177 results on '"Hilliard, Massimo"'
Search Results
2. Modular transient nanoclustering of activated β2-adrenergic receptors revealed by single-molecule tracking of conformation-specific nanobodies
3. Fusogen-mediated neuron–neuron fusion disrupts neural circuit connectivity and alters animal behavior
4. Phosphatidylserine save-me signals drive functional recovery of severed axons in Caenorhabditis elegans
5. SARS-CoV-2 infection and viral fusogens cause neuronal and glial fusion that compromises neuronal activity
6. The dynamin GTPase mediates regenerative axonal fusion in Caenorhabditis elegans by regulating fusogen levels
7. Epidermal control of axonal attachment via β-spectrin and the GTPase-activating protein TBC-10 prevents axonal degeneration
8. Spectrin forms a periodic cytoskeleton within the epidermis to preserve axonal integrity
9. Additional file 1 of Impaired signaling for neuromuscular synaptic maintenance is a feature of Motor Neuron Disease
10. A Core Metabolic Enzyme Mediates Resistance to Phosphine Gas
11. EFF-1-mediated regenerative axonal fusion requires components of the apoptotic pathway
12. The metalloprotease ADM-4/ADAM17 promotes axonal repair
13. Neuron-epidermal attachment protects hyper-fragile axons from mechanical strain
14. The SARS-CoV-2 spike (S) and the orthoreovirus p15 cause neuronal and glial fusion
15. TDP-43 Mutation Affects Stress Granule Dynamics in Differentiated NSC-34 Motoneuron-Like Cells
16. The Zona Pellucida domain containing proteins, CUT-1, CUT-3 and CUT-5, play essential roles in the development of the larval alae in Caenorhabditis elegans
17. Roles for Caenorhabditis elegans rad-51 in meiosis and in resistance to ionizing radiation during development
18. Fusogen-mediated neuron−neuron fusion disrupts neural circuit connectivity and alters animal behavior
19. Axonal repair by fusion: pitfalls, consequences and solutions
20. Axonal degeneration and regeneration: a mechanistic tug-of-war
21. How the Australian Functional Genomics Network (AFGN) contributes to improved patient care
22. Mutations in Caenorhabditis elegans neuroligin-like glit-1, the apoptosis pathway and the calcium chaperone crt-1 increase dopaminergic neurodegeneration after 6-OHDA treatment
23. Disruption of RAB-5 Increases EFF-1 Fusogen Availability at the Cell Surface and Promotes the Regenerative Axonal Fusion Capacity of the Neuron
24. Axonal fusion: An alternative and efficient mechanism of nerve repair
25. Neuronal sub‐compartmentalization: a strategy to optimize neuronal function
26. Defects in synaptic transmission at the neuromuscular junction precede motor deficits in a TDP-43Q331K transgenic mouse model of amyotrophic lateral sclerosis
27. RAB-5 regulates regenerative axonal fusion by controlling EFF-1 endocytosis
28. 6-OHDA-induced dopaminergic neurodegeneration in Caenorhabditis elegans is promoted by the engulfment pathway and inhibited by the transthyretin-related protein TTR-33
29. Defects in synaptic transmission at the neuromuscular junction precede motor deficits in a TDP‐43 Q331K transgenic mouse model of amyotrophic lateral sclerosis
30. Phosphatidylserine save-me signals drive functional recovery of severed axons inCaenorhabditis elegans
31. 6-OHDA-induced dopaminergic neurodegeneration in C. elegans is promoted by the engulfment pathway and inhibited by the transthyretin-related protein TTR-33
32. The Heterochronic Gene lin-14 Controls Axonal Degeneration in C. elegans Neurons
33. A multi-trap microfluidic chip enabling longitudinal studies of nerve regeneration in Caenorhabditis elegans
34. Cell-cell fusion in the nervous system: Alternative mechanisms of development, injury, and repair
35. Cortical synaptic and dendritic spine abnormalities in a presymptomatic TDP-43 model of amyotrophic lateral sclerosis
36. Cortical synaptic and dendritic spine abnormalities in a presymptomatic TDP-43 model of amyotrophic lateral sclerosis
37. Neuron-specific knock-down of SMN1 causes neuron degeneration and death through an apoptotic mechanism
38. The Apoptotic Engulfment Machinery Regulates Axonal Degeneration in C. elegans Neurons
39. Defects in synaptic transmission at the neuromuscular junction precede motor deficits in a TDP-43Q331K transgenic mouse model of amyotrophic lateral sclerosis.
40. Loss of MEC-17 Leads to Microtubule Instability and Axonal Degeneration
41. A multi-channel device for high-density target-selective stimulation and long-term monitoring of cells and subcellular features in C. elegans
42. Rapid and Permanent Neuronal Inactivation In Vivo via Subcellular Generation of Reactive Oxygen with the Use of KillerRed
43. A dominant mutation inmec-7/β-tubulinaffects axon development and regeneration inCaenorhabditis elegansneurons
44. Laterally Orienting C. elegans Using Geometry at Microscale for High-Throughput Visual Screens in Neurodegeneration and Neuronal Development Studies
45. The Heterochronic Gene lin-14Controls Axonal Degeneration in C. elegansNeurons
46. LIN-44/Wnt Directs Dendrite Outgrowth through LIN-17/Frizzled in C. elegans Neurons
47. Wnt signals and Frizzled receptors regulate dendrite formation in C. elegans
48. Axonal regeneration proceeds through specific axonal fusion in transected C. elegans neurons
49. Axonal degeneration and regeneration: a mechanistic tug‐of‐war
50. Femtosecond laser nanosurgery in microfluidic devices and its emerging role in nerve regeneration studies
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