinase (PKR), which is activated by double stranded RNA, induces inflammation by regulating NF-κB signaling. The NLR family pyrin domain-containing 3 (NLRP3) inflammasome also modulates inflammation in response to infection. Porphyromonas gingivalis (P.gingivalis) is an oral bacterium which is implicated in the pathogenesis of periodontal diseases. We previously reported that PKR is a key modulator of bone metabolism and inflammation in the periodontal tissue. PKR was also reported to induce inflammation in response to microbes by regulating the NLRP3 inflammasome, suggesting that PKR could affect inflammation along with NLRP3 in periodontal diseases. In this study, we investigated the effects of PKR on NLRP3 expression and NF-κB activity in P. gingivalis infected osteoblasts. We first constructed a SNAP26b-tagged P.gingivalis (SNAP-P. g.) and traced its internalization into the cell. SNAP-P. g. increased the activity of PKR and NF-κB and also induced NLRP3 expression in osteoblasts. Inhibition of NF-κB attenuated SNAP-P. g.-induced NLRP3 expression. The knockdown of PKR using shRNA decreased both the activity of NF-κB and the expression of NLRP3 induced by SNAP-P.g.. We therefore concluded that in osteoblasts, P. gingivalis activated PKR, which in turn increased NLRP3 expression by activating NF-κB. Our results suggest that PKR modulates inflammation by regulating the expression of the NLRP3 inflammasome through the NF-κB pathway in periodontal diseases.

Published
2017

36. Erratum to: Evaluation of bleeding on probing and gingival crevicular fluid enzyme activity for detection of periodontally active sites during supportive periodontal therapy

Author

Ito, Hiroshi, Numabe, Yukihiro, Sekino, Satoshi, Murakashi, Etsuko, Iguchi, Hitomi, Hashimoto, Shuichi, Sasaki, Daisuke, Yaegashi, Takashi, Kunimatsu, Kazushi, Takai, Hideki, Mezawa, Masaru, Ogata, Yorimasa, Watanabe, Hisashi, Hagiwara, Satsuki, Izumi, Yuichi, Hiroshima, Yuka, Kido, Jun-Ichi, and Nagata, Toshihiko

Published
2014
Full Text
View/download PDF

37. S100A9は、骨細胞様細胞においてMAPKsおよびSTAT3シグナル伝達経路を介してIL-6およびRANKLの発現を増加させる

Author

Takagi, Ryosuke, Sakamoto, Eijiro, Kido, Jun-ichi, Inagaki, Yuji, Hiroshima, Yuka, Naruishi, Koji, and Yumoto, Hiromichi

Subjects
Calprotectin, IL-6, RANKL, Osteocyte, S100A9
Abstract

Objective: Calprotectin is hetero-complex of S100A8 and S100A9 and mainly secreted from neutrophils, monocytes and chondrocytes in inflammatory condition. Calprotectin binds to RAGE and TLR4, and induces the expression of pro-inflammatory chemokines and cytokines in various cells. Periodontitis is chronic inflammatory disease to lead gingival inflammation and alveolar bone resorption. Calprotectin levels in gingival crevicular fluid of periodontitis patients are higher than healthy patients. In the present study, the effects of S100A8 and S100A9 on the expressions of pro-inflammatory cytokines and bone metabolism related factor in mouse osteocyte like cells (MLO-Y4-A2) were investigated. Design: MLO-Y4-A2 cells were treated with S100A8 and S100A9, and the expressions of RAGE, TLR4, RANKL and several inflammatory cytokines were analyzed by PCR and Western blotting or ELISA methods. To investigate the intracellular signaling pathways, phosphorylation of MAPK and STAT3 was determined by Western blotting, and chemical specific inhibitors and siRNAs were used. Results: Expressions of IL-6 and RANKL were increased by treatment with S100A9 but not S100A8. However, both S100A8 and S100A9 did not changed expression of IL-1β, IL-8 and TNF-α. Although RAGE and TLR4 expressions were not up-regulated by S100A9 treatment, transfection of siRNA for RAGE and TLR4 significantly decreased IL-6 and RANKL expressions. In addition, S100A9 activated p38, ERK and STAT3 signaling pathways, and inhibitors for these factors significantly decreased S100A9 induced IL-6 and RANKL expressions. Conclusions: These results indicated that S100A9 induces IL-6 and RANKL production via engagement with RAGE and TLR4 signalings in osteocytes and suggested that S100A9 may play important roles in the periodontal alveolar bone destruction.

Published
2020

42. Correlation Between Gingival Crevicular Fluid Hemoglobin Content and Periodontal Clinical Parameters

Author

Ito, Hiroshi, primary, Numabe, Yukihiro, additional, Hashimoto, Shuichi, additional, Sekino, Satoshi, additional, Murakashi, Etsuko, additional, Ishiguro, Hitomi, additional, Sasaki, Daisuke, additional, Yaegashi, Takashi, additional, Takai, Hideki, additional, Mezawa, Masaru, additional, Ogata, Yorimasa, additional, Watanabe, Hisashi, additional, Hagiwara, Satsuki, additional, Izumi, Yuichi, additional, Hiroshima, Yuka, additional, Kido, Jun-Ichi, additional, Nagata, Toshihiko, additional, and Kunimatsu, Kazushi, additional

Published
2016
Full Text
View/download PDF

46. 79

Author

Hiroshima, Yuka, primary, Hsu, Kenneth, additional, Tedla, Nicodemus, additional, Chow, Sharron, additional, Kawaguchi, Naomi, additional, and Geczy, Carolyn L., additional

Published
2014
Full Text
View/download PDF

47. 83

Author

Hsu, Kenneth, primary, Hiroshima, Yuka, additional, Endoh, Yasumi, additional, Thomas, Shane R., additional, and Geczy, Carolyn L., additional

Published
2014
Full Text
View/download PDF

48. 62

Author

Geczy, Carolyn, primary, Hsu, Kenneth, additional, Hiroshima, Yuka, additional, and Tedla, Nicodemus, additional

Published
2014
Full Text
View/download PDF

Catalog

Books, media, physical & digital resources
See catalog results